Symptoms Of Migratory Arthritis

3 Steps to Permanent Relief for SI Joint and Pelvic Pain

Today we are going to cover the 3 steps topermanent relief for pelvic and SI joint pain. Now what is SI joint painé The SI joint, ifyou do have SI joint pain, is pain on either side of the tailbone where the tailbone connectsto the pelvis. So this is a drawing of the pelvis. This is your tailbone. This isyour lumbar spine or your lower back, the bones in your lower back called vertebraesitting on top of your tailbone. Then on either side of that you have your right hip or yourleft hip if we are looking at a person from the front. When we see somebody with SI jointpain, they will typically have pain on that given side. 70% of the time, it is on theright side. Don't ask me why that is but

70% of the time we know it is on the right. You canalso get shooting groin pain, buttocks pain and symptoms down the outside of the leg thatis affected. It might be pain, numbness, tingling, burning. Typically, that personsuffers from a heaviness as well. It can also cause some incontinence and leaking issuesas along to effect sexual performance as well. It can be absolutely devastating ifthe pelvic problem is bad enough. Fortunately, most people just have pain on one side. Sowhat can you do about ité That person who has SI joint problems typically has troublerolling over in bed, getting in and out of a car, crossing one leg over the other. Usuallythey have trouble sitting for long periods

of time especially if it is on a couch or a softsurface. So it can be absolutely devastating. Now what are the steps to permanent relieffor thaté Number 1 is this. Most people when we seethem if they are going to try exercises on their own and they only have a little bitof SI joint pain or pelvic pain, they are doing stretches. Yes, there is a very basic stretchwhich most people show me on Day 1. It is a piriformis stretch which is where you grabthe knee and pull it up and across to the opposite shoulder. It will give the personwho suffers pelvic or SI joint problems and pain temporary relief. However, long termwe don't want to stretch. We want to

strengthen or stabilize. Why is thaté Whensomebody has an SI joint problem, so the center of gravity of your entire body is inthe center of your pelvis. That needs to move the right way and it needs to be stable whenyou are doing day to day activities. If it is not stable enough, what will happen is thejoint will move so far that it becomes stuck. So if we are looking at the tailbone, we havethe tailbone and we have the pelvis and you get a normal gliding day to day. If morestress, more force is put through the pelvis than the pelvis itself can handle, that SIjoint can handle, what will happen is you actually become stuck. Now that creates allsorts of problems for the muscle around it.

What most people do is try to stretch theirway out of it. The problem is not stretching and it is not flexibility. It is actuallya stability issue. If we want to stabilize, we need to do strengthening exercises. If you go on ourYouTube channel, on the Madden PT official YouTube channel, there is a tutorialcalled Top 3 Exercises for SI Joint and Pelvic Stability. They are very low grade, kindergartenlevelexercises where you can at least begin stabilizing the muscles that controlyour pelvis. But we don't want to stretch, we want to think stabilize. Step 2 is we want to take a look at our habits.So what are some habits that lead to

pelvic instability and problemsé Number oneis standing with the weight shifted. So if I'm standing like this with my weight shiftedside to side for long periods of time, what that does is over years and years and yearsit weakens the ligaments which are tissue that hold joints together that we can't control.Think about somebody with an ankle sprain. They sprained a ligament. They stretchedthe ligament but it is not a muscle. It is different than a muscle. There are littlehabits that we do. If I stand like this and let's say I weigh 175 pounds and if I'm standinglike this, I have 150 pounds on one side and 25 pounds on the other. Where if I'm standingbalanced, I have 87 Â½ pounds on each

Rheumatic fever heart disease

â€œRheumatismâ€� is used to describe inflammationin the joints, muscles, and the fibrous tissue, so rheumatic fever is a type of inflammatorydisease that can damage the heart tissue, and lead to rheumatic heart disease. Rheumatic fever develops after streptococcalpharyngitis, inflammation of the throat due to Streptococcus pyogenes where pyogenes literallymeans â€œmakes pusâ€�. The bacteria is sometimes referred to as â€œGroup A beta hemolyticâ€�streptococcus, and the infection itself is most often just called Strep throat. Thisparticular group of streptococcus has an antigen that lumps it into a group called â€œgroupAâ€�, and it also produces an enzyme called

streptolysin, that completely lyses nearbyred blood cells, or causes them ruptureâ€”rupturing red blood cells is called hemolysis, rightéAnd when those red blood cells rupture and are destroyed, it's called betahemolysisâ€”asopposed to alphahemolysis, where cells aren't actually destroyed, they're just damagedor bruised. Some of these strep bacteria have a proteinon their cell wall called â€œM proteinâ€�, and this particular protein is highly antigenic,meaning the immune system sees it and recognizes it as a foreign molecule, and mounts an immuneresponse, which rightfully so, produces antibodies against these proteins. Those antibodies,though, are thought to crossreact with proteins

on some of our body's own cells, like cellsin the myocardium (or heart muscle) and heart valves, but also cells in the joints, theskin and the brain. This phenomenon, where antibodies accidentally target proteins onour own cells because they look like the proteins on foreign cells, is called molecular mimicry,and is an example of what's called a type 2 hypersensitivity reaction. Once bound tocardiac tissue, the antibodies activate nearby immune cells, which causes a cytokinemediatedinflammatory response and tissue destruction. Obviously though, not everyone that gets strepthroat gets rheumatic fever, and it's actually only a small minority that get it, estimatedaround 3%, and it's more likely in children

or people in areas of poverty and crowding. A lot of patients that do have rheumatic feverfrom strep, sometimes called acute rheumatic fever, will have a variety of of alfindings. The most common of which is migratory polyarthritis of the jointsâ€”where multiplelarge joints become inflamed, swollen and painful, one after another, although thisdamage isn't permanent. Secondly, some patients have pancarditis, or inflammation of all threelayers of the heart tissue. The first, endocarditis, is inflammation of the inner lining whichincludes the valves. The mitral valve is most commonly affected, although the aortic valvemay also be affected. The next is myocarditis,

inflammation of the myocardium, or heart muscle.Inflamed areas in the myocardial tissue are called Aschoff bodies, which are areas withfibrinoid necrosis, with immune cells like T cells and these characteristic Anitschkowcells, enlarged macrophages which have characteristic caterpillarlooking nuclei. It turns out thatmyocarditis is the most common cause of death in acute rheumatic fever because this inflammationand necrosis makes the heart wall unable to contract with full force, which results inheart failure. Finally there's pericarditis, or inflammation of the outer covering of theheart called the pericardium, which can cause pain as well as a friction rub from the inflamedvisceral pericardium rubbing against the inflamed

parietal pericardium which can be heard witha stethoscope. In addition to joint and heart problems, thehypersensitivity reaction in rheumatic fever can affect other tissues as well. Patientscan develop subcutaneous nodules, these firm lumps under the skin made up of collagen.They might also have erythema marginatum, a reddish rash that shows up as rings on thearms or trunk. Also, Sydenham's chorea, which is a set of rapid movements of the faceand the arms, from an autoimmune reaction against basal ganglia of the brain, and thisone typically won't appear until late in the disease, at least 3 months after infection.These five signs constitute the major diagnostic

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