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Those with the most severe symptoms are the proportion of the population officially diagnosed with ASD, the scientists from the University of Bristol, Harvard and MIT and Massachusetts General Hospital found.'Once we had measurable genetic signals in hand - both polygeneic risk and specific de novo mutations known to contribute to ASD - we were able to make an incontrovertible case that the genetic risk contributing to autism is genetic risk that exists in all of us, and influences our behaviour and social communication.'Co-author, Dr Elise Robinshon, from MGH, said: 'We can use behavioural and cognitive data in the general population to untangle the mechanisms through which different types of genetic risk are operating.'Our study shows that collecting and using phenotypic and genetic data in typically developing children can be useful in terms of the design and interpretation of studies targeting complex neurodevelopmental and psychiatric disorders,' said another of the study's authors, Dr Beate St Pourcain, from the University of Bristol and the Max Planck Institute for Psycholinguistics.'Based on the genetic link between population-based social-communication difficulties and clinical ASD, we may now gain further phenotypic insight into a defined set of genetically-influenced ASD symptoms.… continue reading »