Friday, October 23, 2015

How obvious is it that the very definition or type of "Lyme disease" that was EXCLUDED by the Dearborn "two-tiered" case definition is the one caused by the OspA fungus vaccine (Chronic Neurologic)? Lyme was never about arthritis and neither was LYMErix.

NINDS’ MS-Lyme Group, talking about how the TLR2/1 agonists like Pam3Cys or OspA delivered on blebs (shed by the spirochetes) or OspA vaccination inflames the brain while causing humoral immunosuppression:

”The spirochete Borrelia burgdorferi is the agent of Lyme disease, which causes central nervous system manifestations in up to 20% of patients. We investigated the response of human brain microglial cells, glial progenitors, neurons, astrocytes, as well as peripheral blood monocytes to stimulation with B. burgdorferi. We used oligoarrays to detect changes in the expression of genes important for shaping adaptive and innate immune responses. We found that stimulation with B. burgdorferi lysate increased the expression of Toll-like receptors (TLRs) 1 and 2 in all cell types except neurons. However, despite similarities in global gene profiles of monocytes and microglia, only microglial cells responded to the stimulation with a robust increase in HLA-DR, HLA-DQ, and also coexpressed CD11-c, a dendritic cell marker. In contrast, a large number of HLA-related molecules were repressed at both the RNA and the protein levels in stimulated monocytes, whereas secretion of IL-10 and TNF-alpha was strongly induced. These results show that signaling through TLR1/2 in response to B. burgdorferi can elicit opposite immunoregulatory effects in blood and in brain immune cells, which could play a role in the different susceptibility of these compartments to infection.”http://www.ncbi.nlm.nih.gov/pubmed/16783164

NIH in a patent saying these OspA-ish, TLR2/1 agonist, fungal triacyl lipopeptides like LYMErix are delivered to the brain as blebs or exosomes (just like OspA as a vaccine would):

"The invention relates to novel antigens associated with Borrelia burgdorferi which are exported (or shed) in vivo and whose detection is a means of diagnosing Lyme disease. The antigens are extracellular membrane vesicles and other bioproducts including the major extracellular protein antigen. Another object of the invention is to provide antibodies, monoclonal and/or polyclonal, labeled and/or unlabeled, that are raised against the antigens. A further object of the invention is to provide a method of diagnosing Lyme disease by detecting the antigens in a biological sample taken from a host using the antibodies in conventional immunoassay formats. Another object of the invention is to provide kits, for the diagnosis of Lyme disease, comprising the antibodies and ancillary reagents. The advantage of the antibodies used in the invention is that they react with the antigens from geographically diverse strains of Borrelia burgdorferi, but do not react with antigens from related Borrelia spirochetes."http://patft.uspto.gov/netacgi/nph-Parser…

"The point that I wanted to make in regard to the study is that there is very heavy dependence on serologic confirmation. And when we start thinking about the adverse events, *** it was stated originally when we got the overview of the disease that the disease is really quite protean. And actually the adverse events are very similar to what the disease manifestations are.**** And if you start to, as I think Dr. Hall was eluding to -- if you start to kind of say well how often do you actually become seropositive, you can start to have a different take on when someone has an adverse event or whether it is disease specific or infection specific versus vaccine specific. And I think that that is an important issue that we have to deal with. ..."http://www.fda.gov/ohrms/dockets/ac/98/transcpt/3422t1.rtf

CADAVID on shed blebs with OspA on them travel to the brain to inflame it:

”The current view is that bacteria need to enter the brain to cause inflammation. However, in mice infected with the spirochete Borrelia turicatae, we observed widespread cerebral inflammation despite a paucity of spirochetes in the brain parenchyma at times of high bacteremia. Here we studied the possibility that bacterial lipoproteins may be capable of disseminating from the periphery across the blood-brain barrier to inflame the brain. For this we injected normal and infected mice intraperitoneally with lanthanide-labeled variable outer membrane lipoproteins of B. turicatae and measured their localization in blood, various peripheral organs, and whole and capillary-depleted brain protein extracts at various times. Lanthanide-labeled nonlipidated lipoproteins of B. turicatae and mouse albumin were used as controls. Brain inflammation was measured by TaqMan RT-PCR amplification of genes known to be up-regulated in response to borrelial infection. The results showed that the two lipoproteins we studied, LVsp1 and LVsp2, were capable of inflaming the brain after intraperitoneal injection to different degrees: LVsp1 was better than LVsp2 and Bt1 spirochetes at moving from blood to brain. The dissemination of LVsp1 from the periphery to the brain occurred under normal conditions and significantly increased with infection. In contrast, LVsp2 disseminated better to peripheral organs. We conclude that some bacterial lipoproteins can disseminate from the periphery to inflame the brain.”http://www.ncbi.nlm.nih.gov/pubmed/20431027

Lyme criminals Dave Persing and Robert Schoen say in their RICO patent that you can’t tell the difference between “multisystem,” “protean” LYMErix outcomes from chronic neurologic Lyme:

"Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure....http://patft1.uspto.gov/netacgi/nph-Parser…

Latov on how LYMErix causes the same disease as chronic neurologic Lyme:

”Neurological syndromes that follow vaccination or infection are often attributed to autoimmune mechanisms. We report six patients who developed neuropathy or cognitive impairment, within several days to 2 months, following vaccination with the OspA antigen of Borrelia burgdorferi. Two of the patients developed cognitive impairment, one chronic inflammatory demyelinating polyneuropathy (CIDP), one multifocal motor neuropathy, one both cognitive impairment and CIDP, and one cognitive impairment and sensory axonal neuropathy. The patients with cognitive impairment had T2 hyperintense white matter lesions on magnetic resonance imaging. The similarity between the neurological sequelae observed in the OspA-vaccinated patients and those with chronic Lyme disease suggests a possible role for immune mechanisms in some of the manifestations of chronic Lyme disease that are resistant to antibiotic treatment.”http://www.ncbi.nlm.nih.gov/pubmed/15363064

