The Vaccine Damage – Science

This listing takes a look at some of the unbiased selections of vaccine science that show an entirely different story than the vaccines are safe and effective jargon that we are typically told. This selection explores the propaganda filled myths and as well the vaccine related misinformation we have repeatedly been fed.

Also see here in the below vacfacts link, more lists of vaccine studies showing the harm of vaccines and the connection to ASD, and much more.

Slow CCL2-dependent translocation of biopersistent particles from muscle to brain

Abstract

Background

Long-term biodistribution of nanomaterials used in medicine is largely unknown. This is the case for alum, the most widely used vaccine adjuvant, which is a nanocrystalline compound spontaneously forming micron/submicron-sized agglomerates. Although generally well tolerated, alum is occasionally detected within monocyte-lineage cells long after immunization in presumably susceptible individuals with systemic/neurologic manifestations or autoimmune (inflammatory) syndrome induced by adjuvants (ASIA).

Methods

On the grounds of preliminary investigations in 252 patients with alum-associated ASIA showing both a selective increase of circulating CCL2, the major monocyte chemoattractant, and a variation in the CCL2 gene, we designed mouse experiments to assess biodistribution of vaccine-derived aluminum and of alum-particle fluorescent surrogates injected in muscle. Aluminum was detected in tissues by Morin stain and particle induced X-ray emission) (PIXE) Both 500 nm fluorescent latex beads and vaccine alum agglomerates-sized nanohybrids (Al-Rho) were used.

Results

Intramuscular injection of alum-containing vaccine was associated with the appearance of aluminum deposits in distant organs, such as spleen and brain where they were still detected one year after injection. Both fluorescent materials injected into muscle translocated to draining lymph nodes (DLNs) and thereafter were detected associated with phagocytes in blood and spleen. Particles linearly accumulated in the brain up to the six-month endpoint; they were first found in perivascular CD11b+ cells and then in microglia and other neural cells. DLN ablation dramatically reduced the biodistribution. Cerebral translocation was not observed after direct intravenous injection, but significantly increased in mice with chronically altered blood-brain-barrier. Loss/gain-of-function experiments consistently implicated CCL2 in systemic diffusion of Al-Rho particles captured by monocyte-lineage cells and in their subsequent neurodelivery. Stereotactic particle injection pointed out brain retention as a factor of progressive particle accumulation.

Conclusion

Nanomaterials can be transported by monocyte-lineage cells to DLNs, blood and spleen, and, similarly to HIV, may use CCL2-dependent mechanisms to penetrate the brain. This occurs at a very low rate in normal conditions explaining good overall tolerance of alum despite its strong neurotoxic potential. However, continuously escalating doses of this poorly biodegradable adjuvant in the population may become insidiously unsafe, especially in the case of overimmunization or immature/altered blood brain barrier or high constitutive CCL-2 production.

Aluminum Adjuvant in Vaccines Causes Risk to Children According to New Journal Report

A new Canadian study of the mechanisms of aluminum adjuvant toxicity in pediatric patients confirms that immune challenges during early development, including those vaccine-induced, can lead to permanent detrimental alterations of the brain and immune system function. Lucija Tomljenovic, PhD and Christopher A. Shaw, PhD of the University of British Columbia’s evidence-based study was recently published in Lupus, the only fully peer reviewed international journal devoted exclusively to lupus (and related disease) research.

Conclusions/Significance Systemic autoimmunity appears to be the inevitable consequence of over-stimulating the host’s immune ‘system’ by repeated immunization with antigen, to the levels that surpass system’s self-organized criticality.

And if they want to deny where that brain inflammation is coming from in those found with it and having ASD, Pubmed is full of studies showing the inflammatory effect of aluminum adjuvants. That is how they work. Stimulating the immune system to go on high alert to actually recognized a killed/attenuated virus or bactrial pathogen in a vaccine. None of this, is rocket science! What else can adjuvants do? How about cause your immune system to become dysregulated and now be incorrectly recognizing self or organ cells as non self. How about allergens that the immune system was leaving alone; now it is picking that up to. Not in all, but just in some. How many? They will likely never know.

Now, you did not see them mention vaccines as the cause of that said neuroinflamation in the brain. They in this above study, again do not even suggest a cause, and as if they are clueless as to the cause. They have proven that this situation exists; so why are they not at least suggesting a causation?

