Saturday, 1 October 2011

‘Gay’ AIDS and the Hepatitis-B Vaccine: AIDS the Real Story Part 5

The hepatitis-B vaccine was administered to high-risk groups in the West – such as gay males and intravenous drug users – coincident(?) with the onset of AIDS in the U.S.A..[1]

“The AIDS virus [sic] began to appear in homosexuals around 1979. This was immediately following tests of the first hepatitis vaccine,” said J. Anthony Morris, a leading virologist who had worked with the National Institutes of Health (NIH), Walter Reed Hospital and for thirty-five years with the US Food and Drug Administration, in connection with research on vaccines for influenza and other respiratory diseases.

The hepatitis-B vaccine was produced using infected blood taken primarily from homosexual males. Information on HIV in the West prior to the early 1980s comes from the detection of antibodies in blood samples stored as part of a San Francisco study of hepatitis-B among homosexual men.[2]

The first tests of the vaccine were conducted under unusual conditions. Dr W. Schmugner, head of the New York Blood Bank, set up the rules for this study. Only homosexual males between the ages of twenty and forty who were not monogamous could participate in the tests of the vaccine, which took place principally in San Francisco and New York. Soon afterward, AIDS was first detected among gay males in these areas.

The Centres for Disease Control (CDC) reported in 1981 that four percent of those receiving the hepatitis-B vaccine were AIDS infected. This was prior to the advent of testing kits for HIV (Human Immuno-deficiency Virus). In 1984 the CDC reported that sixty percent of those involved in the hepatitis-B program had developed AIDS.[3]

“I wrote a letter to… [the] director of the CDC in Atlanta,” said Dr Morris. “I asked him, with all the evidence that he had at his disposal, to convince me that there is no causative connection between the introduction of the experimental hepatitis vaccine into the homosexual population and the occurrence of AIDS in that same population. He wrote a letter back saying that he believed there was no connection, but that the convincing evidence was just not available.”

The hepatitis-B vaccine was administered to millions of people in high risk groups; particularly gay males and I.V. drug users. There was subsequent debate over whether the HIV retrovirus was properly removed from hepatitis-B vaccine (Heptavox-B, a human blood product). The absence of HIV antibodies in Heptavox-B vaccines recipients “does not rule out the physical presence of AIDS virus antigen in the vaccine,” said Albert L. Maric of Metairie, Louisiana, in the Journal of the American Medical Association (16-1-1987). Other objections rested on the fact that retroviruses can remain dormant (and can’t be found in tests) for periods ranging up to decades.

Early Warning

Is it possible that the World Health Organisation had ample warning of the potential of retroviral contamination of their vaccines as early as 1972? Their own records show that this is the case. Contamination of polio vaccines with the carcinogenic SV-40 virus (see earlier portions of this document) was one precedent in the early 1960s.

In the Bulletin of the World Health Organisation, 1972, there is a pair of Memoranda on Virus-associated immunopathology: animal models and implications for human disease. These memoranda are reports on research – and WHO recommendations for further research – on the “Effects of viruses on the immune system, immune-complex diseases, and antibody-mediated immunologic injury.” This Bulletin reveals much about viruses, but recommendation three (p. 259) deserves scrutiny:[4]

“An attempt should be made to see if viruses can in fact exert selective effects on immune function, e.g…. by affecting T cell function as opposed to B cell function. The possibility should also be looked into that the immune response to the virus may itself be impaired if the infecting virus damages more or less selectively the cells responding to the viral antigens. If this proves to be the case, virus-induced immunodepression might conceivably be highly instrumental in prolonging certain virus infections, such as murine leukaemia, hepatitis, sub-acute sclerosing panencephalitis, or infections caused by LDV, LCMV, or ADV.”

This is a description of something very close to AIDS, if not AIDS itself – and this was written in 1972, years before the advent of AIDS, as a direct recommendation for further research into magnifying the debilitating effects of diseases, by compromising the immune system. A description of AIDS as accurate as this was hard to formulate even in the early 1980s. A few lines later comes the statement: “Recently, it has been shown in animals that viruses can persist in the bloodstream in the form of virus-antibody complexes.” On the next page:

“To date, virus-induced immune-complex disease has been attributed to the deposition of virus-antibody complexes during persistent virus infections.” Were these disease conditions induced in humans by vaccination programs using unsterilised (and blood-contaminated) syringes, filled with usually unrefrigerated serum, contaminated with simian (and other species’) viruses and retroviruses – in Third World populations already affected by endemic diseases?

