"Today, almost half of our population suffers from a degenerative
disease that directly causes Heart Failure, Stroke, Type 2 Diabetes, Kidney
Failure, Impotence, Obesity, Neuropathy, Retinopathy and a host of other similar
symptoms. It is thought to be implicated, but not yet proven to be causal, in
ADHD, ADD and some forms of Cancer. Its beginning symptoms are so mild that
most who exhibit them do not realize that they are under a sentence of premature
death and disability. It affects adults and children of all ages. At the turn
of the last century this disease was so rare that many doctors could not correctly
identify it. Today this disease forms such an important economic pillar in the
medical community that every doctor in the country can easily recognize it at
a glance."

In a time of universal deceit, telling the truth is a revolutionary
act.
George Orwell

This website is about a disease syndrome. It is a disease syndrome
that is so common that all of us either have the disease or we know someone
that does have it. It is a primary, if not the primary, economic support for
a medical community whose policy level management has absolutely no interest
in curing it. This one disease syndrome alone accounted for over 40% of the
deaths in the US from all causes in 1995 (Based on data published by American
Demographics). This disease syndrome currently constitutes one of our most effective
restraints on population growth in the developed nations. At the turn of the
twentieth century, this disease syndrome was so rare that it was considered
a medical curiosity.
Much of the information presented on this website has been excerpted from our
best selling 147 page Special Investigative Report "Insulin: Our Silent
Killer." This report was written by a former victim of this disease who
discovered what causes it, when and how and why it became epidemic and how to
sucessfully reverse it completely, permanently, economically, naturally and
quickly in the large majority of cases. The alternative information needed to
cure this disease is not available from the orthodox medical community; indeed,
there is some evidence that it has been actively suppressed. Much of this needed
information is posted on this website. Additional information, including how
to order the special report, that provides substantial additional detail, is
provided on our more information page.
What's new
Today an epidemic of incredible proportions rages through the country. It directly
affects over half the population and incapacitates almost twenty percent of
us. Over ten percent us are completely dependent upon synthetic medication and
live under constant medical supervision because of this crippling drug dependence.
There are few of us in America who are not affected directly or indirectly by
this disease. It has been known since the 1950's as Insulin Resistant Diabetes,
Hyperinsulinemia, or Insulin Resistant Hyperinsulinemia. It is known to the
medical community by the symptoms that it produces. Some of these symptoms are:
Atherosclerosis, Vascular disease, Diabetes type 2, Impotence, Kidney Failure,
Heart Failure, Liver Damage, Stroke, Obesity, Neuropathy, Retinopathy and Gangrene
to name but just a few. We have separately discussed, each on its own page,
the connection of each of these symptoms to the underlying endocrine disorder,
Insulin Resistant Hyperinsulinemia.

This disease has so many life threatening symptoms that it influenced
an extensive reorganization of the medical disease classification system in
1949. This was the year the medical community defined many of these symptoms
into the independent medical specialty diseases that ravage America today. This
reorganization was promoted as a means of focusing medical activity more clearly
on the underlying disease syndrome. However, in practice it has resulted in
the widespread compartmentalization of the medical community into many different
competing medical specialties according to the presentation of the differing
"proprietary symptom sets" in different patients.

Doctors now are trained to deal only with their proprietary symptom
set according to prescribed protocols. The existence of these numerous new aliases
for this disease syndrome became a real problem for the scientific community
who has a real interest in understanding cause and effect. Their solution to
this "problem of too many aliases" is illuminating. It can be found
on our History page.

Also, this compartmentalization resulted in the establishment of powerful
economic disincentives to the promotion of a cure for the disease as competing
medical specialists vied for market share.

This compartmentalization has obscured both the cause of this disease
and the incredible scope of the epidemic that it produces. However, none of
these newly formed "medical compartments"are trained to deal with
the underlying cause of this disease. As all focus on actually curing anything
became hopelessly lost, the orthodox medical community prospered as never before
in all of its history. Today America has fallen well below the norm even for
western developed nations in the quality and quantity of medical care it dispenses.
Here in the US, doctor caused death is the third leading cause of death from
all causes. However, the American medical community still retains its lead as
the wealthiest and most prosperous medical community in the world.

This compartmentalization resulted in the establishment of powerful
economic disincentives to the promotion of a cure for the disease as competing
medical specialists vied for market share. Clearly the medical reorganization
of 1949, while not all benefiting the patient, did indeed benefit the medical
community.

The disease itself that underlies and causes many of these symptoms
is discussed on our page on Hyperinsulinemia. Early warning indicators often
include elevated Cholesterol Levels, elevated Trigylycerides, poor HDL/LDL ratio,
High Blood Pressure and overweight problems. Some of these early warning indicators
of disease are discussed on our fats page.

This form of Hyperinsulinemia is also thought to be implicated in several
forms of Cancer and in the epidemic of Attention Deficit Hyperactivity Disorder
(ADHD) raging through our schools today; although with these latter two diseases,
the evidence while compelling is not yet completely conclusive. As we develop
the evidence for these connections, we will post them on this site.

Symptoms are valuable warning signs that something is wrong and that
quick effective attention is required. When they are deliberately suppressed
without curative action being undertaken to cure or reverse the disease, the
disease will make more rapid progress. Now that this policy of criminal suppression
of symptoms and careful avoidance of cure is being widely exposed, it is now
possible to look forward to something other than a rapid decline to invalid
status and an early painful death.

One would logically think that this disease would be cured by now since
it has been increasingly well understood since about 1950. However, as we shall
demonstrate on our history page, there are sound economic reasons why the disease
can never, even in principle, be cured by existing medical institutions even
though the cure is well understood in the research community. For the 50% or
so of people who have the disease in its various stages, it is necessary to
seek alternative medical treatment or remain sick.

The good news is that although this disease accounts for almost half
of the annual death toll from all causes, it is, in most cases, curable, permanently,
quickly, economically, completely and often easily and by natural means. That
means little or no reliance upon synthetic designer drugs and no ineffective
medical treatments for symptoms while causal agents remain untreated.

The bad news is that the orthodox medical community cannot afford to
cure this disease. It forms a financial backbone to the entire orthodox medical
establishment. Should an effective cure be popularized, the resulting financial
impact to the medical business, the drug business, several of our tax free foundations
and a large part of our food processing industry would be severe. Many hospitals
would close and many doctors, notably heart surgeons, would soon need to find
another line of work. Many drug companies and a large part of the food industry
would have to either shut down or greatly change their way of doing business.
A discussion of these economic forcing functions is included in our history
page.

This disease originated as an epidemic in the late 1920's and became
the focus of considerable medical attention in the early 1930's. It grew exponentially
through the 1940's and 1950's and is today accepted as a ubiquitous piece of
America's medical wallpaper. From a per capita incidence of 0.0028% at the turn
of the century, it has ballooned to 10% under doctors care; another 10% who
should be under doctors care except they are "coping" and don't yet
realize that they are diseased; and another 30% that exhibit clear symptoms
which they ignore because the disease does not yet interfere with life. In 1995,
forty percent of the death certificates in this country listed one or more symptoms
of this disease. Although much has been learned about this disease, dating back
to the 1930's, including how to easily cure it, it remains the underlying cause
of an incredible annual death toll.

A "death certificate shuffle" exists that acts to obscure
the cause of death and to camoflage the incredible epidemic that rages among
us. The shuffle works like this. Prior to the reorganization of the medical
community in 1949 all of the diseases listed above were known to be but symptoms
of what was then called Diabetes. After the reorganization these "symptoms"
became diseases in their own right. Each group of related symptoms became the
province of the specialist that presumably had expertise with that particular
symptom set. For example, if the disease had progressed to the point where the
patient experienced heart problems, he would be referred to a Heart Specialist.
If he died while treated by the the Heart Specialist, his death certificate
would say "Heart Failure." If the disease progressed to the point
when Kidney function was destroyed before heart problems occured, the death
certificate filled out by the Kidney Specialist would say "Kidney failure."

In this way we have defined a "top ten" category of killer
diseases. Many of these killer diseases are but differing symptoms of a single
underlying disease with numerous confusing alias's. This underlying disease
is now known to be caused by specific poisons and inadequacies in our food supply.
For those who would like to see more about the origins of this disease we have
included a brief history. As we show on our history page, this disease began
to become epidemic shortly after poisonous food was introduced.

This underlying disease is a severe unbalance of the endocrine system
that destroys our ability to metabolize food. The unbalance results in elevated
levels of certain control hormones as the body strives to correct a systemic
problem that it cannot correct without the missing essential elements of nutrition.

Although this is a relatively large site with a considerable amount
of useful information, we recognize that many who are either affected by this
disease or who have the responsibility to treat others may want to study additional
information in report form. For this reason we have compiled a Special Report
written for the layman, but which includes an extensive list of scientific cites
and references. This report includes a specific, non-drug protocol that can
reverse the disease fairly quickly and easily for the large majority that suffer
from it. It includes a Glycemic index with complete instructions for use and
a Bibliography and a Glossary. This special report contains far more information
than we can conveniently present on a webpage. Ordering instructions for this
Special Report are also included on our more page.

The results compiled on this web site and in our Special Report are
the result of a four year study undertaken by this writer who, having the disease,
was faced with the need to find a cure for it because his doctor couldn't or
wouldn't cure it. This Special Report presents, not only an effective cure protocol
for the disease, but helpful information on how to naturally manage its damaging
symptoms during the cure process. It also includes the information needed to
reverse much, but unfortunately not all, of the collateral damage that this
disease causes when it is encouraged to run rampant with ineffective orthodox
medical treatment. Our more page contains much additional descriptive information
about our special report along with ordering information.