Marks, one of the OspA trial administrators on how OspA vaccination causes the same disease we know of as chronic neurologic Lyme:

”A wide range of neurological complications have been reported via the medical literature and the VAERS system after vaccination with recombinant outer surface protein A (OspA) of Borrelia. To explore this issue, 24 patients reporting neurological adverse events (AE) after vaccination with Lymerix, out of a group of 94 patients reporting adverse events after Lymerix vaccination, were examined for causation. Five reports of cerebral ischemia, two transient Ischemic attacks, five demyelinating events, two optic neuritis, two reports of transverse myelitis, and one non-specific demyelinating condition are evaluated in this paper. Caution is raised on not actively looking for neurologic AE, and for not considering causation when the incidence rate is too low to raise a calculable difference to natural occurrence.”http://www.ncbi.nlm.nih.gov/pubmed/21673416

But no "doctor" in America or anyone in the entire hhs.gov knows what Lyme or OspA is. Seriously. Think about it. What is ILADS treating if injecting people with the fungal toxin LYMErix causes the same chronic neurologic disease?

Lyme is a do-over of Tuskegee "Bad Blood." The only "case" you are allowed to have it the one that does not need to be treated: Autoimmune arthritis. This is what is alleged by the CDC and Yale. But in the end all they did was expose the mechanism of the Autism pandemic- fungal antigen induced immunosuppression in the presence of live viruses (or latent ones, in the case of Lyme and CFIDS).

So, a vaccine, they, Yale and the CDC, wanted for a disease that causes no disease. Turns out that was the disease - OspA induced post-sepsis; fungal toxin induced post-sepsis with the reactivated viruses etc of all kinds. Something seen in parallel in so many other circumstances: Burkitt's Lymphoma, Vaccine Autism, the failed Tuberculosis vaccines, CFIDS,... The fact that one infection helps establish another is so well known, doctors give children antibiotics for a cold because that treatment was intended to prevent the secondary bacterial infections that cause otitis.

Thursday, August 20, 2015

This is a great web site to find out exactly what you need to know about beating disease. Please read about FstZ it is the answer we are looking for. Here's the SITE- http://www.alternavita.com/ This has been a huge game changer for me as well as a few thousand others that understand this. I can help with Q's. Naturals for Lyme and Company a Facebook Group. It is closed so PM me for admission or ask to join. Wish you well, Pat

PROTOCOL/SYMBIOSIS/DISEASE

This is the main protocol: Keep it simple.
1. Fenbendazole: (Safeguard, Panacur, same ingredient, different names possibly per country)
Equine Paste or Liquid for Goats. 1 ml. per 50 lbs. body weight. Or squeeze
into a capsule after eyeing up initial dose if using paste. Dosage is usually
once per day. Proceed at your comfort level and convenience. No one can
judge your level of infection. Average time to see major improvement is 4
months.Available at: Amazon, Tractor Supply, Agway, Feed Store, WalMart, etc.
Paste is approx. $10, Liquid is approx. $20.Fenbendazole is a benzimidazole anthelmintic, that has broad spectrum,and a wide safety margin. It binds to 3-tubulin astructural protein that blocks polymerizationof tubulin into microtubules, which damages the integrity and the transportfunction of cells in parasites. The reason behind the wide safety marginis due to its affinity to the parasitic tubules rather than mammals.The drug is minimally absorbed after it is given orally. It is metabolized to the activecompound oxfendazole sulfoxide and sulfone.Fenbendazole is excreted in the feces and urine. Formula C15H13N3O2S
2. Reishi Capsules or Tincture. Most here are using Half Hill Farms tincture
or Aloha Brand Capsules. As per dosage on label. Proceed at your comfort
level, less or more.
3. Clean Water. Spring as verified by microscopy to be free of cyanobacteria
or ‘organisms’. Most here are ozonating. Distilled and cold holds ozone best. Food grade
peroxide is an option after boiling but it must remain in the water for at least 12 hours to neutralize
toxins. Both ozone and food grade peroxide will dissipate leaving you with just pure water free
of pathogens. Both use a form of oxygen to clean water. A2Z on Amazon is a popular choice,
approx. $70. Clean Food. Washing or ozonating fruits and veggies as per directions on
your unit or with food grade peroxide at minimum. Ozonating other foods such as flour, is optional,
place dry ingredients in cannister or bag, insert airstone, time depends on your unit, must follow
directions as per your unit. All ozonators are different. We can not give more than general
instructions for ozonators, you must follow directions for your unit.
4. Optional: For lesions, toothpaste with peroxide and baking soda. Apply to
lesion, let dry, do not touch. See Steve for further options on lesions such as
zinc or cortisone.
5. For the home: Borax or OxyClean for clothes washing or cleaning. Turn
inside out to dry high heat. Pine Sol for cleaning.
That’s it, no real need to add anything further. You must keep the bowels moving. Some suggestions,
fruit juice, citric acid, ice cream. Ozonated Magesium, Mag 07 is a popular brand or ozonated Natural
Calm as it seems to work very well and gives additional support. How you do that is up to you. Fat
increases the ability of Fenbendazole to do it’s job. If you begin to notice fewer bowel movementsyou may want to back off until the bowels move again. You may want to just start with Reishi as it
is more gentle. Dosage is day or night, empty of full stomach for both, whichever is best or most
convenient for you. Proceed only at your comfort level.Further information about our group and the nature of chronic disease and symbiosis or‘fungal’ synergy’ with links for further reading. The term ‘fungus’ is used but we believeone of the most pathogenic organisms is cyanobacteria. It is a true misnomer to use theterm fungus, we must demand that the dangers of cyanobacteria in our water berecognized. We must acknowledge that some true pathogenic algae has been misidentifiedas Candida. We use the term fungal or fungus until all of these pathogenic cyanobacteriaare identified and for ease of understanding.
This is the quintessential root of your 'label'. Or as close as I can find in published literature
that is fairly easy to grasp for the average person. If I could find more to add that is easy
to grasp and understand I would but that is very difficult. And to my knowledge no one
quite understands the synergy but the root is becoming established science more by the
day. Just not recognized by the medical establishment to the extent it must be for everyone's
sake. We will also set out to prove the synergy of pathogens and ‘fungi’ and the role that
plays in chonic disease.http://cdn.intechopen.com/pdfs/29065/InTech-Endotoxin_tolerance_as_a_key_mechanism_for_immunosuppression.pdf
And a quote from the queen of Lyme crime. And another quote. OspA can and does
occur in NATURE. They likely don't even know the number of pathogens/organisms that
are able to induce this. Some have not even been identified (but you
should know they have been and are seeking to reclassify spirochetes into the family of
cyanobacteria after DNA analysis).
You may likely never trace the source, or you may. If it is in your water and I think we've
shown, it is, why keep adding to it? If in your food, the same. If passed generationally,
we should be told. If it contaminated vaccines and it very well could have, whether
intentionally or unintentionally the average person can never uncover (but a few know
enough to guess or know and they are not being truthful but that is their crime) and not
the focus of this site, albeit a VERY IMPORTANT piece of the puzzle. I think we've also
shown how one safe drug (fenbendazole, benzimidazole class) works
across the myriad of 'labels' by reversing the root of your 'label' in addition that Steve to
my knowledge never denied or disagreed to the underlying immune dysfunction or how
these pathogens use the same mechanism to evade the
immune system. And we can not paste all the 'labels' from one source (or all the known
pathogens that can cause this but the treatments (benzimidazoles) also include autoimmune,
heart disease, cancer in addition to anti parasite, anti viral, anti fungal, anti microbial,
anti tumoral. Now that is simply the truth. The truth.

