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Retroviral Talk: Is HIV Just in Lesions?

International Aids Day Special - Listening to medical propaganda, one would think a retrovirus is a very bad thing - something to be killed immediately wherever it is found. But nothing could be further from the truth.

Just like cholesterol has been condemned as "bad" and this lie has given rise to a billion-dollar industry of useless and even damaging cholesterol lowering (statin) drugs, so have retrovira been condemned, giving rise to a billion-dollar industry of Aids research and pharmaceutical intervention, to cocktails of "anti-retroviral" drugs they tell us are a must for anyone reacting to that very unspecific test that shows up stressed proteins associated with HIV. The retrovirus being targeted, HIV, incidentally has never been shown to do anything that could be construed as destroying the human immune system. As a matter of fact, it seems to be a necessary component of healing wounds and lesions, as Cal Crilly has discovered.

Cal is one of the few people who are able and willing to look at these details. His latest brainchild is a question: "Is HIV to be found just in lesions?"

Good reading!

I've been looking at this subject for nearly 14 years now, I guess I stumbled in on a mess.

I often say if you analyse gibberish you may be talking gibberish so anything I write you can decide.

The main thing to know is that everything written about HIV in the last 26 years by the mainstream is complete rubbish and utterly dangerous and if you don't question it you'll miss out on seeing the panic as people realise it's gibberish.

I've talked to many friends who got tagged by the HIV test and the incredible mess the medical profession put them through.

Some did not survive and this is entirely due to medical incompetence.

But this incompetence goes to the very top and they are the ones who need to step out of the way and admit they stuffed up.

For as long as the HIV debacle goes on as is no one in the medical profession can hold their head up high.

Unless they were one of the few that spoke out.

These were my conclusions after a year or two of looking into the heart disease aspect of HIV 'infection'.

We are overall looking at heart disease, leukemia and cancers and the immune failure seen with AIDS is a result of medical incompetence, adding in drug users simply doesn't count.

Caveolin is a lipid raft involved in cell movement, signalling and of course smooth muscle cell movement in lesions.

We have a lot of fat in our bodies, things like caveolin help it hold together.

And it looks like HIV is needed for that process.

The immune system also relies on these fat or lipid rafts to send signals.

Once HIV has bonded to caveolin to make the lipid raft stick to cells then the HIV has done its job and will not be easy to detect in blood samples in healthy people without lesions.

This is why caveolin is a HIV inhibitor.

Once HIV sticks to caveolin it's done its job.

Muscle and wound repair needs both fat as Cholesterol and Selenium along with Vitamin C and Lysine.

Anyone with low fat has low CD4 T-cells as CD4 T-cells respond to cholesterol but also anyone with low fat and lesions will not be able to repair wounds and HIV will remain loose until there is enough fat to make a lipid raft for the HIV to bind to a cell wall.

This is why Antiretroviral drugs dissolve and wreck fat and leave fat cells blobbing all over the place.

The loose fat raises T-cells counts but because the antiretrovirals stop HIV from doing it's job the lesions remain in AIDS patients and they die.

HIV is also just one of the retroviruses in our bodies.

Our genome has 8% retrovirus.

No one knows what AIDS drugs do to all of our retroviral functions because the entire medical profession is bent on destroying them even if they kill the patient.

"Caveolin-1 is a scaffolding protein that organizes and concentrates specific ligands within the caveolae membranes. We identified a conserved caveolin-1 binding motif in the HIV-1 transmembrane envelope glycoprotein gp41 and designed several synthetic peptides, referred to as CBD1, corresponding to the consensus caveolin-1 binding domain in gp41."

"Interestingly, gp41 exists as a stable complex with caveolin-1 in HIV-infected cells."

"Interestingly, gp41 exists as a stable complex with caveolin-1 in HIV-infected cells."

Why didn't anyone ask why it's a stable complex?

Caveolin once bound to HIV then stops HIV jumping around looking for caveolin to bind to.

"Caveolin-1 (Cav-1) is a major protein constituent of caveolae, a type of plasma membrane raft. We observed that coexpression of human Cav-1 with human immunodeficiency virus type 1 (HIV-1) blocked virion production from cells that are ordinarily highly permissive. Further investigation showed that this effect is specific, occurs at low ratios of Cav-1 to HIV-1 DNA, depends on expression of Cav-1 protein, and involves severely impaired expression of HIV-1 proteins."Blockade of Human Immunodeficiency Virus Type 1 Expression by Caveolin-1

"In a previous study we showed that budding of HIV-1 particles occurs at highly specialized membrane microdomains known as lipid rafts. These microdomains are characterized by a distinct lipid composition that includes high concentrations of cholesterol, sphingolipids, and glycolipids. Since cholesterol is known to play a key role in the entry of some other viruses, our observation of HIV budding from lipid rafts led us to investigate the role in HIV-1 entry of cholesterol and lipid rafts in the plasma membrane of susceptible cells."Lipid Rafts and HIV Pathogenesis: Host Membrane Cholesterol Is Required for Infection by HIV Type 1

Of course the thinking in the mainstream is if they reduce all of our cholesterol then HIV will go away.