History

In the 1880's German scientists wondered what was the function of the
Pancreas in anatomy. When they removed the Pancreas from a dog, they noted that
its blood sugar rose uncontrollably. Thus the disease that produced high blood
sugar came to be known as Diabetes and became identified with a nonfunctioning
or improperly functioning Pancreas. Later when Insulin was identified as the
causal agent in controlling blood sugar, a search was launched to isolate and
synthesize Insulin.

In 1922, three Canadian nobel prize winners, Banting, Best and Macleod
succeeded in saving the life of a fourteen year old diabetic girl in Toronto
General Hospital with injectable Insulin. Eli Lilly was licensed to manufacture
this new wonder drug and the medical community basked in the glory of a job
well done. For a time Diabetes, as a disease, was controllable if not curable.
No one liked the idea that the diabetic patient had to be supplied with Insulin
all of his life, but the idea became accepted as better than premature death.

It wasn't until 1933 that rumors about this disease surfaced in a paper
presented by Joslyn, Dublin and Marks and printed in the American Journal of
Medicine. This paper "Studies on Diabetes Mellitus", discussed a major
epidemic of a disease that looks very much like the Diabetes of the early 1920's
only it does not respond to the wonder drug, Insulin. Even worse, sometimes
Insulin treatment kills the patient. This disease became known as Insulin Resistant
Diabetes because it had the symptoms of Diabetes, but did not respond well to
Insulin therapy. Treatment of this disease by diet was started during this period.

Diabetes, which had a per capita incidence of 0.0028% at the turn of
the century, had by 1933, zoomed 1000% to become a disease faced by many doctors.
This disease was destined to go on to affect half of our population and to incapacitate
almost 20% by the 1990's.

It wasn't until 1950 that the medical community was able to perform
serum Insulin assays. This quickly revealed that the disease wasn't Diabetes,
at least not in the accepted classical understanding of Diabetes. This new disease
was characterized by sufficient, often excessive Insulin in the blood. The problem
was that the Insulin didn't seem to work to reduce blood sugar; it was ineffective.
But, since the disease had been known as Diabetes for almost twenty years it
was renamed Type II diabetes to distinguish it from the earlier Type 1 diabetes
characterized by insufficient insulin production by the pancreas.

In 1949, faced by what appeared to be an uncontrollable epidemic of
the so called Insulin Resistant Diabetes, the medical community reorganized
itself into the competing medical specialties that we see today. Thus the "Heart
Specialist", "Endocrinologist", "Allergist", "Intestinal
Disease Specialist", the "Cancer Specialist" and many others
started. Of course, all of these symptoms of this new disease became diseases
in their own right. Heart failure for example, which had been previously understood
often to be but a symptom of Diabetes, now became a disease not directly connected
to Diabetes. It became fashionable to think that diabetes "increased"
Cardio-vascular risk. The causal role of a failed Blood Sugar Control System
(BSCS) in Heart Failure became obscured.

A year later, in 1950, a search was launched for another "wonder
drug" to deal with the Type II diabetes problem. The ideal drug would be,
like Insulin, effective in remitting obvious adverse symptoms of the disease,
but not effective in curing the underlying disease. It would be needed continually
for the remaining life of the patient. It would have to be patentable; that
is, it could not be a natural medication because these are non-patentable. Like
Insulin, it would be economical to manufacture and distribute. Mandatory goverment
approvals would be required to stimulate the use by physicians as a prescription
drug. Testing required for these approvals would have to be enormously expensive
to prevent other, unapproved medications from becoming competitive. If the drug
had unexpected side effects, they could always be explained away by the fact
that the disease was worse than the side effects. If the patient died, well,
we did our best but this is after all a dangerous disease.

Consider; any drug that would really cure the disease would also put
the drug manufacturer out of business in short order due to a lack of customers.
Also any drug that was a natural agent, that could not be patented, was not
only not a suitable drug company investment but had to be suppressed as unacceptable
competition. The reason for this is the huge advertising budget for a natural,
unpatented product could not be protected from other companies simply selling
the product also without incurring any advertising costs. If the natural competing
drug actually worked better than the synthetic drug, all the more reason to
suppress it by force of law and to jail its proponents as quacks.

So it was important that the drug be patentable and not natural, effective
enough to relieve symptoms, but not effective enough to cure the disease. The
medical community also benefited from this approach, not only through the prescription
monopoly that they enjoyed, but because the strategy produced the repeat business
that they also needed in order to prosper economically.

Thus, was implemented the classic medical protocol of "treating
the symptoms". By doing this, both the drug company and the doctor could
stay in business and the patient, while not being cured, was often relieved
of his symptoms. Enough money changed hands to make the American medical establishment
the richest in the world. Unfortunately their patients have not been served
as well.

It is important to note that prior to this time the medical goal was
to cure disease because the patients usually would not accept anything less.
After the introduction of Insulin sucessfully re-trained patients not to expect
to be cured, the same commercial technique was applied to the new Hypoglycemic
agents. Today all of the Hypoglycemic agents marketed to "control"
Type II diabetes meet the original commercial criteria that we have listed above.

In 1955 the oral hypoglycemic drugs were introduced that have been
with us for almost five decades. Some of these drugs, notably Rezulin, have
been known to kill patients; yet, they remain on the market for unexplainable
lengths of time with regulatory agency approval.

Today almost half of the people in the country suffer from one or more
symptoms of this disease and it has become well known to physicians asType II
diabetes, "Insulin Resistant Diabetes, Insulin Resistance, or more rarely
Hyperinsulinemia. One wonders why this "stealth disease" has so many
aliases.

When research scientists tried to make sense out of these disease aliases
they found conventional medical terminology too confusing to allow useful communication
among themselves about the disease. How can you discuss a disease that had as
many aliases as there are medical specialties? How does one discuss a disease
with their peers that has literally dozens, if not hundreds, of major symptoms
and no apparent causative mechanism? This alias, or AKA, type communication
problem was resolved when the scientific community abandoned all of the convential
medical aliases and identified the disease simply as Syndrome X. It is called
a syndrome because it has such a huge number of symptoms. The symptom set includes
all of the listed diseases on our home page and many more as well. The basic
underlying disorder, Syndrome X, is a derangement of the Blood Sugar Control
System by poisonous fats and oils. It is exacerbated and complicated by the
near universal lack of other essential nutrition that the body needs to cope
with the metabolic consequences of these poisons.

Elevated Cholesterol levels, high Triglyceride levels, Hypertension
(High Blood Pressure), inability to metabolize fats and oils, all of the symptoms
listed on this home page as well as the well known inability to metabolize Carbohydrates
are all symptomatic of this disease. According to the American Heart Association,
almost 50% of Americans suffer from one or more of these symptoms. We discuss
this from a different perspective on our Hyperinsulinemia page.

When I discovered that this disease appeared quite suddenly in the
early 1930's, I wanted to know what else had happened then that might be studied
as a causal agent for the disease. It turns out there is a causal agent that
originated during the 1920's and which modern biochemists have now connected
to the extraordinary disease epidemic in which we are involved. This causal
agent is the systematic corruption and commercialization of our food supply
starting with our fats and oils.

As early as 1901, efforts had been made to manufacture and sell food
products by the use of automated factory machinery because of the immense potential
profits that were possible. Most of the early efforts failed because people
were inherently suspicious of food that wasn't farm fresh and because the technology
was poor. Safeway, for example, was reputed to have chosen its name as an assurance
to its customers that it would not sell adulterated food. As long as people
were prosperous, "suspicious food products" made little headway. Crisco,
the artificial shortening, was given away free in 2 1/2 lb cans in an unsucessful
effort to influence the wives of the nation to trust and buy the product in
preference to lard.

Margarine was introduced and bitterly opposed by the dairy states.
With the advent of the depression, Margarine, Crisco and a host of other Refined
and Hydrogenated products began to make significant penetration into the food
markets of the nation. Support for dairy opposition to Margarine faded during
WW II because there wasn't enough butter for the civilian population and the
needs of the military. At this point, the dairy industry having lost much support,
simply accepted a diluted market share and concentrated on supplying the military.
Flax oils and fish oils, which were common in the stores and considered a dietary
staple before our nation became diseased, disappeared from the shelf; the last
supplier of flax oil to the major distribution chains was Archer Dainiels Midland
and they discontinued the product in 1950.

The history of the adulteration of our once clean food supply exactly
parallels the rise of the epidemic Hyperinsulinemia that now lies at the root
of many, if not most, of the degenerative diseases from which we suffer.

On our Hyperinsulinemia page we discuss much more about the nature
of this disease, including effective ways to reverse its progress. More information
is available in our Special Report in hardcopy form about the relationship of
artificial fats and oils to this disease. Our Special Report also provides additional
detail on the food factors that we must guard against and the best way to recognise
the food products that cause this disease.

Diabetes is classically diagnosed as a failure of the body to properly
metabolize Carbohydrates. Its defining symptom is a high blood Glucose level.
Type 1 Diabetes is caused by "insufficient Insulin" production by
the pancreas. Type 2 Diabetes, which constitutes about 95% of all the cases,
is caused by normal, or sometimes excessive production of "ineffective
Insulin". In both types, the blood Glucose level remains elevated. Neither
insufficient Insulin nor ineffective Insulin can limit post prandial (after
eating) blood sugar to the normal range; in established cases of Type 2 Diabetes,
these elevated blood sugar levels often result in chronically elevated Insulin
levels.

The ineffective insulin is no different from normal insulin. Its ineffectiveness
lies in the failure of our cell population to respond to it not in any biochemical
change in the insulin itself. Therefore it is appropriate to note that this
disease is a disease that affects almost every cell in our body. The biochemistry
of our cellular metabolism is changed from the normal.

