"The Convention allows defensive biological work, such as the development of vaccines. However, the line between defensive and offensive work is very thin; in order to make a vaccine or an antidote, one must first learn how a pathogen works, and that information could be put to offensive use"

The spread of human, animal or crop disease can be made to look like an "act of God" with no one able to trace the perpetrator(s)

Abacá, ah buh KAH, (Musa textilis)
is a species of banana native to the Philippines, grown widely as well in Borneo and Sumatra. The plant is of major economical importance, being harvested for its fibre, called Manila hemp, extracted from the large, oblong leaves and stems. On average, the plant grows about 20 feet (6 metres) tall. The fibre is used for making twines and ropes. The plant's name is sometimes spelt Abaká
.
[edit] Composition
The leaves grow from the trunk of the plant, and the bases of the leaves form a sheath (covering) around the trunk. These sheaths contain the valuable fibre. The coarse fibres range from 5 to 11½ feet (1.5 to 3.5 metres) in length. They are composed primarily of the plant materials cellulose, lignin, and pectin. After the fibre has been separated, it is sold under the name Manila. The fibre gets its name from the capital of the Philippines.
**************************************

The superficial isobaric gas counterdiffusion phenomenon, which produces dermal lesions and lethal gas embolism, was investigated at sea level and 2 ATA for effects on the eye of the New Zealand White rabbit. The animals breathed an N2O-O2 mixture by mask and were surrounded by a He environment. There was no gas lesion formation in the conjunctiva or cornea and no gas bubble formation in the anterior compartment of the eye in any of the experimental animals, even at the maximal 8-h exposure. In contrast to the eye, the skin of these animals showed progressive gas-filled lesions after 3 h at 1 ATA. Reasons for the relative insensitivity of the structures of the eye to the counter-diffusion process are discussed.

"Chitridiomycosis was diagnosed in native Arizona
Leopard frogs (Rana yavapiensis and Rana
chiricahuensis). Recently, there has been high mortality in
adult frogs of these species. The lesions noted included
cutaneous erythema and edema, especially of the ventrum.
Microscopically, there was chronic, hyperplastic dermatitis
with hyperkeratosis and intracorneal fungal organisms.
Consultation with the Armed Forces Institute of Pathology
and the Department of Pathology of the National Zoological
Park resulted in identification of the organism as a
species of chitrid fungi. Until the last few years, these
organisms had not been recognized as an agent of disease
in vertebrate animals. They normally live on decaying
vegetation. The factors (environmental, stress, toxic) that
lead to infection with the organism have not been identified
but the infection is being recognized with increasing
frequency in many parts of the US and Australia."

The botanical genus "Microcystis" of unicellular colonial freshwater
plankton species is very well supported by phylogenetic reconstruction, as is the genus Trichodesmium of filamentous, nonheterocystous nitrogen-fixing species typical from oligotrophic marine plankton of the tropics. The picture that emerges from these studies is that sufficient knowledge of ecological and physiological characteristics can lead to a taxonomic system that is largely congruent to the 16S rRNA phylogeny.
A different principle of conversion of light energy into chemical energy is found in the Halobacteria.These archaea are largely confined to surface layers of hypersaline aquatic environments and grow predominantly by chemoorganoheterotrophy with amino or organic acids as electron donors and carbon substrates, generating ATP by respiration of molecular oxygen. In the absence of oxygen, several members are capable of fermentation or nitrate respiration. At limiting concentrations of oxygen, at least three of the described species of Halobacteria (Halobacterium halobium, H. salinarium, H. sodomense) synthesize bacteriorhodopsin (Oesterhelt and Stoeckenius, 1973), a chromoprotein containing a covalently bound retinal. Bacteriorhodopsin is incorporated in discrete patches in the cytoplasmic membrane ("purple membrane"). However, these prokaryotes have only a very limite d capability of light-dependent growth. Only slow growth and one to two cell doublings could be demonstrated experimentally (Hartmann et al., 1980; Oesterhelt and Krippahl, 1983). The fact that rhodopsin-based photosynthesis has been found only in the phylogenetically tight group of Halobacteria may indicate that, because of its lower efficiency, this type of light utilization is of selective advantage only under specific (and extreme) environmental conditions.

