The CDC addressed the slim potential for these viruses posing a public health threat in their 2014 Bat Flu FAQ.

How could bat flu viruses become capable of infecting and spreading among humans?

Because
the internal genes of bat flu viruses are compatible with human flu
viruses, it is possible that these viruses could exchange genetic
information with human flu viruses through a process called
“reassortment.”

Reassortment occurs when two or more flu viruses infect a
single host cell, which allows the viruses to swap genetic information.
Reassortment can sometimes lead to the emergence of new flu viruses
capable of infecting humans.

However, the conditions needed for reassortment to occur
between human flu viruses and bat flu viruses remain unknown. A
different animal (such as pigs, horses, dogs or seals) would need to
serve as a “bridge,” meaning that such an animal would need to be
capable of being infected with both this new bat flu virus and human flu
viruses for reassortment to occur. Since the discovery of bat flu, at
least one study has been conducted to assess the possibility of
reassortment events occurring between bat flu and other flu viruses (3).

So far, the results of these studies continue to indicate that bat flu
viruses are very unlikely to reassort with other flu viruses to create
new and potentially more infectious or dangerous viruses. In their
current form bat flu viruses do not appear to pose a threat to human
health.

Although unlikely, one plausible scenario would involve the reassortment of a bat and a mammalian virus in an intermediate host, such as a pig.

However, in 2014 we saw a report (see Nature Comms: A Chimeric Bat Flu Study) that reassuringly suggested that the H17N10 subtype may have a difficult time making the leap from chiropterans to humans.

Today we've a report published in the Journal of General Virology, where researchers in China forced the reassortment of the NS1 gene from H17N10 bat flu virus with a human H1N1 virus, proving reassortment is possible, although still very unlikely to happen in nature.

Influenza A viruses have the potential to
cause pandemics due to the introduction of novel subtypes against which
human hosts have little or no pre-existing immunity. Such viruses may
result from reassortment between human and animal influenza viruses.
Recently, new influenza-like viruses were identified in bats, raising
the concern for a new reservoir of potential harmful influenza viruses
that could form reassortants with categorized human influenza A viruses.

However, up till now, no one was able to generate a recombinant
reassortant virus containing a single functional gene or domain from
H17N10 that could propagate.

Here, we demonstrate that a recombinant
A/Puerto Rico/8/1934 (H1N1) virus with NS1 gene from H17N10
influenza-like virus can be successfully rescued. Furthermore, we used
luciferase reporter assays and qRT-PCR to show that the NS1 protein from
H17N10 inhibited Sendai virus (SeV) induced activation of IFN-β
expression with an efficiency similar to NS1 from an H5N1 strain.
Moreover, the crystal structure of the NS1 (H17N10) RBD is also similar
to other NS1s.

These results demonstrate that H17N10 influenza-like
virus indeed contains functional genes that are compatible with
categorized influenza A viruses.

Although the chance of this particular
event occurring in nature seems negligible, further research is needed
to address the possibility of the natural formation of reassortments.

Between the emergence of SARS and MERS (both linked to bats), Nipah, Ebola, and now the discovery of bat flu, the past couple of decades have turned out to be busy ones for Chiroptologists (scientists who study bats).

These winged mammals are increasingly viewed as naturals hosts
for, and potential vectors of, a number of newly recognized emerging
pathogens.

All of which makes bats, and bat habitats, something to be avoided. To learn how you can stay safe around bats, the CDC offers the following advice.