However, many other studies we can find do show that this said repeat and chronic activation of the brains microgilia, is caused by aluminum adjuvants and thus the vaccines! That includes the published papers of Dr. Russell Blaylock, and studies a swell from several other sources. Sources that indeed are accepted and peer reviewed from real and substantial health care journals, but just not from a pharma connected journal. Neurology and toxicology related journals are the only place you will find them.

So, now they are calling all these things, Dysregulation Spectrum Syndromes. They seems to be claiming the cause is no known, but als claim without reference, that it is believed developed the disease in-utero as an inflammatory syndrome of sorts, similar to fibromyalgia and many other syndromes. WHERE is their evidence of that, in significant numbers of people? It is a guess??? Well, it sure looks like that is what it is, only a guess. How do they actually disregard the fact that entirely bright and healthy children exist, and then when give such as the MMR and/or a load of other vaccines, that all of a sudden the child is very ill, and then starts the progression to all of the symptoms that end up even lost speech, totally disconnected from functional reality, flapping arms, out of control AUTISM??? They want to entirely overlook that in their scenario of situation.

Here is a second part to what I just stated above, and involves another study finding the same said over activation brain of the microglia cells and a resulting chronic level of brain inflammation, in typical cases of ASD.

As said and as you can witness two right here on this pages listings; several independent studies exist that clearly show the said brain inflammation to be directly caused by vaccines, and specifically aluminum adjuvants combined secondarily with antigen. and as found as well in this below Journal of Neuroinflammation 2013 study, as well is what Dr. Russell Blaylock has pointed out for already years now, and as he outlines in his website, under the section, published papers. Vaccination, and repeat vaccination can cause over activation of the brains microglia cells, (which are essential part of the brains separate immune system), and thus additionally can cause chronic levels of brain inflammation.

Some studies have shown the direct link to vaccines, and other studies like this one have simply pointed out the finding of this inflammation in the brain of those with ASD, and as well ADHD. Interestingly in neither of the two studies I find addressing only the inflammation found in ASD; did they ever in that study even suggest a causation, other than this one here, and in which they refer to the likely causation as likely to be environmental. Why is this so called inflammation syndrome not found in the never vaccinated, then? Are they studying that at all. Nope. All they seem to be concerned with is novel treatments and or development of drugs to treat the condition with. In fact in the conclusion of this study they state the following:

[Conclusions
The prevalence of ASD continues to rise, but there is no clinically effective drug for the core ASD symptoms. Unfortunately, the lack of distinct pathogenesis and biomarkers makes it difficult to develop effective treatments. Stimulation of mTOR, which is already activated due to PTEN mutations, by NT, CRH and/or IL-33, could serve as novel targets for drug development. NTR and CRHR-1 antagonists could, therefore, be used in ASD, along with luteolin.

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How simple is this in its basic concepts; and the CDC is actually unaware of the findings and this, the available science? They have not yet addressed this inflammation issue on any level. They refuse to acknowledge any harm done by vaccines, period, and they will never admit to any of this; nor as to the need of larger studies. Larger studies that they know all to well would destroy them and the existing CDC multiple vaccines given, program. They have to much and everything to lose, if the truth be known and admitted to. ]

Abstract
Increasing evidence indicates that brain inflammation is involved in the pathogenesis of neuropsychiatric diseases.

Autism spectrum disorders (ASD) are characterized by social and learning disabilities that affect as many as 1/80children in the USA. There is still no definitive pathogenesis or reliable biomarkers for ASD, thus significantly curtailing the development of effective therapies. Many children with ASD regress at about age 3 years, often aftera specific event such as reaction to vaccination, infection, stress or trauma implying some epigenetic triggers, and may constitute a distinct phenotype. ASD children respond disproportionally to stress and are also affected by foodand skin allergies. Corticotropin-releasing hormone (CRH) is secreted under stress and together with neurotensin (NT) stimulates mast cells and microglia resulting in focal brain inflammation and neurotoxicity. NT is significantlyincreased in serum of ASD children along with mitochondrial DNA (mtDNA). NT stimulates mast cell secretion of mtDNA that is misconstrued as an innate pathogen triggering an auto-inflammatory response. The phosphataseand tensin homolog (PTEN) gene mutation, associated with the higher risk of ASD, which leads to hyper-active mammalian target of rapamycin (mTOR) signalling that is crucial for cellular homeostasis. CRH, NT andenvironmental triggers could hyperstimulate the already activated mTOR, as well as stimulate mast cell and microglia activation and proliferation. The natural flavonoid luteolin inhibits mTOR, mast cells and microglia and could have a significant benefit in ASD.

