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Call your bank
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Zapping Food---when
buying meat or poultry- or any fruits or veges – to increase the safety
measures of eliminating any potential contaminant—microbes—parasites- mold
fungus- bacteria---So to set a “charge “ this will neutralize whatever or
burn out what may be there---so take a zapper –stunner or cattle prod and
place the prods on the meat or produce and let the charge go for a moment—if
the animal protein is large then do it on 2 sides-apply this to fruit and
veges as well—

If
fruit flies infected with wasps tap the alcohol in rotting fruit, it raises
their blood-alcohol levels and their survival rates. The alcohol doesn't
just kill some of the wasps, it essentially liquifies them.ScienceDaily (Feb. 16, 2012) —
Fruit flies infected with a blood-borne
parasite consume alcohol to self-medicate, a behavior that greatly increases
their survival rate, an Emory University study finds.--"We
believe our results are the first to show that
alcohol consumption can have a protective effect against infectious disease,
and in particular against blood-borne parasites,"
says Todd Schlenke, the evolutionary geneticist who led the research.--"It
may be that fruit flies are uniquely adapted to using alcohol as medicine,"
he adds, "but our data raise an important question: Could other
organisms, perhaps even humans, control blood-borne parasites through high
doses of alcohol?"----Current
Biology is publishing the
study, co-authored by Emory graduate student Neil Milan and undergraduate
student Balint Kacsoh.-The results add to the
growing body of evidence that some animals know how to use toxic substances
found in nature as medicine. Drosophila melanogaster, the common
fruit fly that swirls around browning bananas in your kitchen, is an
important biological model system. The Schlenke lab uses D. melanogaster to
study how immune systems adapt to pathogens.
The fly larvae eat the rot, or fungi and bacteria, that grows on overripe,
fermenting fruit. "They're essentially
living in booze," Schlenke says.
"The amount of alcohol in their natural habitat can range from 5 to 15
percent. Imagine if everything that you ate and drank all day
long was 5-percent alcohol. We wouldn't be able to live like that, but fruit
flies are really good at detoxifying alcohol."--Tiny,
endoparasitoid wasps are major killers of fruit flies. The wasps inject
their eggs inside the fruit fly larvae, along with venom that aims to
suppress their hosts' immune response. If the venom is
effective enough, the wasp egg hatches, and the wasp larva begins to eat the
fruit fly larva from the inside out. Eventually, an adult wasp emerges from
the remains of the fruit fly pupa. Some fruit flies, however, can
overcome the effects of wasp venom and mount an immune response against wasp
eggs. The blood cells in these fly larvae swarm
over the wasp eggs and release nasty chemicals to kill them, allowing the
fruit fly larvae to grow into adults.--"A constant
co-evolutionary battle is going on between the immune systems of the flies
and the venoms of the wasps," Schlenke says.
"Any new mechanism of defense that protects flies from wasps will tend to
spread through fly populations by natural selection." Schlenke
wondered if the fruit flies could be tapping the toxic effects of alcohol in
their natural habitat to fight off wasps.

To test the theory,
the researchers used a bisected petri dish filled with the yeast that fruit
flies are normally fed in a lab environment. The yeast on one side of the
dish was mixed with 6 percent alcohol, while the yeast on the other side
remained alcohol-free. The researchers then released fruit fly larvae into
the dish, allowing them to freely move to either side.

After 24 hours,
80 percent of the fruit fly larvae that were infected with wasps were on the
alcohol side of the dish, while only 30 percent of the
non-infected fruit fly larvae were on the alcohol side.---"The strength of
the result was surprising," Schlenke says. "The
infected fruit flies really do seem to purposely consume alcohol, and the
alcohol consumption correlates to much higher survival rates." Infected fruit flies that consumed alcohol beat
out the wasps in about 60 percent of the cases, compared to a 0 percent
survival rate for fruit fly controls that fed on plain yeast.--"The
wasps aren't as good as the flies at handling alcohol," Schlenke says. A
developing wasp knocked out within an alcohol-consuming fly larva dies in a
particularly horrible way, he adds. "The
wasp's internal organs disperse and appear to be ejected out of its anus.
It's an unusual phenotype that we haven't seen in our wasps before,"
Schlenke says.---The lab repeated the experiment using another species of
wasp that specializes in laying its eggs in D. melanogaster, rather than the
generalist wasp used previously. Again, 80
percent of the infected flies wound up on the alcohol side of the dish,
while only 30 percent of the uninfected flies did. But the alcohol diet was far less effective
against the specialist wasps, killing them in only 10 percent of the
cases.---"You would expect this kind of result," Schlenke says,
"since the generalist wasp species can attack plenty of other flies, but the
specialist wasps are under strong pressure to adapt to the alcohol-infused
habitat of D. melanogaster."--The
researchers hope that their data will lead to more studies of how alcohol
may control pathogens in other organisms, including humans.---"Although
many studies in humans have shown decreased immune function in chronic
consumers of alcohol, little attempt has been made to assay any
beneficial effect of acute or moderate alcohol use on parasite mortality or
overall host fitness following infection," Schlenke says. Story Source- The above story is
reprinted from materials provided by
Emory University. Note: Materials may be edited for
content and length. For further information, please contact the source cited
above. Journal Reference-Neil F.
Milan, Balint Z. Kacsoh, Todd A. Schlenke. Alcohol Consumption as Self-Medication
against Blood-Borne Parasites in the Fruit Fly.
Current Biology,
2012; DOI:
10.1016/j.cub.2012.01.045

The B.C.
government is moving forward with plans for high-tech identity cards that
will one day let people access a host of provincial services on the Internet.
The province intends to sign a $20-million, six-year deal with
Toronto-based SecureKey to provide card reader technology, the
government says. It's the latest in a series of
new laws and contracts that pave the way for expanded government Internet
services.--The cards may one day
be used to access government websites for electronic health records,
prescription histories, age verification, driver's licence details,
electronic voting and school registration for children.
Currently, you have to visit a government office in person and show
appropriate ID to access such services.--Health Minister Mike de Jong
announced last May the government was developing new CareCards, with
improved security features, to combat the millions of dollars lost to health
insurance fraud.[U3]--Those
cards should be available in November, and will roll out over the next five
years. They'll be free and mandatory for all British Columbians, requiring
millions of people to re-enrol into the healthcare system. The project is
expected to cost the government about $150 million.--People will also have
the option of combining their new CareCard and driver's licence into one
B.C. Services Card.--The new government cards
will have embedded security chips, similar to certain credit
cards that allow customers to wirelessly make purchases by touching or
waving their credit card in front of a terminal.--The government's contract
with SecureKey will develop B.C. card readers - key-sized devices that plug
into a computer's USB port. People tap their new CareCard or Services
Card onto the SecureKey reader and enter a PIN number on a
government website, to authenticate their identity.---There
won't be any online government services available when the cards launch in
November.
The province has proposed a rollout of features from different ministries
over the next five years.--B.C. said it will directly award SecureKey a
contract because the company already holds a contract with the federal
government and the province wants its system to be compatible with Canada's
service.---NDP critic Doug Routley said the Liberal government has a
dismal track record on protecting privacy and should have asked for other
bids. "I know some pretty good roofers, but I'd still get three quotes on a
roof," he said. "It'd still be prudent to put this out there for
competition."--There remains many privacy
questions about the direction the government is headed, said
Vince Gogolek, executive director of the B.C. Freedom of Information and
Privacy Association. He said the government rushed through legislation last
fall that allows for crossministry sharing of personal information needed by
the new cards.---The government said it is consulting with the independent
privacy commission on the technology.--Identity cards won't store any
information inside their chips, and the SecureKey readers simply create a
secure path and network to approve a person's ID and re-direct them back to
government information, officials said Friday.-The cards can be remotely
cancelled if lost, and will only access the minimum amount of personal
information required for a service - for example, a birth date to prove an
age or a health number for prescriptions.

ScienceDaily (Feb.
16, 2012) — Billions of engineered
nanoparticles in foods and pharmaceuticals are ingested by humans daily, and
new Cornell research warns they may be more harmful to health than
previously thought.---A research collaboration led by Michael
Shuler, the Samuel B. Eckert Professor of Chemical Engineering and the James
and Marsha McCormick Chair of Biomedical Engineering, studied how large doses of polystyrene
nanoparticles -- a common, FDA-approved material found in substances from
food additives to vitamins -- affected how well chickens absorbed iron, an
essential nutrient, into their cells.--The results were reported
online Feb. 12 in the journal Nature Nanotechnology. According to the study, high-intensity, short-term
exposure to the particles initially blocked iron absorption, whereas
longer-term exposure caused intestinal cell structures to change,
allowing for a compensating uptick in iron absorption.--
The researchers tested both acute and chronic nanoparticle exposure using
human gut cells in petri dishes as well as live chickens and reported
matching results.They chose
chickens because these animals absorb iron into their bodies similarly to
humans, and they are also similarly sensitive to micronutrient deficiencies,
explained Gretchen Mahler, Ph.D. '08, the paper's first author and former
Cornell graduate student and postdoctoral associate.--The researchers used
commercially available, 50-nanometer polystyrene carboxylated particles that
are generally considered safe for human consumption.They found that following acute exposure, a few
minutes to a few hours after consumption, both the absorption of iron in the
in vitro cells and the chickens decreased.---But
following exposure of 2 milligrams per kilogram for two weeks -- a slower,
more chronic intake -- the structure of the intestinal villi began to change
and increase in surface area. This was an effective physiological remodeling
that led to increased iron absorption.--"This was a physiological
response that was unexpected," Mahler said.--Shuler noted that in some sense
this intestinal villi remodeling was positive because it shows the body
adapts to challenges. But it serves to
underscore how such particles, which have been widely studied and considered
safe, cause barely detectable changes that could lead to, for example,
over-absorption of other, harmful compounds.[U4]--Human
exposure to nanoparticles is only increasing, Shuler continued.--"Nanoparticles
are entering our environment in many different ways," Shuler said. "We have
some assurance that at a gross level they are not harmful, but there may be
more subtle effects that we need to worry about."--The paper included
Cornell co-authors Mandy Esch, a research associate in biomedical
engineering; Elad Tako, a research associate at the Robert W. Holley Center
for Agriculture and Health; Teresa Southard, assistant professor of
biomedical sciences; Shivaun Archer, senior lecturer in biomedical
engineering; and Raymond Glahn, senior scientist with the USDA Agricultural
Research Service and courtesy associate professor in the Department of Food
Science. The work was supported by the National Science Foundation; New York
State Office of Science, Technology and Academic Research; Army Corp of
Engineers; and U.S. Department of Agriculture.---Story
Source-The above story is
reprinted from
materials provided by
Cornell University, via
Newswise. The original article was written by Anne Ju. --Journal
Reference-Gretchen J. Mahler,
Mandy B. Esch, Elad Tako, Teresa L. Southard, Shivaun D. Archer, Raymond P.
Glahn, Michael L. Shuler. Oral
exposure to polystyrene nanoparticles affects iron absorption.
Nature Nanotechnology,
2012; DOI:
10.1038/nnano.2012.3

ScienceDaily (Feb.
14, 2012) — A person has a tumor removed from her femur. A soldier is struck
by an improved explosive device and loses a portion of his tibia. A child
undergoes chemotherapy for osteosarcoma but part of the bone dies as a
result. Every year, millions of Americans sustain fractures that don't heal
or lose bone that isn't successfully grafted. But a study presented at the
Orthopaedic Research Society (ORS) 2012 Annual Meeting in San Francisco
offers new hope for those who sustain these traumas. Orthopaedic
researchers with the University of California, San Francisco (UCSF),
Orthopaedic Trauma Institute, have found a very
promising, novel way to regenerate bone. "Cartilage
graft induces bone that actually integrates with the host bone and
vascularizes it," said Ralph
S. Marcucio, PhD, Associate Professor, UCSF School of Medicine.--Cartilage
graft is very different than the current methods used for bone grafting --
autograft bone (a person's own bone) or allograft materials (donor bone).
For various reasons, these two grafting techniques can result in poor graft
integration and osteonecrosis.---"With millions of bone grafting
procedures performed every year in just the United States, developing
improved technologies could directly enhance patient care and clinical
outcomes," Dr. Marcucio said.--Chelsea S. Bahney, PhD, Postdoctoral Scholar,
UCSF School of Medicine, concedes their approach is less orthodox. "It is
not the pathway that most people think about, but it made a lot more sense
to follow the normal developmental mechanism.""This
cartilage is naturally bioactive. It makes factors that help induce
vascularization and bone formation," added Dr. Bahney. "When
people use a bone graft, it is often dead bone which requires something
exogenous to be added to it or some property of the matrix in the graft."
Through a process called endochondral ossification,
cartilage grafts produce new tissue that is very similar to the person's own
bone. Without additional properties to it,
the researchers found the cartilage graft
integrated well and was fully vascularized.--"We're
just taking a very similar cartilage that can induce bone formation,
putting it into a bone defect and letting it just do its thing,"
Dr. Marcucio said.---In the study, the researchers chose a non-stabilized
tibial fracture callus as a source of a cartilage graft. "Healing of the
transplanted cartilage grafts supported our hypothesis by producing a well-vascularized
bone that integrated well with the host," Dr. Bahney said.--"A cartilage
graft could offer a promising alternative approach for stimulating bone
regeneration," Dr. Marcucio said. "Future work will focus on developing a
translatable technology suitable for repairing bone through a cartilage
intermediate at a clinical level."--Story
Source--The above story is
reprinted from materials provided by
Orthopaedic Research Society (ORS).

