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Take one last look at your smartphone. You probably regard it as the cutting edge of technology, and those cute icons as portals to an infinite array of virtual interactions.What I see is nothing more than a hand-held, electronic cabinet of wonders; an anthropological museum crammed full of exhibits of the lives we once lived.

There is a kind of uber-story about the future of ageing, regularly featured in the press, that swings through from the lows of crisis reportage (we’ll all be living longer, the pension pots will be empty, health services will crumple under new obligations to the elderly) to the highs of techno-euphoria (gene therapy and nanotech will enrich our late lives, we’ll be able to see our grandchildren’s children, work until we’re 90 and dodge decrepitude).

We are faced with an important question. There are technologies that allow improving a person - just as we change plants and animals. But is it ethical to change the human body, do we have the right to artificially improve ourselves?

Old age is the final period of our life, having limitations in adaptive capabilities and morphological changes in the body. Alas, at the moment this is inevitable. Despair leads to great achievements, many scientists around the world are trying to fight for an increase in the measured period. Perhaps the coveted drug is closer than we think.

The article reviews the aspects of economic feasibility of the development of life extension technologies and suggests the criteria for calculating the economic efficiency and longevity of the environment for the formation of proposals for the emergence and expansion of market technology of life extension strategies. It was determined that the value of life extension technologies affects the social sphere under surplus of indirect type and direct understanding of the social capital of an individ...

It has been widely accepted that 5-methylcytosine is the only form of DNA methylation in mammalian genomes.
Here we identify N6-methyladenine as another form of DNA modification in mouse embryonic stem cells. Alkbh1
encodes a demethylase for N6-methyladenine. An increase of N6-methyladenine levels in Alkbh1-deficient cells leads
to transcriptional silencing. N6-methyladenine deposition is inversely correlated with the evolutionary age of LINE-1
transposons; its deposition is strongly enriche...

In the nearest future I would like to see the achievements of Mr. Lee applied in the real life. In particular, I would like to know more about how AI will be able to work in a mode of tracking vital signs and recovering the damages in the body.