星期五, 7月 26, 2019

摘要显微外科引起冠状假性频尿柱 克里斯蒂塔)无小核系中,宗派细胞因口围带翻袖部小膜涌现形形色色的以任何方式的缺点而领到变形;在细胞研制安息期的群体中,口围带变形率高达50%从一边至另一边;对无小核系600多个刚使完满二离开的前、后费力查出和1 1000多种虫生理残余相蛋白质的银染,除4只二歧化费力查出和3只二歧化费力查出喃喃地说周带非常外,压倒的多数因排队发作后的细胞都具有合格的的胞口建筑物.在另一方面,专一性培育试验更远的显示出,喃喃地说周变形发作在非排队发作阶段。,损失小核的细胞,同时也损失了胞口建筑物的稳定性.To explore the generality of the morphogenetic function of the micronucleus among ciliates, we employed five stocks of hypotrichous ciliate Pseudourostyla 克里斯蒂塔 : 2, 4, 6, 28 and 46, and five a小核 cell lines:2aml, 4aml, 6aml, 28aml and 46aml derived from stocks 2, 4, 6, 28 and 46 respectively, which were generated by amputation of the cell when the micronuclei became aligned on the left side of the amalgamated macronuclei during cell 机关。 小核 cell lines 2ml, 4ml, 28ml and 46ml derived after amputation but retaining the micronuclei were also studied and served as 把持。 For observation of the cortex and nuclei, protargol staining was used 从头到尾。 The evident character of the a小核 cell lines, as compared with the normal and the 小核 cell lines, was that a substantial proportion of cells exhibited an abnormal adoral zone of membranelles （阿兹姆） When the culture medium began to age after 7～10 days, the culture soon entered stationary phase of 增长。 During the period abnormal AZMs were observed in over 50% of a小核 cells in various cell lines, among which the percentage of 6aml cells with defective AZM came up to 98%. In fact, the percentage of cells with abnormal AZM in a culture population was changeable and varied from low to high from exponential growth phase to stationary 阶段。 These abnormal patterns involved various degrees of reduction of membranellar width in the anterior and middle part of the AZM lapel （阿兹姆 L), and in severe cases disruption of AZM continuity due to the loss of some membranelles, and also indentation of the cytoplasm in this 区域。 Morphogenesis of the a小核s in binary fission and in physiological reorganization was 说。 Restructuring of the pre existing oral apparatus, both during binary fission in the putative proter and during physiological reorganization, resulted in the formation of a normal 阿兹姆。 After restoration of normal AZM in binary fission or physiological reorganization, abnormal AZM pat