Scientists Prepare Test-tube
BurgerCLIVE
COOKSON - Financial Times (U.K.)I think there is going to be a tremendous
emphasis on this. It will become a growing trend, because industrial
cattle operations are going to become unprofitable due to the rising cost
of water as it becomes scarcer.
It takes 1,300 gallons of water to produce an eight ounce hamburger.
As affluence increases across the world -- not to be confused with wealth,
only more than subsistence --
eating meat will increase. The demands for ! water and grain (and the
water it requires) will become unsustainable.The world’s first test-tube
hamburger, created in a Dutch laboratory by growing muscle fibres from
bovine stem cells, will be ready to grill in October, scientists
believe.--'I am planning to ask
Heston Blumenthal [the celebrity chef] to cook it,” Mark Post, leader of
the artificial meat project at Maastricht University in the Netherlands,
told the American Association for the Advancement of Science annual
meeting in Vancouver.--Researchers believe that meat grown in factories,
rather than on farms, will be a more sustainable and less environmentally
harmful source of food. Live cattle and pigs are only 15 per cent
efficient at converting vegetable proteins to meat from the grass and
cereals they eat[U5].--
'If we can raise the efficiency from 15 to 50 per cent by growing meat in
the lab, that would be a tremendous leap forward,” Professor Post said.---Starting
with bovine stem cells[U6],
the Dutch researchers have grown muscle fibres up to 3cm long and 0.5mm
thick. The fibres are tethered and exercised as they grow, like real
muscles, by bending and stretching in the culture dishes. They feed on a broth of
vegetable proteins and other nutrients, equivalent to the grass or grain
diet of cattle.[U7]---At
present the fibres are a pallid yellowish-pink colour, rather than the red
of raw ground beef, because they do not contain blood,[U8]
but Prof Post plans to improve their appearance.--Patrick Brown,
biochemistry professor at Stanford University in California, told the AAAS
that global meat consumption was expected to double by 2050, yet
livestock farming already accounted for 18 per cent of man-made greenhouse
gas emissions and threatened biodiversity worldwide[U9].--
To provide fat, an essential element in real burgers, They will be minced
in with bovine
fat cells are also being grown in the lab.the
muscle fibres.---
This project was started about six years ago, and I expect it will be
another 10 to 20 years before we can mass-produce our meat,” Prof Post
said.

300,000 Organic Farmers Sue Monsanto in Federal Court: Decision on March
31st to Go to TrialThe GMO movement is perhaps the leading
example in our culture of how the greed of a single corporation can destabilize and entire ecosystem, thus
effecting the lives of millions;
all so a small group of people already rich can get richer.Nelson states,
'Corporate control of our food system has led to the loss of millions of
family farmers, destruction of our soil…”-Hundreds
of citizens, (even including NYC chefs in their white chef hats) joined
Occupy the Food System groups, ie Food Democracy Now, gathered
outside the Federal Courts in Manhattan on January 31st, to support
organic family farmers in their landmark lawsuit against Big Agribusiness
giant Monsanto. (Organic Seed Growers & Trade Association v. Monsanto)
Oral arguments were heard that day concerning the lawsuit by 83 plaintiffs representing over
300,000 organic farmers, organic seed growers, and organic seed
businesses.---The lawsuit
addresses the bizarre and shocking issue of Monsanto harassing and threatening organic
farmers with lawsuits of 'patent infringement” if any organic farmer ends
up with any trace amount of GM seeds on their organic farmland.[U11]Judge Naomi
Buckwald heard the oral arguments on Monsanto’s Motion to Dismiss, and the
legal team from Public Patent Foundation represented the rights of
American organic farmers against Monsanto, maker of GM seeds, [and
additionally, Agent Orange, dioxin, etc.]---After hearing the arguments,
Judge Buckwald stated that on March 31st she will hand down her decision
on whether the lawsuit will move forward to trial.--Not
only does this lawsuit debate the issue of Monsanto potentially ruining
the organic farmers’ pure seeds and crops with the introduction of
Monsanto’s genetically modified (GM) seeds anywhere near the organic
farms, but additionally any nearby GM fields can withstand Monsanto’s
Roundup herbicides, thus possibly further contaminating the organic farms
nearby if Roundup is used.[U12]---Of
course, the organic farmers don’t want anything to do with that ole
contaminated GM seed in the first place. In fact, that is why they are
certified organic farmers. But now they have to worry
about getting sued by the very monster they abhor, and even have to spend
extra money and land (for buffers which only sometimes deter the
contaminated seed from being swept by the wind into their crop land).
[U13]At
this point, they are even having to resort to not growing at all the
following organic plants: soybeans, corn, cotton, sugar beets, and canola,
…just to protect themselves from having any (unwanted) plant that Monsanto
could possibly sue them over.[U14]

An
Ontario man says he's angry and frightened after discovering someone hid a
GPS tracking device under his vehicle, apparently to secretly monitor his
movements.---“I was doing
just a regular inspection on my truck and I
found this black box under my truck … with flashing lights inside,” Ben
Ferrill of Warsaw, Ont., told Go Public. “I didn’t know what it was. I
didn’t know if it was a bomb. We were scared to death … It felt like a
movie — unreal.”---After making the discovery last fall he reported it the
next morning to the Ontario Provincial Police. Ferrill said the OPP tried
to find out whom the device belonged to to lay a mischief charge,
but were stymied in their investigation.---“I feel powerless. I
can’t do anything about it and I really wish the police did more. I really
wish they would do more — and I’m upset,” Ferrill said. Ferrill said he
and his wife haven’t been able to sleep properly since, because they are worried someone is watching them.[U15]“We spent lots of nights up
until three in the morning crying and talking about it — not sleeping and
looking out the windows and being suspicious of vehicles that drive by,”
Ferrill said. “Are they following me? Are they watching me? Are they going
to try to do something to me? Are they going to try to do something to our
family? The fear in finding something like that is unreal.”Ferrill’s
lawyer, Ian Wilson, said is illegal to put a
tracking device on someone else’s vehicle without their knowledge or
consent unless there is a search warrant.---“The trouble is, we
don’t know who is behind this,” Wilson said.[U16]
The device Ferrill found, Wilson said, was sold by an American company,
U.S. Fleet Tracking, which sells GPS systems to companies
that want to track their fleet vehicles.
The OPP obtained a production order, compelling the Canadian company that
provides the wireless connection, Kore Wireless, to disclose whose device
it is. However, Kore said it didn’t have that
information[U17].
“I’ve [also] tried to get answers from Kore Wireless and their lawyer but
they will tell me nothing,” Wilson said. Kore Wireless president Alex
Brisbourne told CBC News his company only provides "connectivity. We don’t know who the end user is.”He
said U.S. Fleet bills the customer and gives access to its website, where
the customer can track its fleet vehicles in real time. “Our customer
[U.S. Fleet] is in the United States. They
have no responsibility or accountability to provide that information [to
Canadian police],” Brisbourne said. When asked whether he
requested the customer information from U.S. Fleet in this case,
Brisbourne replied it "is not appropriate for us to ask for that. Security
of information is particularly critical.”
U.S. Fleet’s website makes it clear the company does not give up
information easily, even to American authorities. “U.S.
Fleet Tracking will not under any circumstance make your information or
any data specific to your vehicle tracking account available to any third
party —including local, state or federal law enforcement
authorities
[U18]…
Even if presented with a court order, we promise to fight the courts to
keep this information private and respect the privacy of our paying
customers,” the website reads.

Wilson and Ferrill
said the OPP told them it would be too expensive and time-consuming to
pursue the case through U.S. courts. The
OPP told CBC News the file is now
closed.--Ferrill insisted
the device could not have been put there by his wife or any family member.
In addition,
U.S. Fleet sells to to businesses, not individuals.--He
said the only dispute he is involved in is with his former employer, a car
dealership in Peterborough, Ont.--Ferrill worked for a decade as a
mechanic for Holiday Ford Lincoln. Two years ago, he injured his shoulder
on the job. The Workplace Safety and Insurance Board (WSIB) in Ontario
said he needed to be on light, restricted duty for several months.--After
a few months, Holiday Ford fired him for allegedly stealing a bottle of
windshield washer fluid.--A WSIB investigation found it was a pretext –
that Ferrill had been fired because he was injured, which is a violation
of the law. The WSIB report concluded: “A breach of their re-employment
obligation has occurred.”Holiday Ford is appealing that decision, but it
faces significant fines if the ruling stands. It is also facing a
wrongful-dismissal lawsuit filed by Ferrill.--The company has denied
Ferrill's allegations in court filings.--Ferrill said he discovered the
GPS device a few months after filing his lawsuit. He and his lawyer said
they suspect the dealership could be involved, but they have no way of
proving or disproving that.--CBC News left several messages with Ferrill’s
former manager at Holiday Ford, but received no reply.--He has no idea how
long it was there, and said the combined stress has pushed his family to
the edge.--“Losing my job was really stressful, and then this is just a
part of more stress,” Ferrill said. “It’s really testing our family. It’s
the most stress and discomfort we have ever had.” Ferrill and his lawyer want Canadians to realize how
technology can now be used against them and — at least in this case —there
is no recourse.“It’s extremely frustrating to know that
[customer] information exists in the U.S. somewhere … and we simply can’t
get at it,” Wilson said. “[Ferrill] hasn’t had any peace of mind for the
last 2½ years since he lost his job.”--Wilson said he finds it ironic this is happening to his client at the same
time the federal government is talking about allowing police to monitor
people’s internet use without a warrant.--“The same federal
government that is responsible for this crime bill is also responsible for
these wireless companies,” Wilson said.---He said he believes wireless
companies in Canada should be required by law to know who the end users of
the technology are: “This is a changing environment and steps have to be
taken to protect people.” “We want to know who put this on our truck and
why,” Ferrill said. “I’m sure whoever did this wants to do some type of
harm to me, because whoever did this wants to know what I am doing all the
time and wants to follow me for some reason.”

Hindlimb
ischemia was created in rats and treatments were delivered over seven days
with an osmotic pump. The laser doppler imaging above shows the rat's hind
limb prior to treatment (on the left) and with increased blood flow (image
on the right) just seven days after treatment.

ScienceDaily (Feb.
16, 2012) — University of Texas at Austin researchers have demonstrated a new and more effective method for
regrowing blood vessels in the heart and limbs -- a research
advancement that could have major implications for how we treat heart
disease, the leading cause of death in the Western world.---The treatment
method developed by Cockrell School of Engineering Assistant Professor
Aaron Baker could allow doctors to bypass
surgery and instead repair damaged blood vessels simply by injecting a
lipid-incased substance into a patient. Once inside the body,
the substance stimulates cell growth and spurs the growth of new blood
vessels from pre-existing ones.---Aaron Baker is an assistant
professor in the Department of Biomedical Engineering.---The
method has been tested successfully on rats, and findings of the study
were published recently in the Proceedings of the
National Academy of Sciences
--"Others have tried using
growth factors to stimulate vessel growth in clinical trials and have not
been successful," said Baker, a faculty member in the school's Department
of Biomedical Engineering. "We think that a major reason for this is
that previous methods assumed that the diseased tissues retained the
ability to respond to a growth stimulus. Our method basically delivers extra components
that can restore growth factor responsiveness to the tissue of patients
with long-standing clinical disease."—[U19]
The ability to regrow blood vessels could prove crucial to treating
chronic myocardial ischemia disease, which affects up to 27 million
patients in the U.S. and leads to a reduction of blood flow in the heart
and lower limbs -- ultimately causing organ dysfunction and failure.--Central
ischemia, which affects the heart, occurs when the coronary vessels that
feed blood to the heart become blocked or narrow because of a buildup of
fatty deposits called plaques. Such plaques
are typically the result of a prolonged unhealthy diet or smoking, and
factors such as age, high blood pressure and diabetes increase the risks
of the disease, Baker said.

Doctors have
typically treated ischemia by physically opening the closed artery with a
stent or surgically rerouting blood flow to the poorly perfused tissue.
Both methods have limitations, however, and
are not effective long-term.--The new method introduced by
Baker and his research team builds on a promising revascularization
approach that, up until now, has shown limited efficacy in clinical
trials for treating human disease.---The method combines a growth factor -- a substance
capable of stimulating cellular growth, proliferation and cellular
differentiation, as well as healing wounds-- known as fibroblast growth factor 2 (FGF-2)
with a lipid-embedded receptor to enhance its activity.---A
challenge for scientists and engineers, however, has been getting FGF-2 to
bind with cell receptors -- the very molecules often found on the surface
of the cell that receive chemical signals and direct activity in the cell
from outside sources.---To overcome this, Baker's method embeds the growth factors in synthetic
lipid-based nanoparticles containing a coreceptor known as syndecan-4.The nanoparticles containing co-receptors that, when delivered with the
growth factor, enable improved cell binding so that the growth factor can
direct the targeted cell to divide, proliferate and form new cells for
tissue regrowth.---The incased substance was
injected into rats with hindlimb ischemia and stimulated a complete
recovery from the ischemia in just seven days.---"We hope this
research will increase our understanding of how tissues become resistant
to revascularization therapies and may lead to more effective treatments
for this widespread and debilitating disease," said Baker, who was
recognized last year with the National Institutes of Health Director's New
Innovator Award.-Story
Source---The above story is
reprinted from materials provided by
University of Texas at Austin. -Journal
Reference-E. Jang, H.
Albadawi, M. T. Watkins, E. R. Edelman, A. B. Baker. Syndecan-4 proteoliposomes enhance
fibroblast growth factor-2 (FGF-2)-induced proliferation, migration, and
neovascularization of ischemic muscle.
Proceedings of the National
Academy of Sciences, 2012;
109 (5): 1679 DOI:
10.1073/pnas.1117885109

add the whites of
the Citrus -about 8 slices- then add 1 whole apple- add a pectin capsule (
if you wish not mandatory ) add bht 1 capsule-or any other antioxidant you
see fit to add--example vitamin C-add sodium alginate 1 tsp -tumeric
1 tsp- bromelain powder or capsule 1 tablespoon -add 1/8 cup of
water and allow to blend at slow to medium speed til it gelatinizes--once
this is done pour into a glass container and consume several tsp through
out the day and use a good load of water---this will bind to
metals-toxins-will support the liver-digestive system-and potentially
reduce fatty deposits in the gall bladder and liver

[U4]In
other words if you are exposed to to other metals or poisons or
foods that may produce a poison this will allow for a higher uptake
of these contents further damaging the internals—and If there are
air borne particles as well then what you may have is an exacbrated
level of these being absorbed

[U5]Well
I wonder why!! Since the soy shuts down there thyroids and there
pancreas how can the animal process anything accurately???---these
GE or GMO foods are not meant for them to consume either

[U6]Now
am I the only one seeing this GMO MEAT or GE meat—playing with
genetics to alter the dna of this meat and then feed it to the Human
race----we must be less then dust in the eyes of the powers that
be!! Time to send them home!

