These results establish PKR1 via NFATc3 (显示 NFATC3 抗体) as a crucial modifier of mesenchymal-epithelial transition processing to the development of nephron.

show that MRAP2 significantly and specifically inhibits PKR1 signaling.

Data show that the prokineticins and their receptors PROK2 (显示 PROK2 抗体), PKR1 and PKR2 (显示 PROKR2 抗体) contributes to altered sensitivity in diabetic neuropathy and its inhibition blocked both allodynia and inflammatory events underlying disease.

These results suggest PKR1 to be a crucial player in the preadipocyte proliferation and differentiation.

Loss of PKR1 causes renal and cardiac structural and functional changes because of deficits in survival signaling, mitochondrial, and progenitor cell functions in found both organs.

The functional characteristics of coronary endothelial cells depend on the expression of PKR1 and PKR2 (显示 PROKR2 抗体) levels and the divergent signaling pathways used by these receptors.

Study corroborates the clinical relevance of the EG-VEGF (显示 Prok1 抗体) system in human early pregnancy, and provides evidence for the gene-gene interactions of EG-VEGF (显示 Prok1 抗体) and PROKR variants.

hCG (显示 CGA 抗体) increases EG-VEGF (显示 Prok1 抗体), PROKR1 and PROKR2 (显示 PROKR2 抗体) mRNA and protein expression in a dose- and time-dependent manner, demonstrating a new role for hCG (显示 CGA 抗体) in the regulation of EG-VEGF (显示 Prok1 抗体) and its receptors

Findings, together with the detection of sequence variants in PROKR1, PROK1 (显示 Prok1 抗体) and PROKR2 (显示 PROKR2 抗体) genes associated to HSCR (显示 EDNRB 抗体) and, in some cases in combination with RET (显示 RET 抗体) or GDNF mutations, provide evidence to consider them as susceptibility genes for HSCR (显示 EDNRB 抗体).

The number of PKR1 is not reduced in preeclampsia.

Data suggest that smoking targets human Fallopian tubes via nAChRalpha-7 to increase tubal PROKR1, leading to alterations in the tubal microenvironment that could predispose to ectopic pregnancy.

Data show that expression of PK1 (显示 PKLR 抗体) and PKR1 was detected in primary MM cells and myeloma cell lines.

Cow (Bovine) Prokineticin Receptor 1 (PROKR1) interaction partners

PROK1 (显示 Prok1 抗体), acting via PROKR1, may be involved in the recruitment of monocytes to regressing CL and atretic follicles and their consequent activation therein.