“There was significant correlation between
history of cardiovascular events and presence of fluoride uptake in
coronary arteries. The coronary fluoride uptake value in patients with
cardiovascular events was significantly higher than in patients without
cardiovascular events.”

The argument, at the time, was the study was simply about a new
diagnostic technique and shouldn’t be ‘read into,’ and that, presumably,
the increased fluoride uptake value observed in patients with a higher
frequency of cardiovascular events was a an ‘effect’ of the heart
disease itself and not in any way indicative of fluoride’s causative
role as a cardiotoxic agent — despite the fact that fluoride’s cardiotoxicity has already been consistently demonstrated in the biomedical literature.

Now, a provocative new study published in the journal Toxicology not
only provides some vindication for our previous interpretations, but
also raises serious concern over the cardiovascular complications
associated with water fluoridation practices, showing for the first time
that despite exhibiting an anti-calcification effect in vitro (cell
model) fluoride exposure at levels found in people who drink fluoridated
water exhibits artery-calcifying effects in the more important in vivo
(animal) model.