[U7]Vegetable
Proteins—SOY or other GMO and this is going to be used in the
Genetic sequencing----now what does this do to People!!!

[U8]Life
is in the blood so what then is this---the is a design of food for
something other then life as we know it—this is to feed something
else and if people eat this then there will be health hazards

[U9]Another
lie---the green house gasses being emitted are not from the animals
but rather the Bio diversity of the GMOs and there interference with
the balance of this planet

[U10]BG
–Bill Gates---orrr my likely guess since he has financial power or
perhaps the Vatican since they own the gmos and GE through monsanto

[U11]The
Law has to change if a seed is found on a piece of land then
Monsanto is responsible for it so farmer do not lose there lands
because of second hand negligence and this would contain or limit
the amount of cross breeding of the seed with the real crop---the
law is stacked in Monsanto’s favour and this needs to be addressed

[U12]This
is already happening ---you cannot co exist in the real world—the
moment you plant this abomination it corrupts what is naturally
occurring---the biggest lie being perpetrated in the health food
industry next to GMP ---all Crap

[U13]This
would be enough for a lawsuit since it is on the land and there
according to Monsanto illegally even the buffer doe not deter any
kind of genetic anomaly with this barrier

[U14]Not
growing Soy or Canola or corn at this stage of the game may not be a
bad idea

[U16]The
Question everyone should be asking is –what was he hauling that
required him to be under surveillance---what could have been so
important as to have this kind of Black opp to take place---the fact
he was monitored is not an issue –truckers are always monitored but
to have this kind of sophistication –there must have been something
of the utmost importance—maybe a Bio contaminant for military use
–maybe a military equipment needing to be transported
unnoticed---who knows

[U17]Now
does this sound funny---I mean with everything today asking you for
id and wanting to know if you had cornflakes or pancakes in the
morning does this not see a touch odd “ they don’t know”

[U18]I
think this is good actually and more companies need to apply this
kind of security---

[U19]It
is in the delivery ---the fat could be a lipsome to carry to the
area what is required for regenerating the issues

ScienceDaily (Jan. 24, 2012) — Eating food fried in olive or sunflower oil is not
linked to heart disease or premature death, finds a paper
published in the British Medical Journal online (bmj.com).-The
authors stress, however, that their study took place in Spain, a
Mediterranean country where olive or sunflower oil is used for frying and
their results would probably not be the same in another country where solid and re-used oils were used for frying.--In
Western countries, frying is one of the most common methods of cooking.
When food is fried it becomes more calorific because the food absorbs the
fat of the oils.[U1]--While
eating lots of fried food can increase some heart disease risk factors such
as high blood pressure, high cholesterol and obesity, a link between fried
food and heart disease has not been fully investigated.---So the authors,
led by Professor Pilar Guallar-Castillón from Autonomous University of
Madrid, surveyed the cooking methods of 40,757 adults aged 29 to 69 over an
11-year period. None of the participants had heart disease when the study
began. Trained interviewers asked participants about their diet and cooking
methods. Fried food was defined as food for which frying was the only
cooking method used. Questions were also asked about whether food was fried,
battered, crumbed or sautéed.--The participants' diet was divided into
ranges of fried food consumption, the first quartile related to the lowest
amount of fried food consumed and the fourth indicated the highest
amount.--During the follow-up there were 606 events linked to heart disease
and 1,134 deaths.---The authors conclude: "In a
Mediterranean country where olive and sunflower oils are the most commonly
used fats for frying, and where large amounts of fried foods are consumed
both at and away from home, no association was observed between fried food
consumption and the risk of coronary heart disease or death."--In
an accompanying editorial, Professor Michael Leitzmann from the University
of Regensburg in Germany, says the study explodes the myth that "frying food
is generally bad for the heart" but stresses that this "does not mean that
frequent meals of fish and chips will have no health consequences." He adds
that specific aspects of frying food are relevant, such as the type of oil
used.---Story Source- The above story is reprinted from materials
provided by BMJ-British Medical Journal.
Journal References-P. Guallar-Castillon, F. Rodriguez-Artalejo, E.
Lopez-Garcia, L. M. Leon-Munoz, P. Amiano, E. Ardanaz, L. Arriola, A.
Barricarte, G. Buckland, M.-D. Chirlaque, M. Dorronsoro, J.-M. Huerta, N.
Larranaga, P. Marin, C. Martinez, E. Molina, C. Navarro, J. R. Quiros, L.
Rodriguez, M. J. Sanchez, C. A. Gonzalez, C. Moreno-Iribas. Consumption
of fried foods and risk of coronary heart disease: Spanish cohort of the
European Prospective Investigation into Cancer and Nutrition study.
BMJ, 2012; 344 (jan23 3): e363 DOI:
10.1136/bmj.e363---M. F.
Leitzmann, T. Kurth. Fried foods and the risk of coronary heart disease.
BMJ, 2012; 344 (jan23 3): d8274 DOI:
10.1136/bmj.d8274

ScienceDaily (Feb. 21, 2012) — A leaky gut may be the root of some cancers forming
in the rest of the body, a new study published online Feb. 21 in PLoS ONE by
Thomas Jefferson University researchers suggests.---It appears
that the hormone receptor guanylyl cyclase C (GC-C) -- a previously
identified tumor suppressor that exists in the intestinal tract -- plays a key role in strengthening the body's
intestinal barrier, which helps separate the gut world from the rest of the
body, and possibly keeps cancer at bay. Without the receptor,
that barrier weakens.---A team led by Scott Waldman, M.D., Ph.D., chair of
the Department of Pharmacology and Experimental Therapeutics at Jefferson
and director of the Gastrointestinal Cancer Program at Jefferson's Kimmel
Cancer Center, discovered in a pre-clinical study that silencing GC-C in
mice compromised the integrity of the intestinal barrier.
It allowed inflammation to occur and cancer-causing agents to seep out into
the body, damaging DNA and forming cancer
outside the intestine, including in the liver, lung and lymph nodes.---Conversely,
stimulating GC-C in intestines in mice strengthened the intestinal barrier
opposing these pathological changes.---A
weakened intestinal barrier has been linked to many diseases, like
inflammatory bowel disease, asthma and food allergies, but this study
provides fresh evidence that GC-C plays a role in the integrity of the
intestine. Strengthening it, the
team says, could potentially protect people against inflammation and cancer
in the rest of the body.--"If the intestinal barrier breaks down,
it becomes a portal for stuff in the outside world to leak into the inside
world," said Dr. Waldman. "When these worlds collide, it can cause many diseases, like inflammation and
cancer."--The role of GC-C outside the gut has remained largely
elusive. Dr. Waldman and his team have previously shown its role as a
tumor suppressor and biomarker that reveals occult metastases in lymph nodes.
They've used to it better predict cancer risk, and have even shown a
possible correlation with obesity.---Reporting in the Journal of Clinical
Investigation, Dr. Waldman colleagues found that silencing GC-C affected
appetite in mice, disrupting satiation and inducing obesity. Conversely,
mice who expressed the hormone receptor knew when to call it quits at
mealtime.--However, its role in intestinal barrier integrity, inflammation,
and cancer outside the intestine is new territory in the field. A new drug
containing GC-C is now on the verge of hitting the market, but its intended
prescribed purpose is to treat constipation.--This study helps lays the
groundwork, Dr. Waldman said, for future pre-clinical and clinical studies
investigating GC-C's abilities beyond those treatments in humans, including
prevention and treatment of inflammatory bowel disease and cancer."We've
shown that when you pull away GC-C in animals, you disrupt the intestinal
barrier, putting them at risk for getting inflammatory bowel disease and
cancer. And when you treat them with hormones that activate GC-C it helps
strengthen the integrity of the intestinal barrier," Dr. Waldman said. "Now,
if you want to prevent inflammation or cancer in humans, then we need to
start thinking about feeding people hormones that activate GC-C to tighten
up the barrier."--Story Source--The above story is
reprinted from
materials provided by
Thomas Jefferson
University, via
Newswise. ---Journal References---Jieru Egeria Lin, Adam Eugene
Snook, Peng Li, Brian Arthur Stoecker, Gilbert Won Kim, Michael Sullivan
Magee, Alex Vladimir Mejia Garcia, Michael Anthony Valentino, Terry Hyslop,
Stephanie Schulz, Scott Arthur Waldman. GUCY2C Opposes Systemic Genotoxic
Tumorigenesis by Regulating AKT-Dependent Intestinal Barrier Integrity.
PLoS ONE, 2012; 7 (2): e31686 DOI:
10.1371/journal.pone.0031686--Michael A. Valentino, Jieru E. Lin, Adam
E. Snook, Peng Li, Gilbert W. Kim, Glen Marszalowicz, Michael S. Magee,
Terry Hyslop, Stephanie Schulz, Scott A. Waldman. A uroguanylin-GUCY2C
endocrine axis regulates feeding in mice. Journal of Clinical
Investigation, 2011; 121 (9): 3578 DOI:
10.1172/JCI57925

ScienceDaily (Jan. 22, 2012) — A diet supplemented with a specific probiotic
bacterial strain increases the lifespan of mice.---The mammalian
gut is home to hundreds of bacterial species that contribute to food
digestion and, in some cases, inflammatory gut diseases. Probiotics, beneficial bacterial species, can
enhance gut health by keeping the resident bacteria in check.
Now, a team of researchers at the RIKEN Innovation Center in Wako, including
Mitsuharu Matsumoto, report that administration of the probiotic bacterial strain Bifidobacterium animalis
subspecies lactis LKM512 to mice can lengthen their lifespan.
Matsumoto and colleagues previously showed that LKM512 could reduce inflammatory markers in elderly humans and
modify the makeup of intestinal bacteria2, but the effects of it
on lifespan still required investigation. After starting 10-month-old mice
on a diet including LKM512 for 11 months, the researchers found that LKM512-treated mice lived longer, had fewer
skin lesions, and had better hair quality than untreated mice.
Analyses of the gut of these mice revealed elevated gene expression in some
bacterial species compared with control mice, indicating that LKM512 may improve gut health indirectly by
regulating the levels of other bacterial species. The LKM512
treatment also prevented some age-related changes in bacterial composition
of the gut, suggesting that the probiotic
treatment protects the gut from developing characteristics associated with
aging.---Acting as a barrier between the bacteria and food within
the gut and the rest of the human body is an important role of the gut
lining.Breakdown of this lining can cause
infectious or inflammatory diseases. The researchers found that the gut of
LKM512-treated mice served as a stronger barrier than the gut of control
mice. LKM512 seemed to perform this function by increasing the expression of
various proteins that maintain the tight connection between gut epithelial
cells.-Polyamines are chemicals that reduce inflammation, and
their levels decrease as an individual ages. Matsumoto and colleagues
observed increases in intestinal polyamine
levels in LKM512-treated mice, which may be caused by the greater numbers of
bacteria promoted by LKM512. The increase in polyamines caused by LKM512
appeared to reduce inflammation in the body of the mice, as inflammatory
markers in the blood and urine were lower in LKM512-treated mice compared
with controls. In aged mice
treated with LKM512, inflammatory marker levels were similar to those
observed in younger mice, indicating that adults can benefit from probiotics.---"In
future work, we hope to clarify the effectiveness of LKM512 in humans,"
explains Matsumoto. If the findings extend to humans, inclusion of LKM512
into the human diet could enhance overall health and increase the human
lifespan.---Story Source-The above story is reprinted from
materials provided by RIKEN,
via ResearchSEA.

