Science can seek to quantify, qualify and explain what happens but it cannot prove that what happened did not happen.

To deny the experience of another is the work of cults. In order to break down the self of the initiate they confuse them to the point that they no longer trust their own sense of life. The initiate's personal experience is replaced with cult approved dogma.

In the case of modern medicine one way they break that trust is by having their senior members, or priests, wear official looking white robes. They put on the wall the certificates showing the degree to which the "M.D." or priest has progressed. Are they a simple parish priest or are they a bishop or cardinal? They then parade around as if they are "educated" when in fact they are simply trained and programmed. The "M.D.s" then make pronouncements and if the one confessing is not wise to the ways, they will mistake the trained, programmed reactions of the "M.D." for educated, informed, well thought out responses.

Another way they break the self trust of the unsuspecting human is to publicize "scientific studies." These studies, when examined in detail, are found to be simply dogmatic declarations. They are the "scientific" equivalent of discussions about how many angels can stand on the head of a pin but they are presented to the public as "science." They are also used in the training and programming of "M.D.s."

Many people are happy to go along with this. They have a general sense that "science" has given us all the wonders of modern life. Therefore it works and who are we, who have been trained from birth to think we are inferior through public "schooling," aka training and programming, to question those who hand down the word from on high?

Some of us are too smart for this bullshit. We figured out as preteens or early in our teenage years that "polite society" was full of hypocrisy, is in fact, a smoke screen for all the immorality of human avarice. We rebelled while our classmates who were most easily able to receive and accept the programming moved up the ranks with straight A grades. Their accolades, the proof of their fitness for high level work within the cult piled up. Now some of them are "M.D.s" aka "legal drug dealers." Most of us who were too smart, too tuned in to the actual have ended up as illegal drug dealers, or otherwise made our way on the outskirts of the polite society to which no humane, aware human could comfortably belong.

Yet somewhere in us we continued to think that those kids we made fun of for so readily following orders and believing the unexamined words of the "teachers" or programmers now that they have attained higher degrees within this hypocritical system have finally come to understand the actual. We vaccinated our kids because our classmates who got better grades than us and accumulated more accolades demonstrating the depths they could reach within the depraved system told us we should. We fucked up. We failed to trust ourselves. They beat us. We could not absorb the programming to rise (or sink) through the ranks, yet somehow we absorbed the programming that we were inferior because of this inability to completely shut down our innate humaneness. And then we watched the straight A students inject their magic potions into our kids and our kids died, or became maimed.

Some of us could not find the courage within to reclaim our power even then. We continue to believe the straight A students who do as they are told and simply repeat what they have been programmed to say are in some way superior, or "more intelligent" than we are. When they say the thing we observed with our own eyes did not happen we accept it. We even defend their words with vitriol of our own.

Others felt the split and could not deny it. They know what they saw. No amount of cult programming can break through their direct, undeniable experience. They may not have fully realized the depths of deception that serve as the foundation of modern "medicine" but they know that at least in this case they are right and their straight A classmate is wrong.

Within all this there are a handful of actual scientists who somehow manage to use the machinery of "civil society" to do actual science. When they hear from a parent the story of how vaccines damaged their child, they do not repeat what they have been programmed to say. Instead, they hear the words of the human in front of them. They seek to understand what is happening, why, and what to do about it.

Here is a small sample of their work. Thank you Ginger Taylor for collecting these studies.

In 21 published cases of the Court of Federal Claims, which administers the VICP, the Court stated that the petitioners had autism or described autism unambiguously.

