Speciation is an ongoing process, it’s part of evolution, also an ongoing force. As members of a sub-species, better known as race, continue to diverge over time, the characteristic event will be infertility, fertility issues, birth defects and miscarriage. Once it is born, a failure to thrive and reproduce itself would also count as an adverse selection pressure.

My simple question: do we see this?

Oh, boy. Grab a drink, tall one.

The mixed-race dating pool is limited, to the other mixed-race, for example.
This lowers the potential fitness of the organism, compared to its parents’ baseline.

I’ll take a biomedical approach, from the limited information available.

Asians have a lower median birth weight, a racial difference as real as shorter African gestation periods compared to Whites.

“Although past studies have looked at ethnic differences in perinatal outcomes, the majority of research has focused on white- African-American couples. Few studies have focused specifically on Asian-white couples, said El-Sayed, who is also associate chief of maternal-fetal medicine.
…
More specifically, the researchers found that white mother/Asian father couples had the lowest rate (23 percent) of caesarean delivery, while Asian mother/white father couples had the highest rate (33.2 percent). Because birth weights between these two groups were similar, the researchers say the findings suggest that the average Asian woman’s pelvis may be smaller than the average white woman’s and less able to accommodate babies of a certain size.”

“El-Sayed and his colleagues also found that the incidence of gestational diabetes was lowest among white couples at 1.61 percent and highest among Asian couples at 5.73 percent – and just under 4 percent for Asian-white couples. These findings weren’t altogether surprising: past studies have shown an increased risk of diabetes among Asian couples, which researchers attribute to an underlying genetic predisposition. But the interesting finding, El-Sayed said, was that the risk for interracial couples was about the same regardless of which parent was Asian.”

Dominant genes? No!

“Because of the results on Caesarean section rates they adduce that there is a pelvic size difference between Asian women and white women. Objective male observer acquaintances of mine have generally tended to back up this phenotypic difference between the populations.”

They’re shaped like pre-pubescent boys. Why else get surgery?
You should study it formally though. Asians have the lowest sexual dimorphism and it’s important to know the numbers.

“Although births of multiracial and multiethnic infants are becoming more common in the United States, little is known about birth outcomes and risks for adverse events. We evaluated risk of fetal death for mixed race couples compared with same race couples and examined the role of prematurity and low birth weight as potential mediating risk factors.”

Miscegenation doesn’t work, even with modern medicine.
This applies to black-white pairings too.

It is a disgrace adults are marrying without knowledge of the biology involved.

We have anecdotes?https://www.temptasian.com/fyooz/after-3-miscarriages-the-zuckerbergs-are-finally-expecting-a-girl/
“Most people don’t discuss miscarriages because you worry your problems will distance you or reflect upon you — as if you’re defective or did something to cause this.” Mate choice is something you did. The baby didn’t choose to be conceived by you two. Part of your biology must be defective because miscarriage is an outcome of defective conception and/or pregnancy (there are many possible reasons, some environmental, a few random plus ‘stress’). It sounds cruel but yes, medically, something is wrong.

When trying really hard, the only evidence for hybrid vigour in White Americans vs. mulattos, which they sought to prove (scientism) is “relatively small.” …Is it present or not?https://www.nature.com/articles/nature14618
“this study provides evidence [DS: the evidence isn’t proof?] that increased stature and cognitive function have been positively selected in human evolution, whereas many important risk factors for late-onset complex diseases may not have been.”
That’s bullshit, everyone is getting taller and getting better grades.
May not have been? In Nature?
Listen to the twisting in this: http://www.medicaldaily.com/g00/interracial-couples-may-make-taller-smarter-children-due-greater-genetic-diversity-341348“Meanwhile, human evolution is more focused on the ability to create healthy offspring and have them survive infancy to continue raising them.”Yes.