This will simply kill off all our CD4 cells anyway so you see they think in gibberish.

It's a highly developed secret code of a language that no logical people can understand.

Is AIDS then in the main a disease of low Fat and Selenium, Vitamin C and Lysine?

And the HIV tests just pick up people with lesions or not enough fat in their diet.

If you want your T-cells to go up eat more food as long as you have the Vitamin C, Selenium/Glutathione and Lysine in the diet you won't get heart disease as these are the nutrients that cure heart disease.

This is slightly off the fat subject but a descriptive analysis of what the HERV-W retrovirus does in pregnant women.

I think the fact that a retrovirus is so important for a proper pregnancy this is enough reason to completly ban the Mother to Child prevention campaign where they give disgusting drugs like AZT and Neverapine to women and children.

"Oh they must be infected????" is the standard answer... but they don't know why and still are nowhere near finding out why.

So the Human Immune Retrovirus and p24 antigen only appear when HLA-DR are activated in lesions...

But the lesions go away with good nutrition and so does the p24 and HIV that appears with HLA-DR.

You can do it with any decent foods.

"This study demonstrates that DR expressed on CD4+ T cells dramatically increases HIV-1 expression. Virus production in DR+ cells was > 10-fold higher than in DR- cells as measured by p24 production. Moreover, two T cell lines that were derived from the same parental cell, one DR+ and the other DR-, differentially expressed HIV-1 following transfection with full-length HIV-1 clone as determined by luciferase.
Viral expression in DR+ transfectants were up to 20-fold higher than the DR- counterparts."HLA-DR on T cells enhances HIV-1 expression

And you also see HLA-DR and the CD4 T-cells in arterial changes and inflammation and lesions....

And all other lesions such as kidney, liver, lung, skin, causes can be fungus, bacteria like TB, chemicals, solvents like benzene and alcohol, scurvy, smoking, cancer, etc etc have HLA-DR active in repairing the wound and HIV seems to be a part of that.
Therefore HIV is just part of the wound process, if you are unlucky enough to have some form of lesion then HLA-DR+ activated cells appear and that's where HIV is.

You can google 'lesion' with any organ you pick and 'HLA-DR' and the 3 will appear.

HLA-DR is not a racial gene as I once thought but one that kicks in for wound repair.

Africans and blacks in the US etc are just poorer so are battered about more as we all know from poverty.

And they also get tested more.

That is also an aspect of this epidemic, the medical profession have proved themselves to be blind bigots.

Very few doctors have even registered that the presumption that gays, prostitutes, junkies and Africans and Hispanics will be all carrying a sex virus is quite dodgy.

And if you test these groups over and over again with a dodgy test well someone will look positive.

This test that had nothing to do with a sex virus but was trying to pick up the 'leukemia' retroviruses we all get with cancer so of course the 'sex transmission' belief will stuff up all the data.

Intravenous drug addicts have lesions - well duh.

The gays that first appeared with 'AIDS' snorted drugs that made their lungs bleed.

Hemophiliacs bleed.

Only junkie prostitutes get AIDS the others get paid enough to eat properly.

Even sperm uses HLA-DR to attach with???

"Remember that sperm binds to HLA-DR on white blood cells. Interestingly enough, it turns out that the AIDS virus is covered with a protein coat that mimics HLA-DR in several important respects, including its structure. This suggests that sperm are able to attract and acquire AIDS virus particles."

This study may have clicked it all for me.

These African women became HIV after this using drug tenofovir which causes lesions.

The drug causes lesion in all sorts of places like kidney and liver too.

Or skin rashes will do the trick to react with p24.

"Among 210 patients at 2 indigent care HIV clinics, 9 cases of tenofovir-associated skin reactions were reported. (23) Eight patients were diagnosed with tenofovir-related reactions--a pruritic eruption involving the face (n=7), extremities (n=5), trunk (n=4), and entire body (n=2) occurred within the first 2 weeks of tenofovir therapy. One patient developed concomitant urticarial lesions, angioedema, and shortness of breath. In 2 instances, skin lesions first developed after resumption of a previous tenofovir-containing regimen. Six out of the 8 patients were rechallenged with tenofovir or tenofovir/emtricitabine and developed recurrence of skin eruption and pruritus within 24 to 48 hours, resulting in permanent discontinuation of tenofovir. Three of these patients were taking efavirenz as part of their regimen, which is also associated with cutaneous reactions."Cutaneous drug reactions associated with newer antiretroviral agents

Or the standard AZT used in millions of gays, women and children...mice study but really what was anyone thinking?

I think you can try Bromelain, Turmeric, Olive Leaf Extract and and Green Tea, Sage or Lemon Balm to block the antigens.

Maybe not Sage a week before the test as it has estrogens which in theory could stimulate retroviruses.

Green tea definitely is needed.

Eat enough veggies like the brazil nuts, broccoli garlic for selenium, lemons and all the rest of the yum stuff with a folate and B12 supplement or food, that's for the viral load test.