A brief quote from the queen of Lyme Crime Kathleen Dickson.
December 16 at 8:08am å Like
Kathleen Dickson Not necessarily from Lyme, right, could be some other toxic exposure, but the chronicity comes from being unable to reverse fungal antigen tolerance.
December 16 at 8:11am å Edited å Like å 3
Kathleen Dickson Whatever is a TLR2/1-agonist. Could be spirochetes shedding Osps, could be contaminated vaccines,
could be a moldy home... What makes it irreversible is the fungal antigens, such as OspA.
December 16 at 8:08am å Like
Kathleen Dickson Not necessarily from Lyme, right, could be some other toixc exposure, but the chronicity comes from
being unable to reverse fungal antigen tolerance.
December 16 at 8:11am å Edited å Like å 3
Kathleen Dickson Whatever is a TLR2/1-agonist. Could be spirochetes shedding Osps, could be contaminated vaccines,
could be a moldy home... What makes it irreversible is the fungal antigens, such as OspA.
We've also proven the danger and increasing occurence of HABS around the world which
appeared to increase in the 1850's at the end of the end of the mini ice age. And that
while this is known to WHO, (the increasing number of pathogens in
water) the sciences, even the Veterinary Manuals and animal doctors for decades now,
known algal/fungal/cyanobacterial pathogens or toxins are barely on the radar of the
medical establishment and right up there in denial along with vaccines
and chronic Lyme. We are simply questioning if the chronicity is occuring due to chronic
exposure (as some here say) or an inability to reverse due to lack of proper treatment
or improper treatment. That is all. And we are trying to bring awareness to how our 'labels'
are also affecting not only humans and animals, as well as insects but trees and plants
as well (see for yourself the dying trees affected by 'fungi/algae' if you don't believe it).
We are also bringing to light just what the wrong treatment might do (abx, chemo, steroids,
immuno suppressors) if one does have this as it could be unhelpful in the least and
dangerous at most.
Included are some links to the dangers of abx and their role in chronic disease and theinability to keep up with mutations. This includes natural abx. As well as chemical warfarein the environment in the fight against ‘fungus’. Benzimidazoles do not work like abx.They are not a kiling machine. They harmlessly disable by working on a core mechanismof replication. The tubulin-like protein FtsZ is a bacterial division protein which isalso required in plant and algae organelles. Benzimidazoles (Fenbendazole) maydisrupt this protein formation.

Our only agenda is truth and an end to labels when we believe the root is one (the
mechanism of chronic disease) and to draw attention to increasing environmental exposure,
as well as past environmental exposure that has largely been ignored. We as a groupwill always be thankful and grateful to Steve Beddingfield for helping us to lookunder our nose and as when he helped himself, he chose also to help others inspreading the word about these safe effective treatments as well as to others whoare doing their best to expose and explain the ‘fungal synergy’ in chronic disease.We hope to do our part.
As well as hoping to draw attention to safe effective treatments that address the synergy
of chronic disease and are readily available, inexpensive and effective.See patents on benizimidazole, class of drugs which were originally used as anenvironmental fungicide and algicide, the core chemical structure which is a derivativeof Vitamin B12, and are proven to be safe and effective against a wide range of conditions.Some of the first azoles were thiamidazoles which go back as far as the1800’s.