…Yes, it is.
“Whether you come from a genetically diverse background or not, in the end even the most common medical ailments that affect society will affect everyone, with genetic diversity having little to no impact.”
No, genes. The most common fatal medical ailments aren’t a cold, they’re genetic-based, it’s established fact. And if it had no impact, why push it?
“It combines the parents’ genetic material, resulting in offspring that possess a unique set of genetic blueprints that increase their chances of surviving and thriving compared to a population with limited genetic variability.”
No such thing. Limited genetic variability? No such thing. Where is this thing?
They’re just talking absolute crap to cover how their study was a non-result. Every genome is unique, between twins even. Thriving and surviving varies by individual genome, that should be studied by the natal people. You know this. You hide the scant data that is there with delusions. This is propaganda. It continues:
“This encapsulates Charles Darwin’s theory of natural selection,”
No, he wrote a whole book. Look at the subtitle to The Origin of the Species.
Natural selection is about death and mortality, which you have not studied. Disease is not death.
“where individuals with characteristics that increase their probability of survival”
how? like being able to give birth?
“will have more opportunities to reproduce,”
in a limited dating pool
“according to the University of California, Berkeley’s Understanding Evolution.”
If California understood evolution, it would be Alaska.
“As a result, their offspring will benefit from the variants,”
no, not if they’re the more common disadvantageous mutations or if the combination is novel and fatal
“which will spread throughout the population.”
No, you’re assuming they breed. Infertility exists, and it exists on a spectrum.

“This is an increased risk equivalent to smoking, advanced maternal age or obesity.”

“While other research has found the mother’s ethnicity places a role in the risk of a stillbirth, this has largely been put down to factors related to migration and social disadvantage. What our research shows is women born in South Asia and giving birth in Australia are at increased risk even when other factors are taken into account.”

D.N.A.

“There is growing evidence to suggest a mother’s ethnicity influences how fast her placenta ages as her pregnancy progresses.”

Asian placenta is old, got it.

“For some women, they can go into spontaneous labour sooner. In our study, we found South Asian-born women went into labour a median one week earlier than Australian- or New Zealand-born women.”

Racial differences in gestation duration, again.

“However, for others, an ageing placenta cannot meet the fetus’ increasing metabolic needs at term and beyond. And this increases the risk of stillbirth.”

Infertility, insufficient maternal resources for the fetus. That’s a kind of infertility. Considering how skinny they are and how those female curves are supposed to feed a baby, historically, this is not surprising.
Nature is aborting babies that would starve. Before it kills the mother too.

“And the length of telomeres in placentas from pregnancies ending in stillbirth are two times shorter than those from live births. In other words, the placental cells had aged faster.”

Superior Asian genetics people might wanna cover their innocent eyes.

“Some researchers have also studied ethnic differences in placental telomere length.
In an American study, placental telomeres from pregnancies in black women were significantly shorter than from pregnancies in white women (the ethnic backgrounds of the women were not further defined in the study).”

Superior European placentas. As you’d expect for the one race hit hard by an Ice Age. Perhaps this is an unknown r/K variable.

“Whether telomeres are shorter in placentas from pregnancies in South Asian-born women is unknown.”

“However, BMI does not take into account the relative proportions of fat and lean tissue and cannot distinguish the location of fat distribution”

“However, these are based on information derived from the general population, based on risk of mortality, without consideration for racial or ethnic specificity and were not determined to specifically identify those at risk for diabetes. Recently, the U.S. Centers for Disease Control and Prevention presented initial findings from an oversampling of Asian Americans in the 2011–2012 National Health and Nutrition Examination Survey. These data, utilizing general population criteria for obesity, showed the prevalence of obesity in Asian Americans was only 10.8% compared with 34.9% in all U.S. adults (13). Paradoxically, many studies from Asia, as well as research conducted in several Asian American populations, have shown that diabetes risk has increased remarkably in populations of Asian origin, although in general these populations have a mean BMI significantly lower than defined at-risk BMI levels (14,15). Moreover, U.S. clinicians who care for Asian patients have noticed that many with diabetes do not meet the published criteria for obesity or even overweight.”

“In women, the connection between WHR and health measures appears to be hormonal. It is known that ratios of estrogen, progesterone, and prolactin affect all of these features. The “right” balance promotes both health and low WHR. One version of the “attractiveness theory” posits that our attraction to this body shape developed as an indicator of overall health.”

