Inflammation is the foundation for cancer and degenerative/autoimmune diseases. Small changes in diet and exercise, e.g. omega-3 oils, vitamin D, low starch, and maintaining muscle mass, can dramatically alter predisposition to disease and aging, and minimize the negative impact of genetic risks. Based on my experience in biological research, I am trying to explain how the anti-inflammatory diet and lifestyle combat disease. 190 more articles at http://coolinginflammation.blogspot.com

Anti-Inflammatory Diet

All health care starts with diet. My recommendations for a healthy diet are here:

The soybean oil is primarily an inflammatory omega-6 vegetable oil. HFCS is inflammatory both because it causes a rapid spike in blood glucose and insulin, but because the fructose is a sweet sugar that is even more active than glucose in glycation reactions (adding sugars to amino acids or proteins) that produce the advanced glycation end products that menace diabetics and that cross-link collagen and accelerate the aging of skin and connective tissue. The food starch and sugar both contribute to inflammation through rapid spiking of blood glucose and insulin.

This mayonnaise substitute would be expected to be highly inflammatory and I would recommend that anyone trying to minimize chronic inflammation using fish oil omega-3 supplements should stick to the real thing and pay careful attention to any oils added. Mayonnaise can be whipped up from egg yolks and olive oil. Flax seed oil could also be used -- it isn’t very effective as an anti-inflammatory omega-3 oil, but it is safer than the omega-6 rich vegetable oils. The short, 18C, omega-3 fatty acids can be lengthened to anti-inflammatory 20C (EPA) and 22C (DHA) versions, but the omega-6 fatty acids inhibit that conversion. In this context, I think that saturated fats would be safer than the inflammatory omega-6 soybean oil or its even more incendiary cousin, corn oil .

I am very skeptical of the evidence used to advise the use of unsaturated vegetable oil in place of saturated fats for heart health. Lowering LDL and triglycerides by diet or drugs does not apparently lower heart attack risk. I think that the data all point toward chronic inflammation as the actual culprit. All of the treatments that reduce chronic inflammation (most notably diet and exercise, or COX inhibitors) also decrease the risk of heart disease and death.

1 comment:

Anonymous
said...

Once we understand why saturated fat is good, not bad, what more is there to discuss except who's responsible for how many US deaths because lies about diet and the corruption that puts more and more killer products on grocery store shelves.

Government has been lying, government agencies have been lying, congress has been lying and passing laws enshrining the lies. People are sickening and dying, including the young that are the "hope" of US future. Follow the money for crap's sake; congress is in the pocket of anyone who pays them: big farmer, big pharma, and the AMA which used to be a quasi-trustworthy outfit but hasn't been that for decades. Follow the moneyAnd don't keep voting for those in congress who passed the laws that allowed this to happen. Get them all out, it's our only hope, or run for your life because soon you won't be able to buy meat or saturated fats because they'll be illegal.It's just like the savings & loan scam, the sub-prime scam, climategate, and others. When does it stop?!

Listen to my podcast on Jimmy Moore's Livin' La Vida Low Carb Show

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About Me

I grew up in San Diego and did my PhD in Molecular, Cellular and Developmental Biology (U. Colo. Boulder). I subsequently held postdoctoral research positions at the Swedish Forest Products Research Laboratories, Stockholm, U. Missouri -Colombia and Kansas State U. I was an assistant professor in the Cell and Developmental Biology Department at Harvard University, and an associate professor and Director of the Genetic Engineering Program at Cedar Crest College in Allentown, PA. I joined the faculty at the College of Idaho in 1991 and in 1997-98 I spent a six-month sabbatical at the National University of Singapore. Most recently I have focused on the role of heparin in inflammation and disease.