Now on to our collected evidence for benzimidazoles, the proof of ‘fungal synergy’ as
shown now only in known fungal pathogens such as Candida, some other aquatic
organisms and viruses and the role of symbiosis in pathogenicity and infectivity, ‘it takes
two’ (or three if you count toxins as a major component) in this synergy.
Benzimidazole: A short review of their antimicrobial activities
Namrata Singh, Annamalai Pandurangan, Kavita Rana, Preeti Anand, Arsad Ahamad,
Amit Kumar Tiwari
Abstract
Benzimidazole is the heterocyclic compound formed from benzene and
imidazole ring containing nitrogen, oxygen sulphor and its derivatives are of
wide interest because of their diverse biological activity and clinical applications,
they are remarkably effective compounds both with respect to their inhibitory
activity and their favourable selectivity ratio. Reported nucleus is a constituent
of vitamin-B12. Benzimidazoles are regarded as a promising class of bioactive
heterocyclic compounds that exhibit a range of biological activities like antimicrobial,
anti-viral, anti-diabetic, anti-cancer activity, numerous anti-oxidant,
anti-parasitic, anti-helmintics, anti-proliferative, anti-HIV, anti-convulsant, antiinflammatory,
anti-hypertensive, anti-neoplastic, proton pump inhibitor and
anti-trichinellosis. Benzimidazoles exhibit significant activity as potential
antitumor agents, smooth muscle cell proliferation inhibitors, a treatment for
intestinal cystitis, and in diverse area of chemistry. Some of the important
benzimidazole derivatives have been reported as thyroid receptor agonist
gonadotropin releasing hormone receptor antagonists, non-nucleoside HIV-
1 reverse transcriptase inhibitors and interestingly alkynylbenzimidazoles as
modulators of metabotropic glutamate receptors. The imidazole core is a
common moiety in a large number of natural products and pharmacologically
active compounds. The synthesis of novel benzimidazole derivatives remains
a main focus of medicinal research. This comprehensive overview summarizes
the chemistry of different derivative of substituted benzimidazole along with
their anti-microbial activity containing anti-malarial anti-fungal, anti-bacterial,
anti-viral activities.
DOI: http://dx.doi.org/10.3329/icpj.v1i5.10284
International Current Pharmaceutical Journal 2012, 1(5): 119-127
Keywords
benzimidazoles; anti-malarial; anti-fungal; anti-bacterial; anti-viral, anti tumoral
Full Text: PDF

Now on to our contention with articles, that without ‘fungal synergy’ no pathogen is
particularly harmful on it’s own and that ‘fungus’ plays a major part in infectivity and
virulence. It takes two to tango.
It is not just our contention but that of Lynn Margulis, who alluded that a spirochete was
not infective on it’s own and proved that spirochetes drop DNA in relation to the host but
was never able to finish her work due to her death.
Paul Cox, his work on toxins, evironmental exposure, protein formation in the role of ALS
and more. Alan Barbour, who could not figure out why bovine serum made Borrelia
more or less pathogenic and infective. And many others working in symbiotic evolution
and disease.
First infectivity and replication from UGA in malaria.
Athens, Ga. - Long ago, when life on Earth was in its infancy, a group of small single-celled algae
propelled themselves through the vast prehistoric ocean by beating whip like tails called flagella. It's a
relatively unremarkable tale, except that now, more than 800 million years later, these organisms have
evolved into parasites that threaten human health, and their algal past in the ocean may be the key to
stopping them.
The organisms are called apicomplexa, but people know them better as the parasites that cause malaria and toxoplasmosis, serious diseases that infect millions of people every year, particularly in the developing world.
Now, researchers at the University of Georgia have discovered how an important structure inside these parasitic cells, which evolved from the algal ancestor millions of years ago, allows the cells to replicate and spread inside their hosts. Their research may soon lead to new therapies to halt these deadly pathogens before they cause disease.In order to survive, the parasitic apicomplexa must invade an animal or human and force its wayinto the cells of its host. Once inside the host cell, the parasite begins to replicate into numerousdaughter cells that in turn create additional copies, spreading the infection throughout the body.
In their study, published Dec. 11 in PLoS Biology, the researchers demonstrate that, during the processof replication, the parasite cell loads genetic material into its daughter cells via a strand of fiber that connects the two. By altering the genes for the components of the fiber in the laboratory, the researchers discovered that they could prevent parasite replication, making the parasite essentially harmless."These altered parasites can initially infect cells, but once we turn off the fiber genes, theycannot create new daughter cells and spread," said Maria Francia, lead author and doctoralcandidate in the department of cellular biology. "Since it cannot replicate, the parasite eventuallydies without causing serious harm."This replication fiber appears to have evolved from the flagellum that ancient algae used toswim.
"This was a surprising finding," said Boris Striepen, a Georgia Research Alliance Distinguished Investigator
in UGA's Center for Tropical and Emerging Global Diseases. "These parasites no longer use flagellato swim, but they have apparently repurposed this machinery to now organize the assembly of an invasive cell."
During evolution, flagella have been reengineered to serve numerous different functions in animals,
including the sensors that allow us to see and smell. This study suggests that in these parasites structures used to invade host cells may be also derived from flagella.
Current treatments for diseases like malaria are threatened by the parasite becoming resistant to the
drugs, so the need for new therapies is always pressing.This algae-based connective fiber may serve as a promising target for anti-parasitic drugdevelopment, said Striepen, who is also a cellular biologist in the Franklin College of Arts andSciences. He cautions, however, that more work must be done to learn how to manipulate ordestroy the fiber in parasites that have infected humans or animals.
But both Striepen and Francia argue that scientists do well to pay close attention to the evolutionary
history of the organisms they study."It is extremely important to understand the evolution of different organisms, but especially the evolution of pathogens," Striepen said. "The analysis of their evolution produces important opportunities to develop treatments, but it also helps us understand the basic structure of the pathogens that we must fight."
UGA Center for Tropical and Emerging Global Diseases
The University of Georgia Center for Tropical and Emerging Global Diseases draws on a strong foundation of parasitology, immunology, cellular and molecular biology, biochemistry and genetics to develop medical and public health interventions for at-risk populations. Established in 1998, the center promotes international biomedical research and educational programs at UGA and throughout Georgia to address the parasitic and other tropical diseases that continue to threaten the health of people throughout the world. For more information about the center,
see http://ctegd.uga.edu