On page 261 is Recommendation Seven:

“Animals with infections characterized by persistent or recurrent viraemia (e.g. feline leukaemia, African swine fever, hog cholera and avian lymphomatosis) should be examined for anitiviral antibody circulating virus-antibody complexes, and immune-complex nephritis.” Some of these animal diseases recommended for study are caused by retroviruses – but in those days they would have been known (if recognised at all) as ‘slow viruses’ (lentiviruses) or simply RNA viruses, as mentioned on page 269 of the Memoranda. A recommendation notes the “susceptibility to RNA virus” of ‘H-2’, the major immune system of mice. It also mentions studies “in progress” to determine if the corresponding major human immune system (human-leukocyte-associated or HL-A) was affected by diseases caused by similar viruses. These studies were also recommended in a report on a workshop held at the National Institutes of Health in 1970, which was co-sponsored by the WHO.[5]

This report also suggests “a consideration of viral and/or immune response factors leading to disease. Thus, investigation of diseases with suspected viral etiology, such as leukaemia, lymphoma, and other reticuloses (including Burkitt’s lymphoma) is indicated…” These diseases have forms since found to be associated with or caused by retroviruses.

On the next page we find normal scientific procedure meant that some people were to be given placebos instead of vaccine, to provide controls. “Minimum controls should include normal individuals of the same race, age, and environment as the patient population. Family studies are strongly recommended for two reasons, a) the generation of an unaffected sibling control group and b) the correct ascertainment of HL-A inheritance.”

This shows that research sometimes came before patient welfare in the vaccination programs conducted by the WHO in the early 1970s. Unwitting ‘Third World’ control patients who hadn’t been vaccinated were surrounded by people infected with live smallpox virus! The following independent study is published in the same Federation Proceedings:

“Mass immunization of school children against influenza was used successfully (Monto, et al., WHO Bulletin 41: 537, 1969) to reduce the intensity of a subsequent community epidemic;

“In spite of this knowledge, routine immunization of healthy children is still not recommended, largely because even highly purified, whole virus vaccines continue to prove unacceptably reactive in this young age group” (Foy, et al., WHO Bulletin 41: 537, 1969). The age group mentioned is 12 to 48 months.

The Parke, Davis & Company’s own researchers were recommending that whole virus vaccines should definitely not be given to young children – even healthy young children – a fact known to the WHO since 1969. Yet these reports and recommendations went unheeded – at least in WHO programs in the ‘Third World’, where live, whole virus vaccines were extensively given to young children.[6]

While both the above Memoranda and the NIH workshop report were written by committees of researchers from a variety of academic institutions, only one person co-authored all the reports – Howard C. Goodman, Chief Medical Officer, Immunology, WHO. The WHO did have advance warning of many problems caused by vaccination, including contamination of vaccines with dangerous organisms (see prior sections detailing SV-40 in polio vaccines).

One pathology study supported by an NIH grant in 1972[7] set out to determine the “Theoretical Lowest Size Limit of Spherical Animal Viruses”. It did more than that:“Analysis of available electron microscopy, X-ray diffraction and chemical data as well as the geometrical requirements of the icosahedral shell indicates that the lowest size limit for a spherical virus is 142.6 A.U. (Angstrom Units) in maximum diameter (MD). Such a virus would have a 25 A.U. thick protein shell built of a single molecular type having 60 morphological subunits, 30 A.U. MD, protecting a single-stranded RNA core… Without capsomeres, this virus could enter the cell only by phagocytosis and it would probably be defective requiring a larger virus for its replication. The theoretical virus would be visualized in ultra-thin sections (less than 500 A.U. thick)…”

Compare this theoretical 1972 prediction with the following 1987 description of HIV-1 – the AIDS virus:[8]

“It seems that the core shell is composed of protein that is arranged in an icosahedral structure…

“Sifting through hundreds of different electron micrographs of the virus, Marx and Munn proposed that the core shell was I fact a complicated shape called a deltaicosahedron. This is a polygonal structure composed of 60 triangular elements forming a mix of alternating hexagonal and pentagonal structures which partly interpenetrate each other…”

“HTLV-III virion, or virus particle, is a sphere that is roughly 1,000 angstrom units across,” according to Dr Robert Gallo,[9] placing HIV well within an order of magnitude of the NIH-sponsored study’s theoretical virus – and much larger than what the NIH/WHO knew was the minimum possible size for a virus in 1972. Further, the core of the ‘hypothetical’ virus was predicted to be composed of RNA.

