The HepTech System contains three components that will change your life!

HeptoBoost: An antioxidant supplement that contains all 38 nutrients that accelerate the action of the anti-inflammatory/antioxidant enzyme systems.

HeptoShield: A PolyenolPhosphatidyl Choline (PPC) supplement that incorporates into the cell membrane, thus increasing cellular fluidity of the cell membrane and its organelles. This allows enzymes the needed mobility to orient and locate for proper function.

Fibroguard: This suite of herbal remedies stimulates the production of the endogenous antioxidant enzymes, such as: Catalase, Super Oxide Dismutase, and Glutathione Peroxidase. The mechanism of this stimulation is the release of Nuclear Releasing Factor 2, which turns on the genes which transcribe and ultimately assemble the antioxidant enzymes.

HeptoBoost

A Medical Food for the dietary support of increased oxidative stress.

Hepatitis causes an increase in the production of free radical Reactive Oxygen Species (ROS), which manifests at the cellular/biochemical level as increased levels of oxidizing molecules in and around the liver cells. This increased oxidative stress can trigger inflammatory processes, which result in conversion of stellate cells into myofibroblasts, which in turn secrete collagen. The secretion and accumulation of collagen is meant to form scar tissue to repair physical injury such as lacerations or trauma. But, the inappropriate accumulation of collagen in the case of chronic inflammation results in gradual fibrotic deposition and accumulation. If such accumulation continues, the affected liver tissue will eventually be diagnosed as fibrosis and later as cirrhosis.

HeptoBoost is a Medical Food, contains vitamins/nutrients in the full repletion therapeutic doses proven medically effective by valid research in antioxidant enzyme activity. This strategy positions Heptoboost as the most complete antioxidant support system available. Heptoboost provides cofactor and substrate support for enzymes that quench free radicals, detoxify chemicals in the Cytochrome P450 System, and produce energy through the mitochondrial pathways (Krebs and OxPhos). THus, Heptoboost provides support for a liver under attack by toxins (drugs, chemicals, alcohol), viruses (HCV), or fatty deposits (NAFLD/NASH).

HeptoBoost was formulated to quench the free radicals, known as Reactive Oxygen Species (ROS), in the cells of patients with the various forms of Hepatitis. HeptoBoost provides the substrate needed for the production of glutathione, the master antioxidant. Production of this tripeptide requires a full repletion of the rate-limiting precursor amino acids for the assembly of glutathione by the enzyme Glutathione Peroxidase.

Fibroguard also provides the botanical antioxidant polyphenols that act as protective “chaperones” to protect the cell’s vulnerable membranes and proteins. Heptoboost likewise includes herbal remedies which induce of endogenous antioxidant gene expression.

FibroGuard

The name says it all! Fibroguard provides an effective suite of botanical and nutraceutical ingredients for the management of cellular inflammation and fibrogenesis caused by Hepatitis.

Hepatitis is an inflammation of the liver tissue caused by toxins/alcohol, viruses/infection, fatty deposition, autoimmunity, or trauma. Inflammation is the activation of the immune system and the resultant release of ROS and other biochemical processes to inactivate infection, remove damaged tissue, and initiate repair. One important pathway in the inflammatory response is the activation of the gene transcription factor NFkappaB. This molecule triggers nuclear transcription and production of pro-inflammatory peptides NFkappaB activates stellate cell conversion to myofibroblasts, which in turn secrete collagen/fibrin. This fibrous scar tissue produces a continuum of deposition from fibrosis to cirrhosis.

Fibroguard effectively counteracts hepatic inflammation by stimulating production of the antioxidant enzymes, Catalase, SOD, and Glutathione Perioxidase via Nrf2 activation. Quenching the inflammatory ROS fire reduces levels of NFkappaB activation, which in turn does not stimulate conversion of the hepatic stellate cells into myofibroblasts. Without myofibroblast conversion, there is no production of collagen/fibrin, and no production of fibrosis, and no progression to cirrhiosis.

The liver contains enzymes that naturally and continuously break down scar tissue. If the rate of breakdown exceeds the rate of deposition, the body will gradually remove the excess fibrous deposits. If sufficient hepatic tissue and function remains, stopping the damage, and allowing the body’s own fibrin degradation processes to function, the body may be able to reverse the damage produced by the extreme deposition of fibrous tissue seen in cirrhosis. (note: Todd Schneider was diagnosed with cirrhosis, and through years of supplementation with the HepTech System, his liver enzymes and fibroscan have reversed to normal.)

FibroGuard is a medical food consisting of antioxidant support vitamins, and botanical bioflavonoids and polyphenols from spice, plant, berry and seed extracts. The polyphenols in these botanicals activate the body’s own antioxidant and anti-inflammatory properties. These botanicals stimulate the production of the antioxidant pathways through the Nrf2 signaling pathways, producing Catalase, SOD, and Glutathione Peroxidase. The botanical polyphenols also surround and protect (chaperone) the critical structural and enzymatic proteins.

The polyphenols in FibroGuard reduce the damage produced by inflammation. By quenching free radicals, fewer ROS trigger NFkappaB activation. The polyphenols also stabilize the inactive precursor form of NFkappaB, which in turn dampens the broad cascade of inflammatory gene activations initiated by binding of NFkappaB.

HeptoShield

For the management of hepatic cell external/internal membrane phospholipids composition.

At least one genotype of Hepatitis causes disruption of normal hepatic lipid metabolism, which contributes to the accumulation of intracellular lipids, or steatosis. Steatosis correlates clinically with increased fibrosis or cirrhosis in Hepatitis patients. Hepatitis also causes an increase in free radicals and cellular inflammation which combine to produce lipid peroxidation damage to cell membrane phospholipids as well as the interior cellular membranes such as endoplasmic reticulum, golgi apparatus, mitochondria, perinuclear membrane, lysosomes, endosomes and perioxisomes. Phospholipid translocation and loss of membrane fluidity result in decrease of function in membrane proteins.