Alan Barbour among other authors in the infectivity and virulence of Borrelia.In conclusion, our data suggest that variations in BSK medium formulations havesignificant effects on the infectivity and pathogenicity of B. burgdorferi clinicalisolates. The attenuated pathogenicity of B. burgdorferi variants cultured in BSK-Hmedium is not due to the loss of plasmids. Further studies are in progress to comparethe differences in levels of gene expression and in the protein profiles of variants of B.burgdorferi clinical isolates grown in various BSK media.BSK media is bovine serum. Steve and we have shown that cows are increasinglyinfected with ‘pathogenic fungus’. Bovine serum is also used to culture manyvaccines including MMR.http://iai.asm.org/content/72/11/6702.fullI do believe they recognized this phenomenon much earlier as I have earlierabstracts.
Schwan, T. G., W. Burgdorfer, and C. F. Garon. 1988. Changes in infectivity and plasmidprofile of the Lyme disease spirochete, Borrelia burgdorferi, as a result of in vitro cultivation.
Infect. Immun. 56:1831-1836.
A plasmid is a small molecule within a cell that is physically separated from a chromosomal DNA and
can replicate independently. They are most commonly found in as small, circular, double-stranded DNA molecules; however, plasmids are sometimes present archea and eukarotiuc organisms. In nature,
plasmids often carry genes that may benefit the survival of the organism, for example antibiotic resistance. While the chromosomes are big and contain all the essential information for living (an adequate analogy is the hard-drive of a computer), plasmids usually are very small and contain additional information (in this analogy, plasmids are the USB flash drives). Artificial plasmids are widely used as in vectors and molecular cloning , serving to drive the replication of sequences within host organisms. Plasmids are considered replicons, a unit of DNA capable of replicating autonomously within a suitable host. However, plasmids, like viruses , are not considered by some to be a form of life. Plasmids can be transmitted from one bacterium to another (even of another species) via three main mechanisms:transformation ,transduction, and conjugation . This host-to-host transfer of genetic material is called , horizontal gene transfer and plasmids can be considered part of the transduction and conjugation. Unlike viruses (which encase their genetic material in a protective protein coat called a capsid ), plasmids are "naked" DNA and do not encode genes necessary to encase the genetic material for transfer to a new host. However, some classes of plasmids encode the conjugated sex pilus necessary for their own transfer. The size of the plasmid varies from 1 to over 1,000 kbp , and the number of identical plasmids in a single cell can range anywhere from one to thousands under some circumstances.Bacterium, fungus team up to cause virulent tooth decayin toddlers.
Early childhood caries, a highly aggressive and painful form of tooth decay that
frequently occurs in preschool children, especially from backgrounds of poverty,
may result from a nefarious partnership between a bacterium and a fungus, according
to a paper published ahead of print in the journal Infection and Immunity.
The resulting tooth decay can be so severe that treatment frequently requires surgery -- in the operating
room, says corresponding author Hyun (Michel) Koo of the University of Pennsylvania.
"Our data will certainly open the way to test agents to prevent this disease, and even more intriguing,
the possibility of preventing children from acquiring this infection," says Koo.
In the study the investigators showed that infection by S. mutans and C. albicans together doubled the
number of cavities, and boosted their severity several-fold in rats.
Koo, of U. Penn's School of Dental Medicine, has spent 15 years studying how microbes construct the
biofilms, also known as plaque, that have plagued teeth since H. sapiens invented agriculture, bringing
large quantities of starch into the diet. (Caries are common in Neolithic skeletons, but virtually absent
from our Paleolithic ancestors.)http://www.sciencedaily.com/releases/2014/03/140312132625.htm
PLoS Pathog. 2010 Apr; 6(4): e1000886.
Published online 2010 Apr 29. doi:†
PMCID: PMC2861711
Candida albicans Interactions with Bacteria in the Context of Human
Health and Disease
and *
Hiten D. Madhani, Editor
This article has been other articles in PMC.
Humans are colonized by diverse populations of bacteria and fungi when in a healthy
state and in the settings of disease, and the interactions between these microbial
populations can be beneficial or detrimental to the host . Among these microbial
populations, Candida albicans is the fungus most commonly detected in association
with humans , and numerous studies have described C. albicans interactions with
its bacterial neighbors . Here, with a focus on C. albicans, we provide examples of
how bacterial-fungal interactions can influence human health. In addition, we
highlight studies that give insight into the molecular mechanisms that govern the
physical associations, interspecies communication, and changes in microbial behavior
and survival that occur when bacteria and fungi occupy the same sites.http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2861711/Viruses con bacteria into working for themDate:
January 26, 2012Source:
Massachusetts Institute of Technology, Department of Civil and Environmental EngineeringSummary:
Researchers have discovered that certain photosynthetic ocean bacteria need to beware of
viruses bearing gifts. These viruses are really con artists carrying genetic material taken from
their previous bacterial hosts that tricks the new host into using its own machinery to activate
the genes, a process never before documented in any virus-bacteria relationship. The con occurs
when a grifter virus injects its DNA into a bacterium living in a phosphorus-starved region of
the ocean.PMCID: PMC2861711http://www.sciencedaily.com/releases/2012/01/120126123712.htm
A few additional articles on why abx are harmful, we are not against the use of abx just contendthat they may be harmful when synergy or the true cause of our disease is not recognized. Our contentionmirrors that of the CDC among others in the treatment of ‘LYME disease’. We also contend that someanti fungals may be harmful and that anti virals in and of themselves are not effective in treating ‘fungalsynergy’.
A few more articles on:
Candida misidentified as algae and emerging fungal infections such as protothecosis.
And algaemia. As well as evidence of cyanobacteria and pathogen rise in the water in
North America and Europe.
Quick quote:
Scientists at Arizona State University tell us that antibiotic use is known to almost completely
inhibit excretion of mercury in rats due to alteration of gut flora,3 and even with the known
fact that antibiotics are creating powerful resistant bacterial strains does not stop doctors
from using them to their hearts and pharmaceutical companies content.The Impact of Antibiotics on The Gut Lining
The following is excerpted from the book by Dr. Natasha Campbell-McBride. In the chapter titled
"What Can Damage Gut Flora?," Campbell-McBride considers the impact of antibiotics, drugs
such as contraceptive pills and steroids, diet, environmental toxins, and more. She devotes two
pages to the impact of antibiotics. This excerpt is taken from p. 34 and 35.PenicillinsIn this group we have very widely used Amoxicillin, Ampicillin, Flucloxacillin and all otherantibiotics with Lactobacilli and Bifidobacteria, while promoting the growth of the pathogenicProteus family, Streptococci and Staphylococci. This particular group of antibiotics allow bacterianormally found only in the bowel to move up to the intestines, which predisposes the person todevelopment of IBS (Irritable Bowel Syndrome) and other digestive disorders.http://moldrecovery.blogspot.com/2013/04/the-impact-of-antibiotics-on-gutlining.html#.VNP4_d3XJi1Unusual Fungal and Pseudofungal Infections of Humans
Author Affiliations
.
1
Departments of Pathology
.
3
Medicine, College of Medicine
.
2
Department of Epidemiology, College of Public Health, University of Iowa, Iowa City, Iowa
The spectrum of mycotic disease continues to expand well beyond the familiar entities of candidiasis and aspergillosis (, ). The field of medical
mycology has become a challenging study of infections caused by a wide and taxonomically
diverse array of opportunistic fungi.http://jcm.asm.org/content/43/4/1495.fullMulticentre Etest evaluation of in vitro activity ofconventional antifungal drugs against Europeanbovine mastitis Prototheca spp. isolates
.
AbstractObjectives Bovine mammary protothecosis is a serious pathology that entails
high economic losses in the dairy industry. The disease, the frequency of which
has recently been increasing worldwide, is caused by unicellular, achlorophyllous,
yeast-like algae of two species: Prototheca zopfii and Prototheca blaschkeae.
The objective of this study was to investigate the in vitro activity of a panel of
conventional antifungal drugs against Prototheca spp. isolates.http://jac.oxfordjournals.org/content/67/8/1945
Fatal Algaemia in Patient with Chronic Lymphocytic Leukemia
, , , , , , , and
This article has been other articles in PMC.
To the Editor: Prototheca species are achlorophyllic lower algae, ubiquitous in nature,
which can cause human infections, particularly in immunocompromised patients ().
Human protothecosis is mostly caused by P. wickerhamii and P. zopfii. Although
such infections are infrequent, they can manifest themselves clinically as cutaneous
lesions, olecranon bursitis, and, even more rarely, as disseminated or systemic infections
(). These infections occur in severely immunocompromised patients, such as persons
with AIDS, or patients undergoing extensive treatment, such as cancer treatment or
organ transplantation (ñ). We describe a fatal case of P. wickerhamii algaemia in a
patient with chronic lymphocytic leukemia.http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2744238/Human Protothecosis (See site for additional articles as we have many, also anew species was identified in 2010 by a Japanese researcher, this was misidentifiedas Candida up until 1964. It is found world wide, they claim there are only 164known cases).Human ProtothecosisSUMMARY
Human protothecosis is a rare infection caused by members of the genus Prototheca. Prototheca
species are generally considered to be achlorophyllic algae and are ubiquitous in nature. The
occurrence of protothecosis can be local or disseminated and acute or chronic, with the latter
being more common. Diseases have been classified as (i) cutaneous lesions, (ii) olecranon
bursitis, or (iii) disseminated or systemic manifestations. Infections can occur in both
immunocompetent and immunosuppressed patients, although more severe and disseminated
infections tend to occur in immunocompromised individuals.http://cmr.asm.org/content/20/2/230.full
And finally, we contend that this pathogenisis and chronic disease has been
a widespread problem due to harmful cyanobacteria and harmful algal blooms
around the world for over a century. There are many documents by WHO on
this increasing worldwide problem that we strongly suggest that you investigate.
We find daily evidence of harmful cyanobacteria such as aeromonas, those
that cause protothecosis, as well as the most well known harmful cynabacterial
organisms. We include many in our files.http://en.wikipedia.org/wiki/Cyanobacteria