As far back as 1963, scientific institutions knew that a number of cancers were caused by viruses. One 1963 article from the Salk Institute of Biological Studies said that “Viruses are interesting objects for an experimental study of cancer because many aspects of their interaction with the cells are fairly well understood…

“There are two main types of cancer-producing viruses… The viruses of one group contain RNA as the genetic material, the viruses of the other group contain DNA. The RNA-containing viruses produce leukaemias and solid tumors in chickens and rodents… the RNA is surrounded by protein units… included in a membranous, lipid-containing envelope… these viruses perform many unknown functions in the infected cells.

“The DNA viruses are made up of an outer icosahedral shell of protein units…”[10]

If we combine the geodesic shell of oncogenic (carcinogenic) DNA viruses (like SV-40 and some leukaemia viruses) with the outer membrane and core genetic material of the oncogenic RNA viruses (including ‘slow viruses’ like Visna) – we pretty much have the retrovirus known as HIV. HTLV-1, discovered by Gallo in 1978, also has a geodesic shell surrounded by a fatty (lipid) membrane.

With all these requests by the WHO, the NIH and other organisations for research into ‘RNA viruses’ (retroviruses) and their effects on the human immune system from the 1960s, it’s hard to believe such well-directed research could all have come to nothing. Much work was conducted and directed by these bodies on animal virus modeling of human immune deficiency diseases around 1972 - coincidentally the year the CIA destroyed all their biological warfare records, by order of President Nixon.

Lessons of History

If the WHO didn’t realise the potential side-effects of using impure vaccines by the time they started immunising hundreds of millions of people in the developing world, they certainly should have. Vaccines and blood banks should have been screened for organisms whose existence had already been postulated (in NIH/NCI-backed studies). After the debacle of SV-40, they should have learned the lesson of then-recent history.

To this day, vaccines and blood supplies are still only screened for a handful of known organisms.

According to William Campbell Douglass, M.D. (of the National Health Federation, Monrovia, Ca.), the WHO used smallpox vaccine for sibling studies mentioned in the 1972 Proceedings and the geographic sites chosen were Uganda and other African states, Haiti, Brazil and Japan. The epidemiology of AIDS perfectly coincides with the same geographic areas as this program, according to Douglass and others.

These areas also coincide with the area contaminated with adult T-cell leukaemia (ATL), according to Dr Robert Gallo (the official discoverer of HIV). Gallo and his team used the blood of people suffering from this form of leukaemia to manufacture large quantities of the HIV/AIDS retrovirus, which he called HTLV-III. He had already discovered HTLV-1, the ‘first human retrovirus’ considered responsible for ATL, in the blood of Japanese and Haitian ATL sufferers. The spread of HTLV-1 seems to closely match that of HTLV-III/HIV.

The US Public Health Reports for February 1972 mentions a “Survey of 1953-55 Deaths” that related to childhood malignancies:

“A detailed study of official statistics of mortality preceded the survey of 1953-55 deaths. According to these statistics the risk of dying from a malignant disease after the age of 40 has barely altered in recent years, but the risk of children and young adults dying from these diseases has appreciably increased. In particular, children between 2 and 4 years of age have been more affected by the unfavorable trend of leukaemia mortality than any other age group under 70 years.

“…the recent increase in leukaemia deaths happened sooner in technically advanced countries.” And the article goes on to state that the determining factor was not affluence but “the availability of medial services”!

An examination of statistics shows that after a small blip in 1945, the rise of these diseases began in 1953 and kept growing. A 1956-1960 study noted an increase in leukaemia in a group of children under 5 who showed a remarkable prevalence of mongoloids.[11] Were these more early signs of leukaemia viruses introduced into pregnant women and children (affecting chromosome groups 21 & 22 by vaccines and other medical products? If not for the determining factor being access to medical treatment, one could believe that atmospheric nuclear testing was responsible for the increase during the periods under examination.

A controversial 1986 study by British geneticists seemed to demonstrate that HTLV-III (HIV) evolved primarily inside Negroid blood and lymph systems and is therefore well adapted to them. The next section of this work – detailing the advent of AIDS in Africa – may help dispel a few myths and illuminate a few hidden corners of history.

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