Human Syntaxin 1B (STX1B) interaction partners

The data of this study suggested that the STX1B polymorphisms are associated with Parkinson disease etiology.

Data indicate that single nucleotide polymorphism (SNPS) in the 3'-untranslated region of the fucosyltransferase 1 (FUT1 (show FUT1 Antibodies)) gene and intron of the syntaxin 1B (STX1B) gene were the top hits for Kawasaki disease (KD) susceptibility.

STX1B and the presynaptic release machinery may have a role in fever-associated epilepsy syndromes

The STX1B-Delta transmembrane domain is characterized as the first nucleoplasmic syntaxin with no transmembrane domain.

Mouse (Murine) Syntaxin 1B (STX1B) interaction partners

Results demonstrated that STX1B is essential for the survival of neurons in vitro and it is a critical syntaxin protein for the neurotransmission at the mouse neuromuscular junctions

Although STX1A (show STX1A Antibodies) and STX1B share a basic function as neuronal t-SNAREs, STX1B but not STX1A (show STX1A Antibodies) is necessary for the regulation of spontaneous and evoked synaptic vesicle exocytosis in fast transmission.

STX1B was important for neuronal survival, possibly by regulating the secretion of neurotrophic factors