My ( very limited ) understanding of this makes me wonder if the damage at T11 and T 12 is from the osteoporosis and the damage from L2 through S1 is from the osteoarthritis,, This is a conversation I will have with my PC .Something tells me it will be a long conversation ,
The Pain Management Clinic I am going to does acupuncture and I am hoping it is something that can be included in my treatment plan.
I am also hoping that swimming is something we can work into my exercise plan
I see my PC on the 16th and go back to Pain Management on the 23rd so I have much research to do before then.
I am considering Fosamax as an osteoporosis med.Fosamax has been on the market the longest, seems to have the least side effects and is proven to strengthen bone in the spine But I am also wondering if a shot may be better tolerated than a pill because of my digestive issues ..
Lots to think about

I have not seen my PC yet ,, I see him the middle of March.
My ortho read the report said the disc herniation is mild and not currently needing surgery . and asked if I would go to see a back surgeon about the herniated dics ( for a second opinion) , I said no it would be a waste of time since I will not currently consider surgery .
I was sent to the Pain Management Clinic , the doc there referred to it as Degenerative joint disease .He asked if my PC and I have discussed osteoporosis meds I said he has mentioned it and I see him again in March he said "good plan". I thought Degenerative joint disease was associated with osteoarthritis ??
What the report said ::
""There is old anterior wedging of T11 and T 12 , with focal central compression. A few Schomorl's nodes are incidentally noted .
There are degenerative change with endplate change,particularly L5-S1. There is degenerative signal with in the T11-12 and L2-3 through L5-S1 intervertebral discs.
The visualized spinal cord appears unremarkable , with the conus medullaris ending at about the T12-L1 level.
There appears to be bilteral foraminal narrowing at L2-3 through L5-S1.
at L2-3 there is mild disc bulge and hypertrophic changes posteriorly with narrowing of the spinal canal.
At L3-4 there is mild disc bulge and hypertrophic changess posteriorly with narrowing of the spinal canal
At L4-5 there is mild disc bulge and hypertrophic changes posteriorly with narrowing of the spinal canal
At L5-S1there is mild disc bulge , a smal broad-based left lateral disc herniation into the neural foramina on the left, as well as hypertrophic change posteriorly, with narrowing of the spinal canal
Impression .
Mulitlevel disease L2-3 through L5-S1, including small broad -based left lateral disc herniation into the neural foramina on the left at L5-S1 and narrowing of the spinal canal at L2-3 through L5-S1
Apparent biateral neural foraminal narrowing at L2-3 through L5-S1
Mild old anterior wedging T11 and T12 with focal central compression
Degenerative changes""

I am in the US Any doctor can order the test ,,, but chances are a GP is not going to ,nor will they know the correct test to order.
TG AB & TPO AB are the testing for thyroid autoantibodies that my endocrinologist did when she diagnosed my hashmotos . Mine were so high it made my ANA come back with a moderate positive .

Most medications use corn/corn starch . To totally eliminate corn is nearly imposable but doable if you are extremely strict .When I eliminated corn I did not eliminate my medications and was lucky enough that I did not have to .
Bartfull is correct that soy is one of the big 8 allergens so listed on packaging EXCEPT soy oil . There is a loophole in the law that does not require that soy oil be listed as an allergen on packaging

I finally got my DEXA scan results
AP spine L1-L4 -2.1
dual femur neck left -2.6
dual femur neck right -2.0
dual femur total left -2.0
dual femur total right -1.4
dual femur total mean -1.7
not to bad at all
not bad enough to explain the compression fractures,,( I would not think any way ). I see my doc in March
I am still waiting for a copy of the MRI report ,, it will be interesting to see what that says .

to answer your original question ,, yes you can go from hypo to hyper . I have hashmotos and I bounce around . My endo thinks I may be going into graves ,, always something
I would question the xamthum gum

My ( limited ) understanding of lichen sclerosus is autoimmune with a possible connection to thyroid disease where as Dermatitis Herpetiformis ( DH) is an autoimmune disease associated with celiac disease .
It would be possible to have both lichen sclerosus and dermatitis herpetifomis at the same time
And yes it is very possible to have DH ( celics) with a neg blood work and endoscopy

antibodies can stay active under the skin for up to 2 years after going gluten free , this is why the slightest bit of cross contamination can cause a flair in DH.
The trickiest part of getting a diagnosis by skin biopsy is finding a dermatologist that has even a clue about DH.
A diagnosis of DH is a diagnosis of Celiacs
I would suggest thoroughly researching .dapsone before you consider taking it .
Welcome to the forums
edited to add:
I forgot to mention , you must have active lesions to be tested for DH, the doc needs to take the biopsys from the skin NEXT to the lesions not of the lesions them selves .

gilligan, I have been told basically the same thing ,, I honestly dont know what I am going to do .
I am considering the daily dose instead of the 6 month shot. If I react to something I would rather it was out of my body in a day or so not 6 months

I have Hashimotos .I have been on thryoid meds for about the last 20 + years. I have been on name brand Synthroid since 2002 with no problems .I cannot take the generic ( I bounce around like a rubber ball ) it has to be name brand

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