The classification of Diabetes as a failure to metabolize Carbohydrates
is a traditional classification that originated in the early 19th century when
little was known about metabolic diseases or about metabolic processes. Today,
with our increased knowledge of metabolic processes, it would appear quite appropriate
to define Type 2 Diabetes more fundamentally as a failure of the body to properly
metabolize Fats and Oils as well as carbohydrates. This failure results in a
loss of effectiveness of Insulin and in the consequent failure to metabolize
Carbohydrates. Unfortunately, much medical insight into this matter, except
at the research level, remains hampered by its 19th century legacy.

The Type II diabetes and the Hyperinsulinemic symptoms that occur are
system wide symptoms of a basic cellular failure to properly metabolize Glucose.
Each cell of our body, for reasons which are becoming clearer, find themselves
unable to transport Glucose from the blood stream to their interior. The glucose
then either remains in the blood stream or is stored as body fat, or otherwise
disposed of in the Urine.

Until quite recently it was believed that Insulin molecules bound themselves
to Glucose molecules to form a sort of "lock and key" configuration
at the cellular Membrane interface. These two molecules together sort of fit
the Membrane Receptor and allowed the Glucose to be transported into the cell
where it was used for fuel. Many doctors practicing today were taught this concept
in medical school. While this theory is descriptive and attractive in its simplicity
and while it may be adequate for some purposes, the true picture that is emerging
is much more complex and much more revealing.

It appears that when Insulin binds to a Membrane receptor it initiates
a complex cascade of biochemical reactions inside the cell. One of these reactions
causes Glucose transporters known as GLUT4 molecules to leave their parking
area inside the cell and travel to the inside surface of the cell Plasma Membrane.
When in the Membrane, they migrate through the Membrane to special areas of
the Membrane called Caveolae areas. There, by another series of biochemical
reactions they identify and "hook up" with Glucose molecules and transport
them into the interior of the cell by a process called endocytosis. Within the
cells interior, this Glucose is burned for fuel by the Mitochondria to produce
ATP and waste products. The ATP provides energy to power the cellular activity
and the waste products are excreted by other metabolic cellular pathways.

These GLUT4 transporters are responsible for transporting glucose from
the blood stream into all of our peripheral cells. Of the seven glucose transporters
so far identified they have, by far, the greatest ability to quickly reduce
our blood borne post prandial glucose. GLUT1 and GLUT3 are glucose transporters
that facilitate the transport of glucose from the blood stream across the blood
brain barrier to the neuronal cells of the brain. Remember that the brain uses
glucose as a primary fuel almost exclusively. GLUT2 facilitates transport of
glucose from our liver and intestines into the blood stream; it also regulates
insulin release from the pancreatic beta cells. GLUT5 functions in the absorption
of Glucose from the intestine into the blood stream and also in the reabsorption
of glucose from the kidneys back into systemic circulation when we are in a
glucose sparing mode of operation.. Two additional glucose transporters have
been identified but not yet characterized as to their function; they are, as
you might expect, GLUT6 and GLUT7.

Each of the glucose transporters operate most efficiently at different
levels of blood glucose. GLUT4 swiftly reduces very high levels of glucose.
GLUT3 operates efficiently at low blood glucose concentrations in order to keep
the brain supplied when blood sugar is low. GLUT2, in its regulatory function,
has an activity that is linear across a wide range of blood glucose concentrations.
It can thus provide an insulin demand signal to the pancreatic beta cells that
is proportional to the blood borne concentration of glucose. Since our BSCS
functions as a type 0 control system we would expect to find a proportional
sensor somewhere and this seems to be it.

Type 2 Diabetics, perhaps because of the high average Insulin levels,
typically have only about 1200 Insulin Receptors, or less, per cell Membrane.
This is about half of the norm. This appears to be one of the issues involved
in the high average Insulin levels often encountered in Type 2 Diabetes.

Many of the molecules involved in these Glucose and Insulin mediated
pathways are Lipids, that is they are Fatty Acids. A healthy Plasma Membrane,
now known as an active player in the Glucose scenario, contains a complement
of Cis type w=3 unsaturated fatty acids. This makes the Membrane relatively
fluid and slippery. When these Cis fatty acids are chronically unavailable because
of our diet, Trans fatty acids and short and medium chain Saturated Fatty Acids
are substituted in the cell Membrane. These substitutions make the cellular
Membrane stiffer and more sticky and inhibit the Glucose transport mechanism.
The mobility of the GLUT4 transporters is diminished, the interior biochemistry
of the cell is changed and the number of Insulin Receptors on the cell surface
is reduced. Although there remains much work to be done to fully elucidate all
of the steps in these pathways, this clearly marks the beginning of a biochemical
explanation for the known epidemiological relationship between fat metabolism
and the onset of Type 2 Diabetes.

There exists another phenomena peculiar to Type II Diabetes that operates
to keep the blood sugar elevated. It works like this. Remember that normally
the liver stores glucose as glycogen and inhibits the release of glucose when
glucose stimulated insulin levels are high. You may recall from our earlier
discussion that this mechanism keeps our short term, rapidly accessed supply
of glucose replenished. In Type II Diabetes the elevated glucose levels do not
seem to inhibit glucogenesis in the liver as it does in normal systems. Although
all of the evidence is not yet clear, this can quite likely be due to the same
impaired cellular glucose transport at the liver similar to that observed in
the peripheral cells. Also when the system is stressed, the Catacholamines that
are released stimulate additional glucose release by the liver. Sometimes this
effect can be noted by observing a fasting blood sugar in the morning that is
higher than the blood sugar of the previous evening, several hours after the
last meal of the day. Such unexpected over night elevation of the blood sugar
cannot be explained by a meal in the middle of the night when there wasn't one.

It is fashionable for pop medical science to blame "trans fats"
for the failure of the plasma cell membranes in our 67 or so trillion cells
to facilitate the operation of our GLUT 4 transporters to haul glucose into
the cell. To the extent that these trans fats are responsible for stiffening
the cell membrane and reducing its fluidity when they are used in place of the
Cis type w=3 unsaturated acids, trans fats are indeed a culprit. However, if
we didn't eat transfats and ate only "good" short and medium chain
saturated fats, we would still get Type II diabetes. The body limits its use
of either these transfats or saturated fats in cell membrane repair only when
Cis w=3 unsaturated fatty acids are not present in the diet. It is only when
these Cis w=3 fats are chronically unavailable to the body that Trans fats and
Saturated fats appear in excess in the membranes. These important membranes
then stiffen and become sticky, and systemic disease, including Type 2 Diabetes,
manifests.

All of these Cis w=3 unsaturated fatty acids have been completely removed
from our food chain and replaced by their trans isomer counterparts. This was
done because Cis w=3 oils require refrigeration and typically have a fairly
short shelf life. This makes them incompatible with the economics of modern
food distribution. Our convenient grocery store availablity of these Cis type
essential fatty acids ended in 1950 when Archer Daniels Midland, the last supplier,
decided to stop producing this valuable oil and concentrate on processing flour.
Our diet now consists only of saturated fats, transfats and some of the hardier
Cis w=6 and w=9 fats. All of the delicate w=3 Cis fats, uniquely needed by our
cell membranes, because of their poor room temperature shelf life, have completely
disappeared from the local grocery store.

Two of the w=3 Cis fats are called essential because without them our
bodies develop chronic disease and because our bodies cannot synthesize them
from any other food that we eat. These two are: Linoleic acid (LA) and Alpha
Linolenic acid (LNA). A third fatty acid, Arachidonic Acid (AA), was once thought
to be essential. However, recently it has been discovered that a healthy body
can convert LA into AA, so it is no longer believed to be essential for healthy
people.

A major reason for discontinuing the production of the Cis w=3 oils
is that they rapidly become rancid when placed in a transparent bottle on a
room temperature grocery store shelf. The resulting manufacturing and distribution
problems that would ensue would seriously impact the bottom line of profitability.
The trans isomer counterpart to the essential fatty acid we need and do not
get has a very long shelf life. Trans fatty acids present few manufacturing
or distribution problems for the oil makers; this substitution of the trans
isomers for the needed Cis type oils causes Hyperinsulinemia and results in
the many other symptoms of the disease that are curently killing us. This outrageous
fraud is often accompanied by advertising that informs us of the "monounsaturated"
or "polyunsaturated" value of these worthless and damaging trans oils.
The law does not require the oil sellers to state that their oil is a trans
isomer and not the Cis isomer that we desperately need; and, this is their reason
for not doing so.

For more information about these poisonous fats and oils, please visit
our fats page.

This story started to come to light in 1950 when serum Insulin assays
became available to the medical community. It has been actively suppressed since
then; the careers of many ethical scientists have been damaged by trying to
bring this story to the light of day. Many others have been cowed into politically
correct silence. Now the evidence is so overwhelming and the number of people
becoming knowlegable about the matter is so large that it cannot remain hidden
in industry funded, politically correct ivory towers any longer. For additional
information on the effect of these fats and oils, including how to protect ourselves
from the damage they are believed to cause, see our page on Hyperinsulinemia
.

For those that prefer more information in hard copy form, this is available
in our special report. Please visit this page to see if this is something you
would like to know more about.

Hyperinsulinemia is an endocrine disorder characterized by a failure
of our Blood Sugar Control System (BSCS) to work properly. It manifests when
Insulin progressively loses its effectiveness in sweeping the blood Glucose
from the blood stream into the sixty seven trillion or so cells that constitute
our bodies. Insulin levels in the blood rapidly rise to damaging levels and,
together with the resulting elevated Glucose levels, account for much of the
damage to our Arteries and Vascular system. When Insulin loses its effectiveness
this loss is not due to any change in the Insulin produced by the Pancreas.
It is due to a change in the cellular metabolism of almost every cell in our
body. Although our Insulin has not changed, our cell metabolism has changed.
Our cells no longer respond to blood borne Insulin signalling as they should.