“Another crucial part of the attractiveness theory of wait-hip-ratio (WHR) is that this body shape has to be indicative of something related to fertility, or else it wouldn’t have any evolutionary value.

The key feature in a potential mate is biological fitness, that is, the potential to give birth to many healthy and successful offspring.

Desirable females, in the evolutionary sense, are those that are likely to be healthy, fertile, and robust.

Robust = pelvis, btw.
Venus was never a narrow-hipped vixen.
The body acceptance people should really focus on the hips.

A low WHR, it is thought, must correlate with fertility (ability to have children) and/or fecundity (tendency to have large numbers of children).”

There is such a thing as too low. Boyish figures have less fat, fewer curves and narrower hips.
They’re confusing women who have obesity and babies for State money with natural attractiveness, fecundity in the state of nature and blurring BMI with WHR. Nobody said unhealthy (low) WHR is wealthy, for fecundity. That’s a strawman. The hormones and other details, medical details, are better profiled in the most nubile WHR range. It is a range. Don’t line graph me, study.

It doesn’t mention race although many women in the world do not have a figure. Unless you count a figure of 1.

“The waist is one of the distinguishing human features, such as speech, making tools and a sense of humour,’ says Professor Singh. ‘No other primate has one. We developed it as a result of another unique feature – standing upright. We needed bigger buttock muscles for walking on two legs.”

If the waist makes the human, a lot of women are fucked.

“The ideal ratio in healthy pre- menopausal women ranges between 0.67 and 0.8. In terms of the tape measure, this is produced by waists between 24in and 28in with 36in hips, and waists between 27in and 31in with 40in hips.”

A range.
…How many Asian women have a 36″ hip?
The fat ones I’ve seen were pufferfish.

“come puberty, the sex hormones start directing it differently.”

sexual dimorphism

“Oestrogen, the hormone of female sexual characteristics, concentrates it on the buttocks and hips while the masculinising hormone testosterone encourages fat to form around the waist.’ At the same time testosterone encourages fat to be burnt off the buttocks while oestrogen takes it off the abdomen.These characteristically feminine fat stores are used in the last months of pregnancy and during breast-feeding. This is another reason why women who are seriously underweight often stop menstruating – they would not have the resources to support a pregnancy or a baby.”

Confetti time.

“Women with a low ratio, Professor Singh says, tend to start ovulating younger, and those with a high ratio find it more difficult to become pregnant and tend to have children later. [not by choice]
Although a high waist-hip ratio most commonly goes with being overweight, it can also be found in women of normal weight who have high testosterone levels – a condition that is also associated with being hairy, infertile and having a ‘male’ body shape.”

Manly body, fertility problems. Study it. Avert tragedy.

“In a survey of 106 men aged 18 to 22, the favourite was a female of average weight with the classic hour-glass figure. Not only were such women rated as young, sexy and healthy, they were also seen as ideal for childbearing.”

Again, sexy is different from beautiful.
Porn is a lie.

“The young men regarded the underweight women – defined as women of 5ft 5in weighing less than 90lb – as ‘youthful’ but not particularly attractive, especially for childbearing.”

To prefer the obese over the mannish figured for motherhood is huge.
Youthful is code for making them feel like a pedophile.

“In Professor Singh’s other surveys, men of all ages agreed with these findings – thus bearing out her theory of the waist-hip ratio.”

Women dropped the corset to signal they weren’t just baby-making machines.
It’s hard to test low-WHR women in a world of obesity.

“These data indicate that BF% appears to be a strong cue for attractiveness and that the impact of WHR and BMI on attractiveness is dependent, in part, on BF%. The appearance of body fat may provide disruption in the visual cues of both shape and size of the female body, potentially impacting behavior.”

Speciation is determined by biological compatibility in sum. This includes many factors. On none I have seen do Asian-White hybrids succeed over their parental groups’ averages; even IQ gains, if true, would be worse for the individual’s own fertility rate.