Abstract
Type 2 diabetes is a metabolic disorder that is strongly associated with
cardiovascular risk. Inflammation is a potential risk factor for
cardiovascular disease. In this study, hydro alcoholic extract of Nettle (Urtica
dioica) on insulin sensitivity and some inflammatory indicators in type 2
diabetic patients were studied. A randomized double-blind clinical trial on
50 men and women with type 2 diabetes was done for 8 weeks. Patients were
adjusted by age, sex and duration of diabetes, then randomly divided into
two groups, an intervention and control group.
They received, 100 mg kg-1nettle extract or placebo in three portions a day
for 8 weeks[U2].
Interleukin 6 (IL-6), Tumor Necrosis Factor-alpha (TNF-alpha), High
Sensitive C-Reactive protein (hs-CRP) and Fasting Insulin concentration were
measured. Insulin Sensitivity was calculated, at the beginning and the end
of the study. The data were analyzed by SPSS version 18, p<0.05 was
considered significant for all variables. After 8 weeks, IL-6 and hs-CRP showed a significant decrease in the intervention
group compared to the control group (p<0.05). The findings showed that the hydro alcoholic
extract of nettle has decreasing effects on IL-6 and hs-CRP in patients with
type 2 diabetes after eight weeks intervention.--PMID: 22303583 [PubMed
- indexed for MEDLINE]

How to prepare this—take
equal parts of either wine or vodka-or tequila or grappa Add to the slow
cooker-then add equal portion of distilled water or RO water-NEVER TAP
WATER—allow it to simmer for 3 days—strain it and bottle it and use it
accordingly—measure off your body weight and go from there as it is stated
in the comments—take your weight divide by 2.2 and then mutiply by 100mg
this will give dosing for your body weight so in the case of a 180 lb
person—they would go 180/2.2-=== 81 kgs then multiply by 100 and you get
8100 mgs or 8.1 grams which a teaspoon holds 5 grams so you would either do
a tsp and a half+ or do more dosing qith just the one teaspoon—say you take
one dose several times in his case 1. ½ + 3 times a day orr 1 tsp 5 times a
day—so approximate on how you want to utilize this—

The Johnson City Medical Center
staff actually Discovered this and did an in-depth study On it in our ICU.
The two individuals that discovered this then did An article on it, had it
published and have had it incorporated into ACLS and CPR classes. It is very
true and has and does work. It is called cough CPR. A cardiologist says it's
the truth If everyone who gets this sends it to 10 people,

You can bet that we'll save at
least one life. It could save your life Let's say it's 6:15 p.m. And you're
driving home (alone of course), after an usually hard day on the job. You're
really tired, upset and frustrated. Suddenly you start experiencing severe
pain In your chest that starts to radiate out Into your arm and up into your
jaw. You are only about five miles from the hospital Nearest your home.
Unfortunately you don't know if you'll be Able to make it that far. What can
you do? You've been trained in CPR But the guy that taught the course didn't
tell You what to do if it happened to yourself Since many people are
alone when they suffer a heart attack, this article seemed to be in order.
Without help, the person whose heart is beating improperly and who begins to
feel faint, Has only about 10 seconds left before losing consciousness.
However, these victims can help themselves by coughing repeatedly and very
vigorously. A deep breath should be taken before each Cough, and the cough
must be deep And prolonged, as when producing sputum From deep inside the
chest. A breath and a cough must be repeated About every two seconds without
let up Until help arrives, or until the heart is felt to be beating normally
again. Deep breaths get oxygen into the lungs and coughing movements squeeze
the heart and Keep the blood circulating. The squeezing pressure on the
heart also helps it regain normal rhythm. In this way, heart attack victims
can get to a hospital. Tell as many other people as possible about this, it
could save their lives! From Health Cares, Rochester General Hospital
via Chapter 240s newsletter 'AND THE BEAT GOES ON ' (reprint from The Mended
Hearts, Inc. Publication, Heart Response

[U1]The
question here is if this is not affecting the meditereanens then it is
the type of foods perhaps they are frying and perhaps these foods are
not genetically tampered with—this to would make the difference in the
impact of the foods and oils used

[U2]Take
your body weight and divide by 2.2 and then multiply this by 100mgs to
get the total of grams used—180lb person/2.2=81 kgs the Multiply by
100 and you would need approximately 8100mgs or 8.1
grams—approximately 1 tsp and and just slightly more then half

ScienceDaily
(Feb. 28, 2012) — Slowing or preventing the development of Alzheimer's
disease, a fatal brain condition expected to hit one in 85 people
globally by 2050, may be as simple as
ensuring a brain protein's sugar levels are maintained.--That's
the conclusion seven researchers, including David Vocadlo, a Simon
Fraser University chemistry professor and Canada Research Chair in
Chemical Glycobiology, make in the latest issue of Nature Chemical Biology.-The
journal has published the researchers' latest paper "Increasing O-GlcNAc slows neurodegeneration
and stabilizes tau against aggregation."Vocadlo and his
colleagues describe how they've used an inhibitor they've chemically
created -- Thiamet-G -- to stop O-GlcNAcase,
a naturally occurring enzyme, from depleting the protein Tau of sugar
molecules.-"The general thinking in science," says Vocadlo,
"is that Tau stabilizes structures in the brain called
microtubules. They are kind of like highways inside cells that allow
cells to move things around."---Previous research has shown that the linkage of these sugar molecules to
proteins, like Tau, in cells is essential. In fact, says
Vocadlo, researchers have tried but failed to rear mice that don't
have these sugar molecules attached to proteins.---Vocadlo, an
accomplished chess player in his spare time, is having great success
checkmating troublesome enzymes with inhibitors he and his students
are creating in the SFU chemistry department's Laboratory of Chemical
Glycobiology.--Research prior to Vocadlo's has shown that clumps of
Tau from an Alzheimer brain have almost none of this sugar attached to
them, and O-GlcNAcase is the enzyme that is robbing them.---Such
clumping is an early event in the development of Alzheimer's and the
number of clumps correlate with the disease's severity.--Scott
Yuzwa and Xiaoyang Shan, grad students in Vocadlo's lab, found that Thiamet-G blocks O-GlcNAcase from
removing sugars off Tau in mice that drank water with a daily dose of
the inhibitor. Yuzwa and Shan are co-first authors on this
paper.---The research team found that mice given the inhibitor had
fewer clumps of Tau and maintained healthier brains.--"This work
shows targeting the enzyme O-GlcNAcase with inhibitors is a new
potential approach to treating Alzheimer's," says Vocadlo. "This is
vital since to date there are no treatments to slow its
progression.--"A lot of effort is needed to tackle this disease and
different approaches should be pursued to maximize the chance of
successfully fighting it. In the short term, we need to develop better
inhibitors of the enzyme and test them in mice. Once we have better
inhibitors, they can be clinically tested. Story Source-The
above story is
reprinted from materials provided by Simon Fraser University. ---Journal
Reference-Scott A Yuzwa,
Xiaoyang Shan, Matthew S Macauley, Thomas Clark, Yuliya Skorobogatko,
Keith Vosseller, David J Vocadlo. Increasing O-GlcNAc slows
neurodegeneration and stabilizes tau against aggregation.
Nature Chemical Biology,
2012; DOI:
10.1038/nchembio.797

ScienceDaily
(Feb. 28, 2012) — As soon as women quit
hormone therapy, their rates of new breast cancer decline, supporting
the hypothesis that stopping hormones can lead to tumor regression,
according to a report e-published in Cancer Epidemiology,
Biomarkers, & Prevention.---As
part of the national Breast Cancer Surveillance Consortium,
researchers studied 741,681 woman-years of data (with a median of 3.3
years per woman) on 163,490 women aged 50-79 who were Group Health
Cooperative members and had no prior history of breast cancer.--"This
is the first study to look over time at screening mammography use
among individual women by their hormone therapy status linked with
their breast cancer diagnoses," said lead author Diana S.M. Buist,
PhD, a senior investigator at Group Health Research
Institute.--Previous research has shown a rapid decline in new breast
cancers -- and also in use of hormone therapy and of screening
mammography -- since 2002, when the Women's Health Initiative
published that breast cancer rates were
higher in women taking estrogen and progestin than in those taking
either a placebo or only estrogen.---Some have suggested
that the decline in use of hormone therapy may have caused the fall
in the breast cancer rate, perhaps by making tumors regress, Dr.
Buist explained. But others have countered that the explanation for
the declines in both breast cancer and hormone use might instead be
that because former hormone users are less concerned about breast
cancer or see their doctors less often, they may get less screening
mammography than do women who have never taken hormones.--"We set out
to test this idea," Dr. Buist said, "and our results seem to refute
it." Before 2002, former users of hormone therapy had lower rates of
screening mammography than did current users. "But we found that this
is no longer true," she said. Indeed, former users had the same or
even slightly higher screening rates than current users.---"We
concluded that differences in rates of screening mammography don't
explain the declines in rates of the incidence of invasive breast
cancer among women who've stopped using hormone therapy,
Dr. Buist said. The National Cancer Institute, which supports the
Breast Cancer Surveillance Consortium, funded this study. The
collection of cancer data was also supported in part by several state
public health departments and cancer registries throughout the United
States.---In addition to Dr. Buist, the other co-authors were Diana L.
Miglioretti, PhD, Rod Walker, MS, and Erin J. Aiello Bowles, MPH, of
Group Health Research Institute; Walter Clinton, PhD, of Veterans
Affairs (VA) Puget Sound Healthcare System; Patricia A. Carney, PhD,
of Oregon Health & Science University, in Portland; Stephen H. Taplin,
MD, MPH, of the National Cancer Institute; Tracy Onega, PhD, of
Dartmouth Medical School, in Lebanon, NH; and Karla Kerlikowske, MD,
of the University of California, San Francisco.-Story
Source--The above story
is
reprinted from materials provided by
Group
Health Research Institute. \Journal
Reference-Diana S.M.
Buist, Rod Walker, Erin J. Aiello Bowles, Patricia A. Carney, Stephen
H. Taplin, Tracy Onega, Karla Kerlikowske, Walter Clinton, and Diana
L. Miglioretti. Screening
Mammography Use among Current, Former, and Never Hormone Therapy Users
May Not Explain Recent Declines in Breast Cancer Incidence.
Cancer Epidemiol
Biomarkers Prev,
February 1, 2012 DOI:
10.1158/1055-9965.EPI-11-1115

ScienceDaily
(Feb. 8, 2009) — A new study makes important new findings on the role
of hormone use on the risk of breast cancer, confirming that the use of estrogen plus
progesterone increases the risk of both ductal and lobular breast
cancer far more than estrogen-only; suggesting a two-year
"safe" period for the use of estrogen and progesterone; and finding that the increased risk for ductal
cancers observed in long-term past users of hormone replacement
therapy drops off substantially two years after hormone use is
stopped.--Previous studies have shown that hormone
replacement therapy after menopause increases the risk of breast
cancer and that use of a regimen that includes both estrogen and
progesterone is more detrimental for the breast than the use of
estrogen alone. But more data from large prospective studies are
needed to fully characterize the impact of exogenous hormones (Exogenousor
exogeneous) refers to an action or object coming from outside a
system. It is the opposite of endogenous, something generated from
within the system ) on breast cancer incidence by type
of hormone preparation and histology of the cancer.---To investigate
the association in more detail, American Cancer Society
epidemiologists led by Eugenia E. Calle, PhD, did a prospective study
of 68,369 postmenopausal women who were cancer-free at baseline in
1992. They examined the use of estrogen-only and estrogen and
progesterone in current and former users of varying duration, and the
subsequent risk of developing invasive ductal and lobular carcinoma of
the breast. They also looked at whether the risk for each type of
breast cancer and each type of hormone regimen varied by body mass
index (BMI), stage of disease at diagnosis, and estrogen receptor (ER)
and progesterone receptor (PR) status. For the present study, the
follow-up period ended on June 30, 2005.---They
confirmed the findings from previous work that estrogen and
progesterone increases the risk of both ductal and lobular breast
cancer far more estrogen alone.They also found the risk associated with
use of estrogen and progesterone increases significantly and
substantially within three years of beginning hormone use.
The data showed no increased risk for women who used estrogen and
progesterone for less than two years, potentially identifying a "safe"
period for estrogen and progesterone use.---The study also
found no increased risk of breast cancer in women who
had stopped using estrogen and progesterone
two or more years ago, suggesting a
window of two to three years for the risks of estrogen and
progesterone both to become apparent after initial use and to diminish
after cessation. Few estimates of risk within two to three
years of initiation and cessation are available, so these findings
need replication in other large studies. The study found the use of
estrogen and progesterone was associated with a doubling of risk of
lobular cancer after three years of use, and a doubling of risk of
ductal cancer with 10 years of use. Estrogen-only use was not
associated with increased risk of ductal cancer, even after 20 years
of use, but was associated with a 50 percent increase in risk of
lobular cancer after 10 years of use.---Story Source: The above
story is reprinted (with editorial adaptations by ScienceDaily
staff) from materials provided by
American Cancer
Society, via
EurekAlert!, a
service of AAAS. -- Journal Reference: --Eugenia E. Calle et
al. ---Postmenopausal hormone use and breast cancer associations
differ by hormone regimen and histologic subtype. Cancer,
Published Online: January 20, 2008 DOI:
10.1002/cncr.24101