This is why fen-ben and resihi work. Yes, they all rely on an algal mechanism, a protein called ftsz, that is a conserved mechanism in algae, plant organelles, and all harmful bacteria. It was inserted shortly after cyanobacteria in evolution and has never transferred to humans, in millions of years, never. Fenben binds to and only to and disrupts that mechanism. That includes e coli, lyme, malaria, toxoplasmosis, fungi, virus, syphillis, AIDS, ALL, they all rely on it to replicate, transfer, reproduce, create energy, team up likely etc.

Saturday, March 28, 2015

Water is the major contributor to disease along with sanitary conditions. History has proven this. Pure water is imperative to good health. I find distilled is the only pure water I can find today. I would suggest putting your distilled water in a glass container as soon as possible.

Clean Food

Many algae, biotoxins, pesticides, herbicides and funguses are attached to your produce and meats. Cleaning can be accomplished by using CD, Ozone or Hydrogen Peroxide.

Other than Rife this is the most exciting information I have ever came accrossed. While having good and unusually quick success treating Lyme and it's Co-infections with Rife and Herbs I find regaining and regenerating the damaged areas left behind to be a slow and daunting task. Some symptoms, out of over 100, have actually returned and increased. While trying to solve this puzzle I have come accrossed some new information that has shed new light on part of modality I used to treat Lyme. I urge all to look at this as extremely important part of complete healing and as an essential element of my advised FAVORITES.

Listed below are the issues I'm finding were aided by the use of Ivermectin and Fenbendazole along with Reishi. The PROTOCOL below is used as a Morgellons - Algae -Biotoxin - Autism protocol for now but my experience is showing a very wide range of issues that are begin addressed. I did not know this at the time I dealt with Lyme and Company but basically used it with the addition of Rife and Herbs, MMS and Naturals. This I believe is why I had fairly quick results with Lyme and Company. I did use Fenbendazole plus Ivermectin but not as religiously as below. Not being totally satisfied that Lyme etc. could cause some of these more odd symptoms, I've been busy trying to regenerate the parts that were severely damaged by pathogens with Rife and Herbs-Food etc. I'm quite sure the Lyme and Co-infections are gone, yet for the last 6-8 months, after an accident with ROUND-UP and an Adjuvant ( I since read it makes it 2,000 times as toxic and used in agriculture) which sent me spiraling downward in terms of chronic pain along with a few weeks of bloody diarrhea, headache for weeks and severe arthritis pain in places that was not there before. etc., and the gradual return of the below symptoms, the information documented below now seems to be making a lot of sense. An algae problem with my aquarium got me to thinking of my well water and if algae and iron bacteria can live inside us, hence the name of this document OH MY.

