'''[[User:Tanya_E_Raymond|Tanya Raymond]]''' ATM is able to phosphorylate H2AX, but so is ATR and DNA-PK. [1] Do you have any speculation as to why, upon DNA-PK inhibition, there was only diminished phosphorylation signal in the presence of MDC1 tethering?

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'''[[User:Tanya_E_Raymond|Tanya Raymond]]''' ATM is able to phosphorylate H2AX, but so is ATR and DNA-PK. [1] Can you speculate as to why, upon DNA-PK inhibition, there was only diminished phosphorylation signal in the presence of MDC1 tethering?

Revision as of 13:18, 31 January 2013

Tanya Raymond ATM is able to phosphorylate H2AX, but so is ATR and DNA-PK. [1] Can you speculate as to why, upon DNA-PK inhibition, there was only diminished phosphorylation signal in the presence of MDC1 tethering?