ScienceDaily
(May 9, 2006) — Long-term estrogen therapy
may be related to a higher risk of breast cancer among postmenopausal
women who have had a hysterectomy, according to an article
in the May 8 issue of Archives of Internal Medicine, one of the JAMA/Archives
journals. ---Previous studies have linked
the use of hormone therapy to breast cancer among postmenopausal
women,but have primarily focused on the hormone
combination of estrogen plus progestin, according to background
information in the article. Recently released results from the Women's
Health Initiative (WHI), a large clinical trial of hormone therapy,
found no significant link between estrogen therapy and breast cancer
in women who took the hormone for seven years. ---Wendy Y. Chen, M.D.,
M.P.H., Brigham and Women's Hospital and Dana Farber Cancer Institute,
Boston, and colleagues evaluated women who were part of the Nurses'
Health Study, a group of female nurses that have been followed since
1976. In 1980, 11,508 women from the study were postmenopausal and had
had a hysterectomy. Every two years the researchers enrolled all the
additional women who become postmenopausal and had a hysterectomy, so
28,835 women were included by the end of the study in 2002. Women were
asked by questionnaire every two years if they used hormones and
whether they had developed breast cancer.
For women who developed breast cancer, the researchers obtained
permission to review the women's medical records, which they used to
record the hormone receptor information. Tumors were
classified as positive or negative for estrogen receptor or
progesterone receptor based on how they responded to specific hormonal
therapies. ---Throughout the study
period, 934 invasive breast cancers developed, 226 among women who had
never used hormones and 708 among women who were using estrogen at the
time. The longer a woman used
estrogen, the higher her risk of breast cancer. Those who
had been taking estrogen for fewer than 10 years did not appear to
have a higher risk than those who had never taken hormones, but those who had been taking estrogen for
more than 20 years had a significantly increased risk. The
association was strongest for cancers that were estrogen receptor
positive and progesterone receptor positive. The results were
similar when the researchers evaluated only women who were older than
age 60; only women who had begun estrogen therapy after reaching age
50; and only women who were at least age 50 and had undergone a
hysterectomy, even if they had not gone through menopause. ---"In
conclusion, we found that estrogen therapy was associated with an
increased risk of breast cancer with longer-term use," the
authors write. "Although current use of estrogen therapy for less than
10 years was not associated with a statistically significant increase
in breast cancer risk, the WHI has shown
an increased risk of stroke and deep-vein thrombosis in the same time
period. Women who take
estrogen therapy for prevention or treatment of osteoporosis typically
require longer-term treatment and should
thus explore other options, given the increased risk of
breast cancer with longer-term use."---(Arch Intern Med. 2006; 166:
1027-1032. Available pre-embargo to media at
www.jamamedia.org.)
--Editor's Note: This study was supported by a grant from the National
Institutes of Health, Bethesda, Md. ---Story
Source: The above story is reprinted (with editorial adaptations
by ScienceDaily staff) from materials provided by
JAMA and
Archives Journals, via
EurekAlert!, a
service of AAAS ---

ScienceDaily
(Oct. 12, 2010) — Use of estrogen
therapy is associated with an increased risk of developing kidney
stones in postmenopausal women, according to a report in
the October 11 issue of Archives of Internal Medicine, one of
the JAMA/Archives journals.-- "Nephrolithiasis [kidney stones] is a
common condition that affects 5 percent to 7 percent of postmenopausal
women in the United States," according to background information in
the article. "Because the process of
kidney stone formation is influenced by a variety of lifestyle and
other health-related factors, the true impact of estrogen therapy on
the risk of kidney stone formation is difficult to infer from
observational studies." -- Using data from the national
Women's Health Initiative study, Naim M. Maalouf, M.D., of the
University of Texas Southwestern Medical Center, Dallas, examined data
from two trials: 10,739 postmenopausal women with hysterectomy who
received either an estrogen-only treatment or matching placebo and
16,608 postmenopausal women without hysterectomy who received either
an estrogen plus progestin treatment or matching placebo. Data were
collected for an average of 7.1 years in the estrogen-only trial and
5.6 years for the estrogen plus progestin trial. -- A total of 335 cases of kidney stones were
reported in the active treatment groups, while 284 cases
occurred in the placebo groups. The beginning demographic
characteristics and risk factors for kidney stones were similar in the
two groups, and the authors found that
estrogen therapy was associated with a significant increase in risk of
kidney stones. The corresponding annualized incidence rate
per 10,000 women per year was 39 in the treatment group and 34 in the
placebo group. Development of kidney stones was five times more
common in women with a history of kidney stones at the beginning of
the study, but was not significantly altered by estrogen therapy.
In this trial, estrogen therapy increased
the risk of development of kidney stones irrespective of age,
ethnicity, body mass index, prior hormone therapy use or use of coffee
or thiazide diuretics.The authors conclude that their
results "indicate that estrogen therapy increases the risk of
nephrolithiasis in healthy postmenopausal women. The mechanisms
underlying this higher propensity remain to be determined. In view of
the sizable prevalence of nephrolithiasis in this segment of the
population, these findings need to be considered in the
decision-making process regarding postmenopausal estrogen use."---Story
Source:--The above story is reprinted (with editorial adaptations
by ScienceDaily staff) from materials provided by
JAMA and
Archives Journals.--Journal Reference: N. M. Maalouf,
A. H. Sato, B. J. Welch, B. V. Howard, B. B. Cochrane, K. Sakhaee, J.
A. Robbins. Postmenopausal Hormone Use and the Risk of
Nephrolithiasis: Results From the Women's Health Initiative Hormone
Therapy Trials. Archives of Internal Medicine, 2010; 170
(18): 1678 DOI:
10.1001/archinternmed.2010.342

[U1]I can almost bet there will be dilutions and
contaminants in the product as of take over

[U2]Imagine
the amount of people getting the NEW and Improved Pfizer C

[U3]What
is happening is that the pharmaceuticals are going to take over
the Nutraceutical brands—and with all the deregulating and re
regulating going on the entrepreneur who does have a viable
supplement will eventually be either litigated out or squeezed
out---the fact they actually paid this much out to acquire this
companies portfolio is the way it starts and eventually the FDA
–DEA- Codex-EFSA will exterminate the ones that have something
viable that works well with health
conditions

Tribulus-Shilajit-
Deer Antler-- put all in at equal parts---Designed to increase T
levels—Stimulate the ebb and flow—regulate the insulin-regulate the
kidneys-increase blood vessel flexibility—Use By either one of 2
steps---Step 1 blend all contents to a powder and then apply 1 tablespoon
to a 2 cup pot of water ( preferably distilled or reverse osmosis ) and
bring to a boil ---when done pour into a cup and drink at least 3 servings
a day and about 30 minutes before intercource---can be added with the T
Up—Step 2 shake bag contents til there is a total mix---a and add to a 2
cup pot 1-2 tablespoons and bring to boil when that is done then allow the
stove to be off and allow it to simmer for 5 minutes then drink

Salvia officinalis has been used as a traditional remedy against diabetes
in many countries and its glucose-lowering effects have been demonstrated
in animal studies. The
active compounds and their possible mode of action are still unknown
although it has been suggested that diterpenes may be responsible for the
anti-diabetic effect of Salvia officinalis.

AIM OF THE
STUDY--To investigate
whether the reported anti-diabetic effects of Salvia officinalis are
related to activation of the nuclear receptor peroxisome proliferator-activated
receptor (PPAR)γ and to identify the bioactive constituents.

MATERIALS AND
METHODS:

From a
dichloromethane extract of Salvia officinalis able to activate PPARγ
several major metabolites were isolated by chromatographic techniques. To
assess bioactivity of the isolated metabolites a PPARγ transactivation
assay was used.

RESULTS:

Eight diterpenes
were isolated and identified including a new abietane diterpene being the
epirosmanol ester of 12-O-methyl carnosic acid and 20-hydroxyferruginol,
which was isolated from Salvia officinalis for the first time, as well as
viridiflorol, oleanolic acid, and α-linolenic acid. 12-O-methyl carnosic
acid and α-linolenic acid were able to significantly activate PPARγ
whereas the remaining metabolites were either unable to activate PPARγ or
yielded insignificant activation.

CONCLUSIONS:

Selected
metabolites from Salvia officinalis were able to activate PPARγ and hence,
the anti-diabetic activity of this plant could
in part be mediated through this nuclear receptor

Considerable evidence shows that chronic hyperglycemia can cause
pancreatic beta-cell dysfunction, which contributes to progressive
deterioration of glucose homeostasis and overt diabetes.
In the present study, we found that kaempferol, a flavonol compound
present in various Chinese medicinal herbs,
has cytoprotective effects on cultured clonal beta-cells and pancreatic
human islets. Kaempferol treatment dose-dependently promoted
viability, inhibited cellular apoptosis,
and reduced caspase-3 activity in
beta-cells and human islets exposed to chronic high glucose, with 10 μM
kaempferol exerting the maximum effect. In addition, kaempferol
treatment improved the expression of
anti-apoptotic proteins Akt and Bcl-2 that was significantly reduced in
beta-cells and human islets chronically exposed to hyperglycemia.
Furthermore, exposure of beta-cells and human islets to kaempferol
restored high glucose-attenuated intracellular cAMP and ATP production.
Inhibition of protein kinase A or Akt activation ablated the
anti-apoptotic effect of kaempferol. These cytoprotective effects of
kaempferol were associated with improved insulin secretory function and
synthesis in beta-cells and human islets. These findings provide evidence
that kaempferol may be a naturally occurring anti-diabetic compound by
protecting pancreatic beta-cell survival and function in a hostile
environment that would otherwise lead to type 2 diabetes.-----Tea (black) (dry leaf)-126.96---
Tea (green) (dry leaf)

Health Benefits of Kaempferol---Keampferol
is a strong antioxidant and helps to prevent oxidative damage of our
cells, lipids and DNA. Kaempferol seems to prevent arteriosclerosis by
inhibiting the oxidation of low density lipoprotein and the formation of
platelets in the blood. Studies have also confirmed that kaempferol acts
as a chemopreventive agent, which means that it inhibits the formation of
cancer cells.

The flavonoids kaempferol and quercetin seems to act synergistically in
reducing cell proliferation of cancer cells, meaning that the combined
treatments with quercetin and kaempferol are more effective than the
additive effects of each flavonoid. This was a conclusion from a study by
ML Ackland et al (In Vivo, Feb 2005) titled "Synergistic antiproliferative
action of the flavonols quercitin and kaempferol in cultured human cancer
cell lines"".

A study "Inhibition of P-glycoprotein function and expression by
kaempferol and quercetin" by the Chiang Mai University, Thailand, found
that kaempferol can help to fight cancer because it reduces the resistance
of cancer cells to anti-cancer drugs such as vinbalstine and paclitaxel.

Garlic-Sage- Bioflavonoid-Honey—Take
3-4 bulbs of garlic and 1 tablespoon of powdered Sage-8-9 pieces of the
Bioflavonoids from citrus-add to 1-1/12 cup of unpasteurized honey---blend
all the ingredients til there is a total fusion or the honey liquifies all
the components---use 1 tsp as needed---this will clearly remove toxic
waste and particulates that may occur in the system from respiratory to
liver—this will dramatically reduce Sugar and sustain heart balance and
flow---will increase permeability of the blood vessels—this will regulate
the insulin as well from the sage and garlic will have strong
antibacterial-antifungal properties-and potent antioxidant properties—

Would be used for
Respiratory-issues –Congestions and Cloggy lungs—Heart and
Circulation-Permeability and Flow-Anti Plaque and Fat build up in arteries
and liver-Immune Support to resist Bacteria and Fungal
infections-Chelating elements and Pollutants out of the system

Hawthorn Berry – Rose-Honey-
Take ½ cup of Roses and Hawthorn berry and ½ cup of Unpasteurized
Honey—add the Honey and get it going whipping it for about 5 minutes then
add the other 2 ingredients slowly and pause in the loading-allowing the
blender to fuse the honey and the components—then add more –repeat this
til you completely empty your content---allow this to blend for about
15-20 minutes optimum speed or high speed---this will heat and completely
saturate—when done stop blender—add to glass container---

Use 1 tablespoon
several times a day as a means to balance out heart irregularities

Consuming rose and
Hawthorn berry this will stabilize heart and allow for a steady heart
rate---in conjunction to this you may want to utilize Taurine 500 mgs 2-3
times a day Magnesium Citrate 100-200 mgs 2-3 times a day

And utilizing a
good Vitamin E supplement 200-400 IU’s 2 times a day

Consume adequate
Salt and potassium as well to stabilize heart functions—

At the same time
ALL PROCESS SUGARS ARE TO CEASE—NO EXCEPTIONS—NO SYNTHETIC SWEETNERS—NO
EXCEPTIONS-NO SOY BASED FOODS UNDER ANY CIRCUMSTANCE SINCE THIS LEACES OUT
MAGNESIUM AS WELL AS VITAMIN E-NO EXCEPTIONS-ALL GRAINS AND CEREALS ARE TO
CEASE-NO EXCEPTIONS

OBJECTIVE: To investigate the
possible mechanism and the compound(s) responsible for the antiplatelet
and acetylcholinesterase (AChE) inhibitory effects of Areca catechu crude
extract (Ac.Cr).
METHODS: Aqueous-methanol (70%) was used for extraction of plant material
(betel nut). Antiplatelet activity
was measured in human platelet-rich plasma by using a Lumi-aggregometer
while anti-AChE activity was measured spectrophotometrically in vitro. In
an attempt to find the responsible compound(s) in betel nut for antiplatelet and anti-AChE
activities, different commercially available betel nut
compounds were tested.
RESULTS: Ac.Cr inhibited platelet aggregation induced by arachidonic acid
(AA), adenosine diphosphate (ADP), platelet-activating factor (PAF),
epinephrine and Ca(2+)-ionophore. Ac.Cr was the most potent in inhibiting
ADP- and Ca(2+)-ionophore-induced aggregation. In the AChE assay, Ac.Cr showed significant AChE
inhibitory activity with almost complete inhibition of the enzyme.
Out of the tested compounds, none of the compounds in betel nut showed any
antiplatelet effect except for catechin that
was the most potent against epinephrine-induced aggregation.
Catechin was significantly less potent than Ac.Cr, indicating a presence
of additional compound(s) with antiplatelet activity. For the AChE inhibitory effect, only tannic acid,
gallic acid, diosgenin and isoguvacine were found to be active, whereby
tannic acid was more potent than Ac.Cr.
CONCLUSION: This study shows the possible antiplatelet and AChE
inhibitory potential of betel nut while further studies are needed to
confirm and identify more compounds in betel nut for these actions.--PMID:
21669165 [PubMed - indexed for MEDLINE]