This finding calls into question the decisions of the Court of Federal Claims in the Omnibus Autism Proceeding in 2009 and 2010 and the statement of Health and Human Services on its website that “HHS has never concluded in any case that autism was caused by vaccination.”3 http://www.ncbi.nlm.nih.gov/pubmed/19234401

Autism incidence in California shows no sign yet of plateauing. Younger ages at diagnosis, differential migration, changes in diagnostic criteria, and inclusion of milder cases do not fully explain the observed increases. Other artifacts have yet to be quantified, and as a result, the extent to which the continued rise represents a true increase in the occurrence of autism remains unclear.4 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0027897

The odds of receiving education intervention services were approximately nine times as great for vaccinated boys (n = 46) as for unvaccinated boys (n = 7), after adjustment for confounders. 8 http://www.ncbi.nlm.nih.gov/pubmed/21058170

Change points in these countries corresponded to introduction of or increased doses of human fetal cell line manufactured vaccines, while no relationship was found between paternal age or Diagnostic and Statistical Manual (DSM) revisions and autistic disorder diagnosis. Further, linear regression revealed that Varicella and Hepatitis A immunization coverage was significantly correlated to autistic disorder cases. R software was used to calculate change points. Autistic disorder change points years are coincident with introduction of vaccines manufactured using human fetal cell lines, containing fetal and retroviral contaminants, into childhood vaccine regimens. This pattern was repeated in the US, UK, Western Australia and Denmark. Thus, rising autistic disorder prevalence is directly related to vaccines manufactured utilizing human fetal cells.10 http://www.ncbi.nlm.nih.gov/pubmed/16338635

We found that for every 1000 pounds of industrial release, there was a corresponding 2.6% increase in autism rates (po.05) and a 3.7% increase associated with power plant emissions (Po.05). Distances to these sources were independent predictors after adjustment for relevant covariates. For every 10 miles from industrial or power plant sources, there was an associated decreased autism Incident Risk of 2.0% and 1.4%, respectively (po.05).12 http://www.ncbi.nlm.nih.gov/pubmed/18482737

There is a positive correlation between reduced levels of these proteins and loss of previously acquired language skills in children with autism.

Taken together, these studies suggest increased oxidative stress in autism that may contribute to the development of this disease. A mechanism linking oxidative stress with membrane lipid abnormalities, inflammation, aberrant immune response, impaired energy metabolism and excitotoxicity, leading to clinical symptoms and pathogenesis of autism is proposed.15 http://www.ncbi.nlm.nih.gov/pubmed/17090484

This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism.16 http://www.ncbi.nlm.nih.gov/pubmed/16151044

The most replicated finding in autism neuroanatomy-a tendency to unusually large brains-has seemed paradoxical in relation to the specificity of the abnormalities in three behavioral domains that define autism. We now know a range of things about this phenomenon, including that brains in autism have a growth spurt shortly after birth and then slow in growth a few short years afterward, that only younger but not older brains are larger in autism than in controls, that white matter contributes disproportionately to this volume increase and in a nonuniform pattern suggesting postnatal pathology, that functional connectivity among regions of autistic brains is diminished, and that neuroinflammation (including microgliosis and astrogliosis) appears to be present in autistic brain tissue from childhood through adulthood. Alongside these pervasive brain tissue and functional abnormalities, there have arisen theories of pervasive or widespread neural information processing or signal coordination abnormalities (such as weak central coherence, impaired complex processing, and underconnectivity), which are argued to underlie the specific observable behavioral features of autism. This convergence of findings and models suggests that a systems- and chronic disease-based reformulation of function and pathophysiology in autism needs to be considered, and it opens the possibility for new treatment targets.

Oxidative stress, brain inflammation and microgliosis have been much documented in association with toxic exposures including various heavy metals...the awareness that the brain as well as medical conditions of children with autism maybe conditioned by chronic biomedical abnormalities such as inflammation opens the possibility that meaningful biomedical interventions maybe possible well past the window of maximal neuroplasticity in early childhood because the basis for assuming that all deficits can be attributed to fixed early developmental alterations in neural architecture has now been undermined.17 http://www.ncbi.nlm.nih.gov/pubmed/23867105

Vaccines, in several reports were found to be temporally followed by a new onset of autoimmune diseases. The same mechanisms that act in infectious invasion of the host, apply equally to the host response to vaccination. It has been accepted for diphtheria and tetanus toxoid, polio and measles vaccines and GBS. Also this theory has been accepted for MMR vaccination and development of autoimmune thrombocytopenia, MS has been associated with HBV vaccination.21 http://montanaim.com/pubs/Heavy_Metals_Article.pdf

