TRIM proteins trim the balance of homoeostasis by modulating various biological pathways through protein-protein interactions.

MG53/TRIM72 (zeige TRIM72 Antikörper) protein can be directly applied as a therapeutic agent to increase membrane repair capacity of many cell types.

TRIM acts to chaperone CTLA-4 (zeige CTLA4 Antikörper) transport to the cell surface; this function would be required to account for the phenotypic and functional properties of CD8 (zeige CD8A Antikörper)(+) Tregs.