The only other thing I could think of is a study on STD rates between couples.

https://www.ncbi.nlm.nih.gov/pubmed/2117964http://sti.bmj.com/content/87/Suppl_2/ii14http://www.expat.or.id/medical/stds.html
“The association between travel and STDs has been known for centuries”
What’s the Asian version of burn the coal? Pick the chopstick, get ripped?http://global-disease-burden.healthgrove.com/l/24974/Syphilis-in-Southeast-AsiaPrevalance: “fairly common.”
The wages of sin. You can’t blame the white man.https://link.springer.com/article/10.1007/BF02438113Syphilis present in Asian archaeological samples.http://www.scmp.com/news/asia/east-asia/article/2060294/young-women-among-sufferers-japan-records-huge-spike-syphilis
“Endemic syphilis”https://emedicine.medscape.com/article/1952297-overviewhttp://sti.bmj.com/content/76/6/415
‘referred to as “the intraracial network effect,”’
Oh, that’s why they don’t study it.
“suggest that assortative mixing prevents the spread of STI to other subpopulations.”
“A number of studies in the literature, many of which did not measure biomedical markers of STI, suggest that mixing across subpopulations may contribute to spread of STI in the population, particularly across subpopulations.”
If you increase the microbe’s exposure to different parts of the human genome, it will evolve faster. Simple?
Age groups can be a larger factor, since the older immune system is weak and better for the microbe.
“In a recent study conducted in Seattle we found that most of the disease burden for gonococcal and chlamydial infections in both high prevalence and low prevalence subpopulations was attributable to mixing within the subpopulations”
I think we’ve found the reason white women mix out the least. Same reason we don’t like to eat meat raw – to avoid disease.
‘the proportion of infection attributable to indirect mixing, or so called “bridge populations,”
So it is attributable and naturally must inform sexual behaviour.
“While we found that sexual mixing between particular racial ethnic subpopulations increased the risk of STI significantly, the proportion of the population engaging in sexual mixing, and the numbers of sex partners reported by individuals engaging in sexual mixing across racial-ethnic subpopulations were too low for this increased risk to play a major part in disease burden.”
Hybrid vigour, guys!
The risk isn’t the major part, it’s fine! Water’s fine!
“The literature on racial-ethnic differentials in STI rates and the role of racial ethnic mixing on the spread of STI is emergent; many questions still remain unanswered.”

Could the levels be depleted by behaviour somehow?
Such as porn abuse?

Did you check adrenal function? In compulsive masturbators, the system is fine but the head’s wrong and eventually the glands pack up and give up.
The masturbators are not actually frigid, although their nervous system might be damaged with time. The idea that looping any addictive behaviour is fine for your health is Peak Boomer.

And by Jove, that title. It’s so true it’s painful.
The butthurt it shall produce, I should buy shares in painkillers.

“This study aimed 1) to compare vaccinated and unvaccinated children on a broad range of health outcomes, and 2) to determine whether an association found between vaccination and neurodevelopmental disorders (NDD), if any, remained significant after adjustment for other measured factors.”

Okay.

So far, good science.

“In conclusion, vaccinated homeschool children were found to have a higher rate of allergies and NDD than unvaccinated homeschool children. While vaccination remained significantly associated with NDD after controlling for other factors…”
Oh, that’s why they refuse to study it.
Honestly, at least.

“There are very few randomized trials on any existing vaccine recommended for children in terms of morbidity and mortality,” they note, “in part because of ethical concerns involving withholding vaccines from children assigned to a control group.”

And the ethics of not conducting real science to legally allow something?
There is gonna be a huge lawsuit once this finally breaks. Humongous.

Well, the races are biologically different. Men go in for more medical studies, so maybe vaccines have been designed for the typical race involved in these studies: white.