Eat good amounts fat (: that keeps T-cells up and repairs wounds and get enough sun and sleep so you're hormones stay normal.

Cod liver oil if it rains or gets cloudy for days - as low Vitamin D makes viruses express - or sun.

When you stay in front of a computer at night you destroy the melatonin in your pineal gland and that effects things too.

So get good sleep.

Fish may be the best, it also has decent lysine levels, milk and red meat have a molecule we are allergic to and can cause inflammation.

Most of these recommended foods have anti-inflammatory effect.

CD44 is an attachment molecule that always turns up in tumours and is a good indication cancer will spread.

So HIV buds there.

There is a cross over with HLA-DR here, they occur together.

"CD44 was concentrated in two locations: in clathrin coated pits and at the base of budding HIV virions. For many (although not all) virions, CD44 co-located with budding HIV and appears to form a "collar" at the base of the budding virus. We saw no differences in CD44 localization between HIVLAI/III and HIVBAL isolates, nor differences in HIVLAI/IIIB and HIVBAL gp120gp41 "spike and ball" morphology. No significant immunogold staining of ICAM-1 was seen in these same experiments although low expression of ICAM-1 on the cell surface was shown by indirect immunofluorescence and flow cytometry. However, by using AA5 cells with high expression of ICAM-1 on the cell surface, an even distribution of ICAM-1 both HIVLAI/IIIB-infected and uninfected AA5 cells was observed. We saw only rare association of ICAM-1 molecules with HIV virions. Co-localization of CD44 with HIV virions at the site of virus budding supports the hypothesis that CD44 may be involved in HIV budding."Co-localization of CD44 expression with HIV budding in HIV-infected cells.

I think and suspect Bromelain may have an effect on p24 but I may be wrong, I'll work out the maths later.

But it stops HIV budding in CD44.... so therefore the p24 is likely to disappear.

Be careful with bromelain being a blood thinner, it's good to be on lysine first with enough fat foods and make sure you have vitamin k in the diet to not bleed.

Bromelain has never made me bleed, I've been on it 4 years for psoriasis.

These teas below all have effect on wound healing by the way, as does green tea.

Try them all, Sage will stop dementia.

"Extracts from lemon balm (Melissa officinalis L.), peppermint (Mentha × piperita L.), and sage (Salvia officinalis L.) exhibited a high and concentration-dependent activity against the infection of HIV-1 in T-cell lines, primary macrophages, and in ex vivo tonsil histocultures with 50% inhibitory concentrations as low as 0.004%. The aqueous Lamiaceae extracts did not or only at very high concentrations interfere with cell viability. Mechanistically, extract exposure of free virions potently and rapidly inhibited infection, while exposure of surface-bound virions or target cells alone had virtually no antiviral effect. In line with this observation, a virion-fusion assay demonstrated that HIV-1 entry was drastically impaired following treatment of particles with Lamiaceae extracts, and the magnitude of this effect at the early stage of infection correlated with the inhibitory potency on HIV-1 replication. Extracts were active against virions carrying diverse envelopes (X4 and R5 HIV-1, vesicular stomatitis virus, ecotropic murine leukemia virus), but not against a non-enveloped adenovirus."Aqueous extracts from peppermint, sage and lemon balm leaves display potent anti-HIV-1 activity by increasing the virion density

Also use things like turmeric as this is used in leukemia to stop leukemic cells going out of control and any thing that stops leukemia naturally stops a so called "leukemia retrovirus" like HIV and while you're at it use coconut oil in the curries.

Tomatoes are fab too cos of lycopenes, artichoke pastas... actually all the foods with flavonoids have effect... lemons, berries etc..

Mushrooms I believe should thrown in a plenty due to wound healing properties and the sulphurs.. all of them

"RESULTS:
VCO-treated wounds healed much faster, as indicated by a decreased time of complete epithelization and higher levels of various skin components. Pepsin-soluble collagen showed a significant increase in VCO- treated wounds, indicating a higher collagen cross-linking. Glycohydrolase activities were also found to be increased due to a higher turnover of collagen. Antioxidant enzyme activities, and reduced glutathione and malondialdehyde levels were found to be increased on the 10th day after wounding, which were found to have returned to normal levels on day 14 in the treated wounds. The lipid peroxide levels were found to be lower in the treated wounds. A histopathological study showed an increase in fibroblast proliferation and neovascularization in VCO-treated wounds compared to controls."Effect of topical application of virgin coconut oil on skin components and antioxidant status during dermal wound healing in young rats.

"neovascularization in VCO-treated wounds compared to controls."
Means it made new arteries, I think it gives fats for HIV to bind on and finish repairs.

Things like Butyrate are odd, they fix wounds.

"Researchers working with an experimental drug for sickle cell anemia noticed an unexpected side effect: the incidental healing of debilitating leg ulcers in seven patients.

That serendipitous observation led to a 25-patient phase II study in which treatment with the compound, arginine butyrate, healed 17 of 37 leg ulcers. In contrast, among 24 ulcers that received standard local care for wounds, complete healing was documented for one ulcer.