Introduction
Focal brain inflammation
Increasing evidence indicates that brain inflammation is important in the pathogenesis of neuropsychiatric disorders [1,2]. Autism spectrum disorders (ASD) are pervasive neuro-developmental disorders characterized by varying degrees of deficiencies in social interactions, intelligence, and language, as well as the presence of stereotypic behaviors [3-6]. Recent results from the Centers of Disease Control in the USA indicate that as many as 1/80 children have ASD [7]. Many such children regress at about age 3 years, often after a specific event such as reaction to vaccination, infection [8,9], trauma [10,11], toxic exposures [12] or stress [13], implying the importance of some environmental triggers [14,15].

Now, instead of funding their ridiculous and entirely bogus epidemiological, (population) studies at the CDC; why did they not long ago look at and fund some real physiologically based vaccine safety studies? They could do that looking at this data, and compare the entirely unvaccinated with the fully vaccinated on the CDC schedule. Of course they obviously know as well that they would never find a group of autistic children in the unvaccinated, that they could provide a control group from.

Here below is another excerpt from the said study. And that is not significant enough information, for the CDC to take notice? And they at the CDC claim to be and pretend to be all about the science? Really?

[The richest source of mast cells in the brain is the diencephalon [54] that regulates behavior, while the highest concentration of NTR is in the Broca area [55], which regulates language, known to be lost in many children with ASD. Mast cells are responsible for eliciting neutrophil infiltration that promotes inflammation [56]. Mast cell-microglial interactions are important in neuroinflammatory diseases [57,58]. Microglia are the innate brain immune cells that are increasingly implicated in a number of neuropsychiatric diseases [59]. In fact, abnormal microglial growth and activation was recently reported in the brain of ASD patients [60,61]. Microglia express NTR3, activation of which leads to their proliferation [62].

NT has additional actions that are relevant to ASD (Table 1): it induces intestinal secretion and mobility [63], stimulates glial cell proliferation [64], and can facilitate seizures through activation of glutamate receptors [65]. In fact, the glutamate receptor mGluR5 was reported to be overactive in fragile X mice [66,67], a condition associated with high risk of ASD. In other words, NT could contribute to ASD pathogenesis through different mechanisms (Figure 1).]

As the pathogenesis of fibromyalgia continues to raise debate, multiple putative triggers have been implicated. The current review summarizes the available data linking fibromyalgia to either infection or vaccination. Multiple infectious agents have been associated with the development of either full-blown fibromyalgia (e.g. hepatits C), or with symptom complexes extensively overlapping with that syndrome (e.g. chronic Lyme disease). The cases of Lyme disease, mycoplasma, hepatits C and HIV are detailed. Despite the described associations, no evidence is available demonstrating the utility of antibiotic or anti-viral treatment in the management of fibromyalgia. Possible mechanistic links between fibromyalgia and HIV are reviewed. Associations have been described between various vaccinations and symptom complexes including fibromyalgia and chronic fatigue syndrome. The case of Gulf War syndrome, a functional multisystem entity sharing many clinical characteristics with fibromyalgia is discussed, with emphasis on the possibility of association with administration of multiple vaccinations during deployment in the Persian Gulf and the interaction with stress and trauma. Based on this example a model is proposed, wherein vaccinations function as co-triggers for the development of functional disorders including fibromyalgia, in conjunction with additional contributing factors.

Here you have it! Plain and simple. How many angles would this need be approached from before the obvious would and should be evident?

[Based on this example a model is proposed, wherein vaccinations function as co-triggers for the development of functional disorders including fibromyalgia, in conjunction with additional contributing factors.]

Think, their wrong??? And why is it that the entirely unvaccinated never have any of these conditions? All is again just another coincidence, I bet; right?

