Personal experience has proved repeatedly that a few
grams of AA will relieve coughing urgency within a few minutes. Where continued
coughing causes a chain reaction of irritation, stimulated by environmental
causes, the AA works to relieve the itch that stimulates the cough.The “cure” is temporary, more AA is needed
to continue the relief. Eventually the high need for AA diminishes. COPD
exacerbations may be soothed by Liposomal AA and sodium
ascorbate solution in a nebulizer.The most extreme itchy cough is from Whooping Cough, caused by Pertussis
infection.

AGreenMedInfo
paper by Dr. S. Humphriescites AA as a treatment for Pertussis
(Whooping Cough). AA, in high enough multi gram dosages, is an effective acute
neutralizer of all the bacterial toxins generated by the infections that the
DPT vaccine protects against.With AA
powder so effective against viruses and bacteria (in 1-3 hour, repeated,
several-gram oral AA dosages, 24/7 for about a week), many may question the
medical industry’s overstated need for the whooping cough (Bordetella. pertussis
bacteria) vaccine, which actually is reported not to work very well. IV Sodium
ascorbate can achieve even higher antiviral effects.

Oral intake of
a few grams ofLiposomal
AAis even more effective than AA powder capsules against acute
bacterial and viral invasions of the respiratory tract.See
Amazon reviews.

TheOriginal
Omerod Canadian Papershowed low dosages of AA worked to speed up
recovery from whooping cough. The AA intake was at least 1/10th of
an effective sustained medicinal dose: experimental dosage was too little and
not frequent enough to effect a rapid cure. But benefits from even these low AA
dosages were remarkable.

A 1937
Ormerod Follow-up Paperconfirms that 350 – 100 mg/day amounts of AA
temporarily reduces symptoms. It did not go far enough: larger (1-3 gram)
amounts of AA, administered every 1 to 3 hours, would have shown even greater
benefits. Remember AA’s blood half lifetime is ½ hour. So the Ormerod dosages
and lack of frequency must have left the patients with low (unmeasured) AA
antioxidant blood levels for much of each day, allowing the bacteria to
proliferate.

AA is an essential food-element that we do not make,
but need systemically to sustain life and good health. There is no practical
upper limit to its safety. Its blood half lifetime is 30 minutes. When we are
sick we convert antioxidant AA from a helpful form to a dangerous form
(dehydroxy AA) that is rapidly excreted. In the presence of toxins or
allergies, we can easily run out of helpful antioxidant vitamin C (AA).

Vitamin C’s
effectiveness remains “controversial”
because knowledge of its effectiveness, its low cost, its wide availability and
its safety threaten so many other toxic patented (high profit) medicines that
sustain the “ethical” drug industry.It
may make many of them unnecessary.

Nonetheless, AA is still highly useful as
complementary “medicine” to neutralize life-threatening endotoxins released by
dying bacteria and by invaded body cells that have been killed by prescription
antibiotics and oxides. The toxins and low systemic antioxidant AA blood levels
and high concentrations of oxidized AA (DHA) induce an allergic like histamine
flare reaction. Several grams of AA intake will reduce or eliminate the
severity of drug-induced toxemia and allergic flares. Intake ofAA must be repeated frequently, because it
reacts and is consumed, unlike a vitamin that may be persistent for days.

AA reduces or prevents drug and vaccine adverse
reactions that produce toxins, oxides and inflammation. Illness or antibiotic
reactions cause systemic or local toxemia. Frequently repeated AA replenishment
is needed to keep up with the relentless generation of toxins.AA intake must continue until all the
microbes are eliminated and all toxins are flushed from the body.This may take several days to weeks, of
elevated, frequent AA intake.

How
much Vitamin C. AA intake, in amounts 50 times larger than the (~60
milligram) RDA, i.e., several grams per 1-3 hours, when periodically
administered, provides toxin-neutralizing relief. Symptoms do abate within a few minutes.

Repeated intake
of enough AA provides relief from adverse drug reactions, from anaphylaxis-like
toxic shock symptoms, from smoke inhalation and carbon monoxide poisoning, from
snake and insect venoms, from food poisoning toxins, from jellyfish stings and
sunburn, from plant poisons (mushroom and mold mycotoxins and poison ivy), from
alcohols and barbiturate intoxication, and from allergic reactions of every
kind.

