Please, try to understand my q.
See, the antibodies are present in the maternal blood and very less maternal blood mixes with the fetal blood . So, less feto- RBCs would be killed due to direct mixing of blood.
But i wonder if the antibodies may get flushed into the fetal blood-vessels near placenta due to pressure. If the pressure and offcourse the concentration of these Abs in maternal blood is large enough to have enough Abs flushed into fetal blood then fetus may die due to erythroblastosis. But all this is my hypothesis, is it correct??? Have u got my pt...

So, enough no. of Abs get flushed into fetal blood due to pressure through the placental blood vessels and they lead to the erythroblastosis. But , one pt. is that the fetus must not be in a developemental stage to raise immunoresponse agaist the maternal Abs ... otherwise less erythroblastosis will occur . Right???