An Immune Disorder at the Root of Autism,

Excerpts:

So here’s the short of it: At least a subset of autism — perhaps one-third, and very likely more — looks like a type of inflammatory disease. And it begins in the womb.

It starts with what scientists call immune dysregulation. Ideally, your immune system should operate like an enlightened action hero, meting out inflammation precisely, accurately and with deadly force when necessary, but then quickly returning to a Zen-like calm. Doing so requires an optimal balance of pro- and anti-inflammatory muscle.

In autistic individuals, the immune system fails at this balancing act. Inflammatory signals dominate. Anti-inflammatory ones are inadequate. A state of chronic activation prevails. And the more skewed toward inflammation, the more acute the autistic symptoms.

Nowhere are the consequences of this dysregulation more evident than in the autistic brain. Spidery cells that help maintain neurons — called astroglia and microglia — are enlarged from chronic activation. Pro-inflammatory signaling molecules abound. Genes involved in inflammation are switched on.

These findings are important for many reasons, but perhaps the most noteworthy is that they provide evidence of an abnormal, continuing biological process. That means that there is finally a therapeutic target for a disorder defined by behavioral criteria like social impairments, difficulty communicating and repetitive behaviors.

Aluminum has been added to vaccines for about 90 years in the belief it spurs the body to produce disease-fighting antibodies. But aluminum is toxic, and many common vaccines, including pneumonia, tetanus, and HPV, contain large doses. The result is children are getting amounts that are much higher than those considered safe by regulatory agencies, and adults are adding to the lifetime cumulative amounts of aluminum in their bodies. These megadoses can have a devastating effect on the brain, says Newsmax Health expert Dr. Russell Blaylock, causing everything from brain damage in children to Alzheimer’s in adults.

“Aluminum is toxic,” Dr. Blaylock tells Newsmax Health. “Compelling research has demonstrated that aluminum is an accumulative neurotoxin, even in small concentrations. It has a tendency to concentrate in the hippocampus, an area of the brain vital to crucial functions including learning, memory, and behavior.

“Recent articles on aluminum have shown that aluminum in vaccines is producing severe problems in the brains of developing children,” he says. “The evidence is overwhelming, but many officials and doctors ignore it. They refuse to look at the evidence because it scares them — it’s powerful evidence.

“Of the 36 vaccines children get, 18 of them contain aluminum,” says Dr. Blaylock. “One article showed that children get doses 46 times higher than those considered safe by government agencies.”

How much aluminum is in the vaccines. You may be entirely shocked by how much. No safety studies have EVER been done. Aluminum was given an FDA status of (GRAS) generally regarded as safe, in 1929 and even though aluminum has been used as a vaccine adjuvant for decades, NO safety studies have been done.

Amount of aluminum in vaccines – detailed list

“ActHib – 0 mcg

PedVaxHib – 225 mcg

Prevnar – 125 mcg

Daptacel (DTaP) – 330 mcg

Tripedia (DTaP) – 170 mcg

Infanrix (DTaP) – 625 mcg

Recombivax (HepB) – 250 mcg

Engerix B (HepB) – 250 mcg Polio – 0 mcg

MMR – 0 mcg Chic Pox – 0 mcg Hep A – 250 mcg

Combo Vaccines: Comvax (hep B as Recombivax and HIB as PedVaxHIB) – 225 mcg. This particular combo vax has LESS aluminum than getting the shots separately.

Pentacel (DTaP as Daptacel, HIB as ActHIB and polio) – 1500 mcg. This shot has a lot MORE aluminum that the sum of its parts. You actually get 5 times the amount of aluminum than if you were to get the shots separately.

Aluminum toxicity is a widespread problem in all forms of life, including humans, animals, fish, plants and trees, and causes widespread degradation of the environment and health. Over 7000 reference articles on aluminum toxicity exist in various data bases; ( as of 1996 ) all recognizing the toxicity but concluding the mechanism of action is unknown. — [ Search results – scirus.com ]

Aluminium in vaccines can cause serious polio-like damage, damage motorneuron cells and cause brain damage – possibly leading to Alzheimer’s and other forms of dementia and increasing the risk in children of Autistic Spectrum Disorders.