Our Blood Sugar Control System works like any type 0, negative feedback
system to maintain our blood sugar at a predetermined setpoint. This setpoint
is below the threshold where excess Glucose can cause Vascular damage. And the
Insulin required to do this is normally below the threashold where it will cause
Arterial or Vascular damage. When the Blood Sugar Control System is working
right, it automatically, without our concious knowledge, maintains correct blood
sugar with a minimum amount of Insulin whether we have just eaten a meal or
been fasting and exercising for a week.

When our system starts to exhibit Hyperinsulinemia, our Pancreatic
Beta cells simply increase Insulin production and for a time this maintains
our ability to swiftly lower post prandial (after eating) blood Glucose. For
a time this maintains normal Glucose levels, albeit by the secretion of these
abnormally high Insulin levels.

At some point during the progressive loss of effectiveness of Insulin,
our Pancreatic Beta cells may no longer produce enough Insulin to manage normal
post prandial and fasting Glucose. This may occur because our Pancreas becomes
exhausted by trying to maintain abnormally high Insulin levels needed. It may
occur because the progressive failure of our cellular metabolism has created
a chronic demand for Insulin beyond what even a healthy Pancreas can supply.
In either event, when this happens Type 2 diabetes is diagnosed. Of course,
Hyperinsulinemia has been around for some while, often for a long while, by
the time this diagnosis is made.

The fat and oils connection to Hyperinsulinemia, and thus to all of
the diseases mentioned on our home page, clearly parallels the rise of the Hydrogenated
and Refined fats and oils business. Although not well known outside of research
circles, (for reasons that are probably economic), the connection between artificial
fats and oils and the Hyperinsulinemic destruction of vital functions is now
well established. Recent advances in the the study of appropriate cellular biochemical
pathways have been most revealing.

To stop and reverse the progress of Hyperinsulinemia the following
dietary steps are mandatory:

Do not eat any hydrogenated oils or any prepared foods that contain
them as ingredients.
Do not eat any unsaturated or refined vegetable oil that comes in a transparent
bottle on a room temperature grocery store shelf. Cold pressed olive oil, coconut
oil and Sesame seed oil are the only exceptions to this blanket rule.
Do not eat any oil that has been used in deep frying or that has been heated
in cooking except butter and coconut oil. Do not eat any deep fried foods at
all.
Add one or two tablespoons full of cold pressed flax oil or fish oil to the
daily diet. Do not cook with this oil. Keep it refrigerated when not in use.
Supplement the diet with a good vitamin and chelated mineral complex from a
reputable manufacturer. If you don't know one, get a recommendation from your
local Naturopath or Chiropractor. Be sure to get Chromium, Vanadium, Calcium,
Magnesium, and the trace minerals in the mix.
The program outlined above has a track record of reversing Hyperinsulinemia
and the Type 2 diabetes that often accompanies it. In our Special Report, Insulin:
Our Silent Killer, we are able to expand, explain and elaborate upon this protocol
and present useful tricks and techniques to assure its speedy success in the
vast majority of cases.

However, there is another problem that must be faced when curing this
disease. It is this: during that part of the cure cycle when elevated blood
Glucose and Insulin levels are manifest, the body is being slowly destroyed
by these agents. This period may last for many weeks or months dependent upon
how long the disease has been allowed to run rampant before a cure is attempted.
During this period of time it is of the utmost importance to use all measures
available to keep blood sugar and Insulin levels as low as possible so as to
minimize the damage. The following list of techniques can be of considerable
help in doing this:

Select foods with a low Glycemic Index from a glycemic table. Our Special
Report contains a Glycemic Index with instructions for use and much more information
on this. Do not depend upon food exchanges; they don't work. They are based
on the idea that food may be exchanged on the basis of equal calorie content
and ignore that fact that, while calories are important, the glycemic value
is far more important to the diabetic.
Use fiber in the diet as much as practical. It reduces the "rate effect"
of Blood Sugar Control System perturbation and helps keep post prandial Glucose
in check. Our Special Report elaborates upon the types of fiber and on how and
why fiber works to control blood sugar.
Develop the habit of exercise at least twice a day. Work for twenty minutes
or until you begain to perspire. Do not do aerobic exercises if your Arterial
system has been compromised. Our Special Report discusses the additional non-Insulin
dependent pathway that exercise opens for Glucose disposal.
Investigate the use of herbs such as Gymnema Sylvestre to help keep blood glucose
low. We have more on this Herb as well as over thirty additional hypoglycemic
herbs in our Special Report.
Test for allergic reactions when you eat by taking your pulse. When you eat
a food to which you are allergic, your pulse rate will escalate at least 10
beats per minute. Don't eat any food to which you are allergic. This technology,
too, is expanded and elaborated upon in our Special Report.
As a last resort, consult with your doctor regarding the use of Vanadium Sulfate
and other oral hypoglycemic synthetic drugs. Be sure that you understand the
side effects of these before you commit to using them. Our Special Report, Insulin:
Our Silent Killer discusses rather thoroughly the pros and cons of these medications.
It is very useful information to have if you need to discuss these issues with
your physician.
In the beginning of the recovery program is is best to remove fats and oils,
except flax seed oil, or other oil containing a high percentage of W3 fatty
acids, from the diet as completely as possible. Later, when the Hyperinsulinemia
has started to reverse, it is ok to gradually restore fats and oils, good fats
and oils only, to as much as fifteen percent of the calories consumed.

This program will, relatively quickly, reverse Hyperinsulinemia, Type
2 diabetes and some of the other symptomatic diseases caused by Hyperinsulinemia.
In my case, my Type 2 diabetes was reversed in 103 days from start to finish.
At the start my fasting blood sugar was 368mg/dl. At the end of the program
it varied between 75 and 85 mg/dl.

This program will remove much stress from the components of the Blood
Sugar Control System; over a period of time they too will be restored to youthful
function. This includes the Liver, Pancreas, Adrenals, Thyroid and the interior
transport agents in each cell of our body.

This program will slow and in some cases reverse Vascular damage and
Gangrenous damage to our extremities. There are faster ways to reverse this
sort of damage which is thoroughly discussed in our Special Report.

This program will, over time, revcrse much of the Neuropathic damage
to the nervous system.

It will reverse Atherosclerosis too, but may take years to do it. But
here too, there are faster and better ways to reverse Atherosclerosis which
we cover in our Special Report.

Although this brief discussion of Hyperinsulinemia has been carefully
designed to provide the basic information needed to reverse the condition, a
great deal more information is available, in our Special Report, for those who
prefer to have a reference book handy. The theory here is that more detail may
be needed to assure a comfortable understanding than is practical to include
in a web page.

Fats and oils are an important part of any well designed dietary plan.
A good working understanding of just what they are and how they work is an essential
part of any well conceived diet. Fats and oils, certainly as much and perhaps
more than any other single dietary component, directly impact our health in
profound ways.

The difference between fats and oils is in their melting point. Fats
tend to be solids at room temperature; oils tend to be liquid at room temperature.
To turn a fat into an oil, merely raise its temperature above its melting point.
If the temperature continues to increase beyond the melting point to the point
where some smoke becomes evident, the molecular structure of the oils will change
and a number of toxic molecular isomers will be produced in the oil. If the
oil is allowed to cool or to resolidify, the toxic products will remain. The
temperatures where this damage is done to our fats and oils is about half the
temperatures reached in the refining and Hydrogenation processes. Thus, these
processes routinely destroy all of the nutritional value of our fats and oils.
These refined and/or Hydrogenated fats and oils are characterized by an extraordinarily
long shelf life; some are virtually unspoilable.

Naturally occurring fats and oils are Triglycerides. Triglycerides
consist of three fatty acids bound to a Glycerol backbone. Each fatty acid consists
of a Carbon-Hydrogen chain with a Carboxyl group at the end that is attached
to the Glycerol molecule. The other end is typically terminated with a Hydrogen
bond. Unless changed chemically, by artificial technology, this is the natural
form which we find in the fats and oils that are nutritionally useful. The length
of the fatty acid chain as well as its configuration and relative degree of
saturation determine how the fatty acid will act within our body. Some fatty
acids are vitally necessary to life processes; some are poisons.

Fatty acids are also found in other molecules besides Triglycerides.
For example Phospholipids have two fatty acids and a Phosphorus molecule attached
to the Glycerol backbone. Phospholipids too, play an important role in our cellular
health.

Understanding Triglycerides is an important issue that is complicated
by a great deal of pseudo science that is specifically designed to confuse and
mislead. In addition to the Triglycerides that we eat in the form of fats and
oils, we also have Triglycerides formed, within our bodies, from the sugars
and starches that we eat. Much of this Triglyceride load is deposited in our
adipose (fat) cells when we eat too much fat and sugar and some of us become
obese. Some of these Triglycerides are broken down into their fatty acids which
are then used in cell repair. When we lack Cis type w=3's in our diet, most
of the fatty acid load is either trans-fats or saturated fats; these are used
to repair our cell membranes. It is the combined absence of the Cis w=3 fats
and oils and the presence of these saturated and trans-fats and other toxic
isomers that cause these cellular membranes to become stiff and sticky instead
of fluid and slippery. Additional biochemical detail on this cellular membrane
issue is discussed on our diabetes page.

The saturated fat Triglycerides circulate in the blood stream before
finding a home in our Adipose cells (fat cells). They tend to be sticky instead
of slippery and so contribute to the high incidence of Strokes and Atherosclerosis
associated with high levels of Triglycerides in the blood. They make the blood
viscosity thicker and cause the Platelets to tend to stick together. They are
also an essential step in the chain of events that cause obesity.

