Is Diabetes Really A Sugar Problem? No.

Majid Ali, M.D.

New York 212-873-2444

New Jersey . 201-996-0027

No, Diabetes Is Not a Sugar Problem.

It is an insulin Problem.

Unless specified otherwise, the word at this web site is used for Type 2 diabetes.

BEWARE!

If you think, diabetes is a sugar problem, tests done for blood sugar levels for screening for diabetes will be misleading most of the time.

The diagnosis of diabetes will be delayed for five, ten, or more years.

If you are overweight, it will be much more difficult to lose weight.

Unless you are at your optimal weight, undetected insulin toxicity will injure all your body organs to varying degrees until diabetes is diagnosed and treated for years, usually five to ten or more years.

Large Scientific Claims Require Large Scientific Evidence

The Common Diabetes Is Not a Sugar Problem, But An Insulin Toxicity Problem.

· Brain atrophy

· Brain degenerative conditions

· Rising blood creatinine level

· Rising liver enzyme levels

· Rising CRP test results

Blood Cells Tell The Insulin Toxicity Story

Healthy Blood Cells for Comparative Study. Figure 1

Early Stress on Red Blood Cells (lower picture) . Figure 2

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Microplaques in Circulating Blood

When Blood Glucose Level Rises Above 200 mg/dL

Figure 13 (top) and figure 14 (bottom) show two microplaques in a patient who had received three unsuccessful angioplasties for advanced IHD. Photomicrographs were taken the day after a major nosebleed. Note the compaction of necrotic debris and blood elements in microplaques as contrasted with loose structure of microclots in figure 11.

Figure 8 (bottom) illustrates a zone of plasma congealing unaccompanied by any cellular elements of the blood (seemingly a “spontaneous” phenomenon) in a diabetic with IHD. In our view, such congealing represents accelerated oxidative stress on plasma.

Figure 9 (top) shows some needle-like and amorphous granular microclots in a patient with unstable angina.

Figure 11 (top) shows a microclot formed by a large aggregate of platelets and congealed plasma in a patient five days after angioplasty.

Figure 12 (bottom) shows another field from the same smear and illustrates how microclots in oxidative coagulopathy grow in size when oxidative stress persists.

Figure 13 (top) and figure 14 (bottom) show two microplaques in a patient who had received three unsuccessful angioplasties for advanced IHD. Photomicrographs were taken the day after a major nosebleed. Note the compaction of necrotic debris and blood elements in microplaques as contrasted with loose structure of microclots in figure 11.