Polio Notes:

1. Polio is Toxic Poisoning

The Industrial Age was marked by a constant stream of injury and accident, poisoning of the human population by metals, arsenic, and a dozen toxic compounds used in new industry. Chemical poisoning, not ‘viruses’ are the major cause of illness in the modern world:

The Poison Cause of Poliomyelitis And Obstructions To Its Investigationby Ralph R. Scobey, M.D. Syracuse, N.Y.
Statement prepared for the Select Committee to Investigate the Use of Chemicals in Food Products, United States House of Representatives, Washington, D.C.
From Archive Of Pediatrics (April, 1952)

The disease that we now know as poliomyelitis was not designated as such until about the middle of the 19th Century. Prior to that, it was designated by many different names at various times and in different localities.1,2 The simple designations, paralysis, palsy and apoplexy, were some of the earliest names applied to what is now called poliomyelitis.

Paralysis, resulting from poisoning, has probably been known since the time of Hippocrates (460-437 B.C.), Boerhaave,3 Germany, (1765) stated: “We frequently find persons rendered paralytic by exposing themselves imprudently to quicksilver, dispersed into vapors by the fire, as gilders, chemists, miners, etc., and perhaps there are other poisons, which may produce the same disease, even externally applied.” In 1824, Cooke,4 England, stated: “Among the exciting causes of the partial palsies we may reckon the poison of certain mineral substances, particularly of quick silver, arsenic, and lead. The fumes of these metals or the receptance of them in solution into the stomach, have often caused paralysis.”

Colton5 (1850) mentions the case of a patient who swallowed some arsenic accidentally and was admitted to the hospital. The primary effects of the poison had been successfully combated with proper remedies, but seven days afterward he became paralyzed. It is significant to note that there was a latent period of several days before the paralysis appeared since this delayed reaction is comparable to the incubation period in infectious diseases.

Vulpian6 (1879) experimentally produced paralysis of the extensor muscles of a dog by lead poisoning. The lesions, consisting in colloid degeneration and cell atrophy of the anterior horn cells of the spinal cord were pronounced by Vulpian as poliomyelitis. Adamkiewitz7 (1879) reported two parallel cases, one of poliomyelitis and one of lead poisoning.

In 1881, Popow8 of St. Petersburg, published an essay upon the pathological anatomy of arsenical paralysis as produced artificially in animals. The work of Popow was carried out under the guidance of the distinguished neurologist and microscopist, Professor Mierzeyeski. Popow concluded that arsenic, even in a few hours after its ingestion, may cause acute central myelitis or acute poliomyelitis.

During an epidemic of poliomyelitis in Australia in 1897, Altman 9 pointed out that phosphorous had been widely used by farmers for fertilizing that year. This observation may be of significance since in recent years organic phosphorus insecticides, such as parathion, have been suspected as possible causes of poliomyelitis.

Onuff 10 (1900) reported a case of a painter [paints were lead based] with flaccid paralysis of both legs, in whom the autopsy showed lesions characteristic of poliomyelitis.

Gossage13 (1902), writing on infantile paralysis, says: “The nerve cells or fiber may be acutely disabled by the action of some poison circulating in the blood, and it is possible that such poison would only temporarily impair their functions or so seriously affect them that recovery would be impossible.”

Dr. David E. Edsall14 (1907), writing on the pathology of carbon monoxide poisoning in Osler’s System of Medicine, states: “Peripheral neuritis had repeatedly been described and poliomyelitis and disseminated encephalitis have been seen.”

Collins and Martland15 (1908) reported a case of poliomyelitis in a man, 38 years of age, which resulted from the use of cyanide as a silver polish. The illness began with diarrhea, followed by headache and pain and stiffness in the back of the neck. About eight days after the onset of the illness, he became paralyzed. In discussing Collins and Martland’s paper, Larkin stated that he had seen one instance of this disease following potassium cyanide poisoning.