Metals are ubiquitous in our environment, and exposure to them is inevitable. However, not all people accumulate toxic levels of metals or exhibit symptoms of metal toxicity, suggesting that genetics play a role in their potential to damage health. Metal toxicity creates multisystem dysfunction, which appears to be mediated primarily through mitochondrial damage from glutathione depletion.22 http://www.ncbi.nlm.nih.gov/pubmed/21350943

Thimerosal at concentrations relevant for infants' exposure (in vaccines) is toxic to cultured human-brain cells and to laboratory animals.

Our results provide strong evidence supporting a link between autism and the aluminum in vaccines. A literature review showing toxicity of aluminum in human physiology offers further support. Mentions of autism in VAERS increased steadily at the end of the last century, during a period when mercury was being phased out, while aluminum adjuvant burden was being increased. Using standard log-likelihood ratio techniques, we identify several signs and symptoms that are significantly more prevalent in vaccine reports after 2000, including cellulitis, seizure, depression, fatigue, pain and death, which are also significantly associated with aluminum-containing vaccines. We propose that children with the autism diagnosis are especially vulnerable to toxic metals such as aluminum and mercury due to insufficient serum sulfate and glutathione. A strong correlation between autism and the MMR (Measles, Mumps, Rubella) vaccine is also observed, which may be partially explained via an increased sensitivity to acetaminophen administered to control fever.24 http://link.springer.com/article/10.1007%2Fs12026-013-8403-1#page-1

We have examined the neurotoxicity of aluminum in humans and animals under various conditions, following different routes of administration, and provide an overview of the various associated disease states. The literature demonstrates clearly negative impacts of aluminum on the nervous system across the age span. In adults, aluminum exposure can lead to apparently age-related neurological deficits resembling Alzheimer’s and has been linked to this disease and to the Guamanian variant, ALS–PDC. Similar outcomes have been found in animal models. In addition, injection of aluminum adjuvants in an attempt to model Gulf War syndrome and associated neurological deficits leads to an ALS phenotype in young male mice. In young children, a highly significant correlation exists between the number of pediatric aluminum-adjuvanted vaccines administered and the rate of autism spectrum disorders. Many of the features of aluminum-induced neurotoxicity may arise, in part, from autoimmune reactions, as part of the ASIA syndrome.25 http://www.hindawi.com/journals/jt/2014/491316/

There has been an epidemic of inflammatory diseases that has paralleled the epidemic on iatrogenic immune stimulation with vaccines. The epidemic of diabetes/prediabetes appears to be accelerating at a time when the prevalence of obesity has stabilized, indicating that the negative feedback system of the immune system has been over whelmed. The theory of vaccine induced immune overload explains the key observations that have confounded many competing hypothesis. Unfortunately the prospective controlled trials of vaccines performed for licensure are either too small, too short in duration or inappropriately controlled (use other vaccines as controls) to appropriately study the relationship between vaccines and these epidemics. Furthermore most epidemiological studies performed after licensure of vaccines suffer from the same deficiencies.Biochemical Laboratory Research29 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3364648/#!po=2.00000

What is regressive autism and why does it occur? Is it the consequence of multi-systemic dysfunction affecting the elimination of heavy metals and the ability to regulate neural temperature?30 http://www.ncbi.nlm.nih.gov/pubmed/11895129

Recent studies indicate that transition metals act as catalysts in the oxidative reactions of biological macromolecules therefore the toxicities associated with these metals might be due to oxidative tissue damage.31 http://ajcn.nutrition.org/content/80/6/1611.full