From the Transcripts of the US Vaccines and Related Biological Advisory Committee Meeting (VRBAC) and Scientific Papers

Again, if the medical side is ALL about the science; then why do they refuse to look at nor acknowledge the bulwark of vaccine harm science that exists? Just because the CDC refuses to acknowledge those studies, does it make them invalid? And as said, did you know that in 1929 as well that the FDA gave the substance aluminum, a rated status of (GRAS), generally regarded as safe? Even after put in vaccines in the form of an adjuvant, no safety studies have been done, nor does any sufficient safety data exist. The CDC admits to that right in their own documents. In their documents they clearly put out an assessment of need and the request for additional studies and data. It simply has not been happening. They already know what the result would be.

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Research Article The Severity of Autism Is Associated with Toxic Metal Body Burden and Red Blood Cell Glutathione Levels

Abstract

This study investigated the relationship of children’s autism symptoms with their toxic metal body burden and red blood cell (RBC) glutathione levels. In children ages 3–8 years, the severity of autism was assessed using four tools: ADOS, PDD-BI, ATEC, and SAS. Toxic metal body burden was assessed by measuring urinary excretion of toxic metals, both before and after oral dimercaptosuccinic acid (DMSA). Multiple positive correlations were found between the severity of autism and the urinary excretion of toxic metals. Variations in the severity of autism measurements could be explained, in part, by regression analyses of urinary excretion of toxic metals before and after DMSA and the level of RBC glutathione (adjusted in all cases). This study demonstrates a significant positive association between the severity of autism and the relative body burden of toxic metals.

Oxidative stress in autism NYS Institute for Basic Research in Developmental Disabilities

Excerpt: Several studies have suggested alterations in the activities of antioxidant enzymes such as superoxide dismutase, glutathione peroxidase, and catalase in autism. Additionally, altered glutathione levels and homocysteine/methionine metabolism, increased inflammation, excitotoxicity, as well as mitochondrial and immune dysfunction have been suggested in autism

Cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism

Excerpt: These results suggest that the autism LCLs exhibit a reduced glutathione reserve capacity in both cytosol and mitochondria that may compromise antioxidant defense and detoxification capacity under prooxidant conditions.

A significant co-morbidity of Alzheimer’s disease and cerebrovascular impairment suggests that cerebrovascular dysregulation is an important feature of dementia. Amyloid beta protein (Abeta), a relevant risk factor in Alzheimer’s disease, has neurotoxic properties and is thought to play a critical role in the cognitive impairments. Previously, we demonstrated that the 42mer of Abeta (Abeta42) complexed with aluminum (Al-Abeta42) is much more cytotoxic than non-complexed Abeta42. The level of Abeta in the brain is a balance between synthesis, degradation, and fluxes across the blood-brain barrier (BBB). In the present paper, we determined whether complexing with aluminum affected the ability of radioactively iodinated Abeta to cross the in vivo BBB. We found that the rates of uptake of Al-Abeta42 and Abeta42 were similar, but that Al-Abeta42 was sequestered by brain endothelial cells much less than Abeta42 and so more readily entered the parenchymal space of the brain. Al-Abeta42 also had a longer half-life in blood and had increased permeation at the striatum and thalamus. Brain-to-blood transport was similar for Al-Abeta42 and Abeta42. In conclusion, complexing with aluminum affects some aspects of blood-to-brain permeability so that Al-Abeta42 would have more ready access to brain cells than Abeta42.

Abstract Multiple sclerosis (MS) is a chronic, immune-mediated, demyelinating disease of the central nervous system of as yet unknown aetiology. A consensus of opinion has suggested that the disorder is the result of an interplay between environmental factors and susceptibility genes. We have used a battery of analytical techniques to determine if the urinary excretion of i) markers of oxidative damage; ii) iron and iii) the environmental toxin aluminium and its antagonist, silicon, are altered in relapsing-remitting (RRMS) and secondary progressive MS (SPMS). Urinary concentrations of oxidative biomarkers, MDA and TBARS, were not found to be useful indicators of inflammatory disease in MS. However, urinary concentrations of another potential marker for inflammation and oxidative stress, iron, were significantly increased in SPMS (P<0.01) and insignificantly increased in RRMS (P>0.05). Urinary concentrations of aluminium were also significantly increased in RRMS (P<0.001) and SPMS (P <0.05) such that the levels of aluminium excretion in the former were similar to those observed in individuals undergoing metal chelation therapy. The excretion of silicon was lower in MS and significantly so in SPMS (P<0.05). Increased excretion of iron in urine supported a role for iron dysmetabolism in MS. Levels of urinary aluminium excretion similar to those seen in aluminium intoxication suggested that aluminium may be a hitherto unrecognized environmental factor associated with the aetiology of MS. If aluminium is involved in MS then an increased dietary intake of its natural antagonist, silicon, might be a therapeutic option.

