Previous research published earlier this year in Nature Medicine involving University of Minnesota Medical School faculty Paul D. Robbins and Laura J. Niedernhofer and Mayo Clinic investigators James L. Kirkland and Tamara Tchkonia, showed it was possible to reduce the burden of damaged cells, termed senescent cells, and extend lifespan and improve health, even when treatment was initiated late in life. They now have shown that treatment of aged mice with the natural product Fisetin, found in many fruits and vegetables, also has significant positive effects on health and lifespan.

As people grow old, their bones tend to become brittle, their minds can become dull and their organs sometimes slowly fail. But scientists now think that these unpleasant side effects of aging can be stopped—at least in mice.

Researchers at the Mayo Clinic think they have identified a rogue cell, which works like a bacteria in the body, that could cause most age-related illnesses. These cells are called senescent cells, and they build up over time.

If someone has too many of them, these cells can refuse to die and go on a destructive rampage within the human body, killing other cells that they come in contact with. Mayo Clinic scientists found that they could destroy senescent cells to stop and even reverse the ill effects of aging in rodents. The findings published on Monday in Nature Medicine showed that the mice lived up to 36 percent longer.

“This is exciting research,” said Felipe Sierra, director of the National Institute of Aging’s Division of Aging Biology. “Additional research will be necessary to determine if compounds, like the one used in this study, are safe and effective in clinical trials with people."

For decades researchers have worked to shed light on the causes of neurodegenerative disorders, a group of devastating conditions, including Alzheimer’s and Parkinson’s, that involve the progressive loss of neurons and nervous system function. In recent years, numerous factors, from genetic mutations to viral infections, have been found to contribute to the development of these diseases.

Florida is suing the nation's two largest drugstore chains, Walgreens and CVS, alleging they added to the state and national opioid crisis by overselling painkillers and not taking precautions to stop illegal sales.

Bill Faloon is a man with a mission: to fend off aging. As long as possible, and maybe even forever. A recent Popular Science story called him “the forever man.” This is both because Faloon wants to figure out how we can live forever and because he has been working to resolve this problem for what can seem like forever — or at least since the 1970s, which seems to most of us to be pretty much forever.

Although long life tends to run in families, genetics has far less influence on life span than previously thought, according to a new analysis of more than 400 million people. The results suggest that the heritability of life span is well below past estimates, which failed to account for our tendency to select partners with similar traits to our own.

Flies on dietary restriction are protected from leaky gut and systemic inflammation as they age. Conversely, flies on a rich diet are more prone to intestinal permeability, developing gaps in the intestinal barrier which are caused by an age-related increase in the death of intestinal epithelial cells. Researchers also looked at dysbiosis as a contributor to leaky gut and concluded that diet may ultimately be the primary driver in cellular changes leading to intestinal permeability.

It’s possible to boost declining levels of NAD+, a molecule required for fundamental chemical reactions in the human body, with a dietary supplement. A team led by Leonard Guarente of MIT and Elysium Health gave 120 healthy adult volunteers between the ages of 60 and 80 NRPT (known commercially as Basis) over the course of eight weeks. Volunteers taking the regular dose of NRPT had 40 percent more NAD+ compared to their baseline when tested at four weeks and at eight weeks. Others who took a double dose of NRPT had even higher levels of NAD+, while those who took the placebo saw no increase. Research shows that NAD+ levels decline in animals and humans with age. Boosting NAD+ in animals improves age-associated diseases, so researchers now want to understand whether humans, too, may benefit.

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