Coproporphyrin levels were elevated in children with autistic disorder relative to control groups. Elevation was maintained on normalization for age or to a control heme pathway metabolite (uroporphyrin) in the same samples. The elevation was significant (P < 0.001). Porphyrin levels were unchanged in Asperger's disorder, distinguishing it from autistic disorder. The atypical molecule precoproporphyrin, a specific indicator of heavy metal toxicity, was also elevated in autistic disorder (P < 0.001) but not significantly in Asperger's. A subgroup with autistic disorder was treated with oral dimercaptosuccinic acid (DMSA) with a view to heavy metal removal. Following DMSA there was a significant (P = 0.002) drop in urinary porphyrin excretion. These data implicate environmental toxicity in childhood autistic disorder.33 http://www.ncbi.nlm.nih.gov/pubmed/20576582

HgCl2 stimulates VEGF and IL-6 release from human mast cells. This phenomenon could disrupt the blood-brain-barrier and permit brain inflammation. As a result, the findings of the present study provide a biological mechanism for how low levels of mercury may contribute to ASD pathogenesis.39 http://www.ncbi.nlm.nih.gov/pubmed/17454560

There was a significant dose-response relationship between the severity of the regressive ASDs observed and the total mercury dose children received from Thimerosal-containing vaccines.40 http://www.ncbi.nlm.nih.gov/pubmed/18197631

Thiol-modulated mechanisms of the cytotoxicity of thimerosal and inhibition of DNA topoisomerase II alpha.

Overall, mercury inhibition was selective toward the thioredoxin system. In particular, the remarkable potency of the mercury compounds to bind to the selenol-thiol in the active site of TrxR should be a major molecular mechanism of mercury toxicity.

This study is the first to report an association between virus serology and brain autoantibody in autism; it supports the hypothesis that a virus-induced autoimmune response may play a causal role in autism.49 http://www.ncbi.nlm.nih.gov/pubmed/12145534

Our studies demonstrate that the level of MS mRNA in the brain is high during fetal development and gradually decreases with age. This decrease is likely to be important for the normal progression of development, via its influence over DNA and histone methylation. MS mRNA levels in the cortex are abnormally low in autism, which is likely to reflect the presence of oxidative stress. Therapies which counteract oxidative stress and promote methylation may be a useful strategy in treating autism.52 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0068444

ethylmercury not only inhibits mitochondrial respiration leading to a drop in the steady state membrane potential, but also concurrent with these phenomena increases the formation of superoxide, hydrogen peroxide, and Fenton/Haber-Weiss generated hydroxyl radical. These oxidants increase the levels of cellular aldehyde/ketones. Additionally, we find a five-fold increase in the levels of oxidant damaged mitochondrial DNA bases and increases in the levels of mtDNA nicks and blunt-ended breaks.54 http://www.ncbi.nlm.nih.gov/pubmed/22249285

We conclude that exposure of Hepa1-6 cells to a low dose of adjuvanted hepatitis B vaccine leads to loss of mitochondrial integrity, apoptosis induction, and cell death, apoptosis effect was observed also in C2C12 mouse myoblast cell line after treated with low dose of vaccine (0.3, 0.1, 0.05 μg/ml). In addition In vivo apoptotic effect of hepatitis B vaccine was observed in mouse liver.55 http://jcn.sagepub.com/content/21/2/170.abstract

These data suggest that further metabolic evaluation is indicated in autistic patients and that defects of oxidative phosphorylation might be prevalent.

Children who have mitochondrial related dysfunctional cellular energy, metabolism might be more prone to undergo autistic regression between 18 and 30 months of age if they also have infections or immunizations at the same time.56 http://www.ncbi.nlm.nih.gov/pubmed/10759242

Controls were all negative. The sequences obtained from the patients with Crohn's disease shared the characteristics with wild-strain virus. The sequences obtained from the patients with ulcerative colitis and children with autism were consistent with being vaccine strains. The results were concordant with the exposure history of the patients. Persistence of measles virus was confirmed in PBMC in some patients with chronic intestinal inflammation.57 http://www.nature.com/nature/journal/v501/n7465/full/nature12504.html

