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Incidence of ICD in the Russian Federation (2005-2016)
Screening for prostate cancer: modern presentation and organization
Primary multiple transitional cell epithelium
Androgenic screening in men over 50 years

The role of stem cells in the treatment of urinary incontinence
Percutaneous nephrolithotripsy in infected urine
Testicular Prosthesis in Children and Adolescents: Results from a Multicenter Study
Premature ejaculation is the current state of the problem.

The prevalence of symptoms of impairment of the function of the lower urinary tract in men according to the results of a population study
Patient-centered system of organization of medical care in urology using...

Principles of «4P Medicine» in the organization of health care in the context of urological diseases
Changes in the electrolyte composition of urine under the influence of sodium hypochlorite. The possibility
of reducing the risk of recurrence of nephrolithiasis

Expression of molecular markers of the acute kidney injury in the dynamics of experimental ischemia

Nowadays the role of the organ-sparing
kidney operations is growing, which is
related to the prevention of the chronic
kidney disease progression. The
optimization of the diagnosis and
prognosis of the development of acute
kidney injury (AKI) is linked to the
detection of certain biological markers.
Among them are neutrophil gelatinaseassociated
lipocalin-NGAL (lipocalin-2),
interleukin-18, molecule of the renal
damage-1 (3-NHE-3) and the enzymes
localized normally in the brush border
of the proximal channels, and some
other parameters.

The aim of the study was to study the
level of the AKI biomarkers and the
functional parameters of the cortical
cells in the kidneys of rats of different
age and gender in the dynamics of acute
ischemia. In the experiment 130 male
and female inbreed rats were used with
the body mass of 180-200 g (young
animals) and 300-350 g (mature animals).
The IFA analysis was done in the cortical
tissue of the ischemic kidney with
regard to expression of cystatin C,
interleukin-18, lipocalin-2, L-FABP and
KIM-1. It was shown, that in the
cortical tissue of the ischemic kidneys
in young males and old females early in
the course (already 15 min after
beginning) of ischemia accumulate
cystatin C, interleukin-18, and L-FABP,
in old males – this processes occur later
(in 20 min). The alterations of the KIM-1
and lipocalin-2 levels could be observed
even later. It is worth saying, that most
sensitive to the ischemic damage were
young male and old female rats,
following by old males. The most
tolerant were young females.