Hi, still looking at yout links.....NIH and Rochester> amazing what they get away with......but you and I both know the aids virus came from primates! But what I want to know is how those primates got it?? hehe
My link? I'm lost there.....good job skyler!

TamTam,

jeez, I hope so, I hope the eye thing is just a virus......I really do. My eye spots started 5 months before I had my one and only brief conjucntivitis session. It only lasted about 12 hours. started with a mucus or film feeling in one of the eyes. Groce! All crusty lashes....it was sicko. Then as quick as it came on, it left. No meds nothing taken.

Anyway, look at this: This is what I think it to be.....if you look thru article, it even mentions the cochlear implants.....but hey, I hope I'm wrong.

Hey sky,Nad, anyone.....do you remember seeing (when the full blown disease set in) a thing that looked similar to a sunflower seed but had colored (like purple) 1" string coming out of the end or ends??? That was one of the things that Orkin man could not identify. Just curious.

and this is kind of interesting......

Neuronal Control of Skin Function: The Skin as a Neuroimmunoendocrine Organ
Dirk Roosterman, Tobias Goerge, Stefan W. Schneider, Nigel W. Bunnett and Martin Steinhoff
Department of Dermatology, IZKF Münster, and Boltzmann Institute for Cell and Immunobiology of the Skin, University of Münster, Münster, Germany; and Departments of Surgery and Physiology, University of California, San Francisco, California

This review focuses on the role of the peripheral nervous system in cutaneous biology and disease. During the last few years, a modern concept of an interactive network between cutaneous nerves, the neuroendocrine axis, and the immune system has been established. We learned that neurocutaneous interactions influence a variety of physiological and pathophysiological functions, including cell growth, immunity, inflammation, pruritus, and wound healing. This interaction is mediated by primary afferent as well as autonomic nerves, which release neuromediators and activate specific receptors on many target cells in the skin. A dense network of sensory nerves releases neuropeptides, thereby modulating inflammation, cell growth, and the immune responses in the skin. Neurotrophic factors, in addition to regulating nerve growth, participate in many properties of skin function. The skin expresses a variety of neurohormone receptors coupled to heterotrimeric G proteins that are tightly involved in skin homeostasis and inflammation. This neurohormone-receptor interaction is modulated by endopeptidases, which are able to terminate neuropeptide-induced inflammatory or immune responses. Neuronal proteinase-activated receptors or transient receptor potential ion channels are recently described receptors that may have been important in regulating neurogenic inflammation, pain, and pruritus. Together, a close multidirectional interaction between neuromediators, high-affinity receptors, and regulatory proteases is critically involved to maintain tissue integrity and regulate inflammatory responses in the skin. A deeper understanding of cutaneous neuroimmunoendocrinology may help to develop new strategies for the treatment of several skin diseases.

The DOE/EPA and now univieristies with DOE, like MSU. Right on college campus.

"But genome research, like many other forms of biological and chemical research, is "dual use." And the U.S. Government appears to be very interested in its military applications. Note that the government's Joint Genome Institute (JGI) 48 is not under the auspices of the Department of Health and Human Services. It is part of the Department of Energy, which often works hand-in-glove with the Defense Department.

DOE's own explanation for its involvement in the Human Genome Project betrays military roots:

After the atomic bomb was developed and used, the U.S. Congress charged DOE's predecessor agencies (the Atomic Energy Commission and the Energy Research and Development Administration) with studying and analyzing genome structure, replication, damage, and repair and the consequences of genetic mutations, especially those caused by radiation and chemical by-products of energy production. From these studies grew the recognition that the best way to study these effects was to analyze the entire human genome to obtain a reference sequence. Planning began in 1986 for DOE's Human Genome Program and in 1987 for the National Institutes of Health's (NIH) program. The DOE-NIH U.S. Human Genome Project formally began October 1, 1990, after the first joint 5-year plan was written and a memorandum of understanding was signed between the two organizations.49 "

From Dual Use link.......

Anyone remember the secret meetings between Cheney and Abrahams?
back in 2000. No one ever knew or knows what their agenda is. Hiding the free energy?