The goal of acetylcholinesterase inhibitors (AChEIs) used to treat
Alzheimer's patients is an improvement in cholinergic transmission.
While currently available AChEIs have limited success, a huge impediment
to the development of newer ones is access to the relevant brain areas.
Promnesic, anti-amnesic and AChEI properties were identified in a
standardized ethanol extract from Ptychopetalum olacoides (POEE), a
medicinal plant favored by the elderly in Amazon communities. The purpose of this study
was to provide conclusive evidence that orally given POEE induces AChE
inhibition in brain areas relevant to cognition. Histochemistry experiments confirmed that the anticholinesterase
compound(s) present in POEE are orally bioavailable, inducing meaningful
AChE inhibition in the hippocampus CA1 (â¼33%) and CA3 (â¼20%), and
striatum (â¼17%). Ellman's colorimetric analysis revealed that G1 and G4
AChE isoforms activities were markedly inhibited (66 and 72%,
respectively) in hippocampus and frontal cortex (50 and 63%,
respectively), while G4 appeared to be selectively inhibited (72%) in the
striatum. Western blotting showed that POEE did not induce
significant changes in the AChE immunocontent suggesting that its
synthesis is not extensively modified. This study provides definitive
proof of meaningful anticholinesterase activity compatible with the
observed promnesic and anti-amnesic effects of POEE in mice, reaffirming
the potential of this extract for treating neurodegenerative conditions
where a hypofunctioning cholinergic neurotransmission is prominent.
Adequate assessment of the safety and efficacy of this extract and/or its
isolated active compound(s) are warranted.---PMID:
20833520 [PubMed - as supplied by publisher]

Abstract
Garlic has been used safely since ancient times as both food and medicine
in human populations, but studies of its efficacy in the management of
diabetes have yielded conflicting results. This study has evaluated the
potential hypoglycemic effects of garlic in type 2 diabetic patients. The
study was conducted in diagnosed type 2 diabetic patients (n=60) with
fasting blood sugar level above 126 mg/dl to evaluate the effects of
adding garlic tablets with standard antidiabetic therapy on blood sugar.
Patients were divided randomly into 2 groups. Group 1 (n=30) was given
tablet Garlic (KWAI) 300 mg thrice daily + Metformin 500 mg twice daily
and Group 2 (n=30) was given Placebo+Metformin 500 mg twice daily
respectively for 24 weeks. Serum lipids and fasting blood glucose were
measured at week 0, 12 and week 24. Group1 showed significant reduction in
fasting blood sugar at week 24 with a percentage decrease of (-3.12
percent) (P = <0.005) as compared to group 2 (0.59 percent). At the end of
week 24, GR1 group also showed considerable
decrease in mean total cholesterol (6.2 mg/dl, -2.82%, P=<0.005), LDL-C (-3
mg/dl, 2.18% P=<0.005), triglycerides (-5.2 mg/dl, 3.12%, P<0.005) while
HDL cholesterol was significantly increased (2.36 mg/dl, 6.72%, P<0.005)
as compared to GR2 group. Combination of garlic with typical antidiabetic
remedy has shown to improve glycemic control in addition to
antihyperlipidemic activity. Garlic may be a good addition in the
management of patients with diabetes and hyperlipidemia.---PMID: 21959822
[PubMed - indexed for MEDLINE]

Recipe---take
a garlic extract or supplement and add with it juniper berry and bitter
melon---this should in fact increase the efficiency of the organs in
regulating insulin—repairing the pancreas—assisting the liver-----this
should reduce not only the cholesterol but as well the sugar in the system

You can mix equal parts of an
extract or tincture of the juniper 1/3 or 33 % -bitter melon 1/3 or
33%-and garlic 1/3 or 33% in a 1 oz or 2 oz glass container and utilize ¼
tsp 3 times a day either after a meal or before bed

You can as well take the garlic
supplement and utilize the tincture of bitter melon and juniper either
mixed or separate then use ¼ tsp of each and 1 capsule of garlic

You can take a capsule of each 1---
3 times a day—even though they used the KWAI product the Kyolic will do
well formulas like 108-or 110 will be adequate or even just the plain 100

You might think
that the DNA you inherited is one thing that you absolutely can't do
anything about, but in one sense you'd be wrong. Researchers have found that when healthy but inactive men and
women exercise for a matter of minutes, it produces a rather immediate
change to their DNA. Perhaps even more tantalizing, the study suggests that the
caffeine in your morning coffee might also influence muscle in essentially
the same way. ---The
underlying genetic code in human muscle isn't changed with exercise, but the DNA molecules within
those muscles are chemically and structurally altered in very important
ways. Those modifications
to the DNA at precise
locations appear to be early events in the genetic reprogramming of muscle
for strength and, ultimately, in the structural and metabolic benefits of
exercise.--"Our muscles are
really plastic," says Juleen Zierath of Karolinska Institutet in Sweden.
"We often say "You are what you eat." Well,
muscle adapts to what you do. If you don't use it, you lose it,
and this is one of the mechanisms that allows that to happen."---The
DNA changes in question are known as epigenetic modifications and involve the gain or loss of chemical marks on
DNA
over and above the familiar sequence of As, Gs, Ts, and Cs.
The new study shows that the DNA within skeletal muscle taken from people
after a burst of exercise bears fewer chemical marks (specifically
methyl groups) than it did before exercise. Those changes take place in stretches of DNA that are
involved in turning "on" genes important for muscles' adaptation to
exercise.---When the
researchers made muscles contract in lab dishes, they saw a similar
loss of DNA methyl
groups. Exposure of isolated muscle to caffeine had the same effect.--Zierath
explained that caffeine does mimic the muscle contraction that comes with
exercise in other ways, too. She doesn't necessarily recommend anyone
drink a cup of joe in place of exercise. It's
nevertheless tempting to think that athletes who enjoy a coffee with their
workout might just be on to something.---Broadly speaking, the
findings offer more evidence that our genomes are much more dynamic than
they are often given credit for. Epigenetic modifications that turn genes
on and back off again can be incredibly flexible events. They allow the DNA in our cells to adjust
as the environment shifts.---"Exercise
is medicine," Zierath says, and it seems the means to alter our genomes
for better health may be only a jog away. And for those who can't
exercise, the new findings might point the way to medicines (caffeinated
ones, perhaps?) with -Story
Source--The above story is
reprinted from
materials provided by
Cell Press,
via EurekAlert!, a
service of AAAS. --Journal
Reference-Romain Barrès, Jie
Yan, Brendan Egan, Jonas Thue Treebak, Morten Rasmussen, Tomas Fritz,
Kenneth Caidahl, Anna Krook, Donal J. O'Gorman, Juleen R. Zierath. Acute Exercise Remodels Promoter
Methylation in Human Skeletal Muscle.
Cell Metabolism,
2012; 15 (3): 405 DOI:
10.1016/j.cmet.2012.01.001

Polysaccharides from black tea may blunt the spike in sugar
levels after a meal more than similar compounds from green and oolong tea,
and offer potential to manage diabetes, says a new study.

The
black tea polysaccharides also exhibited the greatest activity for
scavenging free radicals, which are linked to development of diseases such
as cancer and rheumatoid arthritis, according to new findings published in
the Journal of Food Science. Interest in tea and its
constituents has bloomed in recent years, with the greatest focus on the
leaf’s polyphenol content. Green tea contains between 30 and 40 per cent
of water-extractable polyphenols, while black tea (green tea that has been
oxidized by fermentation) contains between 3 and 10 per cent. Oolong tea
is semi-fermented tea and is somewhere between green and black tea. The
new research looked at the polysaccharide content of green, black and
oolong tea, and measured their ability to inhibit the effects of
alpha-glucosidase activity. By inhibiting this carbohydrate hydrolyzing
enzyme, it is possible to reduce the spike in glucose levels in the blood
following a meal (postprandial hyperglycemia).
If additional studies support the potential effects of the
polysaccharides, it could see the black tea extracts positioned in the
diabetic supplements market.

Study details

“Many efforts have been made to search for effective glucose inhibitors
from natural materials,”
said lead researcher Haixia Chen. “There is a potential for
exploitation of black tea polysaccharide in managing diabetes.” Researchers from Tianjin University isolated
three polysaccharide-rich fractions from green, black, and oolong tea. The
black tea was found to contain lower molecular weight polysaccharides.
Green tea had a range from 9.2 to 251.5 KDa, while black tea
polysaccharides ranged from 3.8 to 32.7 KDa. When tested for their ability
to inhibit alpha-glucosidase, as well as antioxidant activities relating
to hydroxyl radicals and DPPH radicals, the black tea
polysaccharides were found to produce the best results,
said Chen and co-workers. “The differences in antioxidant activities
and glycosidase inhibitory properties among the three polysaccharide-rich
fractions appeared to be related to differences in monosaccharide
composition and molecular weight distribution of the polysaccharide,”
wrote the researchers. ----“Physicochemical Properties and Antioxidant
Capacity of 3 Polysaccharides from Green Tea, Oolong Tea, and Black Tea”--Authors:
H. Chen, Z. Qu, L. Fu, P. Dong, X. Zhang

Recipe For Sugar Regulating---take
Black Tea and Sage and make a brew out of them—and drink through out the
day to minimize the ravages of sugar---this can assist in the reduction of
the medications and increase the restoration of the Pancreas

Orrrr

Use
Black Coffee and Sage as well---utilize equal parts of both the sage and
the Tea or Coffee

Prophet Mohammad (PBUH)
said “although onion and garlic have a bad smell, they are cures
for 70 different illnesses that cannot be cured by any other means”.
Onion (A. cepa) and garlic (A. sativa) are closely
related vegetables that belong to the Allium class of bulb-shaped plants,
which also includes chives, leeks, and scallions. Garlic is used for
flavoring in cooking and is unique due to its high sulfur content, along with arginine,
oligosaccharides, flavonoids, and selenium, all of which might promote
health [116].
The association between the consumption of Allium vegetables and
the risk for cancer was assessed in several epidemiologic studies, showing
the protective effect of garlic and onion on cancer. In China, high consumption of Allium vegetables was associated with lower incidence of
gastric cancer [117,
118].
Additional studies in the Netherlands suggested
an inverse correlation between the risk of colorectal, breast, and lung
cancers and the consumption of onion and garlic [119].
Steinmetz et al. [120]
studied the association between garlic consumption and the risk of colon
cancer and found that women who consumed high
amounts of garlic had a 50 percent lower incidence of distal colon cancer
compared with women who consumed less garlic [120].
The risk of breast cancer was also found to be
reduced in women consuming greater amounts of fiber garlic and onions
[31],
as well as that of esophageal and stomach cancers
[121]. Similar
findings were noted with reference to the risk of prostate cancer [122,
123], pancreatic
cancer [33],
and other known cancer types [124].
The amount of garlic consumed in the above
studies varied from 2 to 20g
daily (The World Health Organization (WHO) guidelines for
general health promotion for adults recommend a daily dose of
2 to 5g
of fresh garlic). It was noted that although garlic had been
used safely in cooking, excessive consumption can cause some side effects,
in addition to those of strong breath and body odors [125].
The protective effect of Allium vegetables against tumor
progression and against angiogenesis were attributed to its organosulfur
compounds especially allicin (an active compound in garlic) and diallyl
disulfide [126].
Such compounds are able to block the formation of
cancer-causing substances [127],
halt the activation of cancer-causing substances [128,
129], enhance DNA
repair [130],
reduce cell proliferation, or induce apoptosis-programmed cell death
(Table
1 and [126,
131]).

The origin of its
Latin name, Urtica, means “I burn”, indicative of the stings caused by
glandular hairs on the leaves that contain formic acid and histamine, two
agents known to cause the stinging and skin irritation after contact.
U. dioica leaf has a long history as an herbal remedy and nutritious
addition to the diet. Nettle leaves are a rich source of essential amino
acids, ascorbic acid, several mineral element, and vitamins, such as iron,
provitamin A, and vitamin C [113].
Nettle extracts can be used to treat arthritis, hay fever, kidney
problems, pain, and anemia. Nettle extracts possess hypoglycaemic
properties and improve glucose tolerance [35].
U. dioica is believed to be antioxidant, immunesuppressive,
antirheumatoid, antiulcer, anti-inflammatory, and anticarcinogenic [14].
Indeed, its leaf [114]
and roots [115]
extracts were effective against prostate cancer proliferation.

Source

Abstract

The blood glucose lowering effect
of Urtica dioica (Stinging Nettle) as a medicinal plant has been noted in
old writings such as those of Avicenna.
Recently, there has also been other investigators that indicated the
hypoglycemic effect of Urtica dioica. But so far, the mechanism of this
effect has not been deduced. In this report, a perifusion system is
arranged in which an exact number of Langerhans Islets were exposed to
several fractions of extracts of Urtica dioica by TLC. The active ingredient fraction named F(1), caused
a marked increase in insulin secretion. A simultaneous assay
of glucose showed that the increase in insulin level was associated with a
decrease in glucose level. Furthermore, the active component of Urtica
dioica was found to increase the insulin content of blood sera in normal
and streptozotocin diabetic rats that were injected intraperitoneally (i.p.)
with the active ingredient of the extract. The
in vivo studies presented in this report show that not only an increase in
insulin level of blood sera was observed in rats after 30 min from the
initial point of injection but a simultaneous decrease of blood sugar was
detected when similar sera was tested for glucose. The increase in insulin level was six times
during the 120 min of our determination. The decrease in
blood sugar was found to be similar both in the level and time of
initiation. On the basis of our findings, we assume that F(1) is the
active ingredient of plant leaves extract. The results show that the
blood lowering effect of the extract was due to the enhancement of insulin
secretion by Langerhance Isletes.