Abstract In Alzheimer’s disease (AD), oxidative damage leads to the formation of amyloid plaques while low PP2A activity results in hyperphosphorylated tau that polymerizes to form neurofibrillary tangles. We probed these early events, using brain tissue from a rat model for AD that develops memory deterioration and AD-like behaviors in old age after chronically ingesting 1.6 mg aluminum/kg bodyweight/day, equivalent to the high end of the human dietary aluminum range. A control group consumed 0.4 mg aluminum/kg/day. We stained brain sections from the cognitively-damaged rats for evidence of amyloid plaques, neurofibrillary tangles, aluminum, oxidative damage, and hyperphosphorylated tau. PP2A activity levels measured 238.71+/-17.56 pmol P(i)/microg protein and 580.67+/-111.70 pmol P(i)/microg protein (p<0.05) in neocortical/limbic homogenates prepared from cognitively-damaged and control rat brains, respectively. Thus, PP2A activity in cognitively-damaged brains was 41% of control value. Staining results showed: (1) aluminum-loading occurs in some aged rat neurons as in some aged human neurons; (2) aluminum-loading in rat neurons is accompanied by oxidative damage, hyperphosphorylated tau, neuropil threads, and granulovacuolar degeneration; and (3) amyloid plaques and neurofibrillary tangles were absent from all rat brain sections examined. Known species difference can reasonably explain why plaques and tangles are unable to form in brains of genetically-normal rats despite developing the same pathological changes that lead to their formation in human brain. As neuronal aluminum can account for early stages of plaque and tangle formation in an animal model for AD, neuronal aluminum could also initiate plaque and tangle formation in humans with AD.

Excerpted: HPLC analyses confirmed increased glycolytic ATP production in Al-exposed hepatocytes. These findings reveal the ability of Al to create a hypoxic environment that promotes the translocation of HIF-1alpha to the nucleus and stimulates the anaerobic metabolism of D-glucose.

Excerpt: Aluminum is a well known neurotoxic agent that is overaccumulated in the substantia nigra of patients affected by Parkinson’s disease as well as in certain cerebral areas of other neurodegenerative pathologies such as Alzheimer’s disease. Although the role of aluminum in neurodegenerative diseases is yet to be clearly understood, the metal ion is known to substantially alter the activity of several key enzymes in the central nervous system.

The potential implications of aluminum in the etiopathogenesis of neurological disorders are discussed.

Excerpt: CONCLUSIONS: The growth, development and function of human embryonic cerebral neurocytes was inhibited in the high Al group. The neurotoxicity of Al may be caused by lipid peroxidation and the damage of cell membrane.

This investigation gives detailed analysis of peripheral marker enzymes as well as neurobehavioral tests following chronic aluminium (Al) exposure (10 mg/kg b.w. for 12 weeks intragastrically). We observed a significant decrease in the levels of serum cholinesterase after toxicity. The enzymatic activity of cytochrome oxidase (CO), the terminal enzyme of the electron transport chain, was significantly diminished and that of glucose-6-phosphate dehydrogenase (G-6-PD) was significantly enhanced. Neuromuscular co-ordination was assessed using motor and memory function tests. Deficits were observed suggesting a probable model for chronic Al neurotoxicity.

Source:Department of Biochemistry, Aichi Medical University School of Medicine, Nagakute, Aichi 480-1195, Japan.

Abstract Effect of aluminum on the NADPH supply and glutathione regeneration in mitochondria was analyzed. Reduced glutathione acted as a principal scavenger of reactive oxygen species in mitochondria. Aluminum inhibited the regeneration of glutathione from the oxidized form, and the effect was due to the inhibition of NADP-isocitrate dehydrogenase the only enzyme supplying NADPH in mitochondria. In cytosol, aluminum inhibited the glutathione regeneration dependent on NADPH supply by malic enzyme and NADP-isocitrate dehydrogenase, but did not affect the glucose 6-phosphate dehydrogenase dependent glutathione formation. Aluminum can cause oxidative damage on cellular biological processes by inhibiting glutathione regeneration through the inhibition of NADPH supply in mitochondria, but only a little inhibitory effect on the glutathione generation in cytosol.