SOMETHING ABOUT THAT CD-4 gave it away:
Wonder how many versions of HIV-1 and HIV-2 we can get. All they had to do was add the promoter gene. Make it genetic manifestation jetted to us by way of climate change operations.

"The AIDS pandemic continues to spread unchecked in many parts of the world, with greater than 34 million individuals currently infected with human immunodeficiency virus (HIV). While most infections are due to HIV type 1 (HIV-1) strains, HIV-2 represents a significant minority of all HIV infections in some countries, such as Guinea-Bissau and Portugal. While similar in many ways, there are important differences between HIV-1 and HIV-2 that provide insights into virus evolution, tropism and pathogenesis. Major differences include reduced pathogenicity of HIV-2 relative to HIV-1, enhanced immune control of HIV-2 infection and often some degree of CD4-independence. This review discusses the origin of HIV-2 and its relationship to simian immunodeficiency virus and HIV-1, its epidemiology, its pathogenic potential and how its Env protein interacts with cell surface receptors to mediate virus infection. "
for full article:
http://vir.sgmjournals.org/cgi/content/ ... /1253#SEC6and another look at it:
http://www.hiv.lanl.gov/content/hiv-db/ ... /Kent.html

London..... I am not here..I am here. I am not here..I am here...Spies all around us.....YOU ARE ONE, HE is ONE

Moka nada: The universe is ONE.....The world in a tea cup is only as those in the cup may see it.

Tam: I am a scientist , weel not really, I just read alot an dcut and paste abstract thoughts so these idiots will worship me,the jerks....HAHAHAHAHAHAHAAh And they even believe that I am afraid to be known yet my picture is on my cool video that makes no sense. Yet they still worship me..HAHAHAHAHAHA!

I appreciate your taking this information and hope you find it informative. It is my purpose to inform you of what I think is a rapidly emerging infectious disease. I have seen patients with this disease in my practice. Whether or not you know it, you do as well.

One major organ system affected by this disease is the skin. Patients report itching, stinging, and biting sensations. Most report a ‘skin crawling’ sensation. Patients usually have severe excoriations from continued scratching. Many have large open sores. As a pediatrician, scabies would be high on my differential diagnosis list, but many other disorders might also be considered.

What makes this so different is that many patients have also noticed other skin symptoms and changes in their skin and hair. Some of the descriptions are quite bizarre and unlike anything I had ever been taught or observed. Frankly, the descriptions are so far from anything most of us think of as typical or even possible that credibility is quickly strained.

Many patients report they have seen ‘fibers’ come out of their skin. They report seeing these odd fibers in their home and cars and report they have never seen these before. Even more strange, many report seeing living insects emerge from their skin! Many patients, thinking it the appropriate step, collect specimens and present them to the physician they consult---commonly a dermatologist.

Eighteen months ago, I was not familiar with the psychiatric diagnosis Delusions of Parasitosis. The term ‘match-box’ sign was not familiar to me. That is the term used when patients who believe they are infested with parasites collect skin specimens in a container (in the past, this was most commonly an empty matchbox) to show their doctor. This sign is supposedly pathognomic of Ekbom Syndrome, now generally known as Delusions of Parasitosis or DOP.

The dermatology literature very clearly states that DOP is a diagnosis of EXCLUSION. Yet all too many patients have been immediately labeled as psychiatric cases with no lab studies, no skin biopsies, and often not even a thorough skin examination. Their self collected specimens are usually dismissed and trashed.

Even after patients see a psychiatrist and are pronounced entirely sane, dermatologists across the board have ignored the psychiatry opinion and have started patients on pimozide or other potent psychotropic medications. Some patients have had severe adverse reactions.

More persistent patients report multiple consultations with a variety of specialists. Many feel the physicians did not take them seriously and did not listen to their story. Many have lost faith in our medical care system and have turned to alternative medical care or self treatment. Descriptions of their self treatments are frightening. The treatments have included not just topical application but oral ingestion of insect sprays! Some have been victimized by unscrupulous alternative health care providers. These patients’ stories sparked my interest in Morgellons while also making me ashamed of the treatment given them by members of my profession!

