Hypoplastic left heart syndrome informative speech

Echocardiography is the principal method of diagnosing hypoplastic left heart syndrome. For example, some other health problems that might develop include irregular heart beat arrhythmiasincreased risk of infection in the heart muscle infective endocarditisor weakness in the heart cardiomyopathy.

Occasionally, the right ventricle does not function well following the Norwood operation, and in some case, cardiac transplantation may need to be considered. As they age, many problems can occur, and Fontan patients need careful and thoughtful surveillance throughout their lives by experts in complex congenital heart disease.

The subsequent operations in the staged reconstruction plan are: Some Fontan patients may have weakening of their pumping chamber or leakage of one or more valves. If the fetus has been diagnosed before delivery, stabilization measures are started immediately so the newborn does not become unstable.

Close monitoring Hypoplastic left heart syndrome informative speech essential to detect any organ dysfunction and maintain cardiopulmonary stability because infants with this anomaly may be very unpredictable and undergo quite sudden changes.

If a child with hypoplastic left heart syndrome reaches the time for the second stage about 4 to 6 months of age without major complications, the survival through the Glenn and Fontan operations are much better, exceeding 90 percent with current methods. Sleepiness Diagnosis Some CHDs may be diagnosed during pregnancy using a special type of ultrasound called a fetal echocardiogram, which creates ultrasound pictures of the heart of the developing baby.

Catheters are placed, usually in the umbilical blood vessels, which allow medications to be given and blood to be obtained for testing. Even with improved treatments, many people Hypoplastic left heart syndrome informative speech a CHD are not cured, even if their heart defect has been repaired.

Cyanosistherefore, may be the first clue to the presence of a serious underlying cardiac condition. Even though the infant may have low oxygen saturations, supplemental oxygen is avoided since it tends to Hypoplastic left heart syndrome informative speech more blood flow to the lungs, which may steal blood flow from the body and place excessive demands on the already stressed single right ventricle.

This is because all of the blood from the lungs the oxygenated "red" blood mixes together in the single right ventricle before being pumped out of the lungs and body. There are essentially three treatment options that have been proposed for children with hypoplastic left heart syndrome.

Show Infants with hypoplastic left heart syndrome can develop life-threatening shock when the ductus arteriosus begins to close. The ductus venous the very first shunt which encountered and stations oxygenated blood pass the liver. Because of advancements in operative intervention mortality rates in neonates with Hypoplastic Left Heart Syndrome have diminished.

Show The Norwood operation is the most complex and highest risk procedure in the sequence of staged reconstruction for hypoplastic left heart syndrome. For example, certain conditions a mother has, like pre-existing diabetes or obesity, have been linked to heart defects in the baby.

Finally, to provide blood flow to the lungs, a small tube graft is placed either from an artery to the lung vessels called a modified Blalock-Taussig shunt or from the right ventricle to the lung vessels called a Sano modification. Show This heart defect is one of the most readily diagnosed on fetal echocardiograms and is one of the most common cardiac defects picked up on screening obstetrical ultrasounds.

Examples of completed orders. Some babies have heart defects because of changes in their individual genes or chromosomes. Manipulations of medications and respiratory treatments including possible mechanical ventilation are performed to optimally balance the flow of blood to the body and the flow of blood to the lungs.

This syndrome is called a collection of anomalies involving the left heart and connecting structures. Because of the extensive reconstruction of the aorta that must be done, this operation is one of the most challenging heart surgeries in pediatrics. Aortic and mitral valve stenosis or atresia is present and in certain cases atrial septal defect accompanies.

Some can be treated without surgery using a procedure called cardiac catheterization. Today it is rare that a family may choose not to treat a child with hypoplastic left heart syndrome, though in cases when the infant is unable to be satisfactorily stabilized, no treatment may be advised.

CDC Normal fetal circulation includes the use of three shunts that behave as liver, lung and heart bypass so as to provide the systemic flow of blood flow. Respiratory distress difficult or fast breathing is often present because the lungs will tend to receive an excessively large amount of blood flow.

Such early diagnosis of the anomaly allows for prompt intervention for stabilization at the time of birth so that severe shock may be avoided. Management of these patients requires a symphony of multidisciplinary teams including fetal and pediatric cardiologist, cardiac surgeons, neonatologist, pulmonologist, nutritionist and family advisers.

With the Norwood operation, the right ventricle becomes the systemic or main ventricle pumping to the body. Nongenetic risk factors and congenital heart defects.

Some will have low oxygen levels in their blood that may limit their physical abilities, and that may require the blockage of abnormal vessels that were allowing blue blood to mix with red blood.

Current management at major pediatric heart centers has resulted in survival rates of 75 percent or better. Planning to deliver such an infant at a hospital capable of aggressive newborn resuscitation is important in improving the chances for a good outcome.

However, some CHDs are not detected until after birth or later in life, during childhood or adulthood. Almost all children with hypoplastic left heart syndrome will continue to need some cardiac medications to maximize the efficient function of their heart, and all will require regular periodic follow-up visits with their cardiologist to evaluate their cardiac function and detect late complications such as arrhythmias.

Catheterization, though, does play an important role in the evaluation of the cardiopulmonary function and anatomy in older children with hypoplastic left heart syndrome while planning for later stages in the treatment.

Ultimately, severe shock resulting in seizures, renal failure, liver failure, and worsening cardiac function may develop. The rapid stabilization of these infants must begin as soon as the diagnosis is suspected.Redmond P.

Burke (born 4 November ) is a congenital heart surgeon, innovator, software developer, author, inventor, and founder of The Congenital Heart Institute at Miami Children's Hospital in Miami, Florida.

Hypoplastic Left Heart Syndrome: Informative Speech about Hypoplastic Left Heart Syndrome. Specific Purpose Statement: To inform my audience about the major health concern, diagnosis, and approaches to treating Hypoplastic Left Heart Syndrome. Infants with hypoplastic left heart syndrome can develop life-threatening shock when the ductus arteriosus begins to close.

In most cases, however, the ductus arteriosus is widely open at the time of birth, supplying the blood flow to the body, and babies may not be diagnosed right away.

The most common cytogenetic finding seen with hypoplastic left heart (HLH) is a normal karyotype. Turner syndrome (45,X) is among the most common cytogenetic abnormalities seen with HLH, but is still not as common as a normal karyotype.

Hypoplastic left heart syndrome (American Heart Association) The information provided is intended to be informative and educational and is not a replacement for professional medical evaluation, advice, diagnosis or.

Body Anatomy Hypoplastic left heart syndrome is a congenital heart defect where the constructions in the left heart are severely underdeveloped. The left ventricle, aorta and aortic arch are unable to adequately pump and send blood to .