Managing Hepatitis

Managing Alcoholic Fatty Liver Disease

Alcoholic FLD occurs due to excessive caloric intake from excessive alcohol intake. Ethanol is converted into fat by the liver, and some of it stored in the liver. The fatty tissue produces Free Radicals/ROS, which damage the liver cells, and interfere with healthy liver function.

The cure for Alcoholic FLD is to stop drinking, and protect the liver from the ongoing Fat-produced, Free Radical damage. The HepTech Protocol provides a Free Radical shield, which allows the liver to heal, regenerate, and metabolize the excess liver fat.

Managing NASH

Non-Alcoholic SteatoHepatitis is due to hyperalimentation (eating too much compared to the amount of energy needed to fuel the metabolism and activity). Excessive simple sugar intake stimulates insulin production, which drives the sugar molecules into the cells, where they are converted into ATP for energy, and the excess caloric intake above the energy need is stored as fat. Fat is metabolically active, being stored and mobilized continuously, which produces Free Radicals, and damages the cellular organelles and structure, reducing its functionality, and stimulating its repair response to produce collagen. The result is fibrosis and liver dysfunction. NASH affects millions of overweight, low activity, excessive calorie consuming men and women.

Treating NASH requires lowering the caloric intake. But, in addition to changing to a low glycemic load and calorie-appropriate diet, NASH should be immediately treated with the anti-inflammatory HepTech Proocol. It will take time for the liver to metabolize and eliminate the extra fatty burden of the liver. The HepTech Protocol will provide protection to the liver to prevent further damage to the liver from the fatty tissue-generated free radicals. The necessary cure for NASH is a healthy, low glycemic, moderate calorie diet. But, the catabolism of the fatty deposits will take time, and the liver will continue to suffer damage as long as the extra fat remains. When the free radicals are quenched, the liver cell function can improve, and the natural ability of liver cell to regenerate and repair can proceed.

Managing Chronic Viral Hepatitis

Chronic Viral Hepatitis is a liver-centered viral infection that causes metabolic imbalances in those infected. These imbalances result in clinical manifestations of Hepatitis, which can be brought into balance (homeostasis) by the dietary administration of specific nutrients at specific levels. These clinical manifestations are:

Hepatitis causes an increase in the production of free radical reactive oxygen species (ROS) in the mitochondria of cells. The resulting oxidative stress in the cytosol is clinically significant to the pathogenesis of Hepatitis as well as in the generation and progression of fibrosis in Hepatitis

Hepatitis causes increased levels of inflammation in the cells of the liver. This inflammation is clinically significant to the pathogenesis of Hepatitis as well as in the progression of fibrosis in Hepatitis patients.

Hepatitis causes oxidative damage to vulnerable cell membranes, including interior organelle membranes. This is manifested by changes in membrane phospholipid profiles, as well as changes in membrane function, fluidity, and integrity. Hepatitis genotype 3 contributes to steatosis, which is manifested by the abnormal retention of lipids inside the cells of the liver. Steatosis also has metabolic causation, (NAFLD and NASH). Steatosis, regardless of cause, is clinically significant in the pathogenesis of Hepatitis C as well as in the progression of fibrosis in Hepatitis patients.

The combination of the above clinical manifestations of Hepatitis promotes the activation of the gene transcription factor NFkappaB. This activation starts a chain of events that leads the progression of fibrosis to cirrhosis in the liver. Specifically, NFkappaB activation triggers the production of pro-inflammatory peptides and proteins, the activation of stellate cells, their conversion to myofibroblasts, and the secretion and accumulation of collagen (scar tissue) in the spaces of the liver (from fibrosis to cirrhosis).

The HepTech Comprehensive Medical Food Protocol involves the administration of distinctive nutrients that Hepatitis patients require to ameliorate the clinical manifestations of Hepatitis. Amelioration of these specific clinical manifestations of Hepatitis C decreases levels of activation of the metabolic stimulus (NFkappaB) that triggers the generation and accumulation of scar tissue (from fibrosis to cirrhosis) in the spaces of the liver.