Brief report

Although the neuroinflammatory response in the central nervous system is initiated to exert protective effects, it may also exert detrimental effects and widespread inflammation through the blood–brain barrier. In addition, microglia has gradually emerged as a key interface between stress-related signals and neuroimmune consequences of stress, with stressors leading to elevated microglial activity. In psychiatric diseases such as mood disorders or schizophrenia, the inflammatory response system is activated. This important topic is reviewed by Norbert Müller in this issue. Accordingly,…

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