Have you ever seen it stated that vaccines manufactured by use of aborted fetal cells, have residual cells in the vaccine? And then the CDC claims that oh know that is not true, correct? And the CDC they claims to that all of the vaccines have been purified through the step process and all the bad stuff has been screened out, right? The fact is, that both the CDC and the FDA know better than that. They know that such contamination still a risk factor, and that it can happen, and as well they know that situation can play a role in the creation of illness and chronic conditions.

This study is big strike against several of the currently made vaccines that are made by used of aborted fetal cells. Although the study article doesn’t makes reference per say to human diploid cells, it rather refers to human DNA residuals. That to me would be the same thing, or derived from the same thing. Human diploid cells of course can only come from and typically one thing, aborted fetal tissue. The MMR vaccine is made by use of aborted fetal tissue; more specifically, MRC-5 and WI-38.

Results: The average human DNA fragment length in rubella vaccine was 220 base pairs. Out of 1145 hotspots in the X-chromosome, 25 hotspots are located in 5 of 15 X-chromosome AAGs, between the transcription start and end sites. These genes are NLGN3 and NLGN4X (neuroligins involved in synapse formation), AFF2 and IL1RAPL1 (involved in X-linked mental retardation), and GRPR (gastrin releasing peptide receptor).

Conclusions: Autism-associated genes in the X-chromosome contain multiple regions where potential insertion of short, non-host homologous DNA can occur. With new knowledge due to the human genome project, particularly in regards to SNPs and epigenetics, further work must be done to understand the implications of integrated residual human DNA to the etiology of autism. The said study:

There is also shown in the study what they call a Changepoint analysis for US(DOE) and CA(DDS) Autistic Disorder, Fig. 1. You can see the what appear to be direct increases in ASD after the use of vaccines containing human DNA residuals.

The study is said to be only a hypothesis, but none the less comes up with some very significant findings not only in relation to autism, but as well in what would be clear resulting in potential adverse effects on health as well.

The CDC and FDA would have us believe that vaccines go through an adequate step process of purification.

With what we have more recently seen since the days that SV- 40 was known of as a contaminant in the polio vaccine, there has been as well ongoing issues since with such as so called pig virus DNA in Rotarix, and HPV DNA found in such as Gardasil, that DNA is said to be or have been firmly locked onto the aluminum adjuvant. Then we can add this information below as well and piece it together as a conclusion; we then can conclude that in fact that vaccine makers have clearly NOT been able to purify these vaccines, anywhere nearly as well as they would have us believe they are.

The whole picture leaves remaining as to these kinds of vaccines, a showing as to the facts in reality of there being something far less remaining than a picture of proven and unquestioned safety.

So, quite obviously you can see that those so called purification steps used to so called purify vaccines, and of what they at the FDA already knows should not and can not be in them in relation to causation of chronic disorders and illness, is at times still in the vaccines, and clearly as well has been connected in relation to some serious problems. Of course then has arrived from them the FDA, a complete denial that there is any issue nor evidence of any problem.

Officially they have told us that even though aborted fetal tissue is used to make vaccines, that no cells remain in the vaccine after the manufacturing process. Yet how then and do they list diploid cells, and human albumin, in and on the vaccine ingredient list; but they still say there are none in the vaccine?

And here below you can as well see it stated by the FDA that they have been clearly aware of some of the significant risks of foreign DNA contamination left in vaccines. They do not a solution, and they know they don’t

Project Day Lily chronicles the events surrounding the “Gulf War Syndrome” suffered by over 150,000 veterans (and tens of thousands dead) without proper acknowledgment or treatment to keep secret the origin of their illnesses. Were our Armed Forces exposed to chemical and biological toxins that were supplied, in part, by a sinister network of rogue bureaucrats, intelligence operatives and scientists? This is the story of how one of these biological agents was found by two American scientists as part of a massive testing program and how various academic and government employees did everything in their power to keep this information secret.