Abstract------- Urtica dioica
agglutinin (UDA) is a small plant monomeric lectin, 8.7 kDa in size,
with an N-acetylglucosamine specificity
that inhibits viruses from Nidovirales in vitro.
In the current study, we first examined the efficacy of UDA on the
replication of different SARS-CoV strains in Vero 76 cells. UDA inhibited virus replication in a
dose-dependent manner and reduced virus yields of the Urbani strain by 90%
at 1.1 ± 0.4 μg/ml in Vero 76 cells.
Then, UDA was tested for efficacy in a lethal SARS-CoV-infected BALB/c
mouse model. BALB/c mice were infected with two LD50 (575 PFU) of virus
for 4 h before the mice were treated intraperitoneally with UDA at 20, 10,
5 or 0 mg/kg/day for 4 days. Treatment
with UDA at 5 mg/kg significantly protected the mice against a lethal
infection with mouse-adapted SARS-CoV (p < 0.001),
but did not significantly reduce virus lung titers. All virus-infected mice receiving UDA
treatments were also significantly protected against weight loss (p <
0.001). UDA also effectively reduced lung pathology scores.
At day 6 after virus exposure, all groups of mice receiving UDA had much
lower lung weights than did the placebo-treated mice. Thus, our data suggest that UDA treatment of SARS
infection in mice leads to a substantial therapeutic effect that protects
mice against death and weight loss.
Furthermore, the mode of action of UDA in vitro was further investigated
using live SARS-CoV Urbani strain virus and retroviral particles
pseudotyped with SARS-CoV spike (S). UDA specifically inhibited the
replication of live SARS-CoV or SARS-CoV pseudotyped virus when added just
before, but not after, adsorption. These data suggested that UDA likely
inhibits SARS-CoV infection by targeting early stages of the replication
cycle, namely, adsorption or penetration. In addition, we demonstrated
that UDA neutralizes the virus infectivity, presumably by binding to the
SARS-CoV spike (S) glycoprotein. Finally, the target molecule for the
inhibition of virus replication was partially characterized. When UDA was
exposed to N-acetylglucosamine and then UDA was added to cells just prior
to adsorption, UDA did not inhibit the virus infection. These data support the conclusion that UDA might
bind to N-acetylglucosamine-like residues present on the glycosylated
envelope glycoproteins, thereby preventing virus attachment to cells.

Recipe for Nettle--Make a tea
with this by adding ¼ cup of nettle to a pot of water ( distilled ) bring
to a boil and when boiled turn off and pour---you can as well fuse this
with other herbs such as black tea –sage or do all 2 to increase the
immune and sugar regulating functions as well and will synergize t o
increase the potency of the properties of each herb

Ancient Remedial Use of Nettle----Horse
Nettle, Bull Nettle. S. E. United States.---The
fresh juice of the fruit is concentrated by exposure to currents of air at
a low temperature and preserved by the addition of some alcohol. This is
the process. adopted by McHoy, Howe Co., of Indianapolis. They prepare it
double strength. It is recommended largely in
the treatment of epilepsy. Powder of the root has during recent
years gained some repute as a remedy in
infantile and hysterieal convulsions, but chiefly in epilepsy and
paroxysms connected with menstrual derangements. Dose of Pdr.
Root 10 grs. to 1 dr.-----The
roots, leaves and tops are a positive,
stimulating, tonic astringent; a powerful arrester of hemorrhages
whether of the nose, lungs, stomach, bowels, uterus or urinary organs.
It will also stop hemorrhages when applied
locally and relieve painful hemorrhoids. The Fluid Extract when
diluted forms a good wash for some eczemas

§Savory leaves and tender
shoots contain incredibly high quality chemical compounds that are known
to have anti-oxidant, disease preventing and health promoting properties.
In addition, dietary fiber in this herb helps reduce LDL or bad
cholesterol while increasing HDL or good cholesterol levels.

§Savory leaves contain many
essential volatile oils phenols such as thymol
and carvacrol, as well as linalool,
camphene, caryophyllene, terpineol, myrcene, and other terpenoids.

§Thymol,
one of the important essential oils, has scientificaly been found to have
antiseptic, anti-fungal
characteristics.

§In addition, another
phenolic compound, carvacrol in savory inhibits the growth of
several bacteria strains like E. coli and Bacillus cereus. Carvacol,
therefore, has been used as food additive for its anti-bacterial
properties and in addition, it gives pleasant tangy taste and marjoram
like smell to the food.

§Savory herb is an
excellent source of minerals and vitamins that are essential for optimum
health. Its leaves and tender shoots are one
of the richest source of potasium, iron, calcium, magnesium, manganese,
zinc and selenium. Potassium in an important component of cell
and body fluids that helps controlling heart rate and blood pressure.
Manganese is used by the body as a co-factor for the antioxidant enzyme
superoxide dismutase. Iron is required for red blood cell formation.

§The herb is also a rich
source of many important vitamins such as B-complex group vitamins,
vitamin-A, vitamin-C, niacin, thiamin and pyridoxine.

§Dry savory provides 1.810
mg of vitamin B-6 or pyridoxine; furnishing about 130% of RDA.
Pyridoxine keeps up GABA (soothening neurotransmitter) levels in the brain
which has stress buster function.

§Vitamin A is a fat soluble
vitamin and antioxidant that is required maintaining healthy mucus
membranes and skin and is also essential for vision. Consumption of
natural fruits rich in flavonoids like vitamin A, carotenes helps protect
from lung and oral cavity cancers.

§Attributed Medicinal
PropertiesWhile both
varieties are used in cooking, Summer Savory has a much longer tradition
of medicinal use.It has long been reputed to be a general tonic to the
digestive tract and as a powerful antiseptic. Branches of savory were
tossed onto fire to create an aromatic disinfectant. Even today, because
of its pungent oils, it is commonly used in toothpaste and soaps. Active compounds of the
savory leaf include volatile oils (carvacrol, p-cymene, alpha-thujene,
alpha-pinene, beta-myrcene, beta-caryophyllene, terpinene, and thymol),
and tannic acid.
The carvacol and p-cymene content of this herb give it a mild antiseptic
effect. The tannin content is responsible for savory’s astringent
qualities, making
it a popular choice in the relief of diarrhea.
The herb has also been used as a gargle for sore throat. As a digestive
aid, savory is used in cases of indigestion and flatulence. It is often
added as a spice to dishes containing beans for this reason. The most
common medicinal use of savory today is in the treatment of gastrointestinal
enteritis, the inflammation of the intestinal tract. In some folk cultures, savory has been used to
increase libido.

BACKGROUND--Herbal
products are increasingly used for their effects on the immune system. Milk
Thistle, a commonly used herbal product is known
to inhibit growth of certain tumors, although the mechanism of
this effect remains unknown. Previously we have shown that Milk Thistle extracts stimulate neurons in culture.
Since other drugs that affect the neuronal; system also affect the immune
system, we investigated the effects of Milk Thistle on the immune system.--MATERIAL/METHODS-Standardized
Milk Thistle extract was studied in murine lymphocyte proliferation tests
using Concanavalin A (ConA) as mitogen for non-specific stimulation and
mixed lymphocyte culture (MLC) as allospecific stimulation. Th1 and Th2
cytokine levels in MLC were assayed by two antibody capture ELISA technique.
All tests were performed in triplicate and repeated twice.—RESULTS-We found
that Milk Thistle is immunostimulatory in vitro.
It increased lymphocyte proliferation in both mitogen and MLC assays.
These effects of Milk Thistle were associated with an increase in interferon
gamma, interleukin (IL)-4 and IL-10 cytokines in the MLC (table). This immunostimulatory effect increased in response
to increasing doses of Milk Thistle.---CONCLUSIONS-Our
study has uncovered a novel effect of milk
thistle on the immune system. This immunostimulatory effect
may be of benefit in increasing the immunity to infectious diseases.--PMID-12444368-[PubMed
- indexed for MEDLINE]

Abstract--Milk
thistle extracts have been used as traditional herbal remedies for almost 2000 years. The extracts are
still widely used to protect the liver against toxins and to control chronic
liver diseases. Recent experimental and clinical
studies suggest that milk thistle extracts also have anticancer,
antidiabetic, and cardioprotective effects. This article reviews
clinical trials of milk thistle conducted in the past 5 years including
pharmacokinetic and toxicity studies, herb-drug interactions, and other
safety issues. Several trials have studied the effects of milk thistle for
patients with liver diseases, cancer, hepatitis C, HIV, diabetes, and
hypercholesterolemia. Promising results have
been reported in the protective effect of milk thistle in certain types of
cancer, and ongoing trials will provide more evidence about this
effect. In addition, new established doses and improvement on the quality
and standardization of this herb will provide the much-awaited evidence
about the efficacy of milk thistle in the treatment of liver diseases. Milk thistle extracts are known to be safe and well
tolerated, and toxic or adverse effects observed in the reviewed
clinical trials seem to be minimal. The future of milk thistle research is
promising, and high-quality randomized clinical trials on milk thistle
versus placebo may be needed to further demonstrate the safety and efficacy
of this herb.-PMID-17548793-[PubMed - indexed for MEDLINE]

Abstract--The
potential of wild plants in Israel as sources of edible sprouts has not been
investigated until now. Milk thistle (Silybum marianum L.) is native to the
Mediterranean basin and is now widespread throughout the world; its young
fleshy stems are traditionally eaten by the local Arab sector in Israel, and
its sprouts are rich in antioxidants and have been used as a traditional
medicine for diseases of the liver and biliary tract. The active extract of milk thistle, silymarin, is a
mixture of flavonolignans and is a strong antioxidant that has been proved
to promote liver cell regeneration, to reduce blood cholesterol and to help prevent
cancer. The present objective was to investigate the potential of
milk thistle as a source of edible sprouts rich in antioxidants. We
found that seed germination within 3-4 days was high (96%, except for
striated seeds). Exposure to light significantly reduced sprout growth
and significantly increased the polyphenol content and antioxidative
capacity. The polyphenol content was 30% higher in seeds originating
from purple inflorescences than in those from white ones. We thus found milk thistle to be a good candidate
source of healthy edible sprouts.

Silybum marianum or
milk thistle (MT) is the most well-researched plant in the treatment of
liver disease. The active complex of MT is a lipophilic extract from the
seeds of the plant and is composed of three isomer flavonolignans (silybin,
silydianin, and silychristin) collectively known as silymarin. Silybin is
a component with the greatest degree of biological activity and makes up 50%
to 70% of silymarin.Silymarin is found in
the entire plant but it is concentrated in the fruit and seeds.Silymarin acts as an antioxidant by reducing
free radical production and lipid peroxidation, has antifibrotic activity
and may act as a toxin blockade agent by inhibiting binding of toxins to the
hepatocyte cell membrane receptors. In animals, silymarin
reduces liver injury caused by acetaminophen, carbon tetrachloride,
radiation, iron overload, phenylhydrazine, alcohol, cold ischaemia and
Amanita phalloides. Silymarin has been used
to treat alcoholic liver disease, acute and chronic viral hepatitis and
toxin-induced liver diseases.--PMID-20564545-[PubMed - indexed
for MEDLINE]

Recipe for Milk Thistle Usage---Take
Milk thistle in seed form make a tea with it and take in conjunction with it
1000 mgs if Vitamin C and 1000mgs of MSM---this will increase the impact of
the liver by increasing interferon production as welldetox the liver of
harmful chemicals------If you are around solvents then you may want to add
Arginine 500 mgs -1000mgs to the mix or even just add it to the milk
thistle------If you have liver poisoning then add Alpha Lipoic Acid 100-200
mgs with vitamin C 1000mgs and milk thistle------For Excess Fat in the Liver
you may want to increase B5 500-1000mgs and L Carnitine 500-2grams with it
and the use of Taurine 500-1000mgs and Bile Salts (as suggested on the
bottle)-----For issues in the restoring and regenerating of the liver
---utilizing milk thistle with sage in equal parts in water and
drinking---adding things like nettle and dandelion will assist as well in
the blood purifying effect as well----Sprouting the seeds –as well and
consuming them will assist as well with the antioxidant effect of the herb
and may even slow down the effects of aging

this
can be a male testis as well as a female breast having these metals in our
endocrine system so the genders should be aware

