Detoxing Dead Cancer Cells

Methylation Pathways are Essential

Liver detox pathways are essential to detox poisons as well as cancer cells killed by any method. The methylation pathway is one detox system that is dependent on dietary folate that needs to be either converted to MTHF (methylenetetrahydrofolate) or better, taken orally as MTHF.

There has been a lot of press on the link between MTHFR (methylenetetrahydrofolate reductase) gene mutations and chronic disease. This has resulted in a great deal of concern with the particular form of folate that doctors are giving to their patients.

According to a recent study in the American Journal of Clinical Nutrition, the human gut appears to have a very efficient capacity to convert reduced dietary folates to 5-MTHF, but only a limited ability to reduce folic acid (synthetic folate). The risk occurs when there is unmetabolized folic acid present. Unlike natural folates (5-MTHF and 5-formyltetrahydrofolate), folic acid is not a normal metabolite and must be reduced in order to enter the folate cycle. It has been found that chronic liver exposure to folic acid in humans may actually induce saturation. This could very well be the reason why systemic circulation of unmetabolized folic acid has been reported. In addition, there is a growing body of evidence of folic acid supplementation playing a role in promoting the growth and development of certain cancers.
Source: Folic acid handling by the human gut: implications for food fortification and supplementation. Am J Clin Nutr 2014 ajcn.080507; June 18, 2014.

Methylation Pathways are Essential

Liver detox pathways are essential to detox poisons as well as cancer cells killed by any method. The methylation pathway is one detox system that is dependant on dietary folate that needs to be either converted to MTHF (methylenetetrahydrofolate) or better, taken orally as MTHF.

There has been a lot of press on the link between MTHFR (methylenetetrahydrofolate reductase) gene mutations and chronic disease. This has resulted in a great deal of concern with the particular form of folate that doctors are giving to their patients.

According to a recent study in the American Journal of Clinical Nutrition, the human gut appears to have a very efficient capacity to convert reduced dietary folates to 5-MTHF, but only a limited ability to reduce folic acid (synthetic folate). The risk occurs when there is unmetabolized folic acid present. Unlike natural folates (5-MTHF and 5-formyltetrahydrofolate), folic acid is not a normal metabolite and must be reduced in order to enter the folate cycle. It has been found that chronic liver exposure to folic acid in humans may actually induce saturation. This could very well be the reason why systemic circulation of unmetabolized folic acid has been reported. In addition, there is a growing body of evidence of folic acid supplementation playing a role in promoting the growth and development of certain cancers.
Source: Folic acid handling by the human gut: implications for food fortification and supplementation. Am J Clin Nutr 2014 ajcn.080507; June 18, 2014.

Conners Clinic is located on 8519 Eagle Point Blvd #170, Lake Elmo. From Minneapolis−Saint Paul International Airport (MSP) head southwest and continue onto Green Ln. Then keep right and slight right onto Glumack Dr. Next, keep right to stay on Glumack Dr and keep right at the fork and merge onto MN-5 W. After that merge onto MN-5 W and take the exit onto I-494 E. Take exit 58B to merge onto I-94 E toward Madison and take exit 250 for Radio Dr/Inwood Ave toward Washington County 13. Then use the left 2 lanes to turn left onto Inwood Ave N/Radio Dr. Finally, turn right onto Eagle Point Blvd, turn left at Eagle Point Cir and Conners Clinic will be on your right.

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“Cancer is a scary thing. I’m here to remove the confusion and give you guidance. Cancer patients are rarely told that adding natural, alternative therapies to their care can greatly mitigate their adverse reactions, improve the quality of their life, and give them their life back!” –Dr Kevin Conners

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