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Toxic
manifestations may occur when the serum concentration exceeds
10.4mg/L.

A concentration
higher than 25mg/L justifies treatment by dialysis.(1)

The toxic and
therapeutic blood levels are very close, so any activity leading
to loosing much body water may switch a patient taking lithium
carbonate form the therapeutic to the toxic zone, as in sweating
excessively in melting departments of steel mills.

Also many
anti-inflammatory drugs raise lithium blood levels of patients and
may cause the intoxication, one major offender being ibuprofen (
Motrin, Advil). It is important to remember this name because it
may be sold without a prescription.

There is no such
thing as a single case of lithium intoxication described in the
pertaining literature from the use of it in glaze making or from
the use of ceramic wares covered by lithium-containing
glazes.

The only lithium
compound that is reported as a severe hazard is lithium hydride
(LiH), which is used as a condensing agent in chemical synthesis
with acid esters and ketones, as a dessicant (a reducing agent),
and as a hydrogen source.

The hydride is a
severe irritant to skin and mucous membranes because it becomes
lithium hydroxide when in contact with moisture of these
structures.(3)