Here we find that topotecan, a topoisomerase 1 (TOP1) inhibitor, dose-dependently reduces the expression of extremely long genes in mouse and human neurons, including nearly all genes that are longer than 200 kilobases... many high-confidence ASD candidate genes are exceptionally long and were reduced in expression after TOP1 inhibition. Our findings suggest that chemicals and genetic mutations that impair topoisomerases could commonly contribute to ASD and other neurodevelopmental disorders.58 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0058058

administration of THIM to suckling rats in a vaccination-like manner and at doses analogous to those used in paediatric vaccines or higher injures neurons and astroglia in several brain regions.64 http://www.ncbi.nlm.nih.gov/pubmed/21549155

early postnatal THIM administration causes lasting neurobehavioral impairments and neurochemical alterations in the brain, dependent on dose and sex. If similar changes occur in THIM/mercurial-exposed children, they could contribute do neurodevelopmental disorders.65 http://www.ncbi.nlm.nih.gov/pubmed/17114826

Behavioral testing showed motor deficits in the aluminum treatment group that expressed as a progressive decrease in strength measured by the wire-mesh hang test (final deficit at 24 wk; about 50%). Significant cognitive deficits in water-maze learning were observed in the combined aluminum and squalene group (4.3 errors per trial) compared with the controls (0.2 errors per trial) after 20 wk. Apoptotic neurons were identified in aluminum-injected animals that showed significantly increased activated caspase-3 labeling in lumbar spinal cord (255%) and primary motor cortex (192%) compared with the controls. Aluminum-treated groups also showed significant motor neuron loss (35%) and increased numbers of astrocytes (350%) in the lumbar spinal cord. The findings suggest a possible role for the aluminum adjuvant in some neurological features associated with GWI and possibly an additional role for the combination of adjuvants.66 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2957583/

These data document that exposure to thimerosal during early postnatal life produces lasting alterations in the densities of brain opioid receptors along with other neuropathological changes, which may disturb brain development.67 http://www.ncbi.nlm.nih.gov/pubmed/22015705

TM exposure resulted in a delayed startle response in SD neonates and decreased motor learning in SHR male (22.6%), in SD male (29.8%), and in SD female (55.0%) neonates. TM exposure also resulted in a significant increase in cerebellar levels of the oxidative stress marker 3-nitrotyrosine in SHR female (35.1%) and SD male (14.0%) neonates. The activity of cerebellar type 2 deiodinase, responsible for local intra-brain conversion of thyroxine to the active hormone, 3',3,5-triiodothyronine (T3), was significantly decreased in TM-exposed SHR male (60.9%) pups. This coincided with an increased (47.0%) expression of a gene negatively regulated by T3, Odf4 suggesting local intracerebellar T3 deficiency. Our data thus demonstrate a negative neurodevelopmental impact of perinatal TM exposure which appears to be both strain- and sex-dependent.68 http://www.ncbi.nlm.nih.gov/pubmed/19357975

As a result of the present findings, in combination with the brain pathology observed in patients diagnosed with autism, the present study helps to support the possible biological plausibility for how low-dose exposure to mercury from thimerosal-containing vaccines may be associated with autism.69 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3264864/?tool=pubmed

Emerging research suggests that the timing of environmental factors in the presence of genetic predispositions has influenced the increase in autism spectrum disorders over the past several decades. A review of the medical literature suggests that autism may be impacted by environmental toxicants, breastfeeding duration, gut flora composition, nutritional status, acetaminophen use, vaccine practices and use of antibiotics and/or frequency of infections. The author reports her retrospective clinical research in a general pediatric practice (Advocates for Children), which shows a modest trend toward lower prevalence of autism than her previous pediatric practice or recent CDC data. Out of 294 general pediatrics patients followed since 2005 there were zero new cases of autism (p value 0.014). Given the prevalence of autism for that cohort of 1 in 50 children in the United States, it is important to consider implementing strategies in primary care practice that could potentially modify environmental factors or affect the timing of environmental triggers contributing to autism.