While I did not have to deal with Morgellons ( THANK GOD), I did have many parasites exit thru the skin, mainly in the torso. I'm simply skipping over the parts that do not apply, like the fiber issues and sores.

This link was my light bulb moment. Suddenly many things I still questioned were staring me right in the face. This simple chart has put all the hurdles of Lyme and Company plus many other conditions into what I consider a very sensible explanation to a multitude of conditions and idiopathic diseases.

Now on to our contention with articles, that without ‘fungal synergy’ no pathogen is

particularly harmful on it’s own and that ‘fungus’ plays a major part in infectivity and

virulence. It takes two to tango.

It is not just our contention but that of Lynn Margulis, who alluded that a spirochete was

not infective on it’s own and proved that spirochetes drop DNA in relation to the host but

was never able to finish her work due to her death.

Paul Cox, his work on toxins, evironmental exposure, protein formation in the role of ALS

and more. Alan Barbour, who could not figure out why bovine serum made Borrelia

more or less pathogenic and infective. And many others working in symbiotic evolution

and disease.

First infectivity and replication from UGA in malaria.

Athens, Ga. - Long ago, when life on Earth was in its infancy, a group of small single-celled algae

propelled themselves through the vast prehistoric ocean by beating whip like tails called flagella. It's a

relatively unremarkable tale, except that now, more than 800 million years later, these organisms have

evolved into parasites that threaten human health, and their algal past in the ocean may be the key to

stopping them.

The organisms are called apicomplexa, but people know them better as the parasites that cause malaria and toxoplasmosis, serious diseases that infect millions of people every year, particularly in the developing world.

Now, researchers at the University of Georgia have discovered how an important structure inside these parasitic cells, which evolved from the algal ancestor millions of years ago, allows the cells to replicate and spread inside their hosts. Their research may soon lead to new therapies to halt these deadly pathogens before they cause disease.

In order to survive, the parasitic apicomplexa must invade an animal or human and force its way

into the cells of its host. Once inside the host cell, the parasite begins to replicate into numerous

daughter cells that in turn create additional copies, spreading the infection throughout the body.

In their study, published Dec. 11 in PLoS Biology, the researchers demonstrate that, during the process

of replication, the parasite cell loads genetic material into its daughter cells via a strand of fiber that connects the two. By altering the genes for the components of the fiber in the laboratory, the researchers discovered that they could prevent parasite replication, making the parasite essentially harmless.

"These altered parasites can initially infect cells, but once we turn off the fiber genes, they

cannot create new daughter cells and spread," said Maria Francia, lead author and doctoral

candidate in the department of cellular biology. "Since it cannot replicate, the parasite eventually

dies without causing serious harm."

This replication fiber appears to have evolved from the flagellum that ancient algae used to

swim.

"This was a surprising finding," said Boris Striepen, a Georgia Research Alliance Distinguished Investigator

in UGA's Center for Tropical and Emerging Global Diseases. "These parasites no longer use flagella

to swim, but they have apparently repurposed this machinery to now organize the assembly of an invasive cell."

During evolution, flagella have been reengineered to serve numerous different functions in animals,

including the sensors that allow us to see and smell. This study suggests that in these parasites structures used to invade host cells may be also derived from flagella.

Current treatments for diseases like malaria are threatened by the parasite becoming resistant to the

drugs, so the need for new therapies is always pressing.

This algae-based connective fiber may serve as a promising target for anti-parasitic drug

development, said Striepen, who is also a cellular biologist in the Franklin College of Arts and

Sciences. He cautions, however, that more work must be done to learn how to manipulate or

destroy the fiber in parasites that have infected humans or animals.

But both Striepen and Francia argue that scientists do well to pay close attention to the evolutionary

history of the organisms they study.

"It is extremely important to understand the evolution of different organisms, but especially the evolution of pathogens," Striepen said. "The analysis of their evolution produces important opportunities to develop treatments, but it also helps us understand the basic structure of the pathogens that we must fight."

UGA Center for Tropical and Emerging Global Diseases

The University of Georgia Center for Tropical and Emerging Global Diseases draws on a strong foundation of parasitology, immunology, cellular and molecular biology, biochemistry and genetics to develop medical and public health interventions for at-risk populations. Established in 1998, the center promotes international biomedical research and educational programs at UGA and throughout Georgia to address the parasitic and other tropical diseases that continue to threaten the health of people throughout the world. For more information about the center,

see <a>http://ctegd.uga.edu</a>

Alan Barbour among other authors in the infectivity and virulence of Borrelia.

In conclusion, our data suggest that variations in BSK medium formulations have

significant effects on the infectivity and pathogenicity of B. burgdorferi clinical

profile of the Lyme disease spirochete, Borrelia burgdorferi, as a result of in vitro cultivation.

Infect. Immun. 56:1831-1836.

A plasmid is a small molecule within a cell that is physically separated from a chromosomal DNA and

can replicate independently. They are most commonly found in as small, circular, double-stranded DNA molecules; however, plasmids are sometimes present archea and eukarotiuc organisms. In nature,

plasmids often carry genes that may benefit the survival of the organism, for example antibiotic resistance. While the chromosomes are big and contain all the essential information for living (an adequate analogy is the hard-drive of a computer), plasmids usually are very small and contain additional information (in this analogy, plasmids are the USB flash drives). Artificial plasmids are widely used as in vectors and molecular cloning , serving to drive the replication of sequences within host organisms. Plasmids are considered replicons, a unit of DNA capable of replicating autonomously within a suitable host. However, plasmids, like viruses , are not considered by some to be a form of life. Plasmids can be transmitted from one bacterium to another (even of another species) via three main mechanisms:transformation ,transduction, and conjugation . This host-to-host transfer of genetic material is called , horizontal gene transfer and plasmids can be considered part of the transduction and conjugation. Unlike viruses (which encase their genetic material in a protective protein coat called a capsid ), plasmids are "naked" DNA and do not encode genes necessary to encase the genetic material for transfer to a new host. However, some classes of plasmids encode the conjugated sex pilus necessary for their own transfer. The size of the plasmid varies from 1 to over 1,000 kbp , and the number of identical plasmids in a single cell can range anywhere from one to thousands under some circumstances.