“With regard to acute and chronic conditions, vaccinated children were significantly less likely than the unvaccinated to have had chickenpox and pertussis but, contrary to expectation, were significantly more likely to have been diagnosed with otitis media, pneumonia, allergic rhinitis, eczema, and NDD. The vaccinated were
also more likely to have used antibiotics, allergy and fever medications; to have been fitted with ventilation ear tubes; visited a doctor for a health issue in the previous year, and been hospitalized. The reason for hospitalization and the age of the child at the time were not determined, but the latter finding appears consistent with a study of 38,801 reports to the VAERS of infants who were hospitalized or had died after receiving vaccinations. The study reported a linear relationship between the number of vaccine doses administered at one time and the rate of hospitalization and death; moreover, the younger the infant at the time of vaccination, the higher was the rate of hospitalization and death [55].

The hospitalization rate increased from 11% for 2 vaccine doses to 23.5% for 8 doses (r2 = 0.91), while the case fatality rate increased significantly from 3.6% for those receiving from 1-4 doses to 5.4 % for those receiving from 5-8 doses.”

…. Vaccination with PCV-7 has a marked effect on the complete microbiota composition of the upper respiratory tract in children, going beyond shifts in the distribution of pneumococcal serotypes and known potential pathogens and resulting in increased anaerobes, gram-positive bacteria and gramnegative bacterial species”

It’s tempting to point to babies who did die and pretend like there’s something medical science could have done. Some people will always die, some of those will be infants, the death rate will never be zero, but if you compared deaths without excluding illnesses caused by vaccines, those results would be interesting too.

http://www.wakingtimes.com/2017/05/22/gynecologist-gives-10-reasons-women-quit-taking-birth-control-pills/Because it hasn’t been properly tested isn’t enough?
“While this may be a temporary benefit to taking the Pill, long-term, your natural “female” hormones don’t return to normal. This means your hormones stay whacked for decades.”
“The Pill, as opposed to other contraceptive methods can cause permanent delayed conception”
Do your own research, there are many troubling connections. Gut problems, psychiatric risks, cancer… infertility, possibly birth defects or genetic disorders (notice how many Down’s kids we have now?) are possible but under-studied.

The fat thing doesn’t go away either – you’ll have a higher risk of becoming randomly fat into the future – especially pregnancy. The irony of vain women taking it to avoid getting fat….

“It is almost the equivalent to having twice the number of children… Thus assortative mating by ethnicity can have large fitness benefits, the largest derived from choosing mates within geographic races.”

they pass on more of themselves, less genetic death per carrier (child)
May increase the values for Ks in parental investment theory.

“In other words and general terms, a white mother will be almost as twice as closely related to a child with a white father versus a child with a black father.”

Bear in mind, this isn’t opinion. It’s genetic.

“But when I turn to the mirror in my bedroom to admire us together, I am shocked. She seems so alien.”
LOL
The non-white always benefits far in excess due to dominating the white phenotypes.
They’re hoping to reduce their mutation load/disease risk. The white party loses.

“But still, I can’t shake off the feeling of unease. I didn’t realise how much her looking different would matter… When she was born, pale but with lots of dark hair, I asked the midwife if her eyes would stay blue. ‘Asian genes are very strong,’ she said in what I took to be an ominous tone. No more Brady Bunch kids for me. The midwife has been proved right and every day my baby’s eyes get a little darker.””

Wow so Nazi to insist your daughter look like you. /s
WTF do these r-types expect?

“Since parents share fewer genes with mixed-race children, people involved in interracial marriages are short-changing their own genes, which might explain why people engaged in mixed-race relationships often tend to have lower mate value.”

oh yeah
they signal how they can’t get one of their own so they settled
we all know how attractive desperation is, socially and sexually

“In general, mixed-race people have more health problems.”The hybrid vigour thing was always a lie, it’s reduced fitness.According to the field of medicine.

“In other words, an argument could be made that mixed-race families are maladaptive — both for the parents and the children — and undermine one’s genetic interests. As noted by various commenters, multiracial families often do not possess the harmony, cooperation and purposefulness of same-race families, because mixed-race families lack the focus of genetic investment and returns that same-race families possess.”