Abstract It was hoped that following polio eradication, immunisation could be stopped. However the synthesis of polio virus in 2002, made eradication impossible. It is argued that getting poor countries to expend their scarce resources on an impossible dream over the last 10 years was unethical. Furthermore, while India has been polio-free for a year, there has been a huge increase in non-polio acute flaccid paralysis (NPAFP). In 2011, there were an extra 47,500 new cases of NPAFP. Clinically indistinguishable from polio paralysis but twice as deadly, the incidence of NPAFP was directly proportional to doses of oral polio received. Though this data was collected within the polio surveillance system, it was not investigated. The principle of primum-non-nocere was violated. The authors suggest that the huge bill of US$ 8 billion spent on the programme, is a small sum to pay if the world learns to be wary of such vertical programmes in the future.

The effects of whole-cell pertussis vaccine wane after 5 to 10 years, and infection in a vaccinated person causes nonspecific symptoms (3-7). Vaccinated adolescents and adults may serve as reservoirs for silent infection and become potential transmitters to unprotected infants (3-11). The whole-cell vaccine for pertussis is protective only against clinical disease, not against infection (15-17). Therefore, even young, recently vaccinated children may serve as reservoirs and potential transmitters of infection.

Did you read that, and excerpted below??? AND they didn’t tell anyone THIS? And why did they NOT tell anyone THIS??? You have got to be frigen kidding me. While they are all the while, fear mongering about that babies to young to be vaccinated need to be protected, by those who are vaccinated? Are they simply irresponsibly … insane? How many vaccines could this be said for, a person would wonder.

RESULTS: In the 95 previously immunized patients with serologically confirmed pertussis (mean age [+/- SD], 8.9 +/- 4.4 years old; range, 5 to 30 years old), the mean duration from onset of symptoms until the final diagnosis of pertussis was 23 +/- 15 days. The disease was usually atypical and generally mild. All the described patients had cough, usually prolonged, lasting 4 +/- 3.6 weeks. Only 6% had the classic whoop. The mean WBC count was 8.7 +/- 2.6 cells/mm6, and the lymphocyte count was 40 +/- 12%. Two patients were admitted to the hospital for severe pneumonia. Among the reported cases, the proportion of patients between the ages of 10 and 45 years increased from 6.5% during the period from 1971 to 1980, to 26% during the period from 1980 to 1990, and to 38% during a 1989 outbreak.

Pertussis in the Netherlands: an Outbreak Despite High Levels of Immunization with Whole-Cell Vaccine

In 1996, a sudden increase in pertussis incidence was reported in the Netherlands (2.1 per 100,000 in 1995, 18 per 100,000 in 1996). Although not all potential surveillance artifacts could be excluded, it is highly probable that the data reflect a true outbreak. However, the cause of this increase has not yet been determined. Further research is directed to the severity of disease and a possible mismatch between the vaccine and the circulating Bordetella strains.

And the CDC would fear monger the parents and have us believe it is all due to the unvaccinated, when the fact that children have been vaccinated and as having become the persons in the largest portion of the outbreaks. They have as well recommended and done what they call cacoon style vaccinating of whole families and to date it yet has all failed.

Mutation of the virus by the vaccine itself, and as well the causation of para-pertussis to become more prevalent, likely again due to the vaccine itself. Same thing was seen in the clinical trials with Gardasil, other strains becoming more prevalent.

Pertussis containing vaccines (DTaP) are given five times to children under age six, plus booster doses (Tdap) for teenagers and adults. And it is still failing.

DPT was one of the most dangerous childhood vaccines ever known, and resulted in numerous lawsuits and later federal vaccine court awards for the damage. DTaP, although safer, is still not a safe vaccine.

Bringing the ASD Child Back, biomedical treatment, it CAN be done; a parents story of the journey. Please pass this information on to any parent/s that need the help and this very well put together information! Reversing the ASD, while your doctor will only offer prescriptions to cover up the problem, and make sick brainwashed claims and excuses as for why the parents should not be using biomedical treatment. http://www.regardingcaroline.com/index.html

Biomedical treatment in the successful reversal of ASD. If it were not the vaccines, that would not be happening, nor possible. That is why they continue to deny it, they must.

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