Abstract

Clinical aphasiologists have long recognized that repetition is found in the discourse of speakers with acquired neurogenic communication disorders. Examples of repetition associated with pathology may include echolalia, perseveration, stereotypies, false starts, and recurrent digression. These types of repetition are often interpreted as signs of poor inhibition of undesired responses or poor activation of desired responses, e.g., as associated with anomia. What is typically not addressed in clinical research is the relative degree to which similar categories of performance errors are also found in the discourse productions of non-brain-injured communicators.