Results: Three proteins were identified, of which the proinflammatory chemokine CC chemokine ligand 3 (CCL3) and cytokine TNF-a were downregulated and the antiinflammatory cytokine interleukin-1 receptor antagonist was upregulated.

The study, performed in mouse models for asthma research, showed that the synthetic sulfate monosaccharide blocks the interaction between chemokine CCL20-a T-cell signaling protein and heparin sulfate, a molecule that protects and immobilizes CCL20 on epithelial cells in the lung.

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