In the spring of 1930, there occurred in Ohio, Kentucky, Alabama, Mississippi and other states an epidemic of paralysis.16,17 The patients gave a history of drinking commercial extract of ginger [Jaked Ginger, with organophosphates]. It is estimated that at the height of the epidemic there were 500 cases in Cincinnati district alone. The cause of the paralysis was subsequently shown to be triorthocresyl phosphate in a spurious Jamaica ginger. Death resulted not infrequently from respiratory paralysis similar to the bulbar paralysis deaths in poliomyelitis. On pathological examination, the anterior horn cells of the spinal cord in these cases showed lesions similar to those of poliomyelitis.

Vaccination was on the rise in the 30s, 40s; and vaccination for pertussis and other ailments also Cause polio:

Dr. Ralph Scobey:

In 1936, during a campaign to eliminate yaws in Western Samoa by the injection of arsenicals, an epidemic of poliomyelitis appeared simultaneously.23

In one community all of the patients developed paralysis in the same lower limbs and buttocks in which they had received the injections and this pattern was repeated in 37 other villages, whereas there was no paralysis in uninoculated districts. The natives accused the injections as the cause of the epidemic of poliomyelitis. Most of the cases of paralysis occurred one to tow weeks after the injection of the arsenic.

The foregoing reports indicate that poisons can cause poliomyelitis. It would appear that not any one poison in particular would be responsible for all cases of poliomyelitis but the effect of any one of several could produce the same ultimate result. When a disease is known to be caused by a poison, it is obvious that a search for a germ or virus in relation to it would not be made. Conversely, if a so-called virus is believed to be associated with the disease, then the possibility of poisoning as the cause of the disease would not be considered. It will be shown, moreover, that some so-called virus diseases and virus inclusions can be caused by poisons.

All of this was KNOWN. And ignored.

The Vaccine movement, promoted by the FDR Whitehouse, promoted actively, supported DDT spraying and soaking of infant’s rooms, clothes, and of children at the beach:

Following a recent extensive trip through the South, Dr. Mobbs informed me that wherever DDT had been used intensively against polio, not only was there an epidemic of the syndrome I have described but the incidence of polio continued to rise and in fact appeared where it had not been before. This is not surprising since it is known that not only can DDT poisoning produce a condition that may easily be mistaken for polio in an epidemic but also being a nerve poison itself, may damage cells in the spinal cord and thus increase the susceptibility to the virus.

In this connection, an observation reported by Dr. Albert B. Sabin in the Journal of the A.M.A. in June 1947 is significant: “Since the end of combat in the Phillipines, poliomyelitis has been among the leading causes of death in American troops. Even though only the paralytic cases are reported from there, the incidence of poliomyelitis in American troops in the Phillipines has been at least ten times as high as in the Army within the continental limits of the U.S. during the past two years. Actually, I believe that it is even higher because hundreds of cases which would have been diagnosed as dengue-like or sandfly-like fevers… under conditions which , in my opinion, would preclude the occurrence of both dengue and sandfly fever. And yet checks of the surrounding native population revealed no outbreaks of poliomyelitis.”

US Department of Agriculture found that although fodder treated with DDT caused no damage to the cows eating it, the health of their calves was severely impaired, sometimes with fatal results. The DDT was passed along from cow to calf via the milk (Van Nostrand’s Encyclopedia of Science and Engineering, Van Nostrand Reinhold 1995, v 5, p1775). DDT is a neurotoxin and the calves developed something very much like infantile poliomyelitis. Calves weren’t the only infants drinking cow milk during the early 1950s.

In 1908 two scientists working in Austria, Karl Landsteiner and Erwin Popper, reported that they might have found an ‘invisible virus’ that had caused these epidemics. They had made their discovery, they claimed, after making a suspension in water of minced diseased spinal cord from a nine-year-old victim of infantile paralysis. They had tested this noxious suspension by injecting one or two cups of it directly into the brains of two monkeys. The monkeys fell severely ill (as might have been predicted). One died and the other had its legs paralysed. The scientists then dissected the monkeys and found damage in their central nervous tissues similar to that found in human cases of infantile paralysis.