Bacterium, fungus team up to cause virulent tooth decay

in toddlers.

Early childhood caries, a highly aggressive and painful form of tooth decay that

frequently occurs in preschool children, especially from backgrounds of poverty,

may result from a nefarious partnership between a bacterium and a fungus, according

to a paper published ahead of print in the journal Infection and Immunity.

The resulting tooth decay can be so severe that treatment frequently requires surgery -- in the operating

room, says corresponding author Hyun (Michel) Koo of the University of Pennsylvania.

"Our data will certainly open the way to test agents to prevent this disease, and even more intriguing,

the possibility of preventing children from acquiring this infection," says Koo.

In the study the investigators showed that infection by S. mutans and C. albicans together doubled the

number of cavities, and boosted their severity several-fold in rats.

Koo, of U. Penn's School of Dental Medicine, has spent 15 years studying how microbes construct the

biofilms, also known as plaque, that have plagued teeth since H. sapiens invented agriculture, bringing

large quantities of starch into the diet. (Caries are common in Neolithic skeletons, but virtually absent

Department of Epidemiology, College of Public Health, University of Iowa, Iowa City, Iowa

The spectrum of mycotic disease continues to expand well beyond the familiar entities of candidiasis and aspergillosis (, ). The field of medical

mycology has become a challenging study of infections caused by a wide and taxonomically

diverse array of opportunistic fungi.

<a>http://jcm.asm.org/content/43/4/1495.full</a>

Multicentre Etest evaluation of in vitro activity of

conventional antifungal drugs against European

bovine mastitis Prototheca spp. isolates

.

Abstract

Objectives Bovine mammary protothecosis is a serious pathology that entails

high economic losses in the dairy industry. The disease, the frequency of which

has recently been increasing worldwide, is caused by unicellular, achlorophyllous,

yeast-like algae of two species: Prototheca zopfii and Prototheca blaschkeae.

The objective of this study was to investigate the in vitro activity of a panel of

conventional antifungal drugs against Prototheca spp. isolates.

<a>http://jac.oxfordjournals.org/content/67/8/1945</a>

Fatal Algaemia in Patient with Chronic Lymphocytic Leukemia

, , , , , , , and

This article has been other articles in PMC.

To the Editor: Prototheca species are achlorophyllic lower algae, ubiquitous in nature,

which can cause human infections, particularly in immunocompromised patients ().

Human protothecosis is mostly caused by P. wickerhamii and P. zopfii. Although

such infections are infrequent, they can manifest themselves clinically as cutaneous

lesions, olecranon bursitis, and, even more rarely, as disseminated or systemic infections

(). These infections occur in severely immunocompromised patients, such as persons

with AIDS, or patients undergoing extensive treatment, such as cancer treatment or

organ transplantation (ñ). We describe a fatal case of P. wickerhamii algaemia in a

patient with chronic lymphocytic leukemia.

<a>http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2744238/</a>

Human Protothecosis (See site for additional articles as we have many, also a

new species was identified in 2010 by a Japanese researcher, this was misidentified

as Candida up until 1964. It is found world wide, they claim there are only 164

known cases).

Human Protothecosis

SUMMARY

Human protothecosis is a rare infection caused by members of the genus Prototheca. Prototheca

species are generally considered to be achlorophyllic algae and are ubiquitous in nature. The

occurrence of protothecosis can be local or disseminated and acute or chronic, with the latter

being more common. Diseases have been classified as (i) cutaneous lesions, (ii) olecranon

bursitis, or (iii) disseminated or systemic manifestations. Infections can occur in both

immunocompetent and immunosuppressed patients, although more severe and disseminated

infections tend to occur in immunocompromised individuals.

<a>http://cmr.asm.org/content/20/2/230.full</a>

And finally, we contend that this pathogenisis and chronic disease has been

a widespread problem due to harmful cyanobacteria and harmful algal blooms

around the world for over a century. There are many documents by WHO on

this increasing worldwide problem that we strongly suggest that you investigate.

We find daily evidence of harmful cyanobacteria such as aeromonas, those

that cause protothecosis, as well as the most well known harmful cynabacterial

organisms. We include many in our files.

<a>http://en.wikipedia.org/wiki/Cyanobacteria</a>

Charts from WHO and US/EPA documents on water quality.

Our desire and mission is to help as many

as possible recover, spread awareness and

lovingly support each other and as many

efforts as possible to restore balance to

ourselves and our planet. Thank you Steve

Beddingfield and others who help shine

the light in a world of that often seems

full of darkness.

This is why fen-ben and resihi work. Yes, they all rely on an algal mechanism, a protein called ftsz, that is a conserved mechanism in algae, plant organelles, and all harmful bacteria. It was inserted shortly after cyanobacteria in evolution and has never transferred to humans, in millions of years, never. Fenben binds to and only to and disrupts that mechanism. That includes e coli, lyme, malaria, toxoplasmosis, fungi, virus, syphillis, AIDS, ALL, they all rely on it to replicate, transfer, reproduce, create energy, team up likely etc.

About Me

Grew up in a rural area, farming and logging and construction filled my life with many skills. Yet my heart as always followed a simpler life. It has been for the love of nature and all it entails that has led me to happier place full of adventures, great and small, trying never to miss the opportunity to help each and everyone.