Like a kind of …selection…

White British genes best at IVF *hums national anthem*

https://www.sciencedaily.com/releases/2016/08/160818212907.htm
“The study also found that some groups of women including South Asian Bangladeshi, Black African, Middle Eastern, have a significantly lower number of eggs collected than White British women.”
weeaboos lament
“Furthermore, the increased prevalence of polycystic ovary syndrome (PCOS) in south Asian women may have an impact on egg quality and lower implantation rates.”
“The data suggests that ethnicity is a major independent factor determining the chances of IVF or ICSI treatment success.”
No shit Sherlock. Could it be something of a biological thing going on here…?
“While the reason for this association is difficult to explain, the potential factors could be the observed differences in cause of infertility, ovarian response, fertilisation rates and implantation rates, which are all independent predictors of IVF success.”
Difficult? Awkward.
“”Infertility affects 10-15% of the population and more people are seeking fertility treatment.”
Isn’t that approaching the rate of mixed race couples? Coincidence, I’m sure. I’d like to check but nobody gives enough of a damn about them to collect the data.
“The reasons behind the variation need to be looked at in more detail but in the future could potentially help improve success rates amongst all groups of women.””
Not if it’s genetic.

Mixed race babies weaker babieshttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC2867623/
“contribute to the substantially higher demonstrated risk for stillbirth”
it’s healthy! they tell you
only because they aren’t collecting the data to argue otherwise
I would sue because that’s academic neglect and actual systemic racism.

You really have to search even for data on successful births, then there’s NO fitness advantage by rate.http://paa2012.princeton.edu/papers/122287
“This paper investigates whether mixed race couples have different or same fertility level than same race couples”
“Same race couples have on average 1.93 children while mixed couples have 2.05 children. Same race couples have lower fertility because the majority of those couples are white. This data shows that homo and heterogamous unions do not have the same fertility level”
“Mixed race couples have fertility levels that fall in between same race couples; not as high as black or brown couples but not as low as the white couple.”
Regression to the mean, my old friend.

https://www.quora.com/Is-it-harder-for-interracial-couples-to-conceive
“I can’t find any studies on this at all.”
Red flag to ANYONE else?
“I heard from student of medicine that interracial couples could have problem conceiving child because of their racial differences.”
Why not study it?
They’re screwing over the mixed-race kids.
Possibly killing them, in the long run. That’s just sick, not to study it. Parents deserve to know.

The manosphere dudes with Yellow Fever won’t be pleased.http://blogs.discovermagazine.com/gnxp/2008/10/the-prenatal-wages-of-interracial-relationships/https://www.eurekalert.org/pub_releases/2008-10/sumc-acf092508.php
“In both of these cases there isn’t something mystical going on; God is not smiting those who are sinning by crossing racial lines. Human spontaneous abortion rates are high. Much of this might be due to mother-child immune system responses. It shouldn’t surprise anyone that people from very genetically distinct populations have very different immune profiles. …”
It does if they grow up hearing everyone is biologically equal.
“There is some data which might suggest that genetic relatedness increases reproductive fitness, possibly because of reduced risk of immune incompatibilities between mother and fetus when the father is more closely related to the mother. It stands to reason then that as the father becomes more genetically distant the likelihood of incompatibilities might increase. All of this means that genes matter, and they matter in ways we can possibly predict.”

OT: The vaccine brigade would do well to remember the field has problems…

Don’t expect that cure for cancer soon, the charities should be sued for false advertising.

This is mostly a pharma finding.

Something is rotten in the state of biomedical research. Everyone who works in the field knows this on some level. We applaud presentations by colleagues at conferences, hoping that they will extend the same courtesy to us, but we know in our hearts that the majority or even the vast majority of our research claims are false.

“A lot of what is published is incorrect.” I’m not allowed to say who made this remark because we were asked to observe Chatham House rules. We were also asked not to take photographs of slides. Those who worked for government agencies pleaded that their comments especially remain unquoted, since the forthcoming UK election meant they were living in “purdah”— chilling state where severe restrictions on freedom of speech are placed on anyone on the government’s payroll. Why the paranoid concern for secrecy and non-attribution? Because this symposium—on the reproducibility and reliability of biomedical research, held at the Wellcome Trust in London last week—ouched on one of the most sensitive issues in science today: the idea that something has gone fundamentally wrong with one of our greatest human creations…..