Nevertheless, the following year Simon Flexner and Paul Lewis of the illustrious Rockefeller Institute for Medical Research in the US ‘proved’ a similarly made noxious soup was ‘infectious’ by injecting it into the brain of one monkey. They then extracted some fluid from its brain, injected this into another monkey, and so on through a series of monkeys, paralysing all of them in the process. Flexner and Lewis reported: ‘We failed utterly to discover bacteria… that could account for the disease [paralysis]… The infecting agent of epidemic poliomyelitis [probably] belongs to the class of the minute and filterable viruses that have not thus far been demonstrated with certainty under the microscope.’

In other words, we’ve injected a cocktail of viruses, cellular debris and DNA into a series of monkeys, and we believe that a virus, not yet identified within this noxious cocktail, is responsible! The procedure of Flexner and Lewis was just as dubious as their conclusion: they took no account of the contaminants in their mashed-up soup; they presumed what happened in monkeys would be replicated in humans; and surprisingly, given the evidence around at the time, they didn’t inject samples of cyanide or lead into the brains of monkeys to see if they also caused paralysis. In 1910 neonatologist L Emmett Holt reported: ‘Even five years ago if anyone had suggested that the disease under discussion was an infectious or contagious one, it would have been looked upon as a joke.’

From contemporary reports there were nine times more polio cases in 1957 than in 1956, and that they were more serious than ever before. In the first 8 months of 1957 the Public Health Service reported, out of a total of 3,212 polio cases, there were 1,055 cases of paralysis, or 33.5% of the total. From January 1st to August 1958 there was a total of 1,638 cases of polio, with 801 of them paralytic, or 49% of the total.

An Associated Press Dispatch from Boston on August 30, 1955, reported 2,027 cases of polio in Massachusetts against 273 the same time the previous year-representing an increase of 743%. This followed the inoculation of 130,000 Massachusetts children, and the authorities banned the vaccine. Connecticut reported 276 cases in 1955, up from 144 in 1954; Vermont, 55 up from 15; Rhode Island, 122 up from 22, and Maine, 74 up
from 43.

The Washington D.C. Star, September 20, 1955, reported 180 cases in Washington against 136 the same time in 1954; Maryland’s Health Department reported 189 in 1955 to 134 in 1954; New York State, 764 to 469; Wisconsin, 1655 to 326. The Milwaukee Journal, on August 30, 1955, reported that the city’s schools closed indefinitely because of the polio outbreak, following inoculation with the Salk vaccine.

Idaho stopped Salk inoculations completely on July 1, 1955, with this blast from State Health Director Peterson said, “I hold Salk vaccine and its manufacturers responsible for a polio outbreak that has killed 7 Idahoans and hospitalized 79.” By September 14th 1955, that state had 190 cases compared with 132 for the entire year of 1954. Newark, N.J. stopped inoculations in June, 1955, while Utah took similar action on July 12.

An Associated Press dispatch on November 11, 1955, quoted Dr. Herbert Ratner, Health Commissioner of Oak Park, Illinois, who said that “English authorities in July, 1955, canceled the Salk vaccine programs as ‘too dangerous’, and all European countries, with the exception of Denmark, have discontinued their programs.” Canada also postponed its Salk vaccine program on July 29, 1955.

The New York Times on May 11, 1956, reported on Supplement No. 15 of the Poliomyelitis Surveillance Report for the year which showed 12% more paralysis in 1956 than in 1955. By January 1, 1957, 17 states had rejected their supplies of Jonas Salk’s “anti-paralytic” polio vaccine.

During this year very nearly half the paralytic cases and three-quarters of the non-paralytic cases in children between the ages of 5 and 14 years occurred in “vaccinated” children.

“It was noted in the Union of South Africa and in the USA, especially in the course of severe outbreaks in Hawaii and Chicago, that vaccination in the face of an epidemic did not appear to shorten its course. Laboratory and field studies have shown that vaccination does not prevent infection or interfere with dissemination of virus in the community.”

