Intranasal corticosteroid list

Persons who are using drugs that suppress the immune system (., corticosteroids) are more
susceptible to infections than healthy individuals. Chickenpox and measles , for example, can have a
more serious or even fatal course in susceptible children or adults using corticosteroids. In children or
adults who have not had these diseases or been properly immunized, particular care should be taken to
avoid exposure. How the dose, route, and duration of corticosteroid administration affect the risk of
developing a disseminated infection is not known. The contribution of the underlying disease and/or
prior corticosteroid treatment to the risk is also not known. If a patient is exposed to chickenpox,
prophylaxis with varicella zoster immune globulin (VZIG) may be indicated. If a patient is exposed to
measles, prophylaxis with pooled intramuscular immunoglobulin ( IG ) may be indicated (see the
respective package inserts for complete VZIG and IG prescribing information). If chickenpox or
measles develops, treatment with antiviral agents may be considered.

Triamcinolone is used to treat a number of different medical conditions, such as eczema , Lichen sclerosus , psoriasis , arthritis , allergies , ulcerative colitis , lupus , sympathetic ophthalmia , temporal arteritis , uveitis , ocular inflammation , Urushiol-induced contact dermatitis , aphthous ulcers (usually as triamcinolone acetonide ), visualization during vitrectomy and the prevention of asthma attacks. It will not treat an asthma attack once it has already begun. [2] [3] [4] It has also been used off-label for macular degeneration . [5]

Adenotonsillar hypertrophy causes mouth breathing, nasal congestion, hyponasal speech, snoring, obstructive sleep apnoea, chronic sinusitis and recurrent otitis media. Adenoidal hypertrophy results in the obstruction of nasal passages and Eustachian tubes, and blocks the clearance of nasal mucus. Adenotonsillar hypertrophy and obstructive sleep apnoea are associated with increased expression of various mediators of inflammatory responses in the tonsils, and respond to anti-inflammatory agents such as corticosteroids. Topical nasal steroids most likely affect the anatomical component by decreasing inspiratory upper airway resistance at the nasal, adenoidal or tonsillar levels. Corticosteroids, by their lympholytic or anti-inflammatory effects, might reduce adenotonsillar hypertrophy. Intranasal corticosteroids reduce cellular proliferation and the production of pro-inflammatory cytokines in a tonsil and adenoid mixed-cell culture system.