Metals like
aluminum have been linked to breast cancer for some time, but new research
is confirming the existence of an entirely new class of cancer-causing estrogens known as "metalloestrogens,"[U1]
and which are in thousands of consumer products -- some which are even
used in supplements and foods as "nutrients"...----A
new study published in journal Cancer Research
reveals that dietary cadmium exposure increases the risk of
postmenopausal breast cancer, confirming
earlier research that a broad range of metals we are now being
increasingly exposed to represent an emerging class of “metalloestrogens” with the potential to add to
the estrogenic burden of the human breast.
[U2]---In
a 2006 report
published in the Journal of
Applied Toxicology,
researchers found that the following metals were capable of binding to cellular estrogen
receptors and then mimicking the actions of physiological estrogens:
“aluminium,
antimony, arsenite, barium, cadmium, chromium
(Cr(II)), cobalt, copper, lead, mercury,
nickel,
selenite, tin and vanadate.” [U3]----As
we revealed
in an earlier exposé on the use of toxic forms of selenium in USDA certified organic
infant formula, exposure to sodium selenite (and sodium selenate) is
difficult to avoid, as it is the primary source of supplemental selenium
in mass market vitamins, foods, beverages, etc. The same is true for
inorganic forms of chromium, copper, nickel, tin and and vanadium, which
you will find on the
labels of many mass market multivitamins. --Another daily source of
metalloestrogen exposure for millions of consumers is aluminum-based
antiperspirants.
Investigations into the link between these personal care products and
breast cancer risk came to a head earlier this year
in a story we broke, and excerpt from below: --Aluminium salts used as
antiperspirants have been incriminated as contributing to breast cancer
incidence in Western societies. To date, very little or no epidemiological
or experimental data confirm or infirm this hypothesis. We report here
that in MCF-10A human mammary epithelial cells, a well-established normal
human mammary epithelial cell model, long-term exposure to aluminium
chloride (AlCl(3) ) concentrations of 10-300µm,
i.e. up to 100 000-fold lower
than those found in antiperspirants,
and in the range of those recently measured in the human breast, results
in loss of contact inhibition and anchorage-independent growth." [emphasis
added]--If a metal can exhibit carcinogenic properties at a concentration
100,000-fold lower than
presently used in personal care products,
it is critical that there be a paradigm shift in the way toxicological
risk assessments are performed. --Presently, the risk assessments depend
on animal studies, where the goal is to find out how much of a chemical it
takes to acutely kill 50% of an exposed population (LD50). Only then, is
an “acceptable level of harm” extrapolated for humans (as if determining
an “acceptable level of harm” were an ethically neutral objective).
--------------------The problem is that humans are not mice, there are profound synergistic toxicities that
are never accounted for when we are exposed to more than one chemical
simultaneously,[U4]
the LD50 does not indicate what constitutes a chronic/sublethal toxic dose
or an acknowledgment that chronic toxic effects do occur at far lower
doses, and mostly importantly for the purpose of this article,
the concept of increasing toxicity as the concentration decreases is not
even considered. ---This, in
fact, is what happens in certain types of radiation and petrochemical
exposure.
Low-dose radiation may be far more carcinogenic than high-dose radiation,
which is why "early detection" through
x-ray mammography is such a brutally dishonest practice, as it likely
causes much more cancer than it is said to prevent. --This is also what happens with certain
petrochemicals, e.g. bisphenol A, benzene, pthlatates, which is to say,
they may be more toxic at lower concentration than higher ones, due to
their hormone-mimicking, blocking and/or intensifying effects, as well as
their ability to interact on a molecular level with cell surfaces and
structures, which would not be possible were they aggregated in
greater volumes or quantities. The entire field of
nanotechnology, in fact, suffers from this counterintuitive problem:
the smaller the size, the higher the potential toxicity or the greater the
unintended, adverse health effects.------The
problem of aluminum and similar metallogestrogen exposure, however, is
greater than simply reforming manufacturers and consumer products. The
military releases at least 500 tons of
aluminum chaff annually during training within selected military operating
areas in the United States, and only clandestine organizations know how
much metal has been released into the environment in the name of "saving
the earth" through "geoengineering." Indeed, in what is known as
Atmospheric Aerosol Spraying, our
environment has increasingly become saturated through with metal pollution.
The problem has become so bad that biotech corporations have begun
engineering aluminum-resistant plants[U5],
since many soils have become too acidified due to metal contamination to
adequately sustain plants that produce food.

The goal of Calorie Restriction
is to achieve a longer
and healthier
life by

eating fewer calories

consuming
adequate vitamins, minerals, and other essential nutrients

Extensive
scientific research has shown that a CR diet improves the health and
extends the lifespan of every species so far tested, including worms,
spiders, rodents, dogs, cows and monkeys. We believe that people who adopt
a CR diet will see the same results longer life and better health.

Getting Started

Simply eating less
may not improve health or extend lifespan - it can lead to malnutrition.

Replace calorie-dense foods with calorie-sparse, nutrient-dense foods

Before worrying
about how many calories you're eating, make sure that the foods in your
diet provide sufficient nutrition to avoid malnutrition once you begin to
restrict them.

Avoid simple sugars and flours.
Sugars and flours generally contain very little nutrition for their
calorie content. They also have high glycemic indices, which means that
your body absorbs them quickly, leaving you wanting more a short time
later.

Eat both green leafy (salad) and other vegetables.
Vegetables -- both green leafy vegetables and non-leaf vegetables --
contain the highest content of a wide variety of nutrients for their
calorie content. By volume (and often by calories), vegetables are the
major component of many calorie restricted but not nutrient deficient
diets.

Carefully select your protein and fat sources.
Both protein and fat are required macronutrients, but their form can
have a significant influence on a person's risk factors for a wide
variety of diseases.

Make sure your protein intake is sufficient, but not
overly abundant.
Common recommendations for total protein intake range from 0.6 to 0.8 grams per kilogram of body weight,
and some recommendations are much higher. This is probably a
minimum.---another easier way to figure this out is to take either 60%
of body weight or 80 % and multiply it by your weight this will give a
close approximation of the amount of protein you should have---an
example would be say a 110 lb person minimal amount would be 66grams a
day and up to 88gams a day---take the .6 X 110=66--- take .8
X110=88---or multiply the 60% X110 and remove the zero’s---again gives
you 66 same with the 80% X 110 = 88—remove the zeros---will give a good
approximation

Make sure your proteins are complete and balanced.
A complete protein contains all the essential amino acids, while a
balanced protein contains all those amino acids in ratios that are most
useful to human biophysiology.

Most animal proteins are complete and well-balanced
There are very few perfectly balanced proteins in our food supply, but animal proteins tend to be among the most
balanced. Unfortunately, animal proteins also tend to
include undesirable components. For example, red meat is carcinogenic
[PMID: 12376502], and meat (especially red meat) and dairy often
contain large quantities of saturated fats. The nutrient density (as
always, on a per-calorie basis) of meats is often lower than other
choices.—Again this is outdated
badly---red meats should be broiled or cooked thoroughly after
cleaned---but not to subsist on red meat totally which can be
unhealthy—there are other animal proteins that are easier to consume
and digest---this would be easier and more efficient for most

Non-animal proteins can be balanced by combining different food
families
One can get extremely detailed in finding "complementary" foods, but
in general, combining legumes (beans) or seeds or nuts in balance--
will yield a balanced protein, and rice protein can complement the
proteins in vegetables like broccoli, cauliflower, or spinach. Be aware, however, that both rice and
grains (even whole grains) have relatively high glycemic indices and
relatively low nutrient densities.
If you're going to consume these, choose Cassava or Some other form or
Starch where potassium or other nutrients can be uptaken properly

Comment here this information is outdated badly due to the GMO and GE
contamination You may want to reconsider these foods especially the
rice an legumes which are high in starch ( sugars) and will turn to
fat----if consuming to act as a cleanse fashion then it would be best
to utilize the fibres from other foods such as fruits and veges---most
rice today is GMO’d and should be avoided

Select Healthy fats, and
consumesomeOmega-3
fats
Foods containing monounsaturated fats include olive oil, almonds,
hazelnuts, and avocados. Most of your fat intake should be from these
foods. A very small amount of fat should
be in the form of Omega-3 fatty acids, which are found in
fatty fish (e.g. salmon) – (
minimize fish oils due to the mercury loads in these fish )
and flax oil, walnut oil, and perilla. Caution- fatty foods, even
healthy choices, are high in calories so be sure that you carefully
track your intake so as to stay within your Calorie goal.---

Get baseline blood tests done

In order to chart
your progress, and to make sure that you are avoiding nutrient
deficiencies and other hazards, get at least the minimal recommended
blood tests
done. You'll want to know these results from before your
calorie-restricted diet begins, so that you'll have a baseline for
comparison as you move into a calorie-restricted diet.

Reduce calories after improving diet quality (i.e. nutrition)

Once your diet consists primarily of nutrient-dense, calorie-sparse foods,
you can safely begin to reduce your total calorie intake.
There are some things to consider at this point, if you have not already
considered them.

Lose weight slowly----Many
people lose weight simply by changing their diet to include more
calorie-sparse foods. Even this weight loss should not be allowed to
happen too quickly, but by the time you're able to restrict calories, your
weight loss should proceed very slowly. There are two reasons for this:

Pesticides and other toxins are fat soluble.
There are many pesticides and other toxins in our food supply that are
fat soluble. Human bodies store these toxins relatively harmlessly in
body fat. If you lose that fat too quickly, however, all these toxins are flushed into
your bloodstream, and your detoxification mechanisms (i.e., your kidney
and liver) are unable to remove them [B120YD, 78-80]. You may
thus wind up with much higher blood toxin
levels than what people are normally subjected to, which may have any
number of life-shortening effects.

Sudden adult onset Calorie Restriction shortens the
lifespans of mice.---Though
the lifespan enhancing effects of calorie restriction have been known
since the 1930s, it was also known that if adult mice were suddenly put
on a calorie restricted diet, their lifespans were actually shortened.
Dr. Walford found in the 1980s, however,
that if mice were slowly transitioned from an ad lib to a calorie
restricted diet, then their lifespans increased. The time for
this transition recommended by Dr. Walford is a minimum of 6 to 9
months, but preferably 1 to 2 years. [p. 78, BY120YD]

Be
aware of the potential risks in CR dieting!

Any significant
dietary change should be done in consultation with a knowledgeable
physician. There are several
other risks that you
should be aware of.

There are things
needed to be applied In the area of reduction—and with reduction of body
mass, there will be a reduction to the amount of poisons, toxins, build up
and inflammation—So when applying these protocols do them at least once a
month at any time of the month and watch unwanted health conditions
disappear—and a new vigor come back.

Do this for 5-10
days and afterwards if you wish to consume rice or pasta or polenta then
introduce them back slow and minimize the consumption—breads if you
consume then utilize Rye, Barley, or Oat and again Minimally—the idea is
to get off breads and cereals which are actually Fermenting inside and
potentially causing DNA damage and Restructuring--- Potato Can be used as
long as it is not fried---Can be used
if -broiled—boiled—mashed-or baked this will assist in the
cleansing of the colon as well as provide energy

A new study by
researchers at NYU Langone Medical Center reveals that the presence of
Helicobacter pylori (H. pylori) bacteria is associated with
elevated levels of glycosylated hemoglobin (HbA1c),an important biomarker for
blood glucose levels and diabetes.)---ScienceDaily
(Mar. 14, 2012) — A new study by researchers at NYU Langone Medical
Center reveals that the presence of Helicobacter pylori
(H. pylori)
bacteria is associated with elevated levels of glycosylated hemoglobin
(HbA1c), an important biomarker for blood glucose levels and diabetes.
The association was even stronger in obese
individuals with a higher Body Mass Index (BMI).---The
results, which suggest the bacteria may play a role in the development of
diabetes in adults, are available online in The Journal of Infectious
Diseases.---There
have been several studies evaluating the effect of the presence of H. pylori
on diabetes outcomes, but this is the first to examine the effect on HbA1c, an important, objective biomarker for
long-term blood sugar levels, explained Yu Chen, PhD, MPH,
associate professor of epidemiology at NYU School of Medicine, part of NYU
Langone Medical Center. "The prevalence of
obesity and diabetes is growing at a rapid rate, so the more we know about
what factors impact these conditions, the better chance we have
for doing something about it," Dr. Chen said. Looking at the effects of
H. pylori
on HbA1c, and whether the association differs according to BMI status,
provided what could be a key piece of information for future treatment of
diabetes, she explained.---Type II diabetes causes an estimated 3.8
million adult deaths globally. There have been conflicting reports about
the association between H.
pylori infection and type II
diabetes. To better understand the relationship between H. pylori
and the disease, Dr. Chen and Martin J. Blaser, MD, the Frederick H. King
Professor of Internal Medicine and professor of microbiology, analyzed
data from participants in two National Health and Nutrition Surveys (NHANES
III and NHANES 1999-2000) to assess the association between H. pylori and
levels of HbA1c.----"Obesity is an established risk factor for diabetes
and it is known that high BMI is associated with elevated HbA1c.
Separately, the presence of
H. pylori is also associated
with elevated HbA1c," said Dr. Blaser, who has studied the bacteria for
more than 20 years. "We hypothesized that
having both high BMI and the presence of
H. pylori
would have a synergistic effect, increasing HbA1c even more than the sum
of the individual effect of either risk factor alone. We now know that
this is true."----H. pylori lives in the mucous layer lining the stomach
where it persists for decades.
It is acquired usually before the age of 10, and is transmitted mainly in
families. Dr. Blaser's previous studies have
confirmed the bacterium's link to stomach cancer and elucidated genes
associated with its virulence, particularly a gene called cagA.

Regarding H.
pylori's association with elevated HbA1c, Drs. Chen and Blaser believe the
bacterium may affect the levels of two stomach hormones that help regulate
blood glucose, and they suggest that eradicating H. pylori
using antibiotics in some older obese individuals could be
beneficial.---More research will be needed to evaluate the health effects
of H. pylori
and its eradication among different age groups and in relation to obesity
status, the authors noted."If future studies confirm our finding, it may
be beneficial for individuals at risk for diabetes to be tested for the
presence of H. pylori
and, depending on the individual's risk factor profile" Dr. Chen.-----In
an accompanying editorial in
The Journal of Infectious Diseases,
Dani Cohen, PhD, of Tel Aviv University in Israel, pointed out that while
previous studies have addressed the association between type II diabetes
and H. pylori
in small samples, this study analyzed two independent large national
samples of the general population. Dr. Cohen agreed with the study
authors, suggesting that adults infected with H. pylori
with higher BMI levels, even if asymptomatic, may need anti-H.
pylori therapy to control or
prevent type II diabetes. If
the study findings are confirmed, Dr. Cohen wrote, they "could have
important clinical and public health implications."--Story
Source-The above story is
reprinted from
materials provided by
NYU Langone Medical
Center, via
Newswise.

[U4]Again—take
the 28 components of chemtrails---add that to the water you drink if
you are still drinking from the tap---then figire out the levels in
your plants ( organic or otherwise since they will uptake the toxic
metals as well from the ground released by the pesticides and
chemtrails) and you now have a chemical mix killing or debilitating
a human being

[U5]Which
will further increase the levels in our system since these plants
will be able to absorb more of the metal ---it makes them more
resistant –not more healthy—big difference