Polio was relabeled ‘aseptic meningitis,’ and the criteria for defining ‘polio’ was changed, so that it was very difficult to give anyone a ‘polio’ diagnosis: [Krassner]

After 1955, NON-paralytic polio also acquired a new name. It wasn’t until the mid-1950’s that new laboratory techniques of culturing viruses could distinguish THIS polio from its clinical twin, aseptic meningitis. Before 1960, not a single case of “aseptic meningitis” was reported. Then, it was called (non-paralytic) “polio”, and nationally had totaled 70,083 between 1951 and 1960. But from 1961 to 1992, there had been 220,365cases of aseptic meningitis. There were only 589 cases of non-paralytic polio from 1961 to 1982. Not a single case has been reported since.

Non-paralytic polio may have “disappeared”. But thousands of children still experience the same symptoms as non-paralytic polio every year. Except now, it goes by another name.

Besides the name game, the (presumed) decline in polio due to the Salk vaccine was also an artifact of diagnostic methodology. Prior to 1954, the diagnosis of spinal paralytic poliomyelitis followed the World Health Organization definition: “Signs and symptoms of nonparalytic polio, with the addition of partial or complete paralysis of one or more muscle groups, detected on two examinations at least 24 hours apart.” But beginning in 1955 following the introduction of the Salk vaccine, the criteria changed to conform more closely to the definition used in the (fraudulent) 1954 Salk field trials: “Unless there is residual involvement (paralysis) at least 60 days after onset, a case of poliomyelitis is not considered paralytic.” Laboratory confirmation was possible after 1955, but not required for diagnosis. Obviously, more cases of paralysis had a chance to recover within 60 days, than in 24 hours. As intended, paralytic polio decreased by 23,500 cases from 1955 to 1957. However, after 2 years of widespread uptake of the Salk vaccine, paralysis increased about 50% from 1957 to 1958, and about 80% from 1958 to 1959. (The Sabin oral vaccine supplanted Salk’s by 1961.)

It’s necessary to remember that polio was viewed as mostly, 90 to 95%, non-paralytic.

Other holes started to appear in the virus theory. During WWII army doctors found widespread immunity to the suspected poliovirus, and no evidence of infantile paralysis epidemics, in the Middle East, Asia and Africa. In Turkey they found people who called infantile paralysis ‘the American disease’. The doctors were surprised: immunity to the virus presumably meant that it had infected the population. So, how come it caused no epidemics in these countries?

Salk claimed that his vaccine protected ‘30 to 90 per cent’ of those who received it (a remarkably vague statistic). But more than 60 per cent could have been immune already, at least according to the theory of the US federal agency the Centers for Disease Control and Prevention (CDC) that working-class children were already immune as a result of exposure to the virus in dirt. It is not known if Salk ever checked to see if children were already immune before he vaccinated them, but Hilary Koprowski reported in 1957 that the inhabitants of the Congo were 85 per cent immune before they ever saw a dose of polio vaccine. (Amazingly this didn’t stop Koprowski. He went on to uselessly administer to them hundreds of thousands of doses of his experimental vaccine.)

Also, it should be noted that most cases of “polio” today are, in fact, not ‘associated’ with the little stomach bug (a tiny particle, about 1/1000th the size of a cell). These cases, the majority of paralysis occuring, are called “non-poliomyelitis acute flaccid paralysis,” or, Non-polio polio!

But they still don’t want to look at toxicology. Because, as a wise man once said, “You can’t sue a virus.”

So, why do we vaccinated everyone in the world against a bug that doesn’t cause illness?

And what are we ‘vaccinating’ them with?

What are the proteins, contaminants, bacteria, mycoplasma, heavy metals, adjuvants, and other contaminants? And what do they do in the body?

Here’s an answer:

Polio incidence increased after vaccination. Polio Vaccination was considered a morbid disaster of ruinous proportions in the 50s. Paralysis rates shot up exponentially. Children were quickly paralyzed, and in the arm WHERE they were injected.

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Philosophy

Before there is anything and beneath everything - is mystery. As soon as you name it, you lose it. As soon as you define it, it disappears. The reality: we can't know the eternal; we can only live in a system of the gentlest ethics: Be kind to others, try to do no harm to people and the world.