Tragically, it seems the systemic symptoms are not being considered in these patients. This appears to be a multi-system disorder, as a large proportion of patients report neurological, psychiatric, and gastrointestinal symptoms. Almost all experience significant and disabling fatigue, problems with concentration and short term memory as well as other cognitive difficulties. Many experience periods of what is called “brain fog”. Some have been diagnosed with ADHD, Bipolar Disorder, or other psychiatric diagnoses. A significant number have neurologic impairments, including Multiple Sclerosis, ALS, and other neuropathies. Among children with this disease, about half have the diagnosis of ADHD. 10% carry the diagnosis of autism.

The personal medical history of most patients IS bizarre. The symptoms they describe, as well as their observations, seem totally beyond belief. When they occur on one’s own body, it can cause one to question his sanity. I know. I have had this disease since May, 2004. I have seen things happening to my own body which have stretched the limits of belief. I also developed neurologic symptoms early this year which became severe enough I have been unable to work since May, 2005.

Dermatologists use the term ‘folie a’ deux’ when a family member or associate of the patient develops the symptoms. The term essentially means a delusion shared by two. A similar term is used for 3 affected persons, another if 4 persons are affected, etc. The term has even been used to label those physicians who actually looked and who then agreed the patient had unusual findings!

This supposedly explains the fact that several family members may also have the same symptoms. Personally, that only makes sense for people who know each other well and spend a lot of time together. It does not make sense when unrelated patients, in large numbers, who are scattered geographically all over the United States and in at least 15 other countries, report almost exactly the same symptoms and observations. It also does not make sense that very young children—2 or 3 years old----are delusional when they have these symptoms and say they want the bugs off their skin!

This disease is not recognized by the traditional medical care system. The public health system and the CDC are not investigating, even after numerous contacts have reported concern that this is an emerging infectious illness.

Morgellons is the name we are using although others have called it the ‘fiber disease’, ‘Elliot’s Disease’, and other names. Any of those terms in an Internet search engine will yield a surprising number of hits.

The lack of interest in objective research by the public health system and the CDC has been especially disturbing. The CDC was made aware of concerns about this disease at least 5 years ago. Yet there has been no serious investigation by any part of the public health system. Efforts by The Morgellons Research Foundation as well as numerous individuals to interest the CDC in sponsoring research have not been successful.

If you choose to do your own research, you will find many individuals and groups with an agenda. They believe what they believe and want it proved. Some are convinced there is a government conspiracy and the disease is a result of biologic warfare research which escaped into the environment. There is even the alien invasion group. More credible are those physicians who specialize in Lyme disease and believe Morgellons is associated with or a result of chronic Lyme disease. Until the last year, I had no idea of the controversy surrounding Lyme. It seems there are dramatic differences of opinion about almost every aspect of that disease. Even the current testing recommended by the CDC is questioned.

The New Morgellons Order has only one agenda and no pre-conceived ideas about the cause or treatment of this disease. Our purpose is to get this researched and ultimately find a cure!

As an individual, I am sharing this information only to inform. I believe knowledge of this disease must be distributed to medical professionals. I expect nothing from you and sincerely appreciate the time you have taken to read this and review the information I have given you. I know I become passionate about issues in which I believe. But I do not believe I am delusional and my psychiatrist, who I have seen regularly for almost 20 years for my ADHD, also does not believe I am delusional. He says he has no idea what is causing my symptoms, but believes they are real. My neuropathy is certainly all too real.

Since I developed this disease, I looked at rashes in a new light. I began using a magnifying lens and not just my bare eyes to look at rashes. The number of children in my practice who have skin lesions like mine was frightening.

I am afraid. I have a serious health issue which I believe to be related to Morgellons. I believe many, many people have this disease and may develop systemic symptoms in the future. I think Morgellons is an emerging infectious illness which is already widespread.

I have been shocked at the official position of the CDC---the government agency tasked to protecting this nation from infectious agents. I have thought many times of the scenario concerning AIDS described in the book and TV mini-series titled “And the Band Played On”. Both my father and my wife received blood transfusions in the early 1980’s. At that time the CDC was aware of AIDS and knew the virus was transmitted by blood. My loved ones were lucky and did not receive contaminated blood. Many patients were not so lucky. Their lives were profoundly affected and shortened due to a lack of CDC action. Will you and your family be lucky?