All dietary fatty acids may be divided into two categories: Saturated
and Unsaturated. The Unsaturated fats and oils differ from each other in their
configuration and in their degree of unsaturation. Both types of fatty acids
are produced by the the animal and by the vegetable kingdoms, although some
are predominately found in animal sources and some are predominately found in
vegetable sources. Most concentrated vegetable sources are seeds and nuts; most
animal sources are animal body fat. Unrefined fish oils are good sources of
dietary Cis w=3 fats; unrefined Flax seed oil, Hemp seed oil and several others
are good concentrated vegetable sources of Cis w=3 oils.

Saturated fats are characterized by having all of the possible molecular
locations for a Hydrogen bond filled. Thus, at the moludular level, there is
no molecular difference between a saturated vegetable fat or a saturated animal
fat of the same chain length. There also is no molecular difference between
a natural and an artificial saturated fat of the same chain length. Configuration
is not an issue because when all of the bonds are filled there is only one configuration
possible. As the length of the fatty acid chain lengthens the melting point
of the fat increases. Thus fats which are solid at room temperature have longer
chain lengths than fats which are liquid at room temperatures. Our bodies can
readily process short and medium chain fats; but, it processes longer chain
fats with greater difficulty.

However, with animal sources, vegetable sources and even with artificially
made dietary sources, single individual fat molecules are never found. We must
always deal with mixtures of many different fat and oil molecules in the fats
and oils that we consume. All naturally occuring fats and oils are mixtures
of long and short chain saturated fats and mixtures of mono and poly unsaturated
fats of the Cis configuration. Naturally occuring trans-isomers are relatively
rare and do not occur in sufficient abundance to create a health hazard. However
if fats and oils are refined, heated or Hydrogenated, the mixtures are then
made to also include a huge thermodynamic distribution of highly toxic isomers,
including the notorious trans-isomer, along with partially destroyed molecular
fragments, and other toxic products.

All fats and oils differ from each other in the length of the Carbon-Hydrogen
chain; however, unsaturated fats and oils also differ from each other, and from
saturated fats, in that they have one or more vacant Hydrogen sites along their
chain.These unfilled Hydrogen binding sites give the unsaturated fats and oils
a variety of geometries at the molecular level. Some of these geometries, notably
the "Cis" geometries that occur naturally in nature and are designed
so that our metabolism can readiy handle them, in fact, it needs them. Certain
Cis type unsaturated oils, the w=3's, directly constitute an important building
block in all of the sixty seven or so trillion cells in our body, and they cannot
be obtained by our body except from our food supply. In addition, our enzyme
systems use unsaturated fats as building blocks to construct a wide variety
of needed biochemicals

Short and medium chain length saturated animal fats are a very nutritious
food staple and have been for thousands of years. They provide nine calories
per gram and are "good keepers"; in the days before refrigeration,
this "keeping" quality was very important. It meant that the fat would
not spoil or go rancid easily at room temperature. Our body uses saturated fats
as a highly concentrated energy source when carbohydrates not plentiful. Much
of the disease we experience today is the result of a failure of our systems
to properly and safely metabolize fats and oils. Rather than use them for the
highly concentrated energy source that they are, our body uses them in cell
repair because the Cis w=3's are not in our diet.This is now identified as a
major factor in Hyperinsulinemia.

Cholesterol is a fatty substance that is manufactured by our liver.
It is an extremely important building block for many of our vital functions
including our brains, eyes, nervous systems and sexual apparatus (both varieties).
About 85% of the Cholesterol circulating in our bodies is made by the liver.
We have a Cholesterol control mechanism in our bodies that operates to stabilize
Cholesterol at the circulating level that we find. Cholesterol is also contained
in some of the foods that we eat. If we try to reduce our circulating Cholesterol
by excluding high Cholesterol foods from our diet, our liver simply makes more
Cholesterol in an attempt to maintain a homeostasis (normal level) of Cholesterol
in our blood stream. Controlling circulating Cholesterol through diet is like
trying to empty the ocean with a teaspoon; it sounds like a good pop science
theory but it is really not very effective.

As we shall see elsewhere in this website and in our special report
Insulin: Our Silent Killer the best way to reduce Cholesterol levels to normal
is to cure the underlying Hyperinsulinemia. This entails repairing the Automatic
Cholesterol Control System which regulates our Cholesterol homeostasis. This
repair process requires stabilizing our blood Insulin and Glucose levels and
restoring our entire endocrine system to proper balance. This follows automatically
when we stop consuming dangerous, damaged fats and oils and restore other needed
nutrition to our diet.

Cholesterol, being a fat, does not disolve in the blood stream which
is mostly water. In order to be transported around in the blood, it must be
carried by a Lipoprotein carrier which has an affinity for water. When it is
being carried from the liver to the rest of the body, the Lipoprotein involved
is LDL (low density Lipoprotein). When Cholesterol is being carried from the
body back to the liver for recycling, the carrier is HDL (high density Lipoprotein).
Thus LDL which distributes Cholesterol throughout the body came to be known
as the "bad" Cholesterol and HDL which removes it from circulation
came to be known as the "good" Cholesterol. Hyperinsulinemia is characterized
by a reduction in the HDL fraction and an increase in the LDL fraction. Clearly
this sort of phony science that characterizes one essential Lipoprotein as "good"
and another as "bad" is the sort that comes from marketing and sales
departments; certainly it does not originate in reputable scientific laboratories.

Besides being a most important building block in many of our bodily
functions, Cholesterol is one of the important components of the plaque that
occludes our arteries. It is for this reason that it has attracted notice. Our
diseased state is due to the fact that the normal levels of circulating Cholesterol
have been elevated by Hyperinsulinemia. In fact, this elevated level of Cholesterol
is often one of the early warning signs that we are becoming Hyperinsulinemic.
An appropriate way to reduce Cholesterol is to cure the underlying Hyperinsulinemia.

With the advent of artificial fats and oils and Hydrogenated and Refined
products in the 1920's (see history), the Cis type w=3 unsaturated oils started
to disappear from our dietary food chain and were replaced by a large number
of toxic isomers. These toxic isomers are just different geometries of the unsaturated
oil molecules many of which were, before processing, of the CIS type. Long term
consumption of some of these toxic isomers, notably the trans-isomer, has been
identified with many, if not most, of the chronic disease symptoms discussed
on this home page. Of even greater importance, the complete removal of some
of the Cis type w=3 oils from our diet has been found to be causal in many of
our widespread degenerative diseases including Hyperinsulinemia.

Some of the biochemical effects of these toxic isomers are discussed
on the diabetes page.

Much of this came about because of standardized refining processes
that were introduced into the oils manufacturing business. The new rapid high
temperature extraction techniques, introduced in the 1920's lowered the retail
price of oil, gave it a pure pristine appearance when packaged in a transparent
bottle, gave it a uniform clarity, gave it an almost uniform taste, and destroyed
the Cis w=3 fatty acids that rapidly spoiled at room temperature. It is the
high temperatures used in the refining process that ruins even previously good
oils. If we find a good oil and refrigerate it, it is still easy to destroy
its nutritional qualities when we cook with it by heating it to the point where
it smokes. When delicate Cis w=3 oils are heated, either in cooking or refining,
the oil undergoes irreversible changes; the Cis configuration is destroyed and
many toxic isomers are generated, including the notorious trans-isomer. All
of the antioxidents, previously a part of the unrefined oil are destroyed. Much
of the oil's original flavor is lost and it tastes like a generic oil.

When cooking with fats and oils it is important to do so in a manner
that does not destroy them. Use only butter, Coconut oil and animal fat for
cooking. These contain a higher proportion of saturated fat and thus are not
destroyed as easily at cooking temperatures. Never consume any deep fried foods;
they are all universally soaked with toxic isomers. If you cook with an oil
like olive oil, be sure to mix some water with it to prevent the oil from getting
too hot. Remember that if the oil starts to smoke it is too hot and it is being
destroyed.

To cure Hyperinsulinemia, Type II Diabetes, Syndrome X and many other
consequential diseases that stem from poisonous fats and oils, it is important
to realize that the chronic ingestion of Refined and Hydrogenated fats and oils
is implicated as a causal agent in these diseases. Margarine, artificial shortenings,
refined oils and all Hydrogenated edible products are long term toxic to the
human metabolism. Any unsaturated fat or oil that does not need constant refrigeration
should be considered unedible. Many saturated fats and oils, while also benefiting
from refrigeration, do not turn rancid nearly so easily as Cis w=3 type unsaturated
fats and oils at room temperature.

An important consideration about these edible oils is a widespread
fraudulent advertising technique that enables the oils manufacturer to sell
known toxic oils to the unsuspecting public without breaking the law. Many refined
vegetable oils are advertised as monounsaturated or as polyunsaturated in order
to confuse the purchaser. Indeed, if these oils were the Cis isomer, they would
be desirable oils from a health standpoint. However, Cis type w=3 oils are inherently
unstable and will go rancid quite rapidly in a transparent bottle on a room
temperature grocery store shelf; their shelf life is on the order of ten hours
or sometimes less. The trans-isomer of these oils has a much longer room temperature
shelf life. There is no law to require the oils manufacturer, or the store,
to advise the consumer that these "monounsaturates" and "polyunsaturates"
are trans-isomers and other toxic byproducts that result from the destruction
of the good edible oils that they think they are getting. Since no law exists
to keep their claims honest, oils manufacturers feel free to deceive with dishonest
claims that few consumers understand. In some circles this is not considered
to be fraud.

In our discussion on Hyperinsulinemia we discuss more about the fat
and oil issue and cover in detail ways we can protect ourselves from the consequences
of the fraudulent advertising claims with which we are constantly bambarded.
We also discuss how to reverse the degenerative process in the event we are
invloved with it.

More information is available in our hardcopy Special Report for those
who have a compelling interest or who simply wish to know more about the health
connection to our dietary fats and oils.

