Abstract

There is in evidence a growing appreciation of the fact that pneumonia is far from being a disease sharply limited within the confines of definite clinical specification. That pneumonia may be caused by a considerable variety of bacterial agents is well known, each organism endowed with different degrees of virulence, which, further, may fluctuate from time to time. So, also, is recognized the modification of symptoms and prognosis possible by quantitative pulmonary and hemic involvement, as well as by such qualitative factors as age, physical condition and individual resistance in the patient. As McKinlay6 remarks, Bullowa and Wilcox1 have well