Dehydroascorbic
acid (DHA) is the oxidized (and oxidizing) form of AA.DHA is produced instantly when AA meets a
toxin molecule and acts to neutralize it.DHA, an oxidant,aH is
toxin-generating in its actions.

An important
critical measurement of vitality is the AA/DHA ratio in the blood. The AA/DHA =
Antioxidant/Oxidant concentrations of ascorbic acid. If AA/DHA is much greater
than one (~7) , one is in good health. If AA/DHA is near or less than 1 and
decreasing, the patient is in a death spiral, characterized in the extreme by
symptoms of anaphylaxis. Measuring this dynamic ratio may be hard to do. But
the symptoms of anaphylaxis have been reversed so many times by the immediate
administration of 6-25 of grams of IV sodium ascorbate (AA), which will tip the
AA/DHA ratio’s balance back to the safe range.

This effect
does not last long, and more IV or oral AA needs to be administered repeatedly
for a long time until the patient is past the time when the toxins(endotoxins, exotoxins or allergens) have
stopped being generated and all are neutralized. This may take days to weeks.
Sometimes the patient recovers normal vitality in a few minutes, and if no more
AA is administered, within hours or days, a rapid severe relapse can result.

Anaphylaxis:

Anaphylaxis is a severe, life
threatening, systemic allergic reaction to a toxin. AA blood levels plummet and
systemic histamine levels rise exponentially. AA/DHA ratio is near or less than
one and decreasing as the allergens cause a toxin release cascade.Bradykenin is one name associated with the
toxin of anaphylaxis.

Pre-loading systemic AA via low
nutritional doses reduces the chance and severity of anaphylaxis and
allergies.In crisis, rapid high
systemic administration of adrenaline causes the release of up to about 6 grams
of AA stored in the adrenal glands, reversing anaphylaxis and severe systemic
allergies, stopping the generation of histamine from mast cells. The AA
released neutralizes the Bradykenin toxin and others that are formed in the
presence of microbes or allergens.Raising AA blood levels may be only temporary unless additional
supplemental AA is administered.

Because shortage of AA and surplus
of DHA are the primary causal factors of anaphylaxis, the administration
oftens of grams ofIV sodium ascorbate may be preferable to use
of adrenaline alone, which has but a one-time and limited effect. Use of both,
together, may be optimal, since repeated intake of AA by oral, parenteral
injectionand IV methods can protect
against the effects of continuously generated/released toxins of every kind.
See References 2,3, and 4, below.

Conclusions:

Intensive care
medicine needs to take heed of the relationship between antioxidant AA and DHA
blood concentrations. When antioxidant AA is depleted and DHA predominates,
then many of the biochemical pathways of the body stop working. Systemic
release of histamine occurs and blood levels of histamine rise exponentially.The eventual result is death.

It is time for
medical doctrine to recognize the need to control the AA/DHA ratio to keep it
in the safe range.Patient outcomes
will necessarily improve in critical cases if AA/DHA is high.AA (sodium ascorbate) administration in tens
of grams per hour can easily do this. It now is time to discard all anti-AA
prejudices in medical teaching, doctrine, and practices.AA
pharmacokinetics and pharmaco dynamics should be taught. AA
biochemistry is highly dynamic and interacts rapidly with vital chemical
pathways.

Routine
emergency administration of IV AA is as important as maintaining proper
electrolyte balances.A lot of
mortality and morbidity will be avoided if AA is used as a first aid toxin
neutralizer.

It should be
recognized that treating AA as a limited intake vitamin, and restricting its
intake to one dose per day can lead to swift and extreme AA depletion.The amount of the water-soluble AA depletion
can be calculated using the ½ hour blood half lifetime.In case of continuously generated toxins,
sudden death may result for cases of high oxidizing stress, toxemia and
anaphylaxis, if frequent AA intake is not provided.

Liposomal AA
(L-AA) can provide 5 times better gut-to-blood transfer compared to water
soluble AA. Its blood retention is longer. It provides higher blood-to-brain
transport efficiencies. L-AA targets microbe and infected body cells having
lipid capsules and it delivers the AA with 7 to 10 times greater clinical
effectiveness compared with IV sodium ascorbate on an AA gram-for-gram basis.