Atherosclerosis is a disease which, through several mechanisms, causes
the arteries to occlude. Arteriosclerosis is a closely related disease with
slightly different causative mechanisms. The occlusion that these diseases cause
effectively reduces the interior diameter of the Artery. Because of this reduced
interior cross section of the Artery, less blood can flow through the Arterial
system at normal blood pressure. The body then either increases the blood pressure
or we find ourselves frequently "out of breath" as we try to "make
do" with a lessened oxygen transport around the body. Eventually, if the
Coronary Artery is involved, as it often is, insufficient oxygen delivery to
the Heart muscle results in Heart Failure. If the Renal Artery is involved insufficient
blood supply to the Kidneys can result in Kidney Failure.

The material that accumulates on the interior walls of the Artery is
a plaque-like substance that typically has a calcium substrate that holds it
in place. Among the mechanisms thought to be causative in the development of
plaque in the Arteries are: Insulin damage to Arterial Endothelium that causes
the bodies Arterial repair mechanism to patch the damage with plaque, stimulation
of uncontrolled replication of Cytomegalovirus infected Arterial Intima cells
by Insulin like growth factor and deposits of Cholesterol on a Calcium substrate
in the Artery.

An elevated Insulin level, Hyperinsulinemia, is directly implicated
in the damage done to the Arterial Endothelium that stimulates the repair response;
it is also responsible for the stimulation of Intima replication and in the
depressing of HDL and the elevation of LDL. This change in the HDL/LDL ratio
results in higher than normal Serum Cholesterol levels. Some of this excess
Cholesterol is thought to constitute the construction material for Arterial
Plaque. To cure the Atherosclerosis requires the removal of the plaque from
all of the Arteries. To prevent a return of the Atherosclerosis it is necessary
to cure the Hyperinsulinemia that is a precipating cause of it. Modern orthodox
medical practice does neither.

Conventional orthodox treatment for Atherosclerosis includes Arterial
replacement, often referred to as "bypass surgery", and balloon angioplasty.
Neither treatment addresses the cause of the disease. Neither treatment fixes
the rest of our Arteries which are also occluded, possibly to a less than an
immediate life threatening extent. Both treatments are expensive, invasive and
very dangerous. Some patients die on the operating table. There are no scientific
studies to support the use of these techniques. In addition, only the Artery,
or Arteries, that causes the most life threatening symptom is treated. After
treatment, the disease in all of our remaing untreated Arteries will continue
to progress; often it will continue at at an accelerated rate. These treatments
are time consuming, very expensive and very dangerous. The few times any of
these treatments have ever been subjected to double blind studies to assess
their usefulness they failed to demonstrate any improvement in the long term
survival rate at all. See the article on Heart failure.

Successful unorthodox treatment must include reversal of the damage
done to the Arteries as well as a reversal of the plaque forming mechanism that
caused the problem in the first place. Chelation therapy has been used very
successfully for about forty years to remove the plaque that has accumulated
in the Arteries, all of the Arteries simultaneously. It is fast, efficient and
economic in approximately 80% of the cases; also, risk is virtually non-existent.
Detailed information about Chelation therapy can be obtained from our reference
#5 listed below. Once the Arterial plaque is removed by Chelation, Hyperinsulinemia,
the systemic disease characterized by the high Insulin levels, must be cured
or the Arteries will simply begin the occlusion process all over again. See
our page on Hyperinsulinemia for more information on this killer; included there
is information on how to avoid it and on how to cure it if you haven't successfully
avoided it.

It is important to note that Atherosclerosis reduces, sometimes greatly,
the delivery of oxygen and other vital nutrients to the sixty seven trillion
or so cells that comprise our body. For this reason many diseases of impaired
circulation, some of them potentially fatal, are a direct result of Atherosclerosis.

For those who are interested in the source data from which these conclusions
are drawn, a starter list of references is included below and much more information
is available in our Special Report. There are hundreds of excellent studies
available to support the conclusions found in this web page.

Technically termed Renal Nephropathy, Kidney Disease is an inevitable
result of the chronically uncontrolled blood Glucose and chronic high Insulin
levels that are associated with Hyperinsulinemia and Type 2 diabetes.

The Kidneys are system filters whose purpose is to filter out the water
soluble waste materials in the blood, to mix them with an appropriate amount
of water to form urine and to send this mixture to the bladder for excretion
from the body. They do this in a way that conserves blood components that may
be needed for future use. Thus the Kidneys return non-waste blood components
to the blood stream to maintain proper blood Homeostasis, they control the blood
PH to within very tight limits (7.35-7.45), they control the water Electrolyte
blood balance and they, through the action of Renin-Angiotensis mechanism and
the excretion of water from the system, play an important role in the operation
of the Blood Pressure Control System (BPCS). This BPCS is another of the bodies'
control systems similar to, but not directly related to, the Blood Sugar Control
System.

In structure the Kidneys consist largely of many fine capillaries through
which the blood flows during the filtration process. A number of complex osmotic
reactions occur during the passage of blood through the Kidneys that are driven
by the Electrolyte balance in the blood and by the osmatic pressure differentials
caused by the Sodium Potassium balance in the blood stream.

When Renal Failure occurs, Dialysis is required or death will ensue
in short order, typically 50% within six months and most by one year. Dialysis
is a procedure whereby the blood is externally filtered through a machine especially
designed for the purpose. When this procedure is done periodically the blood
stream is filtered of impurities. By the use of this method life may be prolonged
after Kidney Failure is experienced.

There are three major damage mechanisms by which progressive deterioration
of the Kidneys occurs.

The first is by direct damage of the fine capilaries by the high Glucose
levels associated with Diabetes and with Hyperinsulinemia. The glucose directly
causes an increased permeability and the capillaries leak and fail to perform
their filtering action. Severe and extensive damage to these fine capilliaries
is thought to be irreversable at this time.

A leading cause of Renal Failure for the Hyperinsulinemic patient is
Renal Thrombosis (blood clotting in the Renal vein).

The high levels of Insulin in the blood directly cause Atherosclerosis
of the Renal artery. This mechanism is discussed on our Atherosclerosis page.

A related complicating factor is caused by the high levels of dietary
protein to which many sufferers from Diabetes and Hyperinsulinemia resort. This
protein, when it is metabolized, results in high levels of Ammonia . Chronic
high levels of Ammonia, mostly changed into Urea by the Liver, nevertheless
directly damage the fine capillaries. Also there are known other negative effects
in the tubular basement membranes of the Kidneys that are directly caused by
certain proteins.

Unless normal blood Glucose levels can be maintained without resorting
to a high protein diet, the progress of Hyperinsulinemia and its related Type
II diabetes invariably leads to Renal (Kidney) failure and the need for Dialysis.
In our special report Insulin: Our Silent Killer we thoroughly discuss the dietary
factors that are especially important to the diabetic. This High Protein-Kidney
Damage relationship is potentially one of the most important to the recovering
diabetic. During the recovery process, which may last for several months, it
isn't smart to wreck the Kidneys by resorting to a high protein diet. This is
uniquely important because, while much of the damage done by Type II diabetes
and Hyperinsulinemia is reversable, all of it is not. In particular when the
basement membranes of the Kidneys are destroyed, the destruction is currently
thought to be irreversable.

High blood pressure, Hypertension, which is often found in progressive
Renal Failure, is also one of the characteristics of Hyperinsulinemia. If there
is any suspicion that Hyperinsulinemia or Type II diabetes, may exist, one should
have a blood test done and obtain medical advice based on the BUN and Creatine
levels found in the test with regard to possible Kidney damage.

On the next page where we discuss Hyperinsulinemia, we discuss ways
in which this disease can be readily prevented from progressing. We include
below a starter list of references for those who want to look at the original
data. Much more information about Hyperinsulinemia is available in our Special
Report than can conveniently be put on this web site.

The Liver is a huge chemical factory that manufactures basic biochemicals
that are used by our entire body. It is central to our digestion of fats, to
metabolism, to detoxification and to many other vital processes. When it fails,
death will typically occur within 24 hours.

Detoxification, one of the many vital functions of the Liver, is performed
through multiple chemical pathways. These pathways chemically alter toxic materials
into harmless substances that are water or fat soluble. The water soluble end
products are sent to the Kidneys for excretion in the Urine. The fat soluble
end products are sent to the small intestine for subsequent excretion.

Virtually all synthetic drugs, as well as many natural drugs and compounds,
are regarded by the Liver as toxic. For this reason many, if not most, prescriptions
for synthetic drugs call for up to 50 times the dosage needed to combat the
symptom treated. The assumption is made that sufficient drug must be introduced
into the system to overcome the detoxification activity of the Liver.

This is one of the reasons why most, if not all, modern synthetic drugs
have one or more undesired side effects. Many of the Hypoglycemic agents (agents
that lower blood sugar) used to treat Type II diabetes and Hyperinsulinemia
are known to cause liver damage. There are so many of these agents, with more
appearing every day, in an exploding drug marketplace, that it isn't possible
to list and discuss them all on this web site. Some of the toxic effects of
these drugs affect other organs of the body in addition to the liver. An increasingly
common side effect is that they kill. In our Special Report we thoroughly discuss
the common side effects of all of the current classifications of Hypoglycemic
agents.

When you contemplate taking a prescripton drug it is very important
to understand the consequences of these drugs. You can visit the library to
look up the drug in the "Physicians Desk Reference" (PDR). Or, alternatively,
if you have our Special Report you can quickly look up the risk factors involved
with the medication you're taking in the appropriate section of the report.
This will eneble you to determine the nature of its side effects. If you are
already experiencing any of these side effects it is time to have a serious
talk with your physician. We cite here side effects for a few of the commonly
prescribed agents used to control blood sugar in Hyperinsulinemic and Type II
diabetic patients so that you can get the idea.

