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As knowledge of the mechanisms regulating cardiac energy substrate metabolism increases, and as alterations in energy substrate metabolism attend the above cardiac pathophysiologies, the modulation and optimization of energy substrate metabolism represents a novel and promising area for therapeutic intervention in ischemic heart disease and heart failure.

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In all experiments, hearts were initially perfused in the absence of drug.

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Additional research is needed to elucidate the mechanisms underlying the complex interactions between myocardial substrate metabolism and NO.

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