P311 may promote the migration of ESCs both in micewith superficial partial-thickness burns and in an injured cell model in vitro, and it may play an important role in wound healing.

Protein HYI may closely bind with protein P311 by an alpha helix in hypertrophic scar fibroblasts.

P311 and ITGB4BP expression was elevated in non-small cell lung cancer, possibly indicative of a new signaling pathway.

P311 may be involved in the pathogenesis of hypertrophic scar via induction of a myofibrobla

role for P311 in inducing TGF-beta1-independent myofibroblast transformation

These results suggest a role for levels of P311 in regulating glioma motility and invasion through the reorganization of actin cytoskeleton at the cell periphery.

P311 expression is tightly regulated during the critical periods of alveolar formation, and that under pathologic conditions, its relative absence may contribute to failure of alveolar regeneration and lead to the development of human emphysema.

P311 is the first protein identified to modulate TGF-beta translation and the first pan-regulator of TGF-beta expression.

P311 may promote the migration of ESCs both in micewith superficial partial-thickness burns and in an injured cell model in vitro, and it may play an important role in wound healing.

role for P311 in inducing TGF-beta1-independent myofibroblast transformation

P311 expression is tightly regulated during the critical periods of alveolar formation, and that under pathologic conditions, its relative absence may contribute to failure of alveolar regeneration and lead to the development of human emphysema.