The Sulfonylureas are a class of drugs that include Orinase, Tolinase,
Diabinase and others. These drugs have been implicated in greatly increased
deaths from Heart Disease and Hepatitis, specifically Granulomatus Hepatitis.

Glyburide, another Hypoglycemic agent has been reportedly associated
with Cholestatic Jaundice, with Hepatitis and with Hypersensitivity Angitis;
all of these are very serious Liver diseases.

Rezulin is an oral Hypoglycemic agent that has come to be associated
with Liver Cancer. It was believed to be a potent Cancer producer by the original
FDA investigating officer who refused to approve it for American use. He was
administratively removed from the case and replaced by another who quickly approved
it. After approval it killed an estimated 100 people before recently being finally
removed from the American market by our FDA. It had previously failed to gain
regulatory approval with England's equivalent of our FDA. This matter is currently
headed for the courts.

In addition to drug induced Liver damage, evidence is slowly accumulating
to suggest that the Hyperinsulinemia itself may cause Liver damage. High levels
of Triglycerides in the blood stream are a known result produced by the development
of Hyperinsulinemia and Hyperglycemia (Type II diabetes). When these Triglyceride
levels become chronic, they induce a condition known as Hepatic Steatosis (fatty
liver). In this condition, the Liver becomes infiltrated with Triglycerides
and much of its normal function is impaired.

In our section on Hyperinsulinemia we present effective ways to deal
with this disease. Below we include a starter list of references for those who
wish to do further investigation. In addition to the contents of this web page,
which have been designed to include the essential information needed, more information
is available in our Special Report on this disease.

Although Impotence is a disease that has a variety of causal agents,
some of them not even medical in nature, Hyperinsulinemia is directly implicated
in Impotence through at least three biochemical pathways. These pathways are
Neuropathy of the affected nerves and ganglia, Atherosclerosis and Vascular
insufficiency. All three of these pathways are characteristic of Hyperinsulinemia
and the diabetes that often occurs along with it.

Some studies show that up to 35% of males who suffer from diabetes
are also functionally Impotent. Of course, Impotence along with its causal agent
Hyperinsulinemia, is readily reversible in the vast majority of cases, without
the need for synthetic drugs of any kind. The 35% figure undoubtedly provides
much of the economic incentive for the recent introduction of the drug Viagra.

Impotence is often the result of the Diabetic Neuropathy that develops
as a result of high uncontrolled Glucose levels. Some of the parasympathetic
nerves involved include the Pacinian Corpuscles of the Penis, the Pudential
nerve that passes to the Dorsal Root Ganglia, the Perivesicular, Prostatic and
Cavernous Plexes and the Postganglion fibers that innervate the smooth muscles
of the Vas Deferens, Seminal Vesicle and the Internal Sphincter of the Bladder.
Any or all of these nerves can and do fail to operate properly when subject
to Neuropathic damage from high Glucose levels. See our page on Neuropathy for
more details.

The erectile mechanism works by increasing blood flow through the right
and left Internal Pudendal Arteries to the right and left Corpora Cavernosa
and by decreasing the Venous blood outflow. The high Insulin levels associated
with Hyperinsulinemia cause Arterial occlusion and result in an insufficient
blood supply to the Corpus Cavernosa. The high Glucose levels associated with
this disease results in increased Venous permeability; thus the blood outflow
from the organ is not sufficiently restricted. See our page on Vascular disease.

Viagra, a popular new synthetic drug, has been introduced by Pfizer
for the specific purpose of improving erectile response in Impotent males. While
it is developing a track record of working in some, but not all cases, there
are some precautions that should be taken and some risks that should be weighed
before embarking upon a program of using this drug. Viagra is known to increase
the Hypotensive effects of Organic Nitrates; if you are taking Organic Nitrates
in any form it is not smart to use Viagra. Also a serious list of side effects
seems to be characteristic of this drug. Some of the most important of them,
together with their relative frequency of occurance are:

Heart failure
Angina pectoris
Hypotension
Face Edema
Allergic Reaction
Vomiting
Colitis
Gastroenteritis
Rectal Hemorage
Diabetes
Arthritis
and many others
When these side effects were listed in the PDR (Physicians Desk Reference) it
was stated, by the manufacturer, that the causal relationship between Viagra
and these adverse reactions has not been clearly established; these reactions
simply occurred to volunteers involved in the clinical tests. About 3700 were
involved to some extent in the tests with 550 involved for more than a year.

For those whose Atherosclerosis involves the Carotid or Coronary arteries,
as it often does, the use of Viagra has the potential of precipitating a fatal
Heart Failure event or stroke; and as a result, the drug should not be used
by these patients.

By far the best and safest approach to the problem of Hyperinsulinemic
caused Impotence is to simply cure the Hyperinsulinemia. This will swiftly arrest
the progress of the Impotence. To restore proper functioning in a wholly natural
and normal manner it is necessary to clean out the clogged Arteries. We introduce
this idea on our page Atherosclerosis. It is thoroughly discussed in our Special
Report; see our more information page.

Although Hyperinsulinemia is not the only cause of impotence, there
are many others, it is a major cause of this malady.

We provide below a starter list of references. A much more extensive
Reference list, together with a Bibliography for further research, can be found
in our Special Report, Insulin: Our Silent Killer.To reverse impotence safely,
it is necessary to reduce the elevated levels of blood Glucose and Insulin that
often are the causative agents and to restore proper operation of our automatic
Blood Sugar Control System. Much more information on this subject is also included
in our Special Report.

Stroke is defined as the sudden rupture or occlusion by clotting of
a blood vessel to or within the brain. Symptoms depend greatly on which blood
vessel is affected.. They can range from minor dizziness or momentary temporary
loss of function that is barely perceptible to paralysis or death.

Hyperinsulinemia and the high blood Glucose levels that come with it
produce two effects that are responsible for inducing Strokes. These effects
are: (1) increased thickening of the blood and increased stickiness which tends
to clot and block the blood vessels and, (2) the increased permeability of the
microvascular system that cause the smaller blood vessels and capilliaries to
leak and rupture.

Ischemic strokes are produced by blood clots that may occur at the
site or may occur because of a clot that, having been formed elsewhere, was
carried to the site by blood flow. When blood becomes thicker and more sticky
this type of stroke becomes more likely. The high levels of Glucose and the
high levels of Triglycerides that characterize both Type II diabetes and Hyperinsulinemia
make the blood sticky, viscous and prone to clotting. This is why these diseases
are often associated with Ischemic Stroke.

Hemorrhagic strokes are caused by rupture or severe leaking of the
blood vessels. This is usually caused by Glucose induced damage to the blood
vessel, particularly when small Veins or Capilaries are involved. Chronic elevated
levels of blood Glucose are usually implicated in Hemorrhagic stroke.

The cause of the blood changes that induce both types of Stroke are
found to be characteristic of the Hyperinsulinemia syndrome. Below we include
a starter list of basic references for those so inclined. More information along
with extensive scientific cites and source references are available in our Special
Report. This report, Insulin: Our Silent Killer discusses much more fully the
Hyperinsulinemia that is so often causally implicated in the incidence of stroke.

When the study for this web site started some years ago, we assumed
that the problem of Obesity, as an issue in Hyperinsulinemia and Type II diabetes,
was one that was relatively well understood and thoroughly researched. We were
surprised to discover that less is known about the weight control mechanism,
the hunger reflex and their relationship to Hyperinsulinemia than is known about
vitually any other aspect of this dangerous disease.

During our research we found many competing theories with various credibility
levels. The best of them, though incomplete, made sense and came from some of
the most reputable research labs around the world; the worst of them made no
sense at all and typically originated in the sales departments of the diet and
weight loss industry.

We present here that which we think makes the most sense and which
will likely remain as a basic fundamental to a full understanding of Obesity,
if and when it ever becomes available.

With Hyperinsulinemia, the excess average Insulin levels experienced
cause an increase of Lipogenesis. Lipogenesis is the Insulin mediated conversion
of Glucose into Triglycerides (fats). The resulting high Serum Triglycerides
are stored in adipose cells throughout the body; these adipose (fat) cells become
distended and as they distend, more Insulin is required to enable them to carry
on their metabolic processes. There is but a finite, fixed number of fat cells
available in each body. This number is changed only with difficulty as we mature.
The excess Triglyceride load that they absorb in Obesity simply makes each adipose
cell larger.

As these fat cells absorb the excess Triglyceride load, they emit a
biochemical into the blood stream called Leptin. This is a biochemical that
causes a reduction in the amount of a Neural Polypeptide called NPY (neural
polypeptide Y) which is produced in the Hypothalamus. The Hypothalamus is the
part of our brain known to mastermind many of the bodies regulatory mechanisms.
The body weight seems to depend upon the ratio of Leptin, emitted by the fat
cells, and NPY produced by the Hypothalamus. When there is too much NPY, and
possibly other more obscure Neural Peptides, Glucose transfer into the cells
is inhibited, Lipogenesis is stimulated and Triglycerides are stored in fat
cells. When there is too much Leptin, fat storage is supposed to be inhibited.

This same Hypothalamus also masterminds the control of the Appestat
(hunger regulator). The role of NPY and Leptin, if any, in the hunger reflex
is not well understood yet. But, clearly there is a connection because the only
way to provide the additional Triglyceride load for the fat cells to store is
to stimulate the hunger reflex to require more eating of carbohydrates and of
fats and oils.

In the case of Obesity, the NPY and Leptin levels remain elevated and
it appears that the appropriate Leptin receptors in the Hypothalamus are desensitized.
The result is that the Hypothalamus continues to insist upon Triglyceride storage
in the adipose cells, by emitting NPY, and it continues to maintain a hunger
reflex beyond any reasonable need to do so. We get fat.

One theory, which has some epidemiological support, may be helpful.
It says that because the food we eat is so empty of needed nutrients, our hunger
reflex remains activated and the role of Leptin remains suppressed. This is
done in the body's expectation that, if we continue to eat, eventually the needed
nutrient will magically appear in the stomach. The assumption here is that when
the needed nutrient appears, the Leptin-NPY axis will again function and we
will lose weight. This theory may be fruitful in the effort to more fully understand
the connection between the Hypothalamus, which is widely considered to be a
lower center control mechanism, and the higher centers of our brain where our
intelligence resides.

At any event, while the connection between Obesity and Hyperinsulinemia,
is not yet fully understood, much more is known about the cause, progress and
cure of Hyperinsulinemia and Type II diabetes.

For those who would like to pursue the study of Obesity beyond these
meager beginnings we include a starter reference list. Judicious use of it can
quickly get you into the mainstream of what's going on worldwide in this area.
More data is included in our Special Report on the Hyperinsulinemia that is
so often causal in our Obesity and weight control issues.

Neuropathy is a generic medical term for a group of diseases of the
nerve. They affect both motor and sensory nerves with varying results. Diabetic
Peripheral Neuropathy is a term used to describe damage done to the nerves by
high Glucose levels in the blood stream. Generally this high Glucose level causes
a deterioration of the Mylin Sheath surrounding the central nerve and leads
to its degeneration.

When the affected nerve is a motor nerve, progressive loss of ability
to stimulate the related muscle structures causes loss of function.

When the affected nerve is a sensory nerve, two different results may
occur. Often only the sensory function is impaired and a gradual deterioration
in feeling and sensation occur. Sometimes, when the Mylin Sheath becomes damaged,
intense pain results which necessitates the use of narcotic pain killers.

When sensation is essential to function, as for example in the male
erectile response, function also becomes impaired. See our page on Impotence.

Sometimes when the damage done to the nerve causes loss of function,
as with a Heart muscle, the consequences can result in Heart Failure and death.

All parts of our complex nervous system can be affected by high average
Glucose levels; this includes the Central Nervous System, the Voluntary Nervous
System and both the Sympathetic and Parasympathetic elements of our Involuntary
Nervous System.

When the Blood Sugar Control System is repaired and again functions
properly, Neuropathy, along with many other symptoms of impaired Glucose control
will reverse. Nerves will regenerate. They will regenerate faster when supplied
with the unique nutrients needed to repair cellular and plasma membrane damage.

This Neuropathy is one of several extremely dangerous side effects
of a Blood Sugar Control System that does not function to maintain normal levels
of both Glucose and Insulin in our bodies. Neuropathy is a normal consequence
of Hyperinsulinemia. Below we include a starter list of references for those
who like to study the original material. Much more information is included in
our Special Report which also offers a methodology that has a track record of
reversing the underlying cause of this disease.

More information
Hyperinsulinemia is an explosive epidemic that currently affects half of us
to a greater or lessor degree. In the middle 1920's this disease, then called
Diabetes, was so rare that many doctors never saw it. Some ethnic communities,
notably the Chinese, had never had a single case reported. Others, like the
American Eskimo, who subsisted on a diet of about 60% saturated fat, had no
incidence of this disease at all. Today, half the American population suffers
one or more symptoms of this disease.

It is caused by nutritionless food. This food is sold to us for the
purpose of making a profit and generating growth in the competitive companies
that constitute the processed food industry. Our food lacks essential nutrition
because the nutritious substances in it have a very short shelf life. Since
this abreviated shelf life is incompatible with modern food merchandising technology,
the nutrition is modified or removed. The food is then sold as "fortified",
or as "new and improved". The direct result of this lack of nutrition
never affects the sale of the food because these consequences are delayed by
months or years. By that time the customer (victim?) has either forgotten the
bad food or been trained to believe that it is innocuous.

All of us, without exception, have been programmed by the intense advertising
and marketing promotions to which we are subjected almost from birth. Much of
this stuff is designed to get into our subconscious by bypassing our normal
conscious censor mechanisms. Once there, these implanted perceptions take on
an emotional value that makes us want to defend them as if they were our own
ideas. While there might once have been some legitimate rationale for merchandizing
to sell products, it has become dangerously ubiquitous, intense, deceptive and
effective. Subliminal advertising practices are widespread in the selling of
food products to America. Without constant, informed effort to maintain our
objectivity, we easily fall prey to deeply held beliefs placed in us without
our consent or knowledge.

Which of us doubts that our food industry really has maintaining our
health as their highest priority instead of maximizing their corporate bottom
line. Yet this semireligious belief is ridiculous on the face of it. Who among
us doubts that the medicines routinely sold to the public have been carefully
and honestly cleared by government regulatory agencies. We have no doubts even
when our newspapers are full of articles about the death dealing side effects
of these drugs. Why do we continue to believe that our regulatory agencies act
without being influenced by commercial interests even when we read horror stories
like the approval of Aspartame. Why, after drugs like "Rezulin", formulated
to treat this disease, has killed well over one hundred trusting people, do
we continue to trust the integrity of the drug manufacturer who continued to
sell it? Even after knowing that our regulatory agencies permitted Rezulin to
remain on the market to kill more long after the lethal nature of the drug was
widely reported, how many of us really suspect that anything is wrong? How many
of us abandon this nonsense and seek out an alternative non-lethal treatment?
It is only when we become directly and personally impacted, as was this writer,
that we begin to study carefully what is really going on in this commercial
arena.

Such is the power of the subliminal advertising to which we are all
exposed every day.

Perhaps some insight may be gained into the scope of the health disaster
in which we are involved by noting the the largest processed food manufacturer
in the world is Kraft Foods. Kraft Foods is owned by Phillip Morris; yes that's
right, its the same Phillip Morris that gave us the tobacco scandals of the
late 1990's.

Although we have been careful to supply the needed information that
can help to completely reverse Hyperinsulinemia and Type II diabetes in this
website, we are aware that some of the information contained here is new for
many of us. Sometimes with new information, especially when it may tend to contradict
previously held beliefs, more support and evidence is required to bring the
new data within our comfort zone.

We have prepared a 147 page Special Report entitled "Insulin:
Our Silent Killer" for that half of our population that is either affected
by or involved with this disease. This report is written in layman's language
intended to be readily understood by anyone without special training in medicine.
It includes a Glossary that carefully defines necessary medical terms. It includes
an extensive list of scientific cites and references of the source data. It
contains a Bibliography for further reading. It contains a Glycemic Index for
those who daily face the consequences of faulty fat and carbohydrate metabolism
but who still have to eat.

The report Insulin: Our Silent Killer provides an easily understood,
workable alternative protocol for reversing this disease quickly, completely,
permanently, naturally and economically without dangerous synthetic drugs in
the vast majority of cases. This Special Report also discusses ways to reverse
much of the collateral damage that unchecked proliferation of this disease causes
in the body. This Special Report also provides effective non-drug therapies
that are needed to control Blood Glucose during the time that the disease is
being reversed but before our Automatic Blood Sugar Control System (BSCS) is
again fully functional. All three elements of this approach are equally vital
to curing this disease.

Insulin: Our Silent Killer relates the experiences I had when I was
faced with this killer disease after I discovered that doctors treat but do
not cure it. Most important, this Special Report contains a full detailed explanation
of just where this disease comes from, what causes it, why it is not being routinely
cured, a discussion of the economics of the disease, a discussion of the consequences
of orthodox medical treatment, and of the reasons why the disease has become
a national disgrace.

Our investigation and our report Insulin: Our Silent Killer has been
featured at length in a very favorable book report in the spring, 1999 edition
of Tom Valentine's "True Health" newsletter (Carotec, Inc., PO Box
9919, Naples, FL 34101 (941) 353-2348) and was given high marks by David Lawrence
Dewey, nationally syndicated columnist, in his popular on line health advisory
column:

http://www.dldewey.com

and, we are currently the subject of favorable articles in other leading
health oriented magazines. We are beginning to be involved with the exposing
of this disgraceful epidemic throughout the media.

More recently, some of the research presented in this report was featured
on the Good Morning America TV show as an investigative expose' of a major cause
of this stealth disease epidemic. We are being very well received on nation
wide talk radio everwhere we have been invited. This area of our work is expanding
on its own momentum largely because of the real need for accurate information
by those who are stricken by this disease.

For those who would like additional investigative leads we are preparing
a links list to facilitate searching the world wide web.

To Order This Special Report

Within the US this report, Insulin:Our Silent
Killer, can be ordered by sending $29.00 US, (we pay all postage and handling),
together with your address to:

Thomas Smith
PO Box 7685
Loveland, CO 80537
If you are not a US resident Email us for the special shipping and payment instructions
required for foreign transactions.
Or you can email us at: Valley@healingmatters.com

Or you can call us at: 1 (970) 669-9176. Sorry, we are not set up to
process credit card transactions.

Links
This page is under construction as we search and locate other websites that
contain useful additional information regarding the disease syndrome discussed
at this site and/or related issues.. As we find and check out quality links
we will post them on this page.

David Lawrence Dewey's column:http://www.dldewey.com

David is a best selling author and syndicated columnist that has a
wealth of good information about the disease epidemic from which we all suffer.
He often writes with wit and insight on health related issues the are of importance
to us all.

This is a nursing power site. It about, for and by the nursing profession
with numerous articles and discussions relative to the technology, art, challenges
and issues of the medical business in general and the nursing profession in
particular. An interesting and useful website maintained in a professional manner.

Shirley's wellness cafe

This is a large well researched site with an abundance of valuable
health information on many topics. The material is well organized and presented
in an easy to assimilate fashion. We link to topics of great